ライフサイエンスコーパス: lactobacilli

1 genes (ISGs) being the most affected by both lactobacilli.

2 teria and levels of Streptococcus mutans and Lactobacilli.

3 or total bacteria, Streptococcus mutans, and Lactobacilli.

4 toxicity, and does not inhibit the growth of lactobacilli.

5 is also highly conserved in streptococci and lactobacilli.

6 onii is a member of the acidophilus group of lactobacilli.

7 ng the rectum may be a reservoir for vaginal lactobacilli.

8 he identification of 97 strains of commensal lactobacilli.

9 edictive of an absence of H(2)O(2)-producing lactobacilli.

10 strains as well as other species of vaginal lactobacilli.

11 unique DNA fingerprint compared to all other lactobacilli.

12 colonized with commensal bacteria, primarily lactobacilli.

13 ilus, are the most common species of vaginal lactobacilli.

14 nowledge about the action mode of antifungal lactobacilli.

15 ift involves organisms other than the MS and lactobacilli.

16 the anti-inflammatory effects of beneficial lactobacilli.

17 en have microbiota that are not dominated by lactobacilli.

18 lly digested patterns of previous reports on lactobacilli.

19 ven rennet was assessed for the existence of lactobacilli.

20 the increasing use of probiotics containing lactobacilli.

21 thesis about the natural history and role of lactobacilli.

22 hibit growth of vaginal protective commensal lactobacilli.

23 (+) T lymphocytes and vaginal H2O2-producing lactobacilli.

24 pecific traits in the genomes of the vaginal lactobacilli.

25 Lactobacilli, a component of the normal vaginal flora, c

26 n a pathogen-free facility were colonized by Lactobacilli, a component of the oropharyngeal flora.

27 All tested lactobacilli able to biosynthesis ZnO-NPs indicated by w

28 91; 20.2%) and those with H(2)O(2)-producing lactobacilli absent (n=756; 79.8%).

29 We grouped samples into those in which lactobacilli accounted for at least 50% of the species p

30 resent (community type L) and those in which lactobacilli accounted for less than 50% of the species

31 seudoalteromonas, Burkholderia, Bacteroides, Lactobacillis, Acetobacter, Allobaculum, Ruminococcus, a

32 Lactobacilli acidify CVM to pH approximately 4 by contin

33 lactobacilli were inversely associated with lactobacilli adhesion to epithelial cells and D-lactate

34 rations, and decreased relative abundance of lactobacilli after one and six months of use, relative t

35 Engineered lactobacilli also caused a modest, but statistically sig

36 Probiotic lactobacilli also differentially modulated the C. albica

37 Lactobacilli also increased tumor necrosis factor-alphaa

38 Lactobacilli also inhibited gonococcal invasion of epith

39 The viability of the microcapsulated lactobacilli also remained high (>7 log cfu g(-1)) for 2

40 ted sustained colonization by H2O2-producing lactobacilli among women already colonized (relative ris

41 factors for an absence of H(2)O(2)-producing lactobacilli among women with bacterial vaginosis.

42 s: a nucleoside deoxyribosyltransferase from lactobacilli and a 5'-monophosphate-2'-deoxyribonucleosi

43 ant aciduric bacteria from root lesions were lactobacilli and A. israelii, while from sound root surf

44 Absence of vaginal lactobacilli and accompanying genital inflammation is as

45 and depletion of indole-metabolite-producing Lactobacilli and Alistipes.

46 l women have a relative depletion of vaginal lactobacilli and an increase in vaginal E. coli compared

47 This correlates with a loss of lactobacilli and an overgrowth of diverse bacteria.

48 to examine the effect of N-9 use on vaginal lactobacilli and bacterial vaginosis.

49 daily dose of the Lab4P probiotic comprising lactobacilli and bifidobacteria (50 billion cfu/day).

