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1 inase (ROP18 and ROP16, respectively) is the key molecule.
2 to future structure-function studies of this key molecule.
3 s elicit pathogenesis and platelet CD40 is a key molecule.
4 adding cytokines they lacked or transfecting key molecules.
5  normal, reversed or bilateral expression of key molecules.
6 "jumpstarted" by the selective activation of key molecules.
7                                            A key molecule, alpha-gustducin, a primarily taste-specifi
8 idation of distinct neuronal roles played by key molecules already well known to immunologists (e.g.,
9 in imaging technology with probes that image key molecules and molecular based events that are fundam
10          These findings provide insight into key molecules and pathways involved in innate recognitio
11 complex remodelling process, with a focus on key molecules and pathways that might be suitable target
12 es is characterized by altered metabolism of key molecules and regulatory pathways.
13                                  Identifying key molecules and specific signaling pathways that regul
14 elegans model should yield new insights into key molecules and their possible implications in parasit
15 3 (IRF3) and nuclear factor (NF)-kB, that is key molecules and transcription factors involved in the
16           Cells in this knot express sets of key molecules, and as such have been proposed to act as
17 ty is associated with selective exclusion of key molecules, and that manipulation of transport can en
18  build a presynaptic terminal has been made, key molecules are recruited to assemble synaptic vesicle
19                                            A key molecule associated with the IS is protein kinase C
20 d alopecia, we examined expression levels of key molecules associated with hair follicle differentiat
21 ionally, these cells exhibited enrichment of key molecules associated with memory potential when comp
22 of the stress response kinase p38-MAPK, both key molecules associated with ultraviolet radiation derm
23                           microRNA-146a is a key molecule associating periodontitis with acute corona
24 nteresting implications for the synthesis of key molecules at the threshold of life.
25 le fundamental regulators, such as SOX9, are key molecules both in mice and humans, the way in which
26 onging to the TNF family (BAFF) represents a key molecule by which macrophages and DCs directly regul
27 ulating the metabolism of lipids through the key molecule carnitine palmitoyl transferase 1 (CPT1), i
28 xp3 and the surface protein CD25 are the two key molecules characterizing Tregs.
29 tor of mitochondrial calcium uniporter, as a key molecule conferring cancer cells with resistance to
30 therapeutic dosing of AE, whereas IGF-1 is a key molecule coupled to gene expression of other molecul
31 eptor interacting protein 3 kinase (RIP3), a key molecule critical for the assembly of the necrosome
32                  Genetic knock-down of these key molecules decelerated the proliferation of lymphoma
33                       Dishevelled (Dvl) is a key molecule downstream of Fz receptors.
34 notherapy and biologic therapies that target key molecules driving the Th2 response are already used
35 creasing evidence have pointed out CD25 as a key molecule during this transdifferentiation process, h
36 ha (P<0.01) and TGF-beta1 (P<0.01) which are key molecules during the onset of pregnancy.
37                    During the last year, one key molecule, dystrophin, has been shown to be disrupted
38                                    Recently, key molecules expressed at the initial-segment synapses
39 ning the GC response in mice have identified key molecules expressed on follicular dendritic cells th
40 f 5hmec in zygotes indicating that TET3 is a key molecule for 5hmec synthesis.
41  Thus, our findings establish IKK-gamma as a key molecule for adapting an oncoprotein-specific signal
42 llular process is still unclear, Beclin 1, a key molecule for autophagy, has been suggested to play a
43 r memory CD8(+) T cells expresses CD40L, one key molecule for CD4(+) T-cell-mediated help.
44 id organ homeostasis and identify RANKL as a key molecule for controlling the plasticity of the immun
45                 In the retina, dopamine is a key molecule for daytime vision.
46 th 5' AMP-activated protein kinase (AMPK), a key molecule for energy sensing that negatively regulate
