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1  bilirubin encephalopathy (formerly known as kernicterus).
2 s and to review conventional MRI findings of kernicterus.
3 ch can further cause permanent brain damage, kernicterus.
4                       There were no cases of kernicterus.
5 jaundiced human newborns or animal models of kernicterus.
6 d hyperbilirubinemia and a life-long risk of kernicterus.
7 ometabolism was found in KMT2B, HPRT1 and CP-Kernicterus.
8 erebellar hypoplasia that we characterize as kernicterus.
9 tudies have shown characteristic findings in kernicterus.
10 permanent brain damage, a condition known as kernicterus Although a large portion of hyperbilirubinem
11                             HPRT1, PANK2, CP-kernicterus and CP-preterm cases had cerebellar and insu
12         Affected individuals are at risk for kernicterus and require lifelong phototherapy.
13 f hyperbilirubinemia in an effort to prevent kernicterus and sequelae from elevated bilirubin levels,
14 -Preterm), neonatal jaundice/kernicterus (CP-Kernicterus) and hypoxic-ischaemic encephalopathy (CP-Te
15 er the risks associated with SNJ, especially kernicterus, and possibly avoiding the morbidity associa
16                Four (14%) neonates developed kernicterus between days 14 and 45 postnatal days of lif
17 nt therapy relies on phototherapy to prevent kernicterus, but liver transplantation presently is the
18 scribe a case of severe neonatal anemia with kernicterus caused by compound heterozygosity for null m
19 ation, and neonatal bilirubin neurotoxicity (kernicterus) continues to occur worldwide.
20  prematurity (CP-Preterm), neonatal jaundice/kernicterus (CP-Kernicterus) and hypoxic-ischaemic encep
21 with Crigler-Najjar syndrome type II in whom kernicterus developed after a laparoscopic cholecystecto
22                                              Kernicterus generally occurs in untreated hyperbilirubin
23 in-induced neurologic dysfunction (BIND) and kernicterus has been used to describe moderate to severe
24 ebral palsy was judged to be consistent with kernicterus if magnetic resonance imaging of the brain r
25  a level that is sufficiently low to prevent kernicterus in infants.
26                                        Thus, kernicterus in this model displays not only axonal damag
27 nagement of hyperbilirubinemia in preventing kernicterus is examined.
28               Cerebral palsy consistent with kernicterus occurred in 3 infants (incidence, 0.57 per 1
29               Cerebral palsy consistent with kernicterus occurred only in infants with 2 or more risk
30                                     The term kernicterus, or bilirubin encephalopathy, is used to des
31  between the NAA and Cho levels in the acute kernicterus patients and the control group (healthy pati
32 h were significantly elevated in the chronic kernicterus patients.
33                                              Kernicterus presents as axonopathy with myelination defi
34 nd the onset of bilirubin encephalopathy and kernicterus result in part from delayed expression of UD
35 bilirubinemia or jaundice is associated with kernicterus, resulting in permanent neurological damage
36     Acute bilirubin encephalopathy (ABE) and kernicterus spectrum disorder (KSD) are common newborn h
37                           The development of kernicterus was the result of a largely preventable seri
38 AL/METHODS: Forty-eight pediatric cases with kernicterus were included in this study.