50 The growth of health-promoting lactobacilli and bifidobacteria is supported by FOS, giv

51 o evidence that a multistrain preparation of lactobacilli and bifidobacteria was effective in prevent

52 ia, while a slight increase on the growth of lactobacilli and bifidobacteria was observed after expos

53 monkeys differ in the relative abundances of Lactobacilli and Bifidobacteria, two taxa predominant in

54 receive either a multistrain preparation of lactobacilli and bifidobacteria, with a total of 6 x 10(

55 nal flora characterized by reduced levels of lactobacilli and concomitant overgrowth of anaerobic bac

56 obial community was found to be dominated by Lactobacilli and Enterobacteria, both typically facultat

57 ired microflora in mayonnaise preserved with lactobacilli and essential oils decreased significantly

58 We conclude that the association between lactobacilli and gonococci is complex and may be subject

59 0) had significantly lower concentrations of lactobacilli and higher concentrations of Gardnerella va

60 Specifically, decreases in lactobacilli and increases in Prevotella, Dialister micr

61 g thus allows high efficiency mutagenesis in lactobacilli and lactococci, and may be used to further

62 Quantitative bacterial counts for lactobacilli and M. hominis are better correlates of CVL

63 sers demonstrated an increased prevalence of lactobacilli and non-albicans yeast, while dapivirine ri

64 aginal bacterial flora with disappearance of lactobacilli and overgrowth of Gardnerella vaginalis and

65 nosis (BV) is characterized by low levels of lactobacilli and overgrowth of potential pathogens in th

66 aracterized by the replacement of beneficial lactobacilli and the augmentation of anaerobic bacteria.

67 aginal microbiota is frequently dominated by lactobacilli and transition to a more diverse community

68 ptide production, which allows overgrowth of Lactobacilli and triggers T regulatory cell expansion in

69 he inverse association between H2O2-positive lactobacilli and vaginal E. coli colonization remained i

70 le to displace pre-coated vaginal protective lactobacilli and we hypothesize this to be a trigger for

71 ensal gut bacteria (e.g., bifidobacteria and lactobacilli) and increase the abundance of enterobacter

72 aginal probiotic (G3, oral placebo + vaginal lactobacilli), and probiotic combination (oral lactic ac

73 nderstood, and although mutans streptococci, lactobacilli, and A. naeslundii have been implicated in

74 onserved in streptococci, staphylococci, and lactobacilli, and are required for bacterial biofilm for

75 totoxicity, lack of activity against vaginal lactobacilli, and effectiveness against both HSV-2 and H

76 s, salivary log10 mutans streptococci, log10 lactobacilli, and fluoride level, did not represent stat

77 olved in fatty acid biosynthesis, amounts of lactobacilli, and saturated LCFA were measured in fecal

78 vels of Bifidobacteria, mutans streptococci, lactobacilli, and yeasts were correlated with each other

79 genous microbes that stably colonize a host, lactobacilli appear to be planktonic, opportunistic sett

80 Lactobacilli are a diverse group of species that occupy

81 h an associated decrease in caries, if these lactobacilli are fed to rats in an established caries mo

82 Lactobacilli are found in a wide variety of habitats.

83 Lactobacilli are low-virulence, commensal organisms of t

84 en who are colonized with H(2)O(2)-producing lactobacilli are more likely to maintain a normal vagina

85 Lactobacilli are non-spore forming, lactic acid producin

86 Lactobacilli are part of the normal bacterial flora of t

87 Some lactobacilli are pervasive in dairy, meat and plant food

88 Bifidobacteria and lactobacilli are purportedly beneficial to human health

89 Lower levels of vaginal lactobacilli are reported more frequently in women of Af

90 Strains of lactobacilli are the most applied probiotics tested in c

91 Probiotic lactobacilli are, therefore, explored to treat symptomat

92 hat reduced levels of core bacteria, such as lactobacilli, are associated with conditions like bacter

93 sufficient data exist to support the use of lactobacilli as candidates for the development of new or

94 was altered tryptophan metabolism in airway lactobacilli associated with reduced indole-3-acetic aci

95 by churning the cream that was fermented by lactobacilli at 37 degrees C for 24 h.

96 bserved in levels of mutans streptococci and lactobacilli at all time-points.

97 s for targeted engineering of the S-layer or lactobacilli bacteria in general.