47                               Cyclin B1 is a key molecule for G2-M-phase transition during the cell c
48                          Thus, Bcl-X(L) is a key molecule for hypoxia selection of death resistance.
49  have previously identified fibronectin as a key molecule for incorporation of LTBP1 and TGF-beta int
50              We identified IL-12/IL-23p40, a key molecule for innate immune responses to commensal ba
51             These results identify SOAP as a key molecule for ookinete-to-oocyst differentiation in m
52 d cyclophilin ligand interactor (TACI), is a key molecule for plasma cell maintenance and is required
53                                IKKalpha is a key molecule for the activation of the alternative NF-ka
54          These findings implicate sFRP2 as a key molecule for the biogenesis of a superior regenerati
55                   Cholesterol proved to be a key molecule for the function of synapses as its depleti
56                    Importantly, STING is the key molecule for the systemic baculovirus-induced IFN-I
57 ne (and related phytochelatins) could act as key molecules for ensuring the effective formation of HM
58 iced leader (SL) RNA and the large rRNAs are key molecules for mRNA maturation and protein synthesis,
59 roteins (TRAPs) that have been implicated as key molecules for parasite motility and adhesion onto ho
60  Ionotropic glutamate receptors (iGluRs) are key molecules for synaptic signaling in the central nerv
61 ese results suggest that IRS-1 and IRS-2 are key molecules for the TbetaR-V/LRP-1-mediated growth inh
62       We propose that myosin VI, by removing key molecules from developing hair bundles, is required
63 ts the acquisition of adaptation by removing key molecules from the hair bundle that serve a temporar
64 ing chemical logic to remind how these seven key molecules function as mobile packets of cellular cur
65    These findings have identified HDAC6 as a key molecule gating the effects of acute stress on synap
66 mRNA changes enabled us to predict ZEB1 as a key molecule governed by Galpha12.
67                            TRPM7 serves as a key molecule governing cellular Mg(2+) homeostasis in ma
68 s, stem cells and mitochondria interact with key molecules governing genome integrity, 'stemness' and
69               Previously, we identified that key molecules (IL6, CSF2, CCL5, VEGFA, and VEGFC) secret
70 owever, the regulatory role of miRNAs on the key molecules implicated in kidney fibrosis remains poor
71                            The expression of key molecules implicated in osteoclastogenesis (NFATc1,
72 ifferences in the expression or induction of key molecules implicated in viral induction of interfero
73         Our results have identified p11 as a key molecule in a specific cell type that regulates stre
74       Hydrogen peroxide (H2O2) is known as a key molecule in a variety of biological processes, as we
75 ol regulatory element-binding protein 2 is a key molecule in aggravating proinflammatory responses in
76 e lectin receptor CLECSF8 (CLEC4D, MCL) as a key molecule in anti-mycobacterial host defense.
77                Hydrogen peroxide (H2O2) is a key molecule in biology.
78 component of eukaryotic cell membranes and a key molecule in controlling membrane fluidity, organizat
79 plasticity, and prostaglandin E2 (PGE2) is a key molecule in COX-2-meduated synaptic modification.
80 , PfCRT, was recently demonstrated to be the key molecule in CQ resistance.
81 and degraded poly ADP ribose polymerase-1 (a key molecule in DNA repair and cell survival), leading t
82 tion of poly-ADP ribose polymerase (PARP), a key molecule in DNA repair, led to ovarian cancer patien
83 osure in CD8 T cells, with STAT4 acting as a key molecule in driving optimal antigen-specific respons
84 in gene, Rax, was previously identified as a key molecule in early eye formation in mice and humans.
85 nicotinamide adenine dinucleotide (NAD(+), a key molecule in energy and redox metabolism) decrease wi
86 n vitro and induce the expression of nNOS, a key molecule in gastrointestinal motility regulation.
87 mania spp., the spliced-leader (SL) RNA is a key molecule in gene expression donating its 5'-terminal
88 report that mice deficient in lymphotoxin, a key molecule in gut immunity, were resistant to DIO.
89 ated candidate genes and select one, EGR1, a key molecule in hippocampus-related learning and memory,
90                                  CTLA-4 is a key molecule in immunosuppression, and CD80 is a costimu
91 urther confirmed that DPP8 but not DPP9 is a key molecule in inducing cell death induced by DPP8/9 in
92 ld the acyl-NAD adduct which is considered a key molecule in INH action, providing a better understan
93 le can directly benefit another, making SP a key molecule in inter-ejaculate interaction.
94 y endocytic proteins, it is not considered a key molecule in mediating the major forms of endocytosis
95 immunoglobulin E (IgE) was identified as the key molecule in mediating what are now described as type
96 ported that S-adenosylmethionine (AdoMet), a key molecule in methylation reactions and polyamine bios
97  can be ameliorated by expression of Drp1, a key molecule in mitochondrial fission.
