ライフサイエンスコーパス: laboratory finding

1 mation, comorbidities, and microbiologic and laboratory findings.

2 scussed, and the results are consistent with laboratory findings.

3 mptoms, has 1 or more designated clinical or laboratory findings.

4 hosphatase in the serum were the most common laboratory findings.

5 od, with some disparity between clinical and laboratory findings.

6 th and without bile duct changes had similar laboratory findings.

7 , familial bone disease, or related abnormal laboratory findings.

8 s had been made on the basis of clinical and laboratory findings.

9 including clinical, chest radiographic, and laboratory findings.

10 luid resuscitation, radiography results, and laboratory findings.

11 rrelated with surgical, histopathologic, and laboratory findings.

12 and could not be predicted by examination or laboratory findings.

13 re were no consistent physical, clinical, or laboratory findings.

14 of outcome of SARS-CoV-2 pneumonia based on laboratory findings.

15 the risk of infections and abnormalities in laboratory findings.

16 thropogenic Sulf(inorg), consistent with our laboratory findings.

17 men; there are no specific clinical signs or laboratory findings.

18 ded to incorporate angiographic features and laboratory findings.

19 re history, clinical presentation, and other laboratory findings.

20 ar demographic composition, symptomology and laboratory findings.

21 le within 1.1 per mille, consistent with the laboratory findings.

22 r US findings in the context of clinical and laboratory findings.

23 and chronic gastritis, based on clinical and laboratory findings.

24 to its variable clinical manifestations and laboratory findings.

25 ated, and hepatic enzymes were normal in the laboratory findings.

26 ed to verify the credibility of quantitative laboratory findings.

27 d EoE-associated endoscopic, histologic, and laboratory findings.

28 ancy outcomes, including uterine Doppler and laboratory findings.

29 On the basis of clinical and laboratory findings, 40 of these patients were treated f

30 tions were mostly performed because of liver laboratory findings (87%, 32 of 37), and 54% (20 of 37)

31 igenic, with recent experimentation from our laboratory finding a direct correlation between large an

32 roid complications, comorbidity, and various laboratory findings, adjusting for the total number of v

33 Sharing of epidemiologic and laboratory findings allowed for the rapid identification

34 Clinical, neurological, and laboratory findings along with the timing of seroconvers

35 We evaluated clinical and laboratory findings among patients with nonsevere or sev

36 toy models evaluated against a checklist of laboratory findings; an approach which evokes Alan Newel

37 mation as to the application of clinical and laboratory findings and bone marrow histopathology as th

38 Clinical and laboratory findings and changes over time were described

39 er understanding of the relationship between laboratory findings and clinical reactivity is needed.

40 o identify associations between clinical and laboratory findings and histological features.

41 While laboratory findings and imaging may prove useful, they r

42 ecessary to confirm potential differences in laboratory findings and in fever intensity/duration.

43 also contributes to bridging the gap between laboratory findings and more real-world applications in

44 al trials, we assessed the clinical history, laboratory findings and muscle strength and function in

45 We analyzed the clinical and laboratory findings and outcome of 173 patients hospital

46 nd distribution of symptoms and impairments, laboratory findings and outcomes are essentially alike.

47 The laboratory findings and physical examination were normal

48 correlation between antemortem clinical and laboratory findings and postmortem culture results, ther

49 veloped following consideration of symptoms, laboratory findings and relevant medical history, and in

50 to collection, disease status, and clinical/laboratory findings and their impact on the leukapheresi

51 ion and clinical complications, and examined laboratory findings and toxic effects.

52 of the inconclusive results of both clinical-laboratory findings and ultrasonography, CT imaging was

53 Reversible hemodilution was apparent in laboratory findings and weight gain.

54 e cases of HIES were scored for clinical and laboratory findings and were genotyped with polymorphic

55 d medical records for clinical presentation, laboratory findings, and biopsy results to confirm prima

56 mptoms weekly and monitored adverse effects, laboratory findings, and cardiovascular parameters.

57 lationships between patient characteristics, laboratory findings, and clinical HIT status.