98 In multivariate analysis, only lactobacilli bacterial counts (P=.006; inverse associati

99 lationship between vaginal colonization with lactobacilli, bacterial vaginosis (BV), and acquisition

100 Lactobacilli-based vaginal probiotics warrant further ev

101 Combinations of Lactobacilli, Bifidobacteria, and Streptococcus salivari

102 S DP (1-4) derived from LBG supported better Lactobacilli biofilm formation as compared to KG hydroly

103 genetic and biochemical characterization of lactobacilli BSHs lend to a model of Lactobacillus adapt

104 ffect vaginal colonization by H2O2-producing lactobacilli but that such use may promote loss of non-H

105 udies have shown that oral administration of lactobacilli can be an efficient approach to treat lacta

106 Lactobacilli can lead to bacteremia and liver abscesses

107 y infectious HIV-1 particles, the engineered lactobacilli can potentially capture HIV-1 of different

108 In addition to the better-known lactobacilli, certain strains of Weissella cibaria, Leuc

109 MDCs activated with lactobacilli clearly skewed CD4(+) and CD8(+) T cells to

110 Lactobacilli colonizing the rectum may be a reservoir fo

111 0%), and L. gasseri (10%) were the prevalent lactobacilli colonizing the rectums of 290 females.

112 ophilus CRL 1014 on microbial metabolism and lactobacilli community composition for improving human h

113 The DGGE obtained showed changes in the lactobacilli community from the colon compartments of th

114 Lactobacilli comprise all bacteria previously assigned t

115 nificantly with any of the preparations, but lactobacilli concentrations decreased transiently.

116 l present at six months and not prevented by lactobacilli consumption.

117 adic reported cases of infections related to lactobacilli containing probiotics.

118 L. monocytogenes infection by treatment with lactobacilli correlates with a decrease in host gene exp

119 obacilli in the vagina, suggest that vaginal lactobacilli could reacidify the vagina at the rate obse

120 ein concentrations were linked to changes in lactobacilli counts (P < 0.05, R(2) = -0.33 for the mode

121 c ablation of the glands markedly suppressed Lactobacilli counts and heightened vulnerability to infe

122 Immobilization of lactobacilli cultures in calcium-alginate beads resulted

123 12 months, the proportion with H2O2-positive lactobacilli decreased (n = 32; 53% vs 27%; P = .03).

124 However, lactobacilli decreased by > or = 10(2) cfu/mL in 9 (56%)

125 lenged IL-10-deficient murine colitis model, lactobacilli demonstrated probiotic effects by direct mo

126 The proportion positive for H2O2+ or H2O2- lactobacilli did not change significantly with any of th

127 Absence of lactobacilli did not increase acquisition of VVC.

128 rmal Nugent score at all visits had a stable lactobacilli dominated microbiota with prevailing Lactob

129 with BV compared with women with a "normal" lactobacilli-dominated microbiota.

130 nd vaginal microbiomes has demonstrated that lactobacilli drive epithelial and immune cell responses,

131 Pathogen displacement of vaginal lactobacilli drives innate immune activation and mucosal

132 pported by the observation of persistence of lactobacilli during and after treatment of CDI.

133 asmid-borne genes mediate the persistence of lactobacilli during olive fermentation and enhance their

134 Lactobacilli effectively fermented MOS to generate aceta

135 Proteinase K treatment of methanol-fixed lactobacilli eliminated the inhibitory effect, suggestin

136 Lactobacilli encompass more than 300 species, spanning 2

137 Diabetic rats were fed daily with human lactobacilli engineered to secrete GLP-1(1-37).

138 PCRs to detect bifidobacteria, bacteroides, lactobacilli, Escherichia coli, Clostridium difficile, a

139 have shown nonoxynol-9 (N-9) to be toxic to lactobacilli, especially to strains that produce H2O2.

140 with the ultimate goal of restoring vaginal lactobacilli following microbiological disturbances.

141 ilm community, and successfully compete with lactobacilli for dominance in the vaginal environment.

142 ial efficacy of an anti-ICAM Ab delivered by lactobacilli for use as an anti-HIV-1 microbicide.

143 c acid bacteria and bifidobacteria + vaginal lactobacilli), for 4 months.

144 the assertion from other investigators that lactobacilli found in the GI tract originate in the oral

145 We isolated 80 lactobacilli from South African women with non-optimal (

146 d 900 native microbial isolates including 18 Lactobacilli from the farmed salmon intestines.

