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1 vide insight into the immune pathogenesis of keratoconjunctivitis sicca.
2 d efficacy in a clinically relevant model of keratoconjunctivitis sicca.
3 canine model of aqueous tear deficiency and keratoconjunctivitis sicca.
4 enatal death, vulva and clitoral defects and keratoconjunctivitis sicca.
5 moderate to severe dry eye disease caused by keratoconjunctivitis sicca.
6 ocular surface inflammation that mimic human keratoconjunctivitis sicca.
7 1 may play a key role in the pathogenesis of keratoconjunctivitis sicca.
8 ations, including inflammatory arthritis and keratoconjunctivitis sicca.
10 the immunopathogeneses of conditions such as keratoconjunctivitis sicca, allergic keratoconjunctiviti
11 s may play a key role in the pathogenesis of keratoconjunctivitis sicca and may be a therapeutic targ
12 mmon autoimmune rheumatic disease, refers to keratoconjunctivitis sicca and xerostomia resulting from
13 autoimmune disease that is characterized by keratoconjunctivitis sicca and xerostomia resulting from
15 are extreme tiredness, along with dry eyes (keratoconjunctivitis sicca) and dry mouth (xerostomia).
17 bow/hip dysplasia before osteoarthritis, and keratoconjunctivitis sicca before corneal ulcer, which a
18 eatures of ocular graft versus host disease (keratoconjunctivitis sicca, cicatricial conjunctivitis,
20 xperimental dry eye models and patients with keratoconjunctivitis sicca due to the induction of inter
22 s which included failed bleb, corneal edema, keratoconjunctivitis sicca, filamentary keratitis, corne
23 degeneration, neuroophthalmic disorders, and keratoconjunctivitis sicca in patients with HTLV-1-assoc
26 ces in pathogenesis include the concept that keratoconjunctivitis sicca is a condition of abnormal gr
27 to generate an inducible autoimmune model of keratoconjunctivitis sicca (KCS) for study of pathogenes
28 une; Sandoz, Basel, Switzerland) in treating keratoconjunctivitis sicca (KCS) include an effect on th
30 ta, and MMP-9, similar to that seen in human keratoconjunctivitis sicca (KCS) or "dry eyes." Therefor
31 surface epithelial changes resembling human keratoconjunctivitis sicca (KCS) that are exacerbated by
32 initiating inflammatory responses leading to keratoconjunctivitis sicca (KCS), autoimmunity, and Sjog
35 control subjects (C), 73 with non-Sjogren's keratoconjunctivitis sicca (non-SS KCS) and 21 with Sjog
36 ssment of 210 people, 130 with non-Sjogren's keratoconjunctivitis sicca (non-SS KCS), 32 with Sjogren
40 e implant was effective in a canine model of keratoconjunctivitis sicca, with all animals able to dis