58 ographic data, cardiac injury markers, other laboratory findings, and comorbidity details were collec

59 is clinical treatment failures, interpreting laboratory findings, and correlating the 2 clearly remai

60 imed to describe the clinical presentations, laboratory findings, and endoscopic patterns of affected

61 conjunction with the clinical presentation, laboratory findings, and epidemiological information, th

62 Data on presenting signs and symptoms, laboratory findings, and hospital course were collected.

63 ify EMD, as defined by physical examination, laboratory findings, and imaging results.

64 study evaluated the clinical manifestations, laboratory findings, and molecular genetic results of 21

65 The neurologic manifestations, laboratory findings, and outcome of patients with West N

66 re available on its clinical manifestations, laboratory findings, and outcomes of those patients requ

67 Screening tools, laboratory findings, and physical findings can be helpfu

68 ion of thienopyridine exposure, clinical and laboratory findings, and survival were recorded.

69 ores were correlated with clinical features, laboratory findings, and treatment responses.

70 Patient history, physical examination, laboratory findings, and US results were used to create

71 This article will look at case reports, laboratory findings, animal studies, environmental facto

72 When laboratory findings are discordant with expected clinica

73 The characteristic laboratory findings are elevated markers of inflammation

74 To exclude cirrhosis, combinations of normal laboratory findings are most useful.

75 ny of these conditions overlap, clinical and laboratory findings are necessary to support the imaging

76 Since the clinical and laboratory findings are nonspecific, imaging tests play

77 The clinical manifestations and laboratory findings are reported of 6 consecutive patien

78 Our laboratory findings are supported by clinical samples, w

79 The laboratory findings are supported by recent field observ

80 These laboratory findings are used to interpret observations o

81 ytosis, and abnormal liver enzyme tests were laboratory findings associated with lead toxicity.

82 cognitive outcomes in survivors and identify laboratory findings associated with neurologic injury.

83 ls (n = 29), from patients with diagnoses or laboratory findings associated with serologic cross-reac

84 estations of HIV infection, the physical and laboratory findings associated with them, and the therap

85 020, were analyzed for baseline clinical and laboratory findings at admission and during the disease.

86 d clinical symptoms, as well as clinical and laboratory findings at the time of diagnosis, were recor

87 Clinical and laboratory findings at the time of presentation were eva

88 es, face scale, findings of nasal cavity and laboratory findings before start of therapy and 12 month

89 differences in clinical characteristics and laboratory findings between children infected with Macro

90 There were no significant differences in laboratory findings between the two groups, and many neo

91 not associate with demographic, clinical, or laboratory findings, but they significantly associated w

92 These studies showed the validity of laboratory findings by supporting the idea that tea cons

93 esenting symptoms, clinical examination, and laboratory findings can guide selection of diagnostic im

94 BEST PRACTICE ADVICE 4: Laboratory findings can include elevated folate and, les

95 g treatment of HIV according to clinical and laboratory findings, cancer treatment plan (chemotherapy

96 often present with nonspecific symptoms and laboratory findings, cardiac imaging has emerged as an i

97 As anatomy, physiology, and even laboratory findings change during pregnancy, the clinici

98 m the following 5 categories: radiologic and laboratory findings, clinical and functional status meas

99 and nonspecific clinical, radiological, and laboratory findings commonly lead to misdiagnosis as pne

100 available for further testing, but none had laboratory findings confirming reinfections.

101 ia within 48 h of infusion with clinical and laboratory findings consistent with cytokine release syn

102 utoinflammatory rash, and other clinical and laboratory findings consistent with PLAID were observed

103 munological correlates of protection and how laboratory findings correspond to clinical effectiveness

104 of dilution and amplification supported our laboratory findings, demonstrating that the results are

105 eight; other vital signs and cardiometabolic laboratory findings did not differ between groups.

106 Key clinical, imaging, and laboratory findings differentiate these disorders, allow

107 iagnostic term when clinical, histologic and laboratory findings do not allow for specific categoriza

108 The patient's clinical and laboratory findings during his rapidly fatal course, aut

109 S-A more often manifest certain symptoms and laboratory findings early during hospitalization.

110 Clinical and demographic characteristics, laboratory findings, echocardiographic data, and CTPA im

111 tic resonance imaging [MRI] T2 lesions), and laboratory findings (eg, cerebrospinal fluid-specific ol

112 rogressions article is a story of how single laboratory findings evolve through time to be confirmed,

113 Measures included imaging and laboratory findings, extent of surgery, pathologic diagn

114 0, 2019, and April 14, 2020, with a positive laboratory finding for SARS-CoV-2.