147 The absence of lactobacilli from the vaginal flora was associated with

148 Lactobacilli from women with non-optimal produced less l

149 Lactobacilli, Gardnerella vaginalis, and Mycoplasma homi

150 HIV RNA, lactobacilli, Gardnerella vaginalis, and Mycoplasma homi

151 Pectic oligosaccharides also enhanced lactobacilli growth during mixed batch faecal fermentati

152 Probiotic lactobacilli harbor a considerable number and diversity

153 Exploiting lactobacilli has been complicated by metabolic diversity

154 ural explanation for the mechanisms in which lactobacilli have adapted to their host niche by maximiz

155 Lactobacilli have been associated with dental caries for

156 uated the hypothesis that women colonized by lactobacilli have decreased acquisition of vaginal infec

157 The effect that vaginal lactobacilli have on trichomoniasis is not known.

158 Recently, lactobacilli have shown promise for acting as probiotics

159 Thus, vaginal lactobacilli have the ability to inhibit gonococci at tw

160 sociated with lack of vaginal H2O2-producing lactobacilli (hazard ratio [HR] = 4.0, P < .001) or pres

161 .001) or presence of only non-H2O2-producing lactobacilli (HR = 2.2, P = .02).

162 of vaginal epithelial cells with any of the lactobacilli immediately following exposure to douching

163 otal levels of LCFA correlated with those of lactobacilli in fecal samples from patients with active

164 are necessary for sustained colonization of lactobacilli in humans: 1) a stagnant, retentive niche t

165 s emphasize a potentially important role for lactobacilli in modulating immunological functions of DC

166 tobacillus after using probiotics containing lactobacilli in the course of her treatment of Clostridi

167 foundation for further exploring the role of lactobacilli in the ecological dynamics of vaginal micro

168 and Lactobacillus jensenii, the most common lactobacilli in the female genital tract, inhibit gonoco

169 ed how glycogen could be utilized by vaginal lactobacilli in the genital tract.

170 ially fermented by mainly bifidobacteria and lactobacilli in the human gut.

171 s pronounced differences were observed among lactobacilli in the induction of IL-6 and IL-10.

172 to reflect on the health implications of the lactobacilli in the mouth and downstream GI and to ponde

173 iginate in the oral cavity by proposing that lactobacilli in the oral cavity arise from caries lesion

174 n by vaginal epithelial cells in response to lactobacilli in the presence and absence of Gardnerella

175 ined with an estimate of the total number of lactobacilli in the vagina, suggest that vaginal lactoba

176 High numbers of lactobacilli in the vaginal tract have been correlated w

177 terial counts (G. vaginalis, M. hominis, and lactobacilli) in our model improved the sensitivity and

178 ict failure to become colonized by probiotic lactobacilli include exposure to semen, vaginal intercou

179 cantly more among subjects with NC-LC, while Lactobacilli (including L. fermentum) were more among su

180 genes conserved in L. crispatus and related lactobacilli, including an oleate hydratase (ohyA) and p

181 All lactobacilli induced inflammatory cytokine production by

182 CncC-dependent fashion, and may function in lactobacilli-induced AMP proliferation and intestinal ti

183 Lactobacilli-induced ROS is strictly excluded from defin

184 heir selective effects on bifidobacteria and lactobacilli, influence many aspects of bowel function t

185 m scores were recorded at baseline and after lactobacilli ingestion.

186 capacity for biotransformations catalysed by lactobacilli is an untapped biotechnology resource.

187 e investigate whether the anti-HIV effect of lactobacilli is mediated by extracellular vesicles (EVs)

188 differentiation of group B streptococci from lactobacilli is not possible, but lactobacillus cells al

189 amples produced under laboratory conditions, lactobacilli is one of the dominant groups in all the re

190 tic dogma that the mere absence of "healthy" lactobacilli is the sole mechanism that creates a permis

191 cterial microbiome of the stomach, including lactobacilli, is vital in promoting colonization resista

192 Vaginal lactobacilli isolated from 215 sexually active women wer

193 blood were phylogenetically inseparable from Lactobacilli isolated from the associated probiotic prod

194 Whole-genome-based phylogeny showed that Lactobacilli isolated from treated patients' blood were

195 with HIV-1 and treated with EVs released by lactobacilli isolated from vaginas of healthy women.

196 Vaginal lactobacilli may be associated with trichomoniasis in wo

197 on of "controlled" inflammation by probiotic lactobacilli may be beneficial to improve neutrophil fun

198 Thus, absence of H2O2-positive lactobacilli may be important in the pathogenesis of rec

199 Absence of H2O2-producing lactobacilli may contribute to the development of cervic

200 We hypothesize that commensal lactobacilli may enhance growth of gonococci in vivo by

201 een suggested that vaginal colonization with lactobacilli may reduce the risk of vulvovaginal candidi

202 ifies a family of compounds by which vaginal lactobacilli mediate host immune homeostasis and highlig

203 at predict sustained colonization by vaginal lactobacilli, microbiologic, behavioral, and demographic

204 nges in pH, concentration of viable cells of lactobacilli, microbiological, and organoleptic characte

205 ed Atopobium vaginae, Gardnerella vaginalis, lactobacilli, Mycoplasma hominis, and the human albumin

206 en, vaginal intercourse, and the presence of lactobacilli of the same species at enrollment.

