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1  boutons in laminae I-II, and 5% of those in lamina III.
2 erneurons in laminae I-II and 9% of those in lamina III.
3 nse band of punctate labeling was visible in lamina III.
4 ere was a shrinking gap between lamina I and lamina III.
5  in lamina II and short-latency responses in lamina III.
6  ventral lamina II, but were also present in lamina III.
7 ocytes, which decreased neuronal activity in lamina III and IV and impaired mechanosensation associat
8   The phrenic afferents activated neurons in lamina III and IV of areas 3a and 3b.
9 primed mice activates a subset of neurons in lamina III and IV of the dorsal horn that coexpress PAX2
10 ifically labels astrocyte populations within lamina III and IV of the dorsal spinal cord.
11 tate staining, mainly in the ventral part of lamina IIi and lamina III, than in material fixed with 4
12 e neuropil of the spinal cord, especially in lamina III and the area surrounding the central canal.
13 laminas I-III), 25-30% with anti-GluR2/3 (in laminas III and IV), and 5% with anti-GluR2 (in laminas
14 naptophysin and GluR2/3 are predominantly in laminas III and IV, whereas puncta immunopositive for sy
15 in laminas I and II and with anti-GluR2/3 in laminas III and IV.
16 d throughout the inner half of the SG (i.e., lamina IIi) and sent dense clusters of terminals well in
17 ity was the most intense in inner lamina II, lamina III, and lamina X, and it was the least intense i
18 rokinin 1 receptor are found in lamina I and lamina III, and PKCgamma was present in 22% and 37% of t
19 labeled, Zif268-positive neurons appeared in lamina III Around P16, however, many immunoreactive neur
20 put from the superficial laminae, especially lamina IIi, as well as the II/III border region.
21 did cause substantial cell death not only in lamina III but also in laminae I and II.
22  one-half of the Abeta-fiber terminations in lamina III contain N-cadherin; none contain E-cadherin.
23 ls and a defective presynaptic inhibition of lamina IIi interneurons.
24 ely to nearby dendrites of 135,000 simulated lamina III-IV cells whose dendritic surface area distrib
25 ry input to the superficial dorsal horn from lamina III-IV was identified in a subset of the vertical
26                                         Most lamina III/IV NK1r cells at both levels projected to LPb
27                                              Lamina III/IV NK1R+ neurons and lamina I NK1R- neurons h
28 e AMPA receptors are located on lamina I and lamina III/IV NK1R+ neurons postsynaptic to primary affe
29  (NK1R-) neurons including interneurons, and lamina III/IV NK1R+ neurons, believed to contribute to t
30                     We found that 85% of the lamina III/IV NK1r-immunoreactive neurons in C6 and 17%
31                            Most lamina I and lamina III/IV NK1r-immunoreactive spinothalamic neurons
32 elimination of NK1R+ neurons in lamina I and lamina III/IV of the dorsal horn also suppresses develop
33 (Adelta/C fiber) monosynaptic input, whereas lamina III NK1R+ neurons received low-threshold (Abeta f
34                        In lamina I NK1R- and lamina III NK1R+ neurons, disinhibition enhanced control
35                                 For both the lamina III NK1r-immunoreactive projection neurons and pr
36 routing Abeta fibers normally terminating in lamina III occurs after sciatic nerve neuroma formation.
37 properties similar to low-threshold EPSPs in lamina III of control slices.
38 d behaviors, as shown by c-Fos expression in lamina III of the dorsal horn and the expression of ChR2
39 ribution and nature of these interactions in lamina III of the dorsal horn.
40 ely labeled by the lectin IB4 and project to lamina IIi of the dorsal horn.
41 vity is mostly restricted to interneurons in lamina IIi of the medullary dorsal horn, where they cons
42                  Large projection neurons in lamina III of the rat spinal cord that express the neuro
43 f CGRP-expressing excitatory interneurons in lamina III of the spinal cord dorsal horn and trigeminal
44                                              Lamina IIi PKCgamma interneurons were shown to be activa
45 aise the general issue of synaptic inputs to lamina IIi PKCgamma interneurons.
46 ndicates a highly selective targeting of the lamina III projection cells by glutamatergic neurons tha
47  noxious stimulation, this suggests that the lamina III projection cells receive powerful monosynapti
48 efore likely that polysynaptic inputs to the lamina III projection neurons are recruited during the d
49 munoreactive axons form numerous synapses on lamina III projection neurons that possess the neurokini
50 eshold mechanoreceptors (LTMRs) terminate in lamina IIi raise the general issue of synaptic inputs to
51 mainly in the ventral part of lamina IIi and lamina III, than in material fixed with 4% paraformaldeh
52 rs resulted in the loss of 25% of neurons in lamina III, the major site of termination of large Abeta
53 ollowing this study, the responses of single lamina III-V dorsal horn neurons to an innocuous A beta
54 embranes of neurons in the deep dorsal horn (lamina III-V) and the ventral horn (lamina VI-IX).
55 in the majority of deep dorsal horn neurons (laminas III-VI; 83 of 139 cells), but only in a minority
56 noreactivity in the protein kinase Cgamma-IR lamina IIi was quantified and found to increase.
57 n, besides KChIP3 in the inner lamina II and lamina III, we detected DPP10 in most projection neurons
58 e majority of monosynaptic EPSPs recorded in lamina III were elicited by low-threshold nerve stimulat
59                          Afferent boutons in lamina III were often surrounded by several presynaptic
60 r part of lamina II (IIo) and dorsal part of lamina IIi, whereas the B subunit of the cholera toxin (