ライフサイエンスコーパス: keratinize

1 anatomy of the area, including the width of keratinized and attached gingiva, the facial-to-lingual

2 The vaginal epithelium, in contrast, is not keratinized and can allow absorption of other molecules.

3 Both keratinized and nonkeratinized surfaces of esophagi were

4 The protein is expressed in keratinizing and nonkeratinizing stratified squamous epi

5 sociated with HPV, however, only a subset of keratinizing and verrucous penile carcinomas is positive

6 In Mode 3B, keratinized apoptosed epithelial cells became permanent

7 ha protein, and expression was restricted to keratinized areas.

8 se to at least six different cell types that keratinize as they move up the hair shaft and inner root

9 and mandibular symphyses that probably bore keratinized beaks.

10 condition and finally becomes a part of the keratinized cells of the cornified epidermal layer.

11 inactive condition and becomes a part of the keratinized cells of the cornified layer.

12 ermal cells to a fully inactive state in the keratinized cells of the cornified layer.

13 Ac-T antibody was observed in the late-stage keratinized conjunctival epithelia of patients with OCP.

14 eir distribution with that in pathologically keratinized conjunctival epithelia.

15 epithelia, including the stratified but non-keratinized corneal, limbal and conjunctival epithelium,

16 The lower telomerase activity in the keratinized (differentiated) vagina was probably due to

17 Olmsted syndrome (OS) is a rare keratinizing disorder characterized by excessive epiderm

18 Pachyonychia congenita (PC) is a keratinizing disorder predominantly caused by mutations

19 EPPK) is one of >30 autosomal-dominant human keratinizing disorders that could benefit from RNA inter

20 y pattern was observed in samples from other keratinizing disorders, demonstrating that loss of LEKTI

21 on detection strategy for ongoing studies of keratinizing disorders.

22 differentiation switch to express markers of keratinized epidermis); cornea stroma changes including

23 a major component of cornified envelopes in keratinized epidermis, were substantially up-regulated i

24 Cytokeratins typical of keratinized epithelia (K1, K2, and K10) and the keratini

25 opulated ecological niche that coats all non-keratinized epithelia, and plays a critical role in prot

26 The mice developed severe hypertrophy of all keratinized epithelia, but no malignancies were observed

27 ce that expressed the E6 and E7 oncogenes in keratinized epithelia.

28 opriate development of stratified, squamous, keratinizing epithelia, such as the epidermis and oral e

29 limb abnormalities as a result of defects in keratinizing epithelia.

30 nown as 14-3-3sigma) show similar defects of keratinizing epithelia.

31 olite, produced in large amounts in squamous keratinizing epithelia.

32 the cornea consists of a unique type of non-keratinized epithelial cells arranged in an orderly fash

33 creased incidence of squamous cell tumors of keratinized epithelial cells of the skin and esophagus.

34 zed in transgenic mice overexpressing NGF in keratinized epithelium (e.g. skin, tongue and oral cavit

35 Tumors occurred in regions that had keratinized epithelium and were subjected to repeated me

36 lobal change in the tumorigenic potential of keratinized epithelium in Atp2a2+/- mice.

37 rging functions in the genesis of stratified keratinized epithelium, hair follicles, and squamous cel

38 for maintenance of colonization at sites of keratinized epithelium, such as the vagina, or for adher

39 n (MSW), a group likely exposed primarily at keratinized epithelium, using data from the National Hea

40 ified squamous epithelium to a nonsecretory, keratinized epithelium.

41 ne expression during malignant conversion of keratinized epithelium.

42 physiological and pathological remodeling of keratinized epithelium.

43 sure at nonkeratinized (mucosal) compared to keratinized epithelium.

44 psies appeared as normal mucoperiosteum with keratinized epithelium.

45 g epithelium of attached gingiva and the non-keratinizing epithelium lining the upper two-thirds of t

46 These results demonstrate that both the keratinizing epithelium of attached gingiva and the non-

47 al epithelium, in concert with the epidermal keratinized eyelid epithelium, function together to main

48 ssion of myeloperoxidase was observed in the keratinized eyelid margins of SJS/TEN to validate the pr

49 eks was compared to the mean width of buccal keratinized gingiva (KG) of adjacent teeth.

50 Width of keratinized gingiva (KG) was determined at baseline and

51 ingival tissue thickness (GTT), and width of keratinized gingiva (KG) were assessed at baseline, and

52 cluded: percent root coverage, the amount of keratinized gingiva (KG), and probing depth (PD).

