ライフサイエンスコーパス: ketogenic

1 (BCAAs) by replacing carbohydrate calories (ketogenic).

2 dipose tissue, with predominant increases in ketogenic amino acid abundance and alterations to metabo

3 energy imbalance by reducing metabolites in ketogenic amino acid and nucleotide metabolism.

4 th reduced fatty acid oxidation and enhanced ketogenic amino acid metabolism, together with alteratio

5 e body metabolism, along with an increase in ketogenic amino acids and glycine.

6 fatty liver morphology and higher levels of ketogenic and branched-chain amino acids (BCAA).

7 play key roles in metabolic pathways (e.g., ketogenic and gluconeogenic AAs), pointing to potential

8 Frequent switching between ketogenic and nonketogenic states may optimize health by

9 hepatocytes failed to express gluconeogenic, ketogenic, and urea pathway enzymes.

10 es to a high-protein, low-carbohydrate [(LC) ketogenic] and those to a high-protein, medium-carbohydr

11 trast, we observed an increased abundance of ketogenic beta-hydroxybutyryl-CoA, in association with i

12 butyrate (BHB), a metabolite produced during ketogenic conditions, is also an FFA3 agonist.

13 enesis in the liver under chronic fasting or ketogenic conditions, leading to hepatosteatosis; liver-

14 cterial species that produce stearic acid in ketogenic conditions, whereas consumers were depleted, r

15 Ketogenic conditions-starvation or ketogenic diet-increa

16 A commonly used rodent ketogenic diet (Bio-Serv F3666) that is very high in fat

17 Although caloric restriction (CR) and a ketogenic diet (KD) are often thought to limit tumour pr

18 A ketogenic diet (KD) can induce weight loss and improve g

19 The full anticonvulsant effect of the ketogenic diet (KD) can require weeks to develop in rats

20 ed clinical evidence to support the use of a ketogenic diet (KD) for cancer patients.

21 The high-fat ketogenic diet (KD) has become an increasingly popular d

22 A ketogenic diet (KD) has been reported to enhance respons

23 Weight loss by ketogenic diet (KD) has gained popularity in management

24 Assess the tolerability of a ketogenic diet (KD) in patients with relapsing multiple

25 We asked whether a high-fat ketogenic diet (KD) increases UCP levels and activity in

26 A ketogenic diet (KD) is a high-fat, low-carbohydrate diet

27 A ketogenic diet (KD) is a high-fat, low-carbohydrate meta

28 The ketogenic diet (KD) is a high-fat, low-carbohydrate, and

29 The ketogenic diet (KD) is a normocaloric diet composed of h

30 The ketogenic diet (KD) is an effective treatment option for

31 gated whether an isocaloric low-carbohydrate ketogenic diet (KD) is associated with changes in EE, re

32 An attenuating effect of the ketogenic diet (KD) on CRC cell growth has been previous

33 Previous studies have shown that animals fed ketogenic diet (KD) perform better in learning tasks tha

34 sarcoidosis, requiring preparation with the ketogenic diet (KD) to achieve myocardial glucose suppre

35 aOHB), the main ketone body (KB) produced in ketogenic diet (KD), is neuroprotective in aralar-knock-

36 s with AAAs received either a standard diet, ketogenic diet (KD), or exogenous KBs (EKB).

37 increases with fasting and consumption of a ketogenic diet (KD).

38 g or treatment with the high-fat antiseizure ketogenic diet (KD).

