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1 8 encoding the intermediate filament protein keratin 8.
2 s remained unaltered, including cyclin E and keratin 8.
3 ine-to-histidine mutations at position 53 of keratin 8.
4 es subapically, together with the IF protein keratin 8.
5    Tumors were further classified by markers keratin 8/18 (K18, KRT18), keratin 14 (K14, KRT14) and e
6 n the tumor that express both luminal marker keratin 8/18 and basal marker keratin 5.
7     Ballooned hepatocytes were quantified by keratin 8/18 and ubiquitin (K8/18/Ub) staining.
8 e effect of small heat shock proteins on the keratin 8/18 intermediate filament cytoskeleton using a
9          Consistent with DNAJB6 dysfunction, keratin 8/18, a DNAJB6 client also accumulated in DNAJB6
10 rates expression of NP markers FoxF1, Pax-1, keratin-8/18, carbonic anhydrase-12, and NC markers brac
11                                              Keratins 8/18 (K8/18) are phosphoglycoproteins and form
12                                              Keratins 8/18 (K8/K18) are established hepatoprotective
13 otein aggregates consisting of ubiquitinated keratins 8/18 (K8/K18).
14  blocked normal differentiation, and induced keratin 8, a marker of malignant conversion, but did not
15                                              Keratin 8 and 18 (K8/18) phosphorylation plays a signifi
16                                              Keratin 8 and 18 (K8K18) mutations are found in patients
17 runcates the last 14 amino acids; 8 missense keratin 8 and 18 alterations; and several new polymorphi
18                       We identified 10 novel keratin 8 and 18 heterozygous variants in 44 of 467 expl
19                     Liver disease-associated keratin 8 and 18 mutations modulate keratin acetylation
20  a time-dependent disassembly-degradation of keratin 8 and 18 proteins, which was associated with an
21                                              Keratin 8 and 18 variants in 17 of 467 liver disease exp
22                     The overall frequency of keratin 8 and 18 variants was 12.4% in 467 liver disease
23  and small pancreatic ducts, as evidenced by Keratin 8 and CAM5.2 staining.
24  cytoskeletal intermediate filament proteins keratin 8 and keratin 18 (K8/K18) is associated with cir
25               We tested for mutations in the keratin 8 and keratin 18 genes in purified genomic DNA i
26 osis, but the importance of mutations in the keratin 8 and keratin 18 genes in such patients is uncle
27  of TEC subsets during ontogeny, we analyzed keratin 8 and keratin 5 expression at several stages of
28 duction of epithelial markers E-cadherin and keratin-8 and down-regulation of mesenchymal markers N-c
29 patibility complex transcription factors and keratin-8 and therefore may allow immune evasion and est
30                                              Keratins 8 and 18 (K8/18) are intermediate filament phos
31                                              Keratins 8 and 18 (K8/18) are major constituents of Mall
32                                              Keratins 8 and 18 (K8/18) are simple epithelial cell-spe
33                          Hepatocytes express keratins 8 and 18 (K8/18) as their only cytoskeletal int
34                                              Keratins 8 and 18 (K8/18) heteropolymers may regulate ce
35 eral liver diseases and consist primarily of keratins 8 and 18 (K8/K18) and ubiquitin that are cross-
36                                              Keratins 8 and 18 (K8/K18) are important hepatoprotectiv
37                                              Keratins 8 and 18 (K8/K18) are the intermediate filament
38                                              Keratins 8 and 18 (K8/K18) protect the liver from variou
39 major keratins in the pancreas and liver are keratins 8 and 18 (K8/K18), but their function seemingly
40                     MBs consist primarily of keratins 8 and 18 (K8/K18), require a K8-greater-than-K1
41 -Denk bodies (MDBs), which are aggregates of keratins 8 and 18 (K8/K18), ubiquitin, and the ubiquitin
42 re composed primarily of hyperphosphorylated keratins 8 and 18 (K8/K18).
43  demonstrated the constitutive expression of keratins 8 and 18 and induced expression of keratin 19,
44 n, the transfectants containing vimentin and keratins 8 and 18 demonstrated an increase in focal adhe
45 nimal models, the absence of heteropolymeric keratins 8 and 18 or the presence of mutant keratins in
46                                              Keratins 8 and 18 protect the liver from stress.
47 ich included several keratins, in particular keratins 8 and 18 which are regulated through the ras si
48 h up-regulation of epithelial markers (i.e., keratins 8 and 18) and down-regulation of mesenchymal ma
49 elial tissues express keratins 5 and 14, and keratins 8 and 18, respectively.
50 ns were present in the remaining 10 exons of keratins 8 and 18.
51 5P (of low invasive ability), with cDNAs for keratins 8 and 18.