53 nt level (CAL), probing depth (PD), width of keratinized gingiva (KG), attached gingiva (AG), and rec

54 ession depth, recession width (RW), width of keratinized gingiva (KW), clinical attachment level (CAL

55 ), papilla index score (WDPIS), and width of keratinized gingiva (WDKG) between initial and last meas

56 level (CAL), recession height (RH), width of keratinized gingiva (WKG) and assessment of gingival bio

57 Clinical parameters (width of keratinized gingiva [WKG], facial soft tissue level [FST

58 The standard of care for increasing keratinized gingiva adjacent to teeth that do not requir

59 ican female was referred for augmentation of keratinized gingiva around implants at the right and lef

60 ture as the adjacent tissue, a 1-mm width of keratinized gingiva at 6 months, patient treatment prefe

61 the ability of the LCC to regenerate >/=2 mm keratinized gingiva at 6 months.

62 he compared networks, ResNet50 distinguished keratinized gingiva at the highest accuracy rate of 91.4

63 Spectral reflection of keratinized gingiva at upper central incisors was measur

64 CDH and improve patient quality of life, by keratinized gingiva augmentation and impact on physical

65 tment; 4) gingival recession; and 5) band of keratinized gingiva for each of the six anterior mandibu

66 After removing the mucosa and keratinized gingiva from the test site, either an LCC or

67 recession depth (RD), recession width (RW), keratinized gingiva height measured apico-coronally (KG)

68 At 6 months, the LCC regenerated >/=2 mm of keratinized gingiva in 95.3% of patients (81 of 85 patie

69 preferentially located in plaque, others in keratinized gingiva or buccal mucosa, and some oligotype

70 Automated keratinized gingiva segmentation with the ResNet50 model

71 Width of keratinized gingiva significantly increased after IMITG

72 As expected, the FGG generated more keratinized gingiva than the LCC (4.57 +/- 1.0 mm versus

73 Results confirm the keratinized gingiva to have increased tensile strength (

74 At that time, 112 sites of inadequate keratinized gingiva were found.

75 terally lacking sufficient zones of attached keratinized gingiva were randomly assigned to soft tissu

76 ness, gingival ridge dimension, and width of keratinized gingiva were the esthetic outcomes reviewed.

77 All patients achieved >/=1 mm keratinized gingiva with the LCC treatment by 6 months,

78 inical attachment level, GR height, width of keratinized gingiva, and assessment of gingival biotype.

79 GR dimensions, amount of keratinized gingiva, and clinical attachment level were

80 statistically better root coverage, width of keratinized gingiva, and complete root coverage.

81 teeth being partially or totally engulfed by keratinized gingiva, causing aesthetic and functional pr

82 the output variables -- changes in width of keratinized gingiva, changes in bucco-lingual width, and

83 milar community compositions: buccal mucosa, keratinized gingiva, hard palate; saliva, tongue, tonsil

84 led a thick biotype with an adequate band of keratinized gingiva, Miller Class I mucogingival defects

85 effective therapy for augmenting the zone of keratinized gingiva.

86 sthesia, incisions were initiated within the keratinized gingiva.

87 All patients had a minimum of 4 mm of keratinized gingiva.

88 This flap design is carried out within keratinized gingiva.

89 rous enlargement of maxillary and mandibular keratinized gingiva.

90 ign, localized or generalized enlargement of keratinized gingiva.

91 Clinical parameters including the keratinized gingival (KG) width, KG thickness, horizonta

92 aluated by the probe transparency method and keratinized gingival thickness measurements; study group

93 e aim of the present study is to compare the keratinized gingival tissue (KT) height labial to the ma

94 Thirty-one patients with inadequate keratinized gingival width (KGW) around mandibular incis

95 depth, clinical attachment level (CAL), and keratinized gingival width (KGW) was recorded at baselin

96 Moreover, the increased value of the keratinized gingival width attested to the positive outc

97 Clinical measurements of keratinized gingival width, probing depth, and recession

98 readily, removal of upper skin layer exposed keratinized hair shafts at the skin surface.

99 ypes: well keratinized (K-SCC), intermediate keratinized (I-SCC), and poorly keratinized (P-SCC).