98  the DRGs of diabetic mice at 8 mo of age, a key molecule in pain signaling, and this effect was also
99                   We thus identify Sdf1 as a key molecule in pigment pattern formation, adding to the
100 r data suggest that myocardial MDA5 may be a key molecule in protecting the heart from direct viral i
101          These data suggest that ABCA12 is a key molecule in regulating keratinocyte differentiation
102 vator receptor (uPAR) has been shown to be a key molecule in regulating plasminogen-mediated extracel
103 helial growth factor A (VEGFA, rs3025033), a key molecule in retinal angiogenesis.
104      Importantly, PRDI-BF1 is defined as the key molecule in silencing CIITA and thus MHC-II in multi
105  These data strongly suggest that Cas-L is a key molecule in T cell migration induced by the ligation
106 r (DR)-dependent signals, it appears to be a key molecule in TG-regulated mitochondrial events.
107                  Par3 therefore emerges as a key molecule in the coupling of the early and late event
108 nsforming growth factor-beta (TGF-beta) as a key molecule in the development and progression of hepat
109 itously expressed cell surface protein and a key molecule in the etiology of Alzheimer disease.
110 ese results demonstrate that galectin-3 is a key molecule in the host defense against pneumococcal in
111 xia-inducible-factor 1alpha (HIF1alpha), the key molecule in the hypoxic response.
112             These data suggest that CD7 is a key molecule in the inflammatory response leading to LPS
113                       APPL1 binds to AKT2, a key molecule in the insulin signaling pathway, thereby e
114                                     Akt is a key molecule in the insulin/insulin-like growth factor s
115 oid protein (Abeta) has been implicated as a key molecule in the neurodegenerative cascades of Alzhei
116 inal development, was studied as a potential key molecule in the pathogenesis of BM.
117               Finally, Jab1 may constitute a key molecule in the pathogenesis of dysmyelinating neuro
118  G protein-coupled receptor (KSHV-GPCR) is a key molecule in the pathogenesis of Kaposi's sarcoma, pl
119                         alpha-Synuclein is a key molecule in the pathogenesis of synucleinopathy incl
120 inositol-4,5-bisphosphate (PtdIns-4,5-P2), a key molecule in the phosphoinositide signalling pathway,
121 lted in expression and activation of Akt1, a key molecule in the PI3 kinase signaling pathway.
122         Our study identifies lanosterol as a key molecule in the prevention of lens protein aggregati
123       L-selectin has become established as a key molecule in the recirculation of naive T cells from
124  expression of Lyn, indicating that Lyn is a key molecule in the regulation of BCR-mediated signaling
125 is a G-protein-coupled receptor (GPCR) and a key molecule in the regulation of energy homeostasis.
126                  Cardiac troponin (cTn) is a key molecule in the regulation of human cardiac muscle c
127                       Nitric oxide (NO) is a key molecule in the regulation of tumor-microenvironment
128 -associated protein of 25 kDa (SNAP-25) is a key molecule in the soluble N-ethylmaleimide-sensitive f
129 f cytokinesis, Plo1p is thus implicated as a key molecule in the spatial and temporal coordination of
130 main-containing adaptor protein (TIRAP) is a key molecule in the TLR4 and 2 signaling.
131  AR signaling and suggest that betaARK1 is a key molecule in the transition of myocardial hypertrophy
132  our data suggest that C5a(desArg) acts as a key molecule in the triggering of local inflammation as
133 ypes in NOD mice and indicate that CCR7 is a key molecule in their development.
134                                            A key molecule in this immune pathway is the intracellular
135                                            A key molecule in this process is the inositol lipid PtdIn
136  adenosine triphosphatase (ATPase) ISWI as a key molecule in this process.
137 kin 1 receptor-associated kinase 1(IRAK1), a key molecule in TLR/IL-1R-mediated signaling, is phospho
138 ynthesis of S-adenosylmethionine (AdoMet), a key molecule in transmethylation reactions and polyamine
139 der (SL) RNA, a trans splicing substrate and key molecule in trypanosome mRNA maturation.
140          Taken together, Smac may serve as a key molecule in vivo to selectively reduce the protein l
141 To validate and quantitate the expression of key molecules in a wide range of samples, we have develo
142  helper T cells and actively decreased these key molecules in antigen-activated T(FH) cells.
143  (cysteine-aspartate-specific proteases) are key molecules in apoptosis and require proteolytic remov
144 ctivated protein kinase, that alterations in key molecules in both GLUT4 trafficking and insulin sign
145             The cytokines IL-1 and IL-18 are key molecules in both the innate and the adaptive immune
146                    To understand the role of key molecules in determining the strength and nature of
147 ng suggested that these processes controlled key molecules in glomeruli and specifically podocytes, i
148      Noncoding RNAs (ncRNAs) are emerging as key molecules in human cancer, with the potential to ser
149 les and identifying specific alterations for key molecules in mechanotransduction.
