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1 K14, involucrin, and TRP63, but negative for keratin 10.
2 cytes by double labeling for WNV antigen and keratin 10.
3 ntified including arginase 1, enolase 1, and keratin 10.
4 nation motif at the end of the rod domain of keratin 10.
5 on 10 in the 1A segment of the rod domain of keratin 10.
6 follicle and cyst, and ectopic expression of keratin 10, a marker of interfollicular differentiation
7 abeling of cell-derived mannose residues and keratin 10 allows the visualization of up to 100 cells o
8                                              Keratin 10 and caveolin-1 transcripts were more abundant
9 ter activity of keratin 5, increased that of keratin 10 and enhanced the effect of a differentiating
10 n in terminal differentiation (ie, increased keratin 10 and filaggrin expression).
11 H18-1 cells form multiple layers and express keratin 10 and filaggrin predominantly in the upper laye
12 s expression of maturation-specific proteins keratin 10 and filaggrin.
13 induction of differentiation markers such as keratin 10 and involucrin.
14 sed expression of differentiation-associated keratin 10 and involucrin.
15 ot altered, nor was expression of endogenous keratin 10 and profilaggrin affected.
16 rin alpha 5 beta 1 and delayed expression of keratin 10 and transglutaminase.
17  cell-type specific differentiation markers (keratin-10 and keratin-14).
18  presence of the hair follicle marker Sox 9, keratins 10 and 14, and normal melanocyte distribution a
19                           Immunostaining for keratins 10 and 16 and for involucrin revealed an initia
20 AFMK-stimulated expression of involucrin and keratins-10 and keratins-14 in the epidermis, indicating
21 rly KC differentiation, including keratin 1, keratin 10, and DSC-1, is reversed by p63 blockade.
22 pidermal differentiation markers involucrin, keratin 10, and filaggrin during tissue reconstruction.
23 l differentiation markers such as keratin 1, keratin 10, and loricrin, with or without the induction
24                                We identified keratin 10 as a marker of suprabasal epithelial cells in
25 sialoprotein-binding protein, fibrinogen and keratin-10 binding surface-anchored protein, fibrinogen-
26 f the differentiation markers involucrin and keratin 10 compared to cells with no cell-cell contact.
27                               A study of the keratin 10 deficient mouse, a model for epidermolytic hy
28 hich would spatially constrain the keratin 1/keratin 10 end domains to allow filament compaction and
29 ression in the stratum granulosum, a loss of keratin 10 expression in the stratum spinosum, and an in
30 ermal growth factor suppresses keratin 1 and keratin 10 expression.
31 inine-rich C-terminal peptide that redirects keratin 10 from the cytokeratin filament network to the
32            Mice heterozygous for a truncated keratin 10 gene exhibit acanthosis and hyperkeratosis as
33            Driven by the suprabasal-specific keratin-10 gene promoter, expression of dnRXR alpha seve
34 inocytes induces expression of keratin 1 and keratin 10 genes, early markers of terminal differentiat
35 ystal structure of wild-type human keratin-1/keratin-10 helix 1B heterotetramer at 3.0 angstrom resol
36 ccur within the mutational "hot spot" of the keratin 10 (K10) 2B rod domain, adjacent to severe EI-as
37 rkers of differentiation, keratin 1 (K1) and keratin 10 (K10) but had a minimal effect on the express
38 zation studies determined that PsrP binds to Keratin 10 (K10) on the surface of lung but not nasophar
39                        Reduced expression of Keratin 10 (K10) resulting from Cdk5 knockdown may be re
40 ese areas are composed of keratin 1 (K1) and keratin 10 (K10), and several K1 and K10 point mutations
41       Moreover, establishing live imaging of Keratin-10 (K10) nascent RNA, which marks the onset of s
42 351), human keratin 1 (K211 and K355), human keratin 10 (K163), bovine tubulin alpha (K60, K336, K163
43 sion of keratinocyte differentiation markers keratin 10 (KRT10) and loricrin (LOR).
44 We previously demonstrated that mutations in keratin 10 (KRT10) cause ichthyosis with confetti (IWC),
45 ligase MDM2 are upregulated concomitant with keratin 10 (KRT10) downregulation.
46 nant negative mutations in the gene encoding keratin 10 (KRT10).
47 sease-causing mutations in the gene encoding keratin 10 (KRT10); all result in frameshifts into the s
48                                              Keratin 10 levels, however, were not reduced.
49 ctivated transglutaminase 1, involucrin, and keratin 10 message and protein levels, demonstrating tha
50 8-1 cells synthesize considerable amounts of keratin 10 mRNA and protein when maintained in either su
51 criptase polymerase chain reaction assay for keratin 10 mRNA was developed to distinguish between exp
52 pressing mutant ras under the control of the keratin 10 or keratin 1 gene promoters, the formation of
53 from mutations affecting the tail domains of keratin-10 or keratin-1, and Suzuki et al. expand the mu
54 amplifying population and integrin-negative, keratin 10-positive cells left the basal layer exclusive
55 ferating compartment and an expansion of the keratin 10-positive layer of cells and was associated wi
56 press excess IGF-II delivered using a bovine keratin 10 promoter (k10Igf2/+) develop a disproportiona
57 connexin 26 [gjb2/connexin 26(D66H)], from a keratin 10 promoter, exclusively in the suprabasal epide
58 wed that SdrF mediates bacterial adhesion to keratin 10 through strong and weak bonds involving the A
59 t of stem cell differentiation, we find that Keratin-10 transcription is highly dynamic and largely p
60 cells staining with antibodies to suprabasal keratin 10, transglutaminase type I, involucrin, and fil
61  reduced tumorigenesis because keratin 1 and keratin 10, two keratins that indicate the commitment of
62 ntiation markers profilaggrin, loricrin, and keratin 10 was considerably downregulated in PF-N domain
63 , expression of the differentiation-specific keratin-10 was shown not to be required for HPV late fun