100 SCC was classified into three subtypes: well keratinized (K-SCC), intermediate keratinized (I-SCC), a

101 The columnar epithelium undergoes keratinizing metaplasia, which is reversed when R is sup

102 need for keratinized mucosa (KM) or immobile keratinized mucosa (i.e., attached mucosa [AM]) for the

103 2 times/year) to examine the significance of keratinized mucosa (KM) and gingival tissue (KT) on peri

104 Whether a minimal width of keratinized mucosa (KM) is required to maintain peri-imp

105 The need for keratinized mucosa (KM) or immobile keratinized mucosa (

106 ciated with peri-implantitis diagnosis risk, keratinized mucosa (KM) width, and vestibular depth (VD)

107 r bleeding index (mSBI), probing depth (PD), keratinized mucosa (KM), implant crown length (IC), dist

108 eeding on probing (BOP), suppuration (SUPP), keratinized mucosa (KM), probing depth (PD), marginal re

109 ied plaque index (mPI), and the width of the keratinized mucosa (WKM) were performed at baseline (at

110 nd between induced defect depth and width of keratinized mucosa at baseline (P = 0.03).

111 P), mucosal redness (MR), suppuration (SUP), keratinized mucosa dimension, and marginal bone loss.

112 The role of keratinized mucosa in promoting peri-implant health is c

113 The mean width of keratinized mucosa measured at the mid-buccal location p

114 tached gingiva of the molars and the lingual keratinized mucosa of the incisor region.

115 Width of keratinized mucosa was measured in the region of each im

116 The keratinized mucosa width (KMW) gain was significantly gr

117 ding PSTD class and subclass, probing depth, keratinized mucosa width (KMW), mucosal thickness (MT) a

118 soft tissue phenotype (PSP) encompasses the keratinized mucosa width (KMW), mucosal thickness (MT),

119 romising alternative for the regeneration of keratinized mucosa without tissue harvesting.

120 clinical (probing depth, recession, width of keratinized mucosa, bleeding on probing, suppuration, im

121 similar healing, with increased peri-implant keratinized mucosa.

122 e active and proliferative state than native keratinized mucosa.

123 ized epithelial layer similar to native oral keratinized mucosa.

124 gold standard for augmenting small areas of keratinized mucosa.

125 dulus of 19.75 +/- 6.20 MPa) relative to non-keratinized mucosal regions, where densely arranged elas

126 ced, nonkeratinized (n = 6), and late-stage, keratinized (n = 8).

127 to the specialized epithelia surrounding the keratinized nail plate.

128 The consequences of Notch1 activation in keratinizing nail cells were investigated in a transgeni

129 30 eyes of 30 patients with SJS-induced dry keratinized ocular surfaces; the patients underwent vari

130 Matriptase was also expressed in keratinizing oral epithelium, where it was required for

131 ctodermal cells subsequently produce various keratinized organs such as nails or claws.

132 tively, revealed extensive detachment of the keratinized outer layer and distention of spinous and ba

133 hological changes included detachment of the keratinized outer layer, distention of spinous and basal

134 intermediate keratinized (I-SCC), and poorly keratinized (P-SCC).

135 to compare the CM versus FGGs for augmenting keratinized peri-implant mucosa based on clinical and hi

136 amine green (LG; 0.15 mul) was placed on the keratinized portion of the central lower lid.

137 osteo-odonto keratoprosthesis surgery in dry keratinized post-SJS eyes.

138 ossly, the CXCR3(-/-) mice presented a thick keratinized scar compared with the wild-type mice in whi

139 Over a human's life span, keratinized skin cells, immune cells, and microbes all i

140 l-fate switch from transparent epithelium to keratinized skin-like epidermis, which led to corneal bl

141 --which turns cornea into a non-transparent, keratinized skin-like epithelium--causes corneal surface

142 Although keratinizing squamous cell carcinoma and verrucous carci

143 ly these tumors consisted largely of focally keratinizing squamous cell carcinoma with high-grade mal

144 avicular lesion and also detected concurrent keratinizing squamous cell metastasis in the right axill

145 EN deficiency accelerated the development of keratinizing squamous metaplasia (KSM).

146 elial hyperplasia and female mice developing keratinizing squamous metaplasia.

147 All biopsies showed a multilayered, keratinized, squamous epithelium.

148 ole in establishing the boundary between the keratinized, squamous esophagus/forestomach and glandula

149 and cytokeratin 10/13, suggested a premature keratinized state.

150 the morphologically different layers of the keratinized stratified epithelium of the dorsal layer of

151 d a chronically inflamed cyst lined by a non-keratinized stratified squamous epithelium.

152 rneal epithelial progenitor cells into a non-keratinizing stratified epithelium.