39 We examined the effect of a low carbohydrate ketogenic diet (LCKD) (80% kcal from fat, ketogenic rati

40 nic diet: OR, 8.6 [95% CI, 3.7-20.0]), while ketogenic diet (OR, 6.5 [95% CI, 2.3-18.0]) and modified

41 inuation rates were significantly higher for ketogenic diet (OR, 8.6 [95% CI, 1.8-40.6]) and modified

42 Additional animals received the ketogenic diet (starting at 20 or 60 d old).

43 iabetes or T2DM followed the well-formulated ketogenic diet (WFKD) and the Mediterranean-plus diet (M

44 nged deprivation of food or consumption of a ketogenic diet activates the IRE1alpha-XBP1 pathway in m

45 The ketogenic diet also may facilitate mechanisms by which t

46 Optimized cyclic ketogenic diet and 1,3-butanediol supplementation regime

47 iagnosis rates for 72-h ketogenic diet, 24-h ketogenic diet and 18-h fasting preparations are as foll

48 pression rates for 72-h ketogenic diet, 24-h ketogenic diet and 18-h fasting preparations are as foll

49 ldren were available for analysis: 54 on the ketogenic diet and 49 controls.

50 78 infants were assigned to a ketogenic diet and 58 to antiseizure medication, of whom

51 73 children were assigned to the ketogenic diet and 72 children to the control group.

52 ystem during protein absorption, following a ketogenic diet and an increased intake of antioxidants,

53 ch for a 4-wk period-an LC (4% carbohydrate) ketogenic diet and an MC (35% carbohydrate) diet-randomi

54 al decreases in total microbial loads on the ketogenic diet and enable us to determine the differenti

55 Dietary therapies, including the ketogenic diet and its variations, have been available f

56 lating FGF21 in response to starvation and a ketogenic diet and that FGF21 is required for behavioral

57 he epigenome gives us an appreciation of the ketogenic diet and the potential for bioenergetic therap

58 expression were observed in mice that ate a ketogenic diet and when fasting, which suggests that FGF

59 sacrifice, kidneys of knockout mice fed with ketogenic diet appeared macroscopically similar to those

60 In addition, the ketogenic diet appears to favor the synthesis of glutami

61 usions, general anesthesia, hypothermia, and ketogenic diet as treatment for uncontrolled status epil

62 In vivo, BHB or a ketogenic diet attenuates caspase-1 activation and IL-1b

63 Ketogenic diet can change the metabolism in the body and

64 activation of RIPK1 or metabolic rescue with ketogenic diet can prevent postnatal lethality and BBB d

65 profiling demonstrates that maintenance on a ketogenic diet causes a 25% reduction of myocardial (13)

66 ulation of most microbial pathways following ketogenic diet compared with baseline and vegan diet.

67 under standard diet, overnight fasting, and ketogenic diet conditions.

68 A ketogenic diet consisting of a high ratio of fat to carb

69 hepatocytes following exercise, fasting, or ketogenic diet consumption.

70 A ketogenic diet could be a treatment option in infants wh

71 e, we have explored whether treatment with a ketogenic diet could lead to a similar rescue through in

72 In this phase 4 trial, a ketogenic diet did not differ in efficacy and tolerabili

73 Notably, although a high-fat ketogenic diet elevates ISC function and post-injury reg

74 Weaning mice to a ketogenic diet extends viability to 8.5 wk.

75 dies required for survival during fasting or ketogenic diet feeding.

76 igh lipid utilization, including fasting and ketogenic diet feeding.

77 s induced by an overnight fast or short term ketogenic diet feeding.

78 However, we found that Pcx(L-/-) mice fed a ketogenic diet for 1 week became severely hypoglycemic,

79 esions in Eker rats (Tsc2+/-) subjected to a ketogenic diet for 4, 6 and 8 months.

80 by parents of some children treated with the ketogenic diet for epilepsy control, although this side

81 s were replicated in Ctns(-/-) rats fed with ketogenic diet from 2 to 8 months of life.

82 the age of 3 years who were treated with the ketogenic diet from April 2004 to June 2014 were retrosp

83 also observed after mice were fed with this ketogenic diet from the age of 6 months to the age of 15

84 h groups (40 [51%] of 78 participants in the ketogenic diet group and 26 [45%] of 58 participants in

85 The ketogenic diet had no effect in these models.

86 Ketogenic diet had significant benefits for preventing k

87 A ketogenic diet has also been found to induce mitochondri

88 The ketogenic diet has been widely and successfully used to

89 creased availability of anticonvulsants, the ketogenic diet has re-emerged as a therapeutic option.

