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1 alcium-independent PLA2 activity from rabbit kidney cortex.
2 idney, lymphatic vessels exist mainly in the kidney cortex.
3 tubular cell structure and function in mouse kidney cortex.
4 o analyze differential protein expression in kidney cortex.
5 rtex and isolated CCDs from mouse and rabbit kidney cortex.
6 downstream events in mRNA translation in the kidney cortex.
7 oduct of ARID1A, than did the matched normal kidney cortex.
8 in G30 fetus, G45 liver, and adult liver and kidney cortex.
9 ho-tuberin and fibronectin expression in the kidney cortex.
10 iabetic rats had more apoptotic cells in the kidney cortex.
11 ortex was significantly reduced in OVE26 RKO kidney cortex.
12 antly lower in OVE26 RKO compared with OVE26 kidney cortex.
13 itochondrial Nox4 expression is increased in kidney cortex.
14 KRD11, and TNRC18 map to active enhancers in kidney cortex.
15 hat show expression changing with age in the kidney cortex.
16 n embryo day 14.5, in adult hepatocytes, and kidney cortex.
17 d show concordant DNA methylation changes in kidney cortex.
18 ons from noninfected LLC-PK(1) cells and rat kidney cortex.
19 NA isolated from LLC-PK1 cells, and from pig kidney cortex.
20 rcalated cells identified in sections of rat kidney cortex.
21 ipids has been recently purified from rabbit kidney cortex.
22 st protein liquid chromatography from rabbit kidney cortex.
23 antly lower levels of IR mRNA and protein in kidney cortex (49-56% of the WT) and medulla (32-47%) ho
24       We found decreased malonylation in the kidney cortex (~80% proximal tubules) of type 2 diabetic
25                   Immunoblot analysis of rat kidney cortex and brain identified immunoreactive protei
26 oducts from RNA isolated from rat and rabbit kidney cortex and cerebellum and rabbit S(2) segments we
27 -salt diet led to lower levels of EGF in the kidney cortex and enhanced the expression and activity o
28 beta expression and mTORC1 activation in the kidney cortex and glomeruli of diabetic mice and rats, r
29 ssion in the streptozotocin-induced diabetic kidney cortex and glomeruli, concomitant with activation
30 argets under physiological conditions in the kidney cortex and in primary renal cell cultures.
31 expressed in the nephrogenic zone of newborn kidney cortex and in the medulla.
32 cinic acid ((99m)Tc-DMSA) accumulates in the kidney cortex and is widely used for imaging of the rena
33 ression of mRNA encoding SDF1 (or CXCL12) in kidney cortex and isolated CCDs from mouse and rabbit ki
34 ntitation of the drug and its metabolites in kidney cortex and liver tissues.
35 -1 versus -2 was 2- and 7-fold higher in the kidney cortex and liver, respectively, whereas expressio
36 expressed at sites of ADMA metabolism in the kidney cortex and liver, whereas EDRF/NO is regulated pr
37 n PFAS concentrations were found between the kidney cortex and medulla and between lung lobes.
38 brane-enriched fraction of brain, liver, and kidney cortex and medulla by radiochemical assay.
39 regionally based surviving fractions for the kidney cortex and medulla in terms of their concentratio
40          Expression microarray data from the kidney cortex and medulla, liver, lungs, and spleen were
41 ial transcriptomic data from the adult human kidney cortex and medulla.
42 cellular (EC) and copper/zinc (CuZn) SOD, in kidney cortex and medulla.
43 s as well as markers of oxidative stress, in kidney cortex and medulla.
44 cultured rat mesangial cells (MC) as well as kidney cortex and microdissected glomeruli were examined
45 brary and sequencing revealed that the human kidney cortex and neuronal beta(3) subunits were identic
46 te that cortactin is highly expressed in the kidney cortex and polarized epithelial cells, and is loc
47 -PCR revealed expression of Gi but not Go in kidney cortex and preglomerular vessels.