153 In the keratinizing stratified squamous epidermis of skin, howe

154 areas of squamous metaplasia in the form of keratinizing stratified squamous epithelium, similar to

155 h from a transparent corneal epithelium to a keratinized, stratified squamous, psoriasiform-like epid

156 m spinosum and stratum granulosum, and a non-keratinizing stratum corneum.

157 Rather, they develop a specialized keratinized structure, called the rhamphotheca, that cov

158 +/- 1.7 mm and 2.2 +/- 1.6 mm), increase in keratinized tissue (0.7 +/- 0.8 mm and 1.2 +/- 1.0 mm),

159 linical attachment level (CAL), and width of keratinized tissue (KG) were evaluated at 12 months.

160 The standard of care for increasing keratinized tissue (KT) and vestibular area is an autoge

161 ngival/mucosal grafts (FEGs) used to augment keratinized tissue (KT) at tooth and implant sites, and

162 The role of keratinized tissue (KT) for maintenance of periodontal h

163 Although the need for "adequate" amount of keratinized tissue (KT) for periodontal health is questi

164 for the purposes of increasing the width of keratinized tissue (KT) is an important aspect of period

165 /- 1.1 mm (74.1%) (both P<0.01), increase in keratinized tissue (KT) of 1.2 +/- 1.3 and 1.6 +/- 1.9 (

166 coverage, recession reduction, and amount of keratinized tissue (KT) were analyzed using descriptive

167 CAF+CM resulted in higher increase in keratinized tissue (KT) width (CAF: 0.3 +/- 0.7 mm; CAF

168 CAF+CM resulted in higher increase in keratinized tissue (KT) width (CAF: 0.3 0.7 mm; CAF + CM

169 dy included 12 patients exhibiting deficient keratinized tissue (KT) width (i.e., <2 mm) at the vesti

170 GR depth, keratinized tissue (KT) width, and probing depth were me

171 Clinical REC, keratinized tissue (KT) width, percentage of root covera

172 lts for root coverage and greater amounts of keratinized tissue (KT) with the acellular dermal matrix

173 ding vertical recession (VR), probing depth, keratinized tissue (KT), and attachment level were recor

174 enotype consists of the bone morphotype, the keratinized tissue (KT), and gingival thickness (GT).

175 t circumstances require an increased zone of keratinized tissue (KT), or is KT important?

176 h, clinical attachment level (CAL) gain, and keratinized tissue (KT).

177 ing depth (PD; 2.3 +/- 0.2 mm), and width of keratinized tissue (KT; 2.4 +/- 0.3 mm); measurements we

178 ), clinical probing depth (PD), and width of keratinized tissue (KTW) were measured preoperatively an

179 ondary efficacy parameters included width of keratinized tissue (KTw), probing depth (PD), clinical a

180 was a statistically significant increase in keratinized tissue (mean 0.88 mm) and tissue thickness (

181 dimension was correlated with the amount of keratinized tissue (P = 0.010) and also with defect cove

182 , clinical attachment level (CAL), height of keratinized tissue (wKT), and percentage of root coverag

183 idth (RW), gingival thickness (GT), width of keratinized tissue (WKT), clinical attachment level (CAL

184 idth (RW), gingival thickness (GT), width of keratinized tissue (WKT), clinical attachment level (CAL

185 Recession height (RECH), width of keratinized tissue (WKT), probing depth (PD), vertical c

186 evel (CAL), probing depth (PD), and width of keratinized tissue (WKT), were recorded presurgery (at b

187 (3) clinical attachment level; (4) width of keratinized tissue (wKT); (5) percentage of root coverag

188 was accompanied by a significant increase in keratinized tissue 6 months after surgery.

189 along with a 0.5-mm decrease in the width of keratinized tissue after healing.

190 l parameters with the exception of amount of keratinized tissue and percent shrinkage of keratinized

191 The GA generated more keratinized tissue and shrank less than the HF-DDS graft

192 were the change in the width of the zone of keratinized tissue and the amount of root coverage achie

193 n terms of root coverage, increased width of keratinized tissue and thickness of the gingival biotype

194 elanosis of the buccal gingiva and a lack of keratinized tissue around implants at sites #4 and #13.

195 procedure designed to increase the amount of keratinized tissue around teeth that do not require root

196 outcomes included change in the thickness of keratinized tissue at 3 months and patient-reported outc

197 owed a significant increase in the amount of keratinized tissue at 9 and 12 months compared to baseli

198 The mean width of keratinized tissue at the beginning of the study was 1.7

199 h good color match and an increased width of keratinized tissue at the surgical site.