150 summary, this study identifies SREBPs as the key molecules in regulating angiogenesis in response to
151 ogical or genetic inhibition of ROCK1 and 2, key molecules in Rho signaling, resulted in neuroblastom
152                    Thus, these cytokines are key molecules in several disease processes.
153 creasingly important to the functionality of key molecules in signaling, cell growth, and cell death.
154 scription initiation sites of genes encoding key molecules in TCR rearrangements.
155 aromatic hydrocarbons (PAHs) are regarded as key molecules in the astrochemical evolution of the inte
156  the intracellular concentrations of several key molecules in the bone formation cascade, we examined
157 ew risk loci could lead to identification of key molecules in the development of Barrett's oesophagus
158                               Antibodies are key molecules in the fight against infections.
159 d protein kinase Clambda (PKC-lambda), three key molecules in the insulin-signaling pathway, and was
160                     Amyloid-beta and tau are key molecules in the pathogenesis of Alzheimer's disease
161 hway and cancer-related genes, we identified key molecules in the selection of TRCs, such as ATF4, SL
162  ENU (N-ethyl-N-nitrosourea), have disclosed key molecules in the TLR signaling pathways and helped u
163 ponses is poorly understood, in part because key molecules in this mode of signal transduction, the m
164 habditis elegans have implicated a number of key molecules in this process, including the nutrient-se
165 netic mechanism studies have identified more key molecules (including Osterix, beta-catenin, and soni
166 tial to life because of its role in numerous key molecules, including DNA and RNA; indeed, organisms
167                         Prognostic genes are key molecules informative for cancer prognosis and treat
168 ip between sleep and beta-amyloid (Abeta), a key molecule involved in AD pathogenesis.
169 l 3,4,5-trisphosphate (PtdIns(3,4,5)P3) is a key molecule involved in cell growth signaling.
170 t also suppressed the expression of IL-7R, a key molecule involved in controlling intestinal ILC home
171 actor, Nr4a1, and its target gene, Cartpt, a key molecule involved in dopamine metabolism.
172                   It was found that STAT3, a key molecule involved in IL-6 signaling, was constitutiv
173              The androgen receptor (AR) is a key molecule involved in prostate cancer (PC) developmen
174  mice showed increased expression of RhoA, a key molecule involved in regulation of vascular tone, co
175                         Since NF-kappaB is a key molecule involved in the regulation of several of th
176                            Recently, another key molecule involved in this pathway was identified, sc
177 ancer metastasis; therefore, identifying the key molecules involved during this process promises to a
178  Macrophages deficient in WASp or Cdc42, two key molecules involved in actin core organization in pod
179 n D supplementation attenuated expression of key molecules involved in adipose tissue browning and am
180  Recent studies have highlighted some of the key molecules involved in all of these pathogenic proces
181 to evaluate the expression and activation of key molecules involved in cell survival and proliferatio
182  and ZAP-70 also associated differently with key molecules involved in cytoskeletal and calcium signa
183 temic immunosuppression by downregulation of key molecules involved in DC differentiation and suppres
184 ess has been made in identifying some of the key molecules involved in each of these steps.
185 harmacological inhibition against a panel of key molecules involved in galvanotaxis further revealed
186                   Sunitinib, an inhibitor of key molecules involved in neoangiogenesis, has an establ
187 mplex and its neurobiology, its relevance to key molecules involved in neurodegeneration and the pote
188 ome biosynthetic aspects of T. cruzi mucins, key molecules involved in parasite protection and virule
189 p70 can bind millions of proteins, including key molecules involved in processes of stemness, tumorig
190 he brain and thus controls the expression of key molecules involved in receptor signaling and spine m
191 t of NM, and (2) the expression of two other key molecules involved in regulating neuronal [Ca(2+)](i
192 r adapters capable of binding to enzymes and key molecules involved in signal transduction, maintaini
193                                 However, the key molecules involved in such regulation and the mechan
194 active zone formation and the scaffolding of key molecules involved in SV recycling.
195 f transcriptional factors, ion channels, and key molecules involved in synaptic transmission.