90 therapy for pediatric epilepsy known as the ketogenic diet has seen a revival in its clinical use du

91 treatments such as the high-fat, antiseizure ketogenic diet have become mainstream, and metabolic sub

92 f this study was to test the efficacy of the ketogenic diet in a randomised controlled trial.

93 Here, we report that a ketogenic diet in Kmt2d(+/betaGeo) mice modulates H3ac a

94 onsible for the antiepileptic effects of the ketogenic diet in mice.

95 f this includes the therapeutic benefit of a ketogenic diet in neurologic diseases, including epileps

96 ning could in part explain the effect of the ketogenic diet in reducing epileptic seizures.

97 Strikingly, a ketogenic diet increased lysine acetylation in Cpt2M(-/-

98 The ketogenic diet induced hepatic lipid oxidation and ketog

99 tion of a high fat, low carbohydrate (HF-LC) ketogenic diet induces hepatic fibroblast growth factor

100 The ketogenic diet is a high-fat, adequate protein, low carb

101 A ketogenic diet is a potential adjuvant cancer therapy th

102 The medium chain triglyceride ketogenic diet is an established treatment for drug-resi

103 ingly, the use of therapeutic hypothermia or ketogenic diet is described as a strategy for super-refr

104 A ketogenic diet is used as anti-seizure therapy i.a. in t

105 Specifically, murine ketogenic diet lacks the complex spectrum of phytochemic

106 Ketogenic diet may provide a partial therapeutic alterna

107 The mechanisms underlying the ketogenic diet might also have roles in other disorders,

108 o mediate the stimulatory effect of an HF-LC ketogenic diet on FGF21 expression.

109 information is available on the effect of a ketogenic diet on plasma lipoproteins in children with d

110 Beneficial effects of ketogenic diet on tubular function were also observed af

111 without stratification, to either a classic ketogenic diet or a further antiseizure medication for 8

112 entified NLRP3 inflammasome blockade using a ketogenic diet or betaHB supplementation as a potential

113 ctive of whether ketosis was achieved with a ketogenic diet or exogenous ketone ester.

114 demonstrate that energy stress induced by a ketogenic diet or fasting can enhance checkpoint blockad

115 Replacing chow by ketogenic diet overrode the macroH2A1.1-dependent metabo

116 es of hepatic oxidative fluxes fueled by the ketogenic diet paralleled lower rates of de novo lipogen

117 and had not been treated previously with the ketogenic diet participated in a randomised controlled t

118 The ketogenic diet produced good seizure control.

119 A high-fat ketogenic diet produced significant increases in the ath

120 bgroup of the 371 patients accepted into the ketogenic diet program between 1994 and 2001.

121 Prolonged feeding of a ketogenic diet promotes the growth of renal tumors by re

122 Anticonvulsant mechanisms of the ketogenic diet remain incompletely understood.

123 A low-carbohydrate ketogenic diet remains one of the most effective (but my

124 All three patients who tried the ketogenic diet responded well to this treatment, and fut

125 is preclinical study, we demonstrated that a ketogenic diet significantly improves colitis in CGD mic

126 At 6 months, the high-fat ketogenic diet significantly increased the mean plasma l

127 consideration of dietary choline content in ketogenic diet studies in rodents to limit hepatic mitoc

128 Analysing medium-chain fatty acids in future ketogenic diet studies will provide further insights int

129 The results from this trial of the ketogenic diet support its use in children with treatmen

130 R in mice suppressed the ability of an HF-LC ketogenic diet to induce hepatic FGF21 gene expression.

131 Depletion of glucose levels using a ketogenic diet to lower lactate production by glycolytic

132 mised cells points to the potential use of a ketogenic diet to treat patients with heteroplasmic mtDN

133 Behavioral assays indicate that ketogenic diet treatment leads to the prolonged rescue o

134 Ketogenic diet treatment of Shank3-deficient mice elevat

135 Ketogenic diet treatment significantly reduced tactile a

136 ave been shown in the rat hippocampus during ketogenic diet treatment.

