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1 rived tubuloids represent a functional human kidney tubule.
2 on of major Na(+) transporters all along the kidney tubule.
3 determinants of Na(+) reabsorption along the kidney tubule.
4 nsor, detecting changes in fluid flow in the kidney tubule.
5 ociated with lower DACH1 expression in human kidney tubules.
6 ferroptotic cell death propagation in female kidney tubules.
7 protein kinase B (also known as AKT1) in the kidney tubules.
8 glomeruli, peri-glomerular regions, and some kidney tubules.
9 ower expression of ACE-2 at the level of the kidney tubules.
10 fying a novel role of forkhead box O4 in the kidney tubules.
11 tions via suppression of mTORC1 signaling in kidney tubules.
12  cells induced the acquisition of functional kidney tubules.
13 nes PKD1 or PKD2 induces cyst formation from kidney tubules.
14  organ ducts such as the digestive tract and kidney tubules.
15 r that is characterized by cyst formation in kidney tubules.
16 efect in oriented cell division in precystic kidney tubules.
17 usters of interstitial cells surrounding the kidney tubules.
18 its differentiation of these precursors into kidney tubules.
19 ns at concentrations present in seawater and kidney tubules.
20 s from different segments and distinct mouse kidney tubules.
21 sely, augmentation of NAD(+) may protect the kidney tubule against diverse acute stressors.
22   Histology revealed a subsequent absence of kidney tubules, an enlarged cardinal vein and expansion
23 .5% ascribed to the fetal lung, 59.4% to the kidney tubule and 6.2% to the small intestine).
24 potassium channel (ROMK) is expressed in the kidney tubule and critically regulates sodium and potass
25 se samples consistently generate fetal lung, kidney tubule and gastrointestinal epithelial organoids
26 all protein), is exclusively produced by the kidney tubule and has specific biochemical properties th
27 lial organoids, manifesting small intestine, kidney tubule and lung identity.
28 it active NaK transport by as much as 50% in kidney tubule and other tissues.
29 e considered tantamount to the injury of the kidney tubule and the epithelial cells thereof (AKI).
30 r plexiform layers of the retina, and in the kidney tubules and collecting ducts.
31 s in the developing skin, as well as dilated kidney tubules and ectatic Bowmans spaces.
32 th endogenous beclin 1 protein expression in kidney tubules and exogenous beclin 1 peptides are kidne
33 lture, wildtype cells differentiated to form kidney tubules and glomerular-like structures, whereas C
34 owed by a chronic reduction of expression in kidney tubules and impairment of glucose metabolism.
35 s in cell lines, in freshly isolated primary kidney tubules and in mouse models of cardiac transplant
36 es, tendon, and the connective tissue around kidney tubules and lung alveoli, which all contain fibri
37           The SUR2B is also expressed in rat kidney tubules and may combine with Kir.1 to form renal
38 c disease characterized by cyst formation in kidney tubules and other ductular epithelia.
39 in mice induced cilium elongation defects in kidney tubules and predisposed animals to cyst developme
40  expressed in each of the 14 segments of rat kidney tubules and used the proteomic data that we obtai
41   We explored whether MNPs can target cystic kidney tubules and whether rapamycin-encapsulated-MNPs (
42 dants, are present at high concentrations in kidney tubules and, when exogenously applied, protect ma
43  EMT in epithelial cells from mammary gland, kidney tubules, and epidermis.
44 as observed for lung alveoli and bronchioli, kidney tubules, and liver sinusoids.
45 stinal tract, the thick straight segments of kidney tubules, and the marrow stromal cells in adults.
46 stimulating factor-1 (CSF-1) is expressed by kidney tubules at the onset of LN, increases with diseas
47 s angiotensin-converting enzyme (ACE) in the kidney tubules but not in other tissues.
48 nd on epithelial cells, such as those of the kidney tubule, but also on nonepithelial cells, such as
49 ssessed multiple large cysts in the proximal kidney tubules, but even in tensin null mice with normal
50 t establishes and maintains the structure of kidney tubules, but the role of this pathway in the path
51 mmals has been attributed to the blockage of kidney tubules by insoluble complexes of melamine with c
52 has been used to determine whether embryonic kidney tubules can be stimulated by cAMP to form cysts.
53  progenitor cells (Six2 (Cre) Dnmt3a/3b) and kidney tubule cells (Ksp (Cre) Dnmt3a/3b).
54                                              Kidney tubule cells (KTC) are targets of T lymphocyte in
55 hed DNA maps of these regulatory regions for kidney tubule cells and glomerular endothelial cells, ma
56 ximately reconstruct the spatial position of kidney tubule cells and to predict corticomedullary gene
57    Single-cell RNA sequencing indicated that kidney tubule cells are an important source of MMP7.
58 olycystic kidney disease (ADPKD) occurs when kidney tubule cells are rendered null for either PKD1 or
59 genetic and immunostaining studies indicated kidney tubule cells as a disease-causing cell type.