200 rnatives to autogenous soft tissue grafts in keratinized tissue augmentation and in root coverage pro

201 e and increase in the width and thickness of keratinized tissue can be achieved at 3 months whether a

202 complete RC (CRC), attachment gain (AG), and keratinized tissue change (KTC) were also calculated.

203 a connective tissue graft tended to increase keratinized tissue compared to ADM (0.52-mm difference;

204 pression and function of matriptase in mouse keratinized tissue development, homeostasis, and maligna

205 tissue graft demonstrated greater amount of keratinized tissue during the 12-month evaluation period

206 Limiting the flap design to keratinized tissue facilitates flap closure and avoids w

207 The mean increase in keratinized tissue for both treatments was 0.80 mm.

208 elimination and augmentation of the zone of keratinized tissue for prosthetic reasons.

209 f the outcomes measured (recession coverage, keratinized tissue formation, probing depths, and clinic

210 cluded recession, clinical attachment level, keratinized tissue height, and plaque index.

211 e amount of root coverage, probing depth, or keratinized tissue in coronally advanced flaps for root

212 There was greater increase in the width of keratinized tissue in the E group (1.5+/-1.1 mm) than th

213 roups were compared, the width of the buccal keratinized tissue in the E group showed an increase of

214 Only keratinized tissue in the test group demonstrated a stat

215 probing attachment level, and the amount of keratinized tissue in the treatment of localized recessi

216 ted in a statistically significant effect on keratinized tissue increase, but no significant effects

217 ary benefit, it is believed that the zone of keratinized tissue is additionally increased after gingi

218 al inflammation, areas with small amounts of keratinized tissue may remain stable over long periods o

219 self-reported discomfort, and the amount of keratinized tissue obtained.

220 There was a mean increase in keratinized tissue of 0.60 mm for test sites and a mean

221 t (FGG), evaluating their ability to augment keratinized tissue or gingiva.

222 p exhibited an average of 1.0 to 1.2 mm more keratinized tissue over time than the test group (P <0.0

223 site-specific phenomenon independent of the keratinized tissue present in the adjacent dentition (NC

224 V infection was greater (31.0%) across the 3 keratinized tissue sites (genital skin, eyebrow hairs, f

225 Changes in keratinized tissue thickness (KTT), keratinized tissue w

226 root coverage, keratinized tissue width, or keratinized tissue thickness (P >0.05).

227 Keratinized tissue thickness gain was significant only i

228 Keratinized tissue thickness was increased by 1.0 +/- 0.

229 ession depth and recession width and gain of keratinized tissue thickness, keratinized tissue width,

230 Keratinized tissue was increased for the ADM group by 0.

231 t coverage of 5 mm along with a 2-mm band of keratinized tissue was obtained at 24 weeks.

232 -1.19 versus 3.50 +/-0.73), and the width of keratinized tissue was significantly increased (2.47+/-1

233 root coverage (CRC), width, and thickness of keratinized tissue were assessed by the same masked exam

234 differences in recession depth and width of keratinized tissue were seen at 5 months between the GP+

235 a mean root coverage of 87.4% and increased keratinized tissue width (1.2 mm) when compared to their

236 CTG resulted in a greater increase in keratinized tissue width (CTG: 0.96 mm vs. XDM: 0.3 mm,

237 erences were observed between the groups for keratinized tissue width (group 1: 4.80 +/- 1.48 mm; gro

238 h (PD), clinical attachment level (CAL), and keratinized tissue width (KT).

239 ded the following: 1) probing depth (PD); 2) keratinized tissue width (KT); 3) tissue biotype (TB); a

240 t (LCC) versus free gingival graft (FGG) for keratinized tissue width (KTW) augmentation in natural d

241 overage (CRC), mean root coverage (MRC), and keratinized tissue width (KTW) changes.

242 Mean keratinized tissue width (KTW) increased at a similar ra

243 Greater gains in the keratinized tissue width (KTW) were found for SCTG + CAF

244 changes in slopes for recession depth (REC), keratinized tissue width (KTW), and clinical attachment

245 recession depth (RD), recession width (RW), keratinized tissue width (KTW), clinical attachment leve

246 bing depth, clinical attachment level (CAL), keratinized tissue width (KTW), horizontal recession (HR

247 anges in keratinized tissue thickness (KTT), keratinized tissue width (KTW), relative gingival recess

248 Clinical measurements (GR length [REC], keratinized tissue width [KT], complete root coverage [C

249 (recession depth, mean root coverage [mRC], keratinized tissue width [KTW], and gingival thickness)

250 erage [MRC%], complete root coverage [CRC%], keratinized tissue width [KTW], gingival thickness [GT]

251 robing depth, clinical attachment level, and keratinized tissue width and thickness were measured at

252 fluence of CTGs on soft tissue thickness and keratinized tissue width are also discussed.