137 Our data revealed that overall, a ketogenic diet was associated with a significant upregul

138 A 4:1 ketogenic diet was maintained for 15 mo, and whole-body

139 mpared to other treatment options, while the ketogenic diet was most effective in maintaining seizure

140 Only a decade ago the ketogenic diet was seen as a last resort; however, it ha

141 e first year of life, while clobazam and the ketogenic diet were effective in long-term seizure manag

142 er barbiturates, vagus nerve stimulator, and ketogenic diet were not associated with outcome.

143 In 2009, two controlled trials of the ketogenic diet were published, as well as an Internation

144 and adults with G1D who were not receiving a ketogenic diet were selected on a first-come, first-enro

145 e report (k=0.88) Conclusion: A 72-h daytime ketogenic diet with 3-day overnight fasting, achieved su

146 superior myocardial suppression versus 24-h ketogenic diet with overnight fasting and 18h-fasting us

147 esponds to dietary manipulation such as the 'ketogenic diet'.

148 Growing evidence suggests that one of the ketogenic diet's main mechanisms of action is reducing i

149 of GLUT1 deficiency has important treatment (ketogenic diet) and genetic counseling implications.

150 (24 h of fasting and a very low carbohydrate ketogenic diet) to demonstrate that heart muscle engages

151 n of the diet (the medium-chain triglyceride ketogenic diet), have been shown to directly inhibit AMP

152 pecific diets (i.e., complex carbohydrate or ketogenic diet), iron supplementation, and albumin infus

153 as the modified Atkins diet) other than the ketogenic diet, (2) the relatively frequent occurrence (

154 CS diagnosis rates for 72-h ketogenic diet, 24-h ketogenic diet and 18-h fasting pre

155 Myocardial suppression rates for 72-h ketogenic diet, 24-h ketogenic diet and 18-h fasting pre

156 et, glucose predominates throughout but in a ketogenic diet, 3-hydroxybutyrate contributes most stron

157 terventions such as caloric restriction, the ketogenic diet, and a high-fat diet systemically alter a

158 tabolic factors such as caloric restriction, ketogenic diet, and hyperglycemia influence the inflamma

159 , not only as the mechanism of action of the ketogenic diet, but also as a potential target of future

160 me modulates the anti-seizure effects of the ketogenic diet, but how specific dietary formulations di

161 dy produced during fasting or adherence to a ketogenic diet, can inhibit the NLRP3 inflammasome and r

162 Ketogenic diet, diazepam, valproate, potassium bromide a

163 Children were randomly assigned to receive a ketogenic diet, either immediately or after a 3-month de

164 ermia, magnesium, pyridoxine, immunotherapy, ketogenic diet, emergency neurosurgery, electroconvulsiv

165 ion, pyridoxine, steroids and immunotherapy, ketogenic diet, hypothermia, emergency resective neurosu

166 es that several mechanisms may exist for the ketogenic diet, including disruption of glutamatergic sy

167 OR inhibition, serum-starvation/fasting, and ketogenic diet, increased ASO-mediated target reduction

168 er 80 years ago with the introduction of the ketogenic diet, is required.