60 t returns urate from the basolateral side of kidney tubule cells back to plasma.
61 kyrin and Fas was confirmed in vivo in mouse kidney tubule cells by coimmunoprecipitation and colocal
62 rn' translatome specifically dysregulated in kidney tubule cells destined to form cysts.
63                          Deletion of CDK5 in kidney tubule cells did not prevent G2/M arrest but did
64 nion of TOR (Rictor) to inactivate mTORC2 in kidney tubule cells of mice.
65                             OPN release from kidney tubule cells triggered lung endothelial leakage,
66                      We identify a subset of kidney tubule cells with a profibrotic-inflammatory phen
67                                  In cultured kidney tubule cells, ERVs elicit the activation of cytos
68  by fluorescence intensity in primary canine kidney tubule cells, Madin-Darby canine kidney cells, an
69  mtROS overproduction and mtDNA release from kidney tubule cells, which BAM15 prevented.
70 entified upregulated blood pressure genes in kidney tubule cells.
71 ANBA is a lysosomal gene highly expressed in kidney tubule cells.
72 nd STING agonists induced PERK activation in kidney tubule cells.
73 rylation, and matrix protein accumulation in kidney tubule cells.
74 important upstream activator of ER stress in kidney tubule cells.
75 AKI, we knocked out IL-22RA1 specifically in kidney tubule cells.
76 king complex is required for ciliogenesis in kidney tubule cells.
77 o paracrine profibrotic signaling in injured kidney tubule cells.
78 on the underlying proliferative potential of kidney tubule cells.
79                                          The kidney tubules constitute two-thirds of the cells of the
80 ined reduced, especially in the medulla, and kidney tubule damage was detected as Kim-1 expression.
81 zed and secreted into the medium by cultured kidney tubules derived from cystic C57BL/6J-cpk mice.
82  Signaling from the primary cilium regulates kidney tubule development and cyst formation.
83 inate from normal and nondilated ADPKD human kidney tubules display normal ciliary expression of the
84  were used to establish the role of C5aR1 in kidney tubules during AKI in germline C5ar1(-/-), myeloi
85 irectional cell growth, including defects in kidney tubule elongation that lead to formation of kidne
86                                    Along rat kidney tubules, endolyn is variously localized to the ap
87 ls of Kid1 mRNA correlate with maturation of kidney tubule epithelia in rat post-natal kidney develop
88 of kidney function heritability localized to kidney tubule epithelial cCREs and an additional 7% to k
89 y to profile gene expression in each type of kidney tubule epithelial cell.
90                              Irradiating rat kidney tubule epithelial cells (NRK52E) with 1-20 Gy gam
91 -sensorial function of the primary cilium in kidney tubule epithelial cells and (ii) the distinct thr
92  promoted the prolonged dedifferentiation of kidney tubule epithelial cells observed in CKD.
93 lia, first cervical vertebra, thyroid gland, kidney tubules, esophagus, stomach, and intestines.
94  These data indicate that C5aR1 signaling in kidney tubules exerts renoprotective effects against tox
95 o create an online information resource, the Kidney Tubule Expression Atlas.
96                                              Kidney tubule fatty acid oxidation (FAO) gain-of-functio
97 uding zebrafish posterior axis formation and kidney tubule formation.
98 -channel activity was significantly lower in kidney tubules from Na(+)-restricted alpha(F2M) mice com
99                        We manually dissected kidney tubules from rat kidneys and subjected samples to
100                        Moreover, PT cells in kidney tubules from TRACK mice exhibit increased genomic
101 t zebrafish organs shows sdf-1 expression in kidney tubules, gills, and skin.
102 macrophages and T cells accumulate at sites (kidney tubules, glomeruli, pulmonary bronchioli, lymph n
103 hepsin-mediated processing of uromodulin for kidney tubule homeostasis and salt sensitivity.
104  colleagues found that deleting Nedd4-2 from kidney tubules in adult mice led to ENaC accumulation, b
105 tudy, we have observed that WNV infection of kidney tubules in mice coincides with the loss of expres
106 n 1 peptides increased cell proliferation in kidney tubules in normal mice.
107     We examined ErbB4 function in developing kidney tubules in vivo with Pax8-Cre-mediated conditiona
108 merular apparatus in vitro, micropuncture of kidney tubules in vivo, and clearance kinetics of plasma
109 ressing epithelial cells after injury in rat kidney tubules in vivo.
110 outperformed either SCr or BUN for detecting kidney tubule injury and TFF3 augmented the potential of
111 epatocytes in the liver, infiltration of the kidney tubule interstitial by chronic inflammatory cells
112 eported Stx binding sites were identified in kidney tubules, intestinal lymphoid aggregates, sinusoid
113 se models that ablate Intu specifically from kidney tubules (Intu KO) were established.