115 AQP4-antibody is now the most important laboratory finding for the diagnosis of NMO.

116 We sought to describe clinical and laboratory findings for a large cohort of patients with

117 noses, evaluated consistency of RI and other laboratory findings for chemicals identified by the RI a

118 ve predictive value of specific clinical and laboratory findings for curable enteric infections excee

119 We evaluated clinical and laboratory findings for patients with erythema migrans w

120 ntified between ethnobotanical knowledge and laboratory findings for studied grasses.

121 In this investigation, we present laboratory findings from an ongoing C. auris outbreak in

122 Insights from clinical and laboratory findings have also been recently harnessed fo

123 miological, clinical, neuroradiagnostic, and laboratory findings have enhanced our diagnostic accurac

124 Demographics, symptoms, radiologic and laboratory findings, hospitalization status, antimicrobi

125 ensitivity, 74%-81%; specificity, 25%-44%]), laboratory findings (human leukocyte antigen B27-positiv

126 ents' race, sex, age, clinical presentation, laboratory findings, imaging tests, treatment, and follo

127 Some laboratory findings imply that imatinib may primarily af

128 he normal range in the blood and is a common laboratory finding in patients.

129 We report the clinical and laboratory findings in 11 patients in whom thrombotic th

130 retinal imaging, ocular disease course, and laboratory findings in 4 patients with submacular fluid

131 one biopsy in 9 patients and clinical course/laboratory findings in 5.

132 , and the need for careful interpretation of laboratory findings in conjunction with clinical signs i

133 This report describes the clinical laboratory findings in golden hamsters experimentally in

134 tes, unique clinical features, MRI and other laboratory findings in NMO have been clarified further.

135 Typical laboratory findings in patients with hyperthyroidism are

136 ncies were categorized based on clinical and laboratory findings in the affected patient.

137 lished studies and argue for the adoption of laboratory findings in the staging systems that are used

138 Clinical and laboratory findings in this disorder are similar to thos

139 ilis by briefly summarizing the clinical and laboratory findings in uncomplicated syphilis.

140 Laboratory findings included elevated C-reactive protein

141 Initial laboratory findings included leukocytosis and elevated l

142 Laboratory findings, including a complete blood count an

143 Laboratory findings, including biochemistry, electrolyte

144 Laboratory findings, including biochemistry, electrolyte

145 0-100 U/L [0.7-1.67 mukat/L]), but all other laboratory findings, including complete blood count, ren

146 40-100 U/L [0.7-1.67 ukat/L]), but all other laboratory findings, including complete blood count, ren

147 Clinical information and laboratory findings, including IL-6 levels, were collect

148 Although none of the laboratory findings, including LPS hyporesponsiveness, i

149 terpreted in the context of all clinical and laboratory findings, including plasma BKPyV-DNAemia.

150 Laboratory findings, including the growth of new blood v

151 te pancreatitis and those without imaging or laboratory findings indicative of pancreatic disease.

152 cs of the history, physical examination, and laboratory findings, individually and in combination, in

153 cording to medical history, medications, and laboratory findings (insulin-like growth factor 1, folli

154 Implementing our laboratory findings into a global modeling framework sho

155 However, translating these laboratory findings into effective clinical strategies c

156 Translating the laboratory findings into the clinical environment where

157 unity-dwelling men to determine whether this laboratory finding is manifest at the population level.

158 of both the patient's clinical situation and laboratory findings is important for tailoring therapy o

159 The correlations among laboratory findings, liver stiffness, and fibrosis score

160 treatment of an in-stent restenotic lesion), laboratory findings (low baseline hemoglobin and reduced

161 and, evolutionary tradeoffs predict that the laboratory findings may not be relevant to human populat

162 oncept that in systemic lupus erythematosus, laboratory findings may not correlate well with the unde

163 The lack of characteristically abnormal laboratory findings may result in a delay in the proper

164 "Symptoms, signs, and abnormal clinical and laboratory findings, not elsewhere classified".