48 otide and amino acid composition, and rabbit kidney cortex and rabbit neuronal GABA(A) receptor beta(
49 he kidney PCR products revealed that the rat kidney cortex and rat neuronal GABA(A) receptor beta(2)
50 he kidney PCR products revealed that the rat kidney cortex and rat neuronal GABA(A) receptor beta(3)
51 ression of SLC26A7 remained unchanged in the kidney cortex and stomach in water deprivation, indicati
52 ract separate factors that correspond to the kidney cortex and the kidney pelvis.
53 ignificant decrease in AQP2 abundance in the kidney cortex and the outer medulla.
54 protocol, 68 ablations were performed in the kidneys (cortex and medulla) and livers of 27 adult pigs
55  marked increase in non-heme iron content of kidney cortex, and a marked increase in the non-heme iro
56 chment of pathways involving NFkappaB in the kidney cortex, and a targeted data mining approach ident
57 urgery produced a metabolic switch in liver, kidney cortex, and fat depots toward decreased mitochond
58 pathways and reduces metabolism in the fetal kidney cortex, and ketamine blocks or ameliorates this r
59 al RNAs were isolated from both duodenum and kidney cortex, and the VDR and calbindin mRNA levels wer
60 duced DC numbers in the healthy and inflamed kidney cortex, and to a lesser degree in the kidney medu
61 Aging Kidney Anatomy study has characterized kidney, cortex, and medulla volumes using a manual image
62 utomated segmentation approach for measuring kidney, cortex, and medulla volumes.
63     At PTPN6/PHB2 cg19942083, methylation in kidney cortex associates with lower renal PTPN6 expressi
64 tribution was highly organ specific with the kidney cortex being the major target organ, followed by
65 RNA-seq) data set and Mendeley Data on human kidney cortex biopsy proteomics were used.
66 nding protein in membranes obtained from the kidney cortex but not in membranes from oocytes.
67 centration of gadolinium was retained in the kidney cortex, but T1-weighted MRI was not sensitive for
68 iency in db/db mice is sufficient to restore kidney cortex C1P content and to protect from DKD.
69 lial cells from ADPKD cysts and normal human kidney cortex cells (HKC) were cultured, and cAMP levels
70 significant polyol pathway activation in the kidney cortex characterized by high levels of aldose red
71                     Gene expression in fetal kidney cortex collected 24 h after the onset of hypoxia
72 protein expression was increased in diabetic kidney cortex compared with non-diabetic controls and wa
73                            RNA-sequencing of kidney cortex demonstrated increased transcripts involve
74                 Immunoblot analysis of human kidney cortex demonstrated that hOAT1 is an 80- to 90-ki
75 liver, bladder, sublinguinal salivary gland, kidney cortex, dermis, and synovial membrane were comple
76 lternative splicing of the DNase I mRNA, rat kidney cortex DNA complementary to RNA library was scree
77 man immune cells are uncommon in the porcine kidney cortex early after xenotransplantation and consis
78 g sequences derived from the purified rabbit kidney cortex enzyme.
79                   Results For all GBCAs, the kidney cortex exhibited higher gadolinium retention at 1
80 rsed the up-regulation of mRNA expression of kidney cortex fibrosis genes (CCN2, Col1a2, TGF-beta1, a
81                           By immunoblotting, kidney cortex homogenate from patients treated with angi
82 cs and phosphoproteomics of freshly isolated kidney cortex identified either reduced expression or lo
83                    Northern blot analysis of kidney cortex in both mice revealed nearly equivalent me
84 ed expression of Smad3 protein levels in the kidney cortex in response to chronic treatment with a hi
85 solution optical cross-section images of the kidney cortex in situ and in real time, which can provid
86           We observed GPR4 expression in the kidney cortex, in the outer and inner medulla, in isolat
87 g the plasma membrane of L2 cells and in the kidney cortex is expressed in glomerular and proximal tu
88 mia-reperfusion-induced serum creatinine and kidney cortex kidney injury molecule-1 levels by 78% and
89                                       In the kidney cortex, kidney medulla, and lungs, the TH-associa
90 scriptome-wide association studies (TWAS) in kidney cortex, kidney tubule, liver, and whole blood and
91 TWAS across four kidney transcriptomes (GTEx kidney cortex, kidney tubules, TCGA-KIRC [The Cancer Gen
92                           By screening a rat kidney cortex library with a CaT1 probe, we isolated a c
93 ere reduced selectively by 35% to 85% in the kidney cortex, liver, and MRVs 72 hours following the co
94 elaxation times were 21% longer (P <.05) for kidney cortex, liver, and spleen and T2 relaxation times
95   Angiotensinogen mRNA and protein levels in kidney cortex, measured by real-time reverse transcripta
96 e presence of rOCT1A mRNA transcripts in rat kidney cortex, medulla, and intestine.