169 d be suppressed by a carbohydrate-restricted ketogenic diet, MEDICA treatment suppressed tumor growth

170 ll now turn to dietary therapies such as the ketogenic diet, medium-chain triglyceride diet, modified

171 rials comparing different dietary therapies (ketogenic diet, modified Atkins diet, and low glycemic i

172 as the perceived absolute requirement for a ketogenic diet, the assumed lack of structural brain def

173 Our findings reveal that on a ketogenic diet, treatment with eFT508 (also known as tom

174 y pharmacological stimulation of PDH or by a ketogenic diet, two treatments used for PDH deficiencies

175 bohydrates are restricted in patients on the ketogenic diet, we evaluated the effects of limiting car

176 g opinions about the anticancer actions of a ketogenic diet, we have shown that this anti-seizure the

177 duals with epilepsy benefit from consuming a ketogenic diet, which is similar to the more commonly kn

178 at liver were dramatically up-regulated by a ketogenic diet, which lowered HNE basal level.

179 tant adjunct to pharmacologic therapy is the ketogenic diet, which often improves seizure control, ev

180 I3K inhibitors is dramatically enhanced by a ketogenic diet, with a proposed mechanism involving diet

181 compared with littermate control mice fed a ketogenic diet, yet it did not improve cardiac hypertrop

182 ncentrations are higher within the hearts of ketogenic diet-fed mice challenged with ketones compared

183 mpared with chow-fed C57BL/6 mice, fasted or ketogenic diet-fed mice displayed increased hepatic TG c

184 Ketogenic conditions-starvation or ketogenic diet-increase K8(+/+) HMGCS2, whereas this res

185 Here we focus on a novel hypothesis that a ketogenic diet-induced change in energy metabolism incre

186 These data suggest that a ketogenic diet-related bleeding tendency occurs in about

187 onent of the medium-chain triglyceride (MCT) ketogenic diet.

188 to respond appropriately to consumption of a ketogenic diet.

189 tely reversed in GF mice by consumption of a ketogenic diet.

190 after C75 injection in rats maintained on a ketogenic diet.

191 nd glycolysis), valproic acid (VPA), and the ketogenic diet.

192 cover possible therapeutic mechanisms of the ketogenic diet.

193 est in alternative therapies, especially the ketogenic diet.

194 with standard diet (P < 0.001), but not with ketogenic diet.

195 exogenous ketone ester supplementation or a ketogenic diet.

196 e reduction and may be a sounder option than ketogenic diet.

197 g translatable approaches such as fasting or ketogenic diet.

198 icarboxylic acid cycle and glutamate under a ketogenic diet.

199 irs ketogenesis in response to fasting and a ketogenic diet.

200 ity and attenuated seizures in animals fed a ketogenic diet.

201 rmacologic inhibition of PI3K and SGLT2 or a ketogenic diet.

202 mice consuming a high-fat, low-carbohydrate ketogenic diet.

203 ic action of a high fat and low carbohydrate ketogenic diet.

204 sant effect of the medium chain triglyceride ketogenic diet.

205 en fasted or fed a high-fat low-carbohydrate ketogenic diet.

206 nt antitumor mechanisms were launched by the ketogenic diet.

207 PHOS deficiency can be bypassed by feeding a ketogenic diet.

208 -intensity exercise, or the low-carbohydrate ketogenic diet.

209 ed Atkins diet: OR, 11.3 [95% CI, 5.1-25.1]; ketogenic diet: OR, 8.6 [95% CI, 3.7-20.0]), while ketog

210 riched exclusively in saturated fatty acids (ketogenic diet; KD) confer a hyper-inflammatory response

211 asting or by maintaining a low-carbohydrate (ketogenic) diet might reduce epileptic seizures and offe

212 ere, we show that a carbohydrate-restricted (ketogenic) diet potentiates CI induced by intermittent h

213 In a murine ketogenic-diet study, we compare microbial loads in lume

214 Here, we report that ketogenic-diet-induced changes in energy status enhance

215 ydrate, 35% fat, 15% protein) and isocaloric ketogenic diets (KD; 5% carbohydrate, 80% fat, 15% prote