165 A risk model based on chest radiographic and laboratory findings obtained at admission was predictive

166 In this study, the radiological and laboratory findings obtained from 31 consecutive patient

167 No specific pattern of signs, symptoms, or laboratory findings occurred with enough frequency to co

168 artate receptor, that is, the characteristic laboratory finding of anti-N-methyl-D-aspartate receptor

169 large health maintenance organization with a laboratory finding of CKD (defined as estimated GFR betw

170 We used machine learning for processing laboratory findings of 110 patients with SARS-CoV-2 pneu

171 We compared the clinical and laboratory findings of 15 patients with sudden-onset syn

172 We examined the clinical and laboratory findings of a consecutive series of patients

173 The patients with clinical or laboratory findings of elevated D-dimer level or elevate

174 40 consecutive patients with no clinical or laboratory findings of hemodialysis access dysfunction.

175 sing Tourette syndrome reflects clinical and laboratory findings of investigations of behavioral, neu

176 ophy in splenic tissues were compatible with laboratory findings of leukopenia, lymphopenia, and thro

177 ility criteria were available cranial US and laboratory findings of maternal Zika virus infection dur

178 study sought to investigate the clinical and laboratory findings of patients affected by sudden-onset

179 the clinical presentation, pathogenesis, and laboratory findings of patients with these two disorders

180 oning of the system and for extrapolation of laboratory findings of stressor impacts on specific comp

181 a routine ophthalmologic consultation after laboratory findings of systemic Candida septicemia, whic

182 pretreatment and posttreatment clinical and laboratory findings of the case series patients.

183 Here, we aimed to translate laboratory findings on enhanced fear extinction with rep

184 a deterioration in the general condition and laboratory findings or appearance of new abdominal compl

185 No distinctive laboratory findings or prognostic worsening were detecte

186 as CMV syndrome (CMV viremia and clinical or laboratory findings) or end-organ disease.

187 were apparent on vital sign determinations, laboratory findings, or electrocardiographic measurement

188 anges from baseline in vital signs, clinical laboratory findings, or electrocardiography findings in

189 ere no major imbalances in adverse events or laboratory findings, or evidence of systemic JAK inhibit

190 t admission, vitals and examination results, laboratory findings, outcomes, blood transfusion status,

191 Pretreatment PSMA PET, laboratory findings, overall survival, a fall in prostat

192 Based on history, clinical examination, and laboratory findings, patients may be placed in three cat

193 althy adolescents is less clear than that of laboratory findings, perhaps due to high night-to-night

194 Clinical and laboratory findings persist despite cessation of vitamin

195 We recorded clinical and laboratory findings prospectively, fitted survival curve

196 Recent laboratory findings provide exciting insights into a bid

197 Laboratory findings ranged widely and did not characteri

198 Of all laboratory findings readily available to the clinician,

199 e adverse drug effects were observed, and no laboratory findings required discontinuation of treatmen

200 Clinical symptoms and laboratory findings resembled those of classic acute sch

201 e some promising theoretical simulations and laboratory findings, results from field trials that eval

202 ncreatitis improved over a few days, and the laboratory findings returned to normal ranges.

203 Laboratory findings revealed an elevated white blood cel

204 Laboratory findings revealed inflammation with an increa

205 Transcriptomic data were corroborated by laboratory findings: SARS-CoV-2 infection increased plas

206 Our laboratory findings serve to reinforce field observation

207 These laboratory findings should not be interpreted as indicat

208 It extended recent laboratory findings-showing an increase in task-focus an

209 We investigated the patient background, laboratory findings, side reactions from their medical r

210 with the aid of specific clinical, MRI, and laboratory findings; studies of people misdiagnosed with

211 rature of > or = 40 degrees C), and abnormal laboratory findings (such as a pH <7.35, a blood urea ni

212 Laboratory findings (such as prothrombin time and biliru

213 Recent laboratory findings suggest that it might be possible to

214 Laboratory findings suggest that the foreskin is enriche

215 one had onward transmission, consistent with laboratory findings suggesting a secondary immune respon

216 ve pure S. aureus culture in urine, and have laboratory findings suggesting systemic infection.