97                         Replenishment of the kidney cortex microvasculature requires 1-5 seconds comp
98 xpression was significantly increased in the kidney cortex of db/db mice, and exposure to palmitate o
99 d restore the lipid metabolic network in the kidney cortex of diabetic (db/db) mice, raising the poss
100 d complex lipid synthesis are altered in the kidney cortex of diabetic patients.
101 NA expression showed significant increase in kidney cortex of heterozygous Akita(+/-) and db/db mice
102  liver of rodents, but in both the liver and kidney cortex of humans and pigs; therefore, the pig was
103 MP, whereas the cAMP effect was abolished in kidney cortex of NHERF1(-/-) mice.
104  was isolated mitochondria obtained from the kidney cortex of rats treated with IFO or IFO + AGM.
105                                       In the kidney cortex of TGF-beta transgenic mice, the significa
106         Although studies in BB isolated from kidney cortex of wild type and NHERF1(-/-) mice have sho
107  observed in human podocytes in vitro and in kidney cortexes of diabetic db/db mice in vivo.
108 ding of intravenously injected F-Dapa in the kidney cortexes of rats and wild-type and Sglt1-knockout
109                                          For kidney cortex, ovoid zones of 1.5 cm +/- 0.1 x 0.5 cm +/
110                                       In the kidney cortex, perturbation of homeostasis by a TLR7-dep
111                                           In kidney cortex, preparations consisting mainly of proxima
112        Primarily CX3CR1-dependent DCs in the kidney cortex processed antigen for the intrarenal stimu
113 s expression system, and the purified rabbit kidney cortex protein exhibit both CaIPLA2 and lysophosp
114 n, revealing significant accumulation in the kidney cortex region close to glomeruli.
115 ound that they were enriched 4.5-fold in the kidney cortex relative to nonspecific control T cells 24
116          Immunofluorescence studies in human kidney cortex revealed that ATP6N1B localizes almost exc
117 lot analysis in cultured mesangial cells and kidney cortex revealed that Nox4 is present in crude mit
118 ate gel analysis of extracts from normal rat kidney cortex revealed the presence of a DNase with an a
119             EPR spectroscopy analysis of rat kidney cortex samples showed that all GBCAs were primari
120  analysis on 404 ccRCC tumors and 167 normal kidney cortex samples using publicly available databases
121 imes (mean +/- SD) at 3.0 T are reported for kidney cortex (T1, 1,142 msec +/- 154; T2, 76 msec +/- 7
122 spite a 50% lower AIF protein content in the kidney cortex, there was no loss of complex I activity o
123 tion of this PLA2 to homogeneity from rabbit kidney cortex through sequential column chromatography i
124 etramer in blood vessels, RidA homotrimer in kidney cortex tissues).
125                                              Kidney cortex volume, but not renal histology parameters
126            Conversely, synthesis of NAADP in kidney cortex was almost undetectable.
127 al imaging; and gadolinium speciation in the kidney cortex was assessed using EPR spectroscopy.
128                        Microsomes from human kidney cortex were found to convert AA to a single major
129 ild type and NHERF1(-/-) ileum and wild type kidney cortex were inhibited by cAMP, whereas the cAMP e
130 2 transcripts are very abundant in liver and kidney cortex, whereas the expression is significantly l

 
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