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1 mat, and cell surface receptors, such as the killer immunoglobulin-like receptors.
2 pecifically downregulates disease-associated killer immunoglobulin-like receptors.
3 munoreceptors such as the T cell receptor or killer immunoglobulin-like receptors.
4 els of the activating receptor NKp46 and the killer immunoglobulin-like receptors 2DL1/S1 and 3DL1, r
6 CD117(+)M-CSFR(+)) showed more expression of killer immunoglobulin-like receptors, a fraction of kill
7 tural killer (NK) cells and their activating killer immunoglobulin-like receptors (aKIRs) influence t
8 rtain the association of natural killer cell killer immunoglobulin-like receptors and human leukocyte
9 ike receptors, mannose-binding lectin, CD14, killer immunoglobulin-like receptors, and matrix metallo
10 e-binding positions 9, 99, 116, and 156, and killer immunoglobulin-like receptor binding position 77
12 sion is closely monitored by NK cells, whose killer immunoglobulin-like receptors encode MHC-I-specif
13 ng patients (n=222) and controls (n=191) for killer immunoglobulin-like receptor gene polymorphisms d
14 tor-ligand interactions (for example, due to killer immunoglobulin-like receptor-HLA mismatch) can tr
15 g/m(2)/d on days -6 through -2), followed by killer immunoglobulin-like receptor-human leukocyte anti
16 regard, NK cells that express an inhibitory killer immunoglobulin-like receptor (iKIR) for which the
17 he role of education through four inhibitory killer immunoglobulin-like receptors (iKIRs) from person
19 ating natural killer (NK) cell receptor, the killer immunoglobulin-like receptor (KIR) 3DS1, and its
20 ells expressing CD94: NKG2A but no change in killer immunoglobulin-like receptor (KIR) expression.
21 d 0.7 to 21 years were enrolled; donors were killer immunoglobulin-like receptor (KIR) favorable base
23 in reaction (PCR) assays to compare NKG2 and killer immunoglobulin-like receptor (KIR) gene expressio
25 -cells played by the inherited repertoire of killer immunoglobulin-like receptor (KIR) genes therefor
27 atural killer (NK) receptors, in particular, killer immunoglobulin-like receptor (KIR) KIR3DL2, a rec
28 ent MHC class I ligands for donor inhibitory killer immunoglobulin-like receptor (KIR) receptors, as
30 Several studies support a role for specific killer immunoglobulin-like receptor (KIR)-HLA combinatio
33 " In humans, interactions between inhibitory killer immunoglobulin-like receptors (KIR) and human MHC
36 een HLA-B27 and immune receptors such as the killer immunoglobulin-like receptors (KIR) found on a ra
38 are transmitted by NK inhibitory receptors (killer immunoglobulin-like receptors, KIR) at the site o
40 ct to two such families of NK receptors, the killer immunoglobulin-like receptors (KIRs) and the kill
41 inhibitory receptors, such as the family of killer immunoglobulin-like receptors (KIRs) and the NKG2
43 I molecules as ligands to NK cell inhibitory killer immunoglobulin-like receptors (KIRs) as a means o
45 city is attenuated by ligation of inhibitory killer immunoglobulin-like receptors (KIRs) by HLA class
46 portant role of NK cells expressing specific killer immunoglobulin-like receptors (KIRs) in the contr
49 argely been attributed to mismatches between killer immunoglobulin-like receptors (KIRs) on NK cells
55 ansplantation, produce cytokines and express killer immunoglobulin-like receptors (KIRs) that regulat
56 h increasing age, T cells gain expression of killer immunoglobulin-like receptors (KIRs) that transmi
57 ortant ligands for inhibitory and activating killer immunoglobulin-like receptors (KIRs) which are fo
58 Instead, CD4(+)CD28(null) T cells express killer immunoglobulin-like receptors (KIRs) with a prefe
59 ors, including the activating and inhibitory killer immunoglobulin-like receptors (KIRs), and human l
60 tural killer (NK) cells possess little or no killer immunoglobulin-like receptors (KIRs), high interf
61 ell checkpoint inhibitor, targets inhibitory killer immunoglobulin-like receptors (KIRs), however it
67 ic factors for improved outcome were missing killer immunoglobulin-like receptor ligand (PFS and OS),
68 ng CBRM1/5-positive granulocytes and missing killer immunoglobulin-like receptor ligand as positive i
69 When the model for OS also included missing killer immunoglobulin-like receptor ligand, human antimo
71 cell-surface dimers that can activate innate killer immunoglobulin-like receptors on CD4(+) T helper
73 immunoglobulin-like receptors, a fraction of killer immunoglobulin-like receptor-positive-expressing