196   Transcriptomics revealed downregulation of key molecules involved in T cell receptor (TCR) signalin
197 NA microarray analysis is starting to reveal key molecules involved in the accumulation of free chole
198 ated in part through increased expression of key molecules involved in the class II MHC pathway via i
199 in the striatum, we suggest that they may be key molecules involved in the expression of the dyskines
200  Wiskott-Aldrich syndrome protein and Cdc42, key molecules involved in the formation of actin-based p
201 on on Th17 cells and inhibited expression of key molecules involved in the generation of pathogenic T
202 aken together, these results have identified key molecules involved in the group II mGluR-induced pot
203                            Almost all of the key molecules involved in the innate and adaptive immune
204              Genes and protein expression of key molecules involved in the insulin signaling cascade
205 gnaling systems as well as the processing of key molecules involved in the pathology of the disease.
206                                 In addition, key molecules involved in the TRAIL/DR5 pathway during D
207 s, and progress has been made in identifying key molecules involved in this signaling.
208 wn mechanisms by which ATP and adenosine are key molecules involved in thrombosis by regulating the a
209 mad signaling pathway, and the production of key molecules involved in tissue repair, such as type I
210 e route to the identification of some of the key molecules involved.
211 hich is abundant in spontaneous tumors, as a key molecule limiting this IFN-induced tumor regression
212              The data suggest that CDX1 is a key molecule linking etiological agents of BM to the dev
213               Data indicate cathepsin B as a key molecule mediating neurodegeneration, opening resear
214                      Laminin appears to be a key molecule mediating the epithelial-mesenchymal intera
215 results indicate that the OAMB receptor is a key molecule mediating the octopamine's signal for appet
216 monstrating that Nemo-like kinase (NLK) is a key molecule modulating disease toxicity in spinocerebel
217 e forkhead transcription factor, FoxP3, is a key molecule necessary and sufficient for Tregs developm
218       Spinal application of retinoic acid, a key molecule necessary for iMF, bypasses NMDA receptor-m
219 r animal models, have identified some of the key molecules necessary for peripheral innervation and f
220 ed that Meis1 regulates the transcription of key molecules necessary for the endosomal machinery.
221 that RB deficiency altered the expression of key molecules needed for proper cellular organization an
222 ic, stochastic noise in small populations of key molecules (notably, Period mRNA near its daily minim
223               The synthesis of cysteine, the key molecule of any thiol metabolism, has been elucidate
224                                          One key molecule of T cell activation is the actin-remodelin
225 ylation of kinesin light chain 2 (KLC2), the key molecule of the kinesin cargo delivery system.
226  cell death converging at anoikis, including key molecules of interaction such as Beclin 1, reactive
227 nosaialic acids, is thought to be one of the key molecules of signal transduction in mammalian cells.
228 d small interfering RNAs (viRNAs), which are key molecules of the antiviral RNAi response.
229 trate a sex-specific interaction between two key molecules of the human serotonergic system, and sugg
230 or, type I (IL1R1) gene, which is one of the key molecules of the inflammatory pathways.
231                                          The key molecules of the oxygen-sensing system include the t
232  for characterizing the interactions between key molecules of the slit diaphragm that control glomeru
233 e of motor neurons in ALS and that targeting key molecules of this cascade may prove to be neuroprote
234 he muscle-specific kinase MuSK is one of the key molecules orchestrating neuromuscular junction (NMJ)
235  to bromodomain, testis-specific (Brdt) as a key molecule participating in chromatin remodeling durin
236 ve response may be determined, in part, by a key molecule, periostin, which maintains the integrity o
237         Tumor-derived PTHrP has emerged as a key molecule playing multiple roles in cachexia, from fa
238         Tumor-derived PTHrP has emerged as a key molecule playing multiple roles in cachexia, from fa
239                         The contributions of key molecules predicted to align chromosomes at the cent
240 rtive activity, suggesting that IGF-2 is the key molecule produced by these cells that stimulates HSC
241                                              Key molecules promoting migration and invasion exist in
242 proach with the kinase hits identified other key molecules putatively involved in tau phosphorylation
243                             AEG-1 might be a key molecule regulating initiation and progression of NA
244     This was spearheaded by the discovery of key molecules regulating body weight homeostasis.
245                                 However, the key molecules regulating homeostasis in central mammalia
246                            MHC class II is a key molecule required for Ag presentation to Th cells, p
247           These findings identify LTBP4 as a key molecule required for the stability of the TGFbeta r
248      Thus, our study has shown that Dcc is a key molecule required for ventral migration of early-bor
249 patients with PIK3CD GOF mutations, identify key molecules required for cell-mediated immunity agains
250 el was developed to simulate the dynamics of key molecules required for LTP and LTD.
