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1 ndent fibrotic signaling within cells of the kidney collecting duct.
2 ant role in regulating acid secretion in the kidney collecting duct.
3 L are coexpressed in epithelial cells of the kidney collecting duct.
4 ence exists for active urea transport in the kidney collecting duct.
5 lated insulin receptor (IR) signaling in the kidney collecting duct.
6 water pores in principal cells that line the kidney-collecting duct.
7 he apical membrane of principal cells in rat kidney collecting ducts.
8 expressing the human proto-oncogene c-MYC in kidney collecting ducts.
9 nes of barrier epithelia such as bladder and kidney collecting duct, a feature essential to barrier f
10                         PAR2 is expressed in kidney collecting ducts, a main site of control of Na(+)
11 agonist-stimulated Na(+) reabsorption in the kidney collecting duct, acting to enhance Na(+),K(+)-ATP
12 ateral plasma membrane of principal cells in kidney collecting duct, airway epithelium, and gastric p
13 lateral plasma membranes of epithelia in the kidney collecting duct, airways, stomach, and colon.
14 a master regulator of gene expression in the kidney collecting duct and in vivo loss of NFAT function
15 ing the epithelial barrier properties of the kidney collecting duct and is important to osmoregulatio
16 d with abnormal cytoskeletal dynamics in the kidney collecting ducts and perturbed calcium and cAMP s
17  in the outer and inner medulla, in isolated kidney collecting ducts, and in cultured outer and inner
18 ulates the osmotic water permeability of the kidney collecting duct by inducing exocytotic insertion
19   Alpha intercalated cells (alphaICs) in the kidney collecting duct (CD) belong to a family of mitoch
20 e uptake of fluorescently loaded ECVs into a kidney collecting duct cell line (mCCDC11) and into prim
21 ly stimulated ENaC-mediated Na+ current in a kidney collecting duct cell line, although GILZ2 and GIL
22 undantly expressed in mouse kidney and mouse kidney collecting duct cells (mpkCCD14).
23           Deleting this gene specifically in kidney collecting duct cells prevents cilia formation an
24 rmines ciliary trafficking of polycystins in kidney collecting duct cells without affecting protein l
25                 Here we showed that in mouse kidney collecting duct cells, claudin-4 functioned as a
26                                           In kidney collecting duct cells, filamentous actin (F-actin
27 ation induced by desmopressin stimulation of kidney collecting duct cells.
28 s as a result of an autoimmune insult on the kidney collecting duct cells.
29 D in association with autoantibodies against kidney collecting duct cells.
30 er endothelial (bEnd.3) or epithelial (mouse kidney collecting duct) cells increases the rate of calc
31 uaporin 2 (AQP2) water channel, expressed in kidney collecting ducts, contributes critically to water
32 wth of cysts generated by Madin-Darby canine kidney collecting duct-derived cells in three-dimensiona
33               Thus, talins are essential for kidney collecting duct development through mechanisms th
34 thelial sodium channel (ENaC) present in the kidney collecting duct, distal colon, and the lung is re
35 ased ENaC current in Fischer rat thyroid and kidney collecting duct epithelia.
36 that KO of either PLEKHA7 or PDZD11 in mouse kidney collecting duct epithelial cells results in incre
37 ressed at the basolateral plasma membrane of kidney collecting-duct epithelial cells.
38 pressed in the epithelial cells of the human kidney collecting duct in response to uropathogens.
39 ostcapillary venules, the epithelial cell of kidney collecting ducts, lung alveoli, and the Purkinje
40                         In this study murine kidney collecting duct (mCCDC11) cells were used to demo
41 sed to investigate the role of miRNAs in the kidney collecting duct of mice.
42 traced the fate of genetically labeled adult kidney collecting duct principal cells after Hes1 inacti
43  plasma membrane accumulation of AQP2 in rat kidney collecting duct principal cells in situ, and in s
44 ion in cultured AQP2-expressing cells and in kidney collecting duct principal cells.
45 sides PT cells, pRCC can also originate from kidney collecting duct principal cells.
46 GTPase, Rac1, as a driver of postobstructive kidney collecting duct repair.
47  of the ureter, which leads to injury of the kidney collecting ducts, results in permanent structural
48      We found that HDAC7 is expressed in the kidney collecting duct, supporting a potential role for
49  normally associated with the differentiated kidney collecting duct system including the water channe
50 ystic dilation of the cortical and medullary kidney collecting duct system, a phenotype resembling in
51 ishable from that in basolateral membrane of kidney collecting duct; the E-face showed corresponding
52 n cornea, oral mucosa, esophagus, intestine, kidney collecting ducts, ureter, bladder, urethra, and t
53 + absorption across epithelia, including the kidney collecting duct, where it plays a critical role i
54 rin 2 (AQP2) is a water channel found in the kidney collecting duct, where it plays a key role in con