207 During follow-up, absence of vaginal lactobacilli on culture was associated with an increased

208 to measure the effects of H(2)O(2)-producing lactobacilli on gonococcal infection.

209 ection was adapted to examine the effects of lactobacilli on gonococcal interactions with endometrial

210 However, the influence of probiotic lactobacilli on immune responses in the context of VVC r

211 onectin and casein, as did CbpG expressed on lactobacilli or as a purified full-length or truncated r

212 P < .001) and with absence of H2O2-positive lactobacilli (OR, 2.9; P = .04).

213 ngs the colonization rates at age 5 weeks of lactobacilli (P < .001) and bacteroides (P = .02) increa

214 emales colonized by other H(2)O(2)-producing lactobacilli (P < .001).

215 increased the bifidobacterial (P < 0.01) and lactobacilli (P < 0.001) populations but significantly d

216 loss of colonization with H(2)O(2)-producing lactobacilli (P=.018), as was antibiotic use (P< or =.00

217 In particular, elevated levels of lactobacilli physically displaced and attenuated C. rode

218 tide-1 content as well as Bifidobacteria and Lactobacilli populations in the caecum.

219 into 2 groups: those with H(2)O(2)-producing lactobacilli present (n=191; 20.2%) and those with H(2)O

220 Lactobacilli present in caries lesions represent both a

221 ined the ability of G. vaginalis to displace lactobacilli previously adhered to HeLa cells.

222 guished CTV-05 from other endogenous vaginal lactobacilli prior to and after vaginal capsule use.

223 ver, until now, most research has focused on lactobacilli probiotics.

224 ever, coliform colonization occurred whether lactobacilli produced H2O2 or not.

225 Lactobacilli produced leucine, isoleucine and valine as

226 t the hypothesis that H2O2-producing vaginal lactobacilli protect against acquisition of BV but do no

227 iological studies suggest H(2)O(2)-producing lactobacilli protect women against gonorrhea.

228 s that although most of the tested probiotic lactobacilli reduced C. albicans-induced vaginal epithel

229 estinal colonization by TNF-alpha-inhibitory lactobacilli reduced intestinal inflammation in H. hepat

230 Thus, while cervicovaginal lactobacilli reduced the production of the majority of i

231 Intranasal inoculation of two airway lactobacilli restored IAA and recapitulated its protecti

232 Priming with live lactobacilli resulted in diminished granulocyte recruitm

233 itially colonized only by non-H2O2-producing lactobacilli resulted in loss of vaginal lactobacilli (R

234 OS promoted the growth of bifidobacteria and lactobacilli, resulting in high levels of short chain fa

235 ing lactobacilli resulted in loss of vaginal lactobacilli (RR, 2.5; 95% CI, 1.2-5.3).

236 V and promotion of vaginal colonization with lactobacilli should be evaluated as potential interventi

237 each sample, we calculated the proportion of lactobacilli species (ie, Lactobacillus crispatus, Lacto

238 Lactobacilli species are considered non-pathogenic organ

239 Fourteen postbiotics derived from various lactobacilli species were screened, and Latilactobacillu

240 In an early study, three Lactobacilli strains (L. farciminis H3 and A11 and L. sa

241 The present study utilizes lactobacilli strains having the potential to accumulate

242 A probiotic containing 2 Lactobacilli strains or placebo was administered for 3 w

243 Selected binary combinations of lactobacilli strains were recently successfully used to

244 ARDRA and RAPD data showed three distinct lactobacilli strains, including L. acidophilus, L. plane

245 ing zinc oxide nanoparticles (ZnO-NPs) using lactobacilli strains.