217 poglycemia, rhabdomyolysis, arrhythmias, and laboratory findings suggestive of a defect in mitochondr

218 ive study, for 57 children with clinical and laboratory findings suggestive of APN, the 2 radiopharma

219 We studied three patients with symptoms and laboratory findings suggestive of human granulocytic ehr

220 ut chest radiographic changes or clinical or laboratory findings suggestive of pneumonia (nonpneumoni

221 k in one male with both a family history and laboratory findings suggestive of XLA.

222 These clinical and laboratory findings support the concept that telomerase

223 Taken together, the clinical analyses and laboratory findings support the interpretation that impr

224 Hyperferritinaemia is a common laboratory finding that is often associated with metabol

225 w of recent clinical studies and correlative laboratory findings that advance our understanding of th

226 Our field results support laboratory findings that caching rates and distances by

227 nd any associated extracutaneous clinical or laboratory findings that may accompany them.

228 ng-term effects of therapy, as well as novel laboratory findings that may alter future treatment stra

229 cent studies assessing clinical features and laboratory findings that may help diagnose psychogenic m

230 fy the specific combinations of clinical and laboratory findings that presumably account for this dia

231 However, no clinical or laboratory findings that reliably distinguish X-linked d

232 hic characteristics, duration of illness and laboratory findings that we were able to obtain.

233 No clinical or laboratory findings that were present at MCD diagnosis p

234 All piglets had appropriate weight gain and laboratory findings throughout the post-operative period

235 functions and allows the generalizability of laboratory findings to be assessed.

236 ities in lung cancer, efforts of translating laboratory findings to clinical tests, and prospective o

237 ed in conjunction with the physical exam and laboratory findings to identify children at risk for IAI

238 o use the history, physical examination, and laboratory findings to identify structural causes and di

239 ong-term datasets, remotely sensed data, and laboratory findings to inform forecasting of insect pest

240 We related the laboratory findings to signs of sympathetic neurocircula

241 Our work connects laboratory findings to the real world and delineates the

242 e are problems involved in generalizing from laboratory findings to the reporting of the symptoms of

243 This translation of neurophysiological laboratory findings to therapy is a clear example of why

244 l manifestations, immunosuppression regimen, laboratory findings, treatment, and outcomes.

245 re characterized by nonspecific clinical and laboratory findings, ultimately requiring a trial of dru

246 On the basis of clinical and laboratory findings, various forms of HES have been defi

247 ent of symptoms, endoscopic, histologic, and laboratory findings was carried out, were included.

248 nable healthand no significant alteration in laboratory findings was noted.

249 ve tuberculin skin test alone among clinical laboratory findings was significantly associated with an

250 Among initial laboratory findings, we analyzed leukocytes, lymphocytes

251 The laboratory findings were analyzed using INLA package to

252 Clinical backgrounds, clinical symptoms, and laboratory findings were compared between neonates with

253 Clinical and laboratory findings were compared between patients who d

254 Clinical and laboratory findings were consistent with the EPO-depende

255 Typical laboratory findings were elevated C-reactive protein, le

256 Primary laboratory findings were hypophosphatemia (100%) and pro

257 The most frequent laboratory findings were lymphopenia, thrombocytopenia,

258 Abnormal laboratory findings were negatively associated with disc

259 Clinical and laboratory findings were non-specific, while imaging fea

260 gher adverse events, or clinically important laboratory findings were observed after the administrati

261 ical outcomes, sociodemographic factors, and laboratory findings were performed.

262 Clinical features and laboratory findings were recorded for all 245 residents

263 Oral and general health data including laboratory findings were recorded from hospital records,

264 Data about clinical manifestations and laboratory findings were retrieved from medical records.

265 Clinical course, imaging, and laboratory findings were retrieved from the electronic m

266 Although laboratory findings were similar for children with oligo

267 e trials and attempt to integrate with basic laboratory findings where relevant to define issues pert

268 Field observations supported our laboratory findings where significant concentrations (42

269 Our field observations are consistent with laboratory findings while shedding light on the phenomen

270 matic hyperuricaemia was considered a benign laboratory finding with little clinical importance in th

271 However, laboratory findings with current methodologies are often

272 Typing is usually based on clinical and laboratory findings with monoclonal gammopathy evaluatio

273 eline characteristics, physical examination, laboratory findings, x-ray or computed tomography scan f