114 hese results demonstrate that mTOR regulates kidney tubule ion handling and suggest that mTOR regulat
115   Hormone regulation of ion transport in the kidney tubules is essential for fluid and electrolyte ho
116           Inactivation of HNF-1beta in mouse kidney tubules leads to early-onset cyst formation and p
117 association studies (TWAS) in kidney cortex, kidney tubule, liver, and whole blood and proteome-wide
118 nism of sodium retention and its location in kidney tubules may vary with time in nephrotic syndrome
119 e effects of serum protein exposure in human kidney tubule microphysiologic systems and with orthogon
120 the target nor the site of ROS production in kidney tubule mitochondria in short-term diabetes.
121 P components in cytoskeletal assembly during kidney tubule morphogenesis in Xenopus laevis and zebraf
122 d the effect of selective mTOR inhibitors on kidney tubule Na+ and K+ transport in WT and Sgk1-/- mic
123 n a wide range of epithelial tissues such as kidney tubules or breast acini, cells organize into bidi
124 ch has used RNA sequencing in microdissected kidney tubules or single cells isolated from the kidney
125         Genetic deletion of cGAS or STING in kidney tubules, or pharmacological inhibition of STING,
126  At the genomic level, female ESR1-deficient kidney tubules partially lose their anti-ferroptotic cap
127 t in the early stage of acute pyelonephritis kidney tubules participate actively in the local host re
128 ifferentiation, while upregulation of FAO in kidney tubules provided protection from kidney fibrosis
129 fen-induced expression of Kim-1 in zebrafish kidney tubules resulted in loss of the tubule brush bord
130 cohort (n = 95) of normal and fibrotic human kidney tubule samples followed by systems and network an
131 f these determinants are expressed in the 14 kidney tubule segments using recently published RNA-sequ
132 ximal tubule and decreased it in more distal kidney tubule segments.
133     After ischemia-reperfusion injury (IRI), kidney tubules show activated transforming growth factor
134 erated a novel mouse model with an inducible kidney-tubule specific knock-in of fpnC326Y, which encod
135 metabolite changes was evaluated in diabetic kidney tubule-specific AMPKgamma2KO (KTAMPKgamma2KappaOm
136 C beta-subunit or alpha-subunit silencing or kidney tubule-specific beta-ENaC or alpha-ENaC knockout
137                                              Kidney tubule-specific C5ar1 knockout mice recapitulated
138 mline C5ar1(-/-), myeloid cell-specific, and kidney tubule-specific C5ar1 knockout mice.
139                                              Kidney tubule-specific Cul3 disruption causes tubulointe
140                                  Conditional kidney tubule-specific ENaC gamma-subunit knockout mice
141 hromatin accessibility assays suggested that kidney tubule-specific Glis3 inactivation resulted in dy
142 dium transporters in wild-type and inducible kidney tubule-specific Hif1a knockout mice.
143                               We report that kidney tubule-specific inactivation of Inpp5b on a globa
144 age in wild-type mice and knockout mice with kidney tubule-specific inactivation of mTORC2.
145                     We also used conditional kidney tubule-specific knockout mice lacking ENaC subuni
146  kidney ischemia/reperfusion injury, whereas kidney tubule-specific knockout of GRK4 decreased injury
147 xamined calcium and phosphate homeostasis in kidney tubule-specific NHE3 knockout mice (NHE3(loxloxPa
148 test this prediction, we generated two novel kidney tubule-specific transgenic mouse strains with ind
149                            Experiments using kidney/tubule-specific Tet2 knockout mice indicated its
150 r kidney transcriptomes (GTEx kidney cortex, kidney tubules, TCGA-KIRC [The Cancer Genome Atlas kidne
151 of 1-3 microM, without affecting other major kidney tubule transporters.
152                          ERVs expressions in kidney tubules trigger RIG-I/STING, and cytokine express
153                                       In rat kidneys, tubules undergoing atrophy late after IRI but n
154 re we examined cytosine methylation of human kidney tubules using Illumina Infinium 450 K arrays from
155 AnkG190, the isoform of ankyrin expressed in kidney tubules, was used as bait in a yeast two-hybrid s
156 divisions maintain the diameter of postnatal kidney tubules, we find that cell divisions are randomly
157  increased oxidative stress was found in the kidney tubules when mitochondria AKT1 was inhibited, sup
158 y were found in the brush border of proximal kidney tubules where megalin is localized.
159 A class are expressed in epithelial cells in kidney tubules, where they are required for the formatio
160 ng saccharides in the epithelium of proximal kidney tubules, whereas scattered glomeruli appeared col
161 r vesicle tRNAs reflects oxidative stress of kidney tubules which might be useful to detect ischemic
162 s disruption of key cytoskeletal elements in kidney tubules, which contributes causally to the injury
163  revealed similar levels of ENaC activity in kidney tubules, while no physiologically relevant differ
164 ltrastructural morphology of mitochondria in kidney tubules without cyst formation.
165 wth is substantial and involves expansion in kidney tubules without growth of new nephrons, which are
166 in development and uptake of proteins by the kidney tubule, yolk sac, and thyroid.

 
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