216 Ketogenic diets (KDs) are popularly used to aid a myriad

217 Intermittent short-term fasting (ISTF) and ketogenic diets (KDs) exert overlapping but not identica

218 High-fat, low-carbohydrate ketogenic diets (KDs) have shown beneficial effects in m

219 Very low-carbohydrate, high-fat ketogenic diets (KDs) induce a pronounced shift in metab

220 Ketogenic diets (KDs)-very-low-carbohydrate and very-hig

221 Ketogenic diets (KGDs) have proven effective for disorde

222 d mortality, whereas carbohydrate-restricted ketogenic diets ameliorate T2D and suppress breast cance

223 As both ketogenic diets and intermittent fasting increase circul

224 gamma coactivator-1alpha pathway activation, ketogenic diets and other new strategies is required.

225 Despite some benefit from ketogenic diets and vagal nerve stimulation, there has b

226 Ketogenic diets are also now being increasingly studied

227 Over 70% of obese humans have NAFLD and ketogenic diets are common weight loss strategies.

228 ite, a response that seems to be absent when ketogenic diets are used.

229 Ketogenic diets are very efficient to control epilepsy a

230 Ketogenic diets are very low carbohydrate, high fat, mod

231 how the well-recognized clinical effects of ketogenic diets can alter human behavior via the same ce

232 Ketogenic diets can improve seizures in children older t

233 he mechanisms through which low carbohydrate ketogenic diets exert their ameliorative effects still r

234 participants sequentially consumed vegan or ketogenic diets for 2 weeks ( NCT03878108 ).

235 The use of ketogenic diets for weight loss is not warranted.

236 Second, common ketogenic diets lead to a therapeutically counterintuiti

237 flammatory effects of caloric restriction or ketogenic diets may be linked to BHB-mediated inhibition

238 Caloric restriction and ketogenic diets may modify the progression of neurologic

239 eckpoint blockade and advocate for combining ketogenic diets or AMPK agonists with anti-CTLA4 immunot

240 liver AQP9 levels were observed in rats fed ketogenic diets or high-protein diets, but AQP9 levels w

241 ate diets offered balanced benefits, whereas ketogenic diets produced greater weight loss but greater

242 Ketogenic diets promote nutritional ketosis and have sho

243 e short term, high-protein, low-carbohydrate ketogenic diets reduce hunger and lower food intake sign

244 However, the effectiveness of ketogenic diets toward alleviating NAFLD remains unclear

245 endurance sports and as medical treatment in ketogenic diets where these diets regulate energy metabo

246 tors, denosumab discontinuation, SARS-CoV-2, ketogenic diets, and extreme exercise, but these account

247 ose and -insulin conditions, associated with ketogenic diets, can reduce the activity of the mechanis

248 fied into 6 overarching thematic groups: (1) ketogenic diets, diabetes, supplements, and statins; (2)

249 High-fat, low-carbohydrate diets, known as ketogenic diets, have been used as a non-pharmacological

250 ch as starvation, diabetic ketoacidosis, and ketogenic diets, play a potentially important role in re

251 iciency on the pleiotropic effects of rodent ketogenic diets, we studied four custom diets that diffe

252 olic and physiologic alterations provoked by ketogenic diets.

253 were 3-5 times those reported with high-fat ketogenic diets.

254 LUT1-DS) who objected to or did not tolerate ketogenic diets.

255 ross LVEF (P(interaction)>0.10), whereas the ketogenic effect diminished at higher LVEF (P(interactio

256 gest that fasting-associated ketosis and the ketogenic effects of SGLT2 inhibitors occur almost entir

257 Methods: KEETO-CROSS (Ketogenic Endogenous versus Exogenous Therapies for myoC

258 We describe the rate-limiting enzyme of the ketogenic energy metabolism pathway, mitochondrial 3-hyd

259 In summary, chronic exposure to ketogenic environment during obesity and NAFLD has the p

260 Under a chronic "ketogenic environment," the hepatocyte diverted more ace

261 teraction between oncogenic BRAF V600E and a ketogenic enzyme 3-hydroxy-3-methylglutaryl-CoA lyase (H

262 cogenic BRAF(V600E) stimulates expression of ketogenic enzyme 3-hydroxy-3-methylglutaryl-CoA lyase an

263 e concomitantly decreasing gluconeogenic and ketogenic enzyme gene expression.