253 ficantly greater root coverage and increased keratinized tissue width compared to XDM for treating mu

254 One study reported an increase in keratinized tissue width post-CAOT plus PhMT-b, while an

255 leeding on probing, plaque index scores, and keratinized tissue width then recorded.

256 Keratinized tissue width was increased by 2.2 +/- 0.2 mm

257 Clinical measurement of recession and keratinized tissue width was standardized using customiz

258 Gingival thickness, keratinized tissue width, and bone morphotype are three

259 th and gain of keratinized tissue thickness, keratinized tissue width, and clinical attachment level

260 there was improvement in gingival thickness, keratinized tissue width, and root coverage.

261 orrelation with percentage of root coverage, keratinized tissue width, or keratinized tissue thicknes

262 lt in additional (or "rebound") increases in keratinized tissue width.

263 DDS graft was safe and capable of generating keratinized tissue without the morbidity and potential c

264 ad significantly more tissue thickness gain (keratinized tissue) than the crestal group compared to b

265 ed that 19 sites showed a slight increase in keratinized tissue, 35 were unchanged (for a total of 54

266 level, recession depth and width, amount of keratinized tissue, and bleeding on probing were obtaine

267 ft exposed resulted in a greater increase of keratinized tissue, and complete coverage of the graft r

268 t the widest point, probing depth, amount of keratinized tissue, and marginal tissue thickness.

269 me with increased gingival tissue thickness, keratinized tissue, and root coverage.

270 ), marginal tissue levels, biotype, width of keratinized tissue, and soft tissue thickness.

271 n terms of root coverage, increased width of keratinized tissue, and thickness of the gingival biotyp

272 : recession depth, recession width, width of keratinized tissue, clinical attachment level, and probi

273 r recession depth, recession width, width of keratinized tissue, clinical attachment level, and probi

274 ificant differences in root defect coverage, keratinized tissue, clinical attachment level, or clinic

275 istically equivalent, including increases in keratinized tissue, esthetic results, and subject satisf

276 n depth, clinical attachment level, width of keratinized tissue, mobility, and plaque score.

277 g depth, clinical attachment level, width of keratinized tissue, percussion sensitivity, pulp vitalit

278 l defect, width (WKT) and thickness (TKT) of keratinized tissue, probing depth (PD), and clinical att

279 Clinical measurements of vertical recession, keratinized tissue, probing depths, and attachment level

280 and slight, but significant improvements in keratinized tissue, tissue thickness, and bone level.

281 o the gingival margin, and with 2 to 3 mm of keratinized tissue.

282 ot coverage with an increase in the width of keratinized tissue.

283 ge, probing attachment levels, and increased keratinized tissue.

284 DM to free gingival graft in augmentation of keratinized tissue.

285 keratinized tissue and percent shrinkage of keratinized tissue.

286 robing depth, recession width, and amount of keratinized tissue.

287 es), and 7 sites showed a slight decrease in keratinized tissue.

288 gingival index, probing depth, and width of keratinized tissue.

289 re common in females, and often found on the keratinized tissue.

290 eans to treat gingival recession and augment keratinized tissue.

291 3) determine the effect of the procedure on keratinized tissue; and 4) evaluate the amount of creepi

292 onal extent of the gingival defect; width of keratinized tissue; probing depth; clinical attachment l

293 erged resulted in better preservation of the keratinized tissues (width and thickness) with similar a

294 root coverage (RC) and ADM-based increase in keratinized tissues to other commonly used mucogingival

295 rsor of the cornified envelopes of mammalian keratinizing tissues.

296 Well keratinized tonsillar SCCs lack HPV DNA and are associat

297 almost exclusively associated with a poorly keratinized tumor histology.

298 Interestingly, a majority of the keratinized tumors expressed high levels of miR-21.

299 These results demonstrate that the non-keratinized vaginal epithelium permits a rapid absorptio

300 s of aire-deficient mice were pathologically keratinized with significant epithelial damage and alter