52 ue-type plasminogen activator (tPA), AFP and keratins 8 and 19 is inhibited, whilst brachyury and myo
53  position 61 (a highly conserved glycine) of keratin 8, and 2 had tyrosine-to-histidine mutations at
54 actor) was both necessary and sufficient for keratin 8 cleavage in chlamydia-infected cells, suggesti
55                       A cleavage fragment of keratin 8 corresponding to the central rod region was de
56  increase in p62 and Hsp25 levels as well as keratin 8 cross-linking that is normally associated with
57            After temporally ablating Pten in keratin 8-expressing luminal cells, luminal-derived Pten
58  the adenoviral vector was validated through Keratin-8 expression and Cre-recombinase activity.
59 ontrols: keratin 18 deletion (delta64-71), a keratin 8 frameshift that truncates the last 14 amino ac
60  others decreased keratin 8 phosphorylation (keratin 8 G433S).
61   Mutations that introduced disulfide bonds (keratin 8 G61C or R453C) decreased keratin solubility, p
62                             Mutations in the keratin 8 gene may predispose people to liver disease an
63                                        Since keratin 8 is a major component of the intermediate filam
64   Furthermore, the C-terminal tail domain of keratin 8 is shown to be essential for this effect.
65                                  Exposure of keratin 8 (k-8) expressing human breast cancer cells (MC
66            The intermediate filament protein keratin 8 (K8) and its cross-linking by transglutaminase
67                                              Keratin 8 (K8) and keratin 18 (K18) are the intermediate
68                                              Keratin 8 (K8) and keratin 18 (K18) form intermediate fi
69                           Human mutations in keratin 8 (K8) and keratin 18 (K18), the intermediate fi
70 ated ectopic expression in cardiomyocytes of keratin 8 (K8) and keratin 18 (K18), two epithelial-spec
71                                              Keratin 8 (K8) and keratin-18 (K18) are the major interm
72 Using stable CACO-2 transfectants expressing keratin 8 (K8) antisense RNA under a tetracycline-respon
73                                   Absence of keratin 8 (K8) in mice leads to colitis, decreased Na/Cl
74                                              Keratin 8 (K8) is a major intermediate filament protein
75            The intermediate filament protein keratin 8 (K8) is critical for the development of most m
76 aining "particles," mostly containing either keratin 8 (K8) or 18 (K18), but not both.
77                                              Keratin 8 (K8) regulated the expression of macrophage ch
78 n kinase C delta-mediated phosphorylation of keratin 8 (K8) Ser-73.
79                                              Keratin 8 (K8) serine 73 occurs within a relatively cons
80 lexes with the intermediate filament protein keratin 8 (K8) thereby preventing its correct traffickin
81 ectopic expression of the Merkel cell marker keratin 8 (K8) throughout the epidermis.
82                                              Keratin 8 (K8) variants predispose to human liver injury
83            We investigated the regulation of keratin 8 (K8), a type II simple epithelial IF, by lysin
84  of intestinal keratins is unknown, although keratin 8 (K8)-null mice develop colitis, hyperplasia, d
85 Keratins are essential for MDB formation and keratin 8 (K8)-overexpressing transgenic mice are predis
86 2 and keratin overexpression, with a greater keratin 8 (K8)-to-keratin 18 (K18) ratio, which are crit
87 ne sites on the simple epithelial IF protein keratin 8 (K8).
88 atin in "simple type" glandular epithelia is keratin 8 (K8).
89                                              Keratins 8 (K8) and 18 (K18) are major components of int
90 l tissues such as liver and pancreas express keratins 8 (K8) and 18 (K18) as their major intermediate
91                     Simple epithelia express keratins 8 (K8) and 18 (K18) as their major intermediate
92                     Simple epithelia express keratins 8 (K8) and 18 (K18) as their major intermediate
93  was not affected, but hsc hsp70, hsp90beta, keratin 8, keratin 18 and caveolin-1 were deregulated fo
94                     Here, we show that human keratin 8 (KRT8) genetic variants were associated with I
95 y molecule-1 (Kim1), lipocalin 2 (Lcn2), and keratin 8 (Krt8)-and of several novel genes (Ahnak, Sh3b
96                                 In contrast, keratin-8 (KRT8), which colocalizes with KRT19 on the su
97 ents in simple epithelial cells, cleavage of keratin 8 may increase the solubility of the host cell c
98 -associated mutations have been described in keratin 8 or 18 (K8/18) which are the major keratin pair
99 ach to identify proteins that associate with keratins 8 or 18 (K8/K18) in a pervanadate-dependent man
100 r oxidative stress, whereas others decreased keratin 8 phosphorylation (keratin 8 G433S).
101 ge-committed basal Lgr5-positive and luminal keratin-8-positive cells of the adult mouse mammary glan
102  the expression from which being driven by a Keratin-8 promoter, will deliver Cre-recombinase specifi
103                     The most common variant, keratin 8 R340H, at the highly conserved R340 was found

 
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