264 scles showed a dramatic up-regulation of the ketogenic enzyme HMGCS2 and the secreted protein FGF21 (

265 eceptor alpha-dependent induction of the key ketogenic enzyme HMGCS2.

266 Patients who received ketogenic feeding had fewer hypoglycaemic events (0.0% v

267 Ketogenic feeding was feasible and may be an interventio

268 Ketogenic feeding was feasible, safe, well tolerated and

269 liver disease (NAFLD) include remodeling of ketogenic flux and sustained tricarboxylic acid (TCA) cy

270 Addition of fiber to a fiber-deficient ketogenic formula restores seizure resistance, and suppl

271 protected mice that are fed fiber-containing ketogenic formulas.

272 ional mechanism, whereas L-FABP can activate ketogenic gene expression in fed mice.

273 nounced defect in ketone body production and ketogenic gene expression on fasting.

274 ation next to PPARA target genes and delayed ketogenic gene expression.

275 ha), the master transcriptional activator of ketogenic genes, and that suppression of NCoR1 (nuclear

276 Here we show that the prolongevity ketogenic hormone fibroblast growth factor 21 (FGF21), a

277 e and esterified PUFAs in hepatocytes, while ketogenic insufficiency depleted PUFAs and increased liv

278 arboxylic acid cycle, coupled with increased ketogenic/lipogenic activity via acetyl-CoA, 3-hydroybut

279 investigated the effects of adaptation to a ketogenic low carbohydrate (CHO), high fat diet (LCHF) d

280 Adaptation to a ketogenic low carbohydrate, high fat (LCHF) diet markedl

281 and biomarker change in adults adhering to a ketogenic low-carbohydrate (KLC) diet or a nonketogenic

282 p analyses explored differences by CRD type (ketogenic, low-carb, and moderate-carb), replacement mac

283 Given that myeloid cells express ketogenic machinery, we hypothesized this pathway may se

284 itially to approximately 22% after 5 days in ketogenic medium and was accompanied by a dramatic impro

285 emonstrate common (fatty acid) and distinct (ketogenic) metabolic signatures across the LVEF spectrum

286 Conversely, the ketogenic module operates via a GR-induced TF cascade, w

287 Therefore, promotion of ketogenic nutrient deprivation signaling by SGLT2 inhibi

288 l-CoA (HMG-CoA) lyase is a key enzyme in the ketogenic pathway that supplies metabolic fuel to extrah

289 by beta-oxidation within the "mitochondrial ketogenic pathway" (MKP) to generate beta-hydroxybutyrat

290 Fatty acids were transferred to the ketogenic pathway, and ketones were used in brown adipos

291 gene expression of fatty acid oxidation and ketogenic pathways.

292 a function causes diversion of liver-trapped ketogenic precursors into triacylglycerol.

293 te ketogenic diet (LCKD) (80% kcal from fat, ketogenic ratio 1.75:1, w/w) compared to a general hospi

294 lation, and hypoglycemia, with an inadequate ketogenic response in adult mice lacking PPARalpha (PPAR

295 These include the ketogenic response of the liver to fasting and diabetes,

296 Here, we directly test the ketogenic role of glucagon in mice, demonstrating that n

297 but the feasibility and safety of inducing a ketogenic state in physiologically unstable patients is

298 treatment with PPARgamma agonists and under ketogenic states.

299 To rescue the deficiency, we added ketogenic substrates to the water in which the embryos d

300 n additional 6 dietitian consultations for a ketogenic very-low-calorie diet (2 formulated meal repla