246 vable microbes, including oral streptococci, lactobacilli, Streptococcus mutans, and Candida, in sali

247 We also demonstrate that commensal lactobacilli support the growth of N. gonorrhoeae in vit

248 udies indicate that probiotics, particularly lactobacilli, suppressCandidagrowth and biofilm developm

249 Overall, lactobacilli suppressed IL-6 (adjusted p < 0.001) and IL

250 detection of H(2)O(2) by a broader range of lactobacilli than a published, widely used agar formulat

251 P < .001) and in women without H2O2-positive lactobacilli than in women with (odds ratio [OR], 4.0; P

252 ntain a normal vaginal flora than women with lactobacilli that do not produce H(2)O(2).

253 We identified new shuffled lactobacilli that grow at substantially lower pH than do

254 esence of novel CRISPR-Cas immune systems in lactobacilli that may be exploited for genome editing.

255 Commensal lactobacilli that produce hydrogen peroxide (H(2)O(2)) i

256 Since strains of lactobacilli that produce hydrogen peroxide inhibit the

257 For differentiating lactobacilli, the bacterial suspension was screened for

258 Although often dominated by lactobacilli, the vaginal microbiota is also frequently

259 The effect of adding the cells of four lactobacilli to a squid pen powder (SPP)-containing medi

260 The goal of this study was to employ these lactobacilli to combat human immunodeficiency virus type

261 teria-free postbiotics, bioactive lysates of lactobacilli, to reduce intracellular S. aureus coloniza

262 longitudinal loss or acquisition of vaginal lactobacilli used as probiotics.

263 Heavy growth of lactobacilli was associated with a lower frequency of va

264 the vagina and rectum by H(2)O(2)-producing lactobacilli was associated with the lowest prevalence o

265 s can naturally or therapeutically encounter lactobacilli, we investigated the effects of several wel

266 Lactobacilli, well known as beneficial bacteria, are als

267 Furthermore, lactobacilli were able to displace adherent gonococci fr

268 females, vaginal and rectal colonization by lactobacilli were assessed by culture methods.

269 Low levels of vaginal lactobacilli were associated with trichomoniasis (odds r

270 Lactobacilli were detected in 64% of subjects, and 42% o

271 No lactobacilli were detected in the lungs of wild-type (WT

272 Salivary levels of mutans streptococci and lactobacilli were enumerated at baseline, 3, 6, and 12 m

273 ariable fragment Abs secreted by transformed lactobacilli were evaluated for their protective efficac

274 cecal crypt communities is diverse and that Lactobacilli were found closer to the lumen than Bactero

275 The longevity-promoting benefits of lactobacilli were hypothesized as early as 1907.

276 Lactobacilli were identified on the basis of DNA homolog

277 A total of 272 isolates of lactobacilli were identified to the species level by use

278 Vaginal and rectal lactobacilli were identified to the species level by usi

279 Cytokine responses to the lactobacilli were inversely associated with lactobacilli

280 Results show that lactobacilli were less abundant in fermentations perform

281 Women with reduced lactobacilli were less likely to regain normal flora tha

282 o-cultivation with C. albicans and probiotic lactobacilli were mostly driven by specific bacterial sp

283 Levels of S. mutans were positively and Lactobacilli were negatively associated with caries.

284 Lactobacilli were observed to be a microbiome component

285 Vaginal lactobacilli were present in 62% of the women and were s

286 Murine-derived probiotic lactobacilli were selected in vitro for their ability to

287 ified polymorphic DNA (RAPD) and distinctive lactobacilli were sequenced.

288 alis and M. hominis and decreasing levels of lactobacilli were significantly associated with BV by Nu

289 The predominant lactobacilli were the species Lactobacillus crispatus (3

290 to regain normal flora than were those whose lactobacilli were unaffected.

291 Lactobacilli, when targeted to the respiratory epitheliu

292 Fermented vegetables contain many lactobacilli, which are also potent Nrf2 activators.

293 biotics, or by endogenous bifidobacteria and lactobacilli, whose metabolic activity and growth may al

294 ; and (2) that toxicological assays of these lactobacilli will show them to be benign.

295 which is indicative of DC maturation, those lactobacilli with greatest capacity to induce IL-12 were

296 ome axis and provide a framework to engineer lactobacilli with improved bile resistance and use probi

297 tobacillus acidophilus BG2FO4 is a strain of lactobacilli with properties of marked intestinal adhere

298 t juice appeared to support the viability of lactobacilli, with higher microorganism numbers observed

299 ample, vaginal suppositories containing live lactobacilli, would alter the microbiomial composition h

300 ontained mainly streptococci, staphylococci, lactobacilli, yeasts, and enterobacteria.