251              Thus, SAP affects expression of key molecules required for T-B cell collaboration by mec
252                               Prestin is the key molecule responsible for OHC electromotility.
253 d phospholipase A2 group IB (PLA2G1B) as the key molecule responsible for the formation of aMMDs.
254                                    CD14 is a key molecule responsible for the innate host inflammator
255 ype NPCs, suggesting that TSP1 is one of the key molecules responsible for astrocyte-induced neurogen
256        However, the mechanosensory cells and key molecules responsible for sensing the physical prope
257                   In this context, CD73 is a key molecule, since via degradation of adenosine monopho
258  apparently result in the down-regulation of key molecules, such as cyclin A, which may be responsibl
259                                              Key molecules, such as the transduction channel, may be
260  our knowledge, cyclin D3 is identified as a key molecule targeted by autoimmunity that plays a nonre
261           Here we demonstrate that IL-9 is a key molecule that affects differentiation of T(H)17 cell
262 e-acetylglucosaminyltransferase (LARGE) is a key molecule that binds to the N-terminal domain of alph
263 retory protein 4 (HE4) is a newly discovered key molecule that causes ECM deposition.
264 e identify PCNA-associated factor (PAF) as a key molecule that controls cancer cell stemness.
265 s identifying adenosine deaminase (ADA) as a key molecule that delineates a human Tfh helper program
266      Studies were designed to characterize a key molecule that is involved in the dysregulation.
267 ty of cyclic AMP-dependent protein kinase, a key molecule that is known to be involved in L-LTP.
268 ogether, our results support that TRAF6 is a key molecule that mediates the interaction between melan
269 S, a member of the CD28 family, represents a key molecule that regulates adaptive responses to foreig
270                               Delineation of key molecules that act epigenetically to transduce diver
271                               Examination of key molecules that are known to be required for limb dev
272         Recent studies indicate that certain key molecules that are vital for various developmental p
273  were combined with RNA-seq data to identify key molecules that associate with long-term durable self
274       Thus, our study has identified several key molecules that control USP33 degradation within the
275                            Identification of key molecules that drive angiogenesis is critical for th
276 istinct microenvironmental niches to provide key molecules that drive innate and adaptive immune resp
277                                           As key molecules that drive progression and chemoresistance
278 terations in the expression of Fgf8 and Shh, key molecules that establish a signaling center critical
279 y novel components of the pathway, including key molecules that function as positive or negative regu
280 ding proteins profilin and thymosin beta4 as key molecules that localize actin monomers to the leadin
281 pe 2 BBZDR/Wor rats changes in expression of key molecules that make up the nodal and paranodal appar
282             The absence of Msh2 or Mlh1, two key molecules that mediate mismatch repair in eukaryotic
283 ppaB kinase alpha (IKKalpha) and IKKbeta are key molecules that predominantly mediate noncanonical an
284 d liver sinusoidal endothelial cells express key molecules that recruit and activate innate and adapt
285             To address this problem, cyclins-key molecules that regulate cell cycle transition-have b
286            Long noncoding RNAs (lncRNAs) are key molecules that regulate gene expression in a variety
287 data demonstrate a functional link between 2 key molecules that regulate hypoxia preconditioning and
288                                  Identifying key molecules that regulate the crosstalk between the im
289 alian target of rapamycin complex 1 serve as key molecules that sense cellular energy and nutrients l
290                                        Three key molecules that sense nutrients and broadly affect ge
291 ll proliferation and differentiation and are key molecules that target retinoid and retinoic acid rec
292 , phosphorylate and regulate the activity of key molecules that ultimately control the expression of
293 depend on the precise and timely delivery of key molecules throughout life.
294 stence in their hosts while also revealing a key molecule to pursue while devising methods to improve
295   Taken together, we propose that Foxc2 is a key molecule to regulate PAI-1 gene expression.
296                                            A key molecule to solve the problem, hexamethylenetetramin
297 gulation factors, such as myosin-1, serve as key molecules to link phosphatidylinositol signals to po
298 of Trk, identifying Rac GTPase as one of the key molecules whose activity is critical for cell surviv
299 r applicability of this approach for linking key molecules with defined cellular functions in another
300 ion pathways and that, specifically, several key molecules within the mitogen-activated protein kinas

 
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