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1 gnoses and 690 with non-PDD diagnoses (e.g., language disorder).
2  factor causes a severe monogenic speech and language disorder.
3 doreductase Subunit A11 (NDUFA11) for speech/language disorder.
4 ctual disability, coordination disorder, and language disorder.
5  chronic serous otitis media, and expressive language disorder.
6  first known monogenic cause of a speech and language disorder.
7 s in adulthood with an isolated, progressive language disorder.
8 ion of the FOXP2 protein causes an inherited language disorder.
9 pmental verbal dyspraxia, a human speech and language disorder.
10 entify a consistent neural substrate for the language disorder.
11 t be central to the FOXP2-related speech and language disorder.
12 umans cause an autosomal-dominant speech and language disorder.
13 at mutations in human FOXP2 produce a severe language disorder.
14 have AD, while the female sib has expressive language disorder.
15 sive aphasia beyond the lens of an exclusive language disorder.
16 nding and perhaps ameliorating developmental language disorders.
17 ically robust risk markers for developmental language disorders.
18 severe, persistent, developmental speech and language disorders.
19  associated with an increased risk of speech/language disorders.
20 lioration and/or prevention of developmental language disorders.
21 tor that plays a role in the pathogenesis of language disorders.
22 pment of standardized clinical assessment of language disorders.
23 ave reported an involvement of this tract in language disorders.
24 o determine its potential as a biomarker for language disorders.
25 ical evaluation to advance identification of language disorders.
26 pathways that may be disturbed in speech and language disorders.
27 actor on 7q31 contributes to both autism and language disorders.
28 ophrenia who had a high rate of prepsychotic language disorders.
29  protein P2 (FOXP2) is associated with human language disorders.
30 EAS), a spectrum of epileptic, cognitive and language disorders.
31 early screening and delineating the roots of language disorders.
32 (95% CI, 0.88-1.16) for developmental speech/language disorder, 0.79 (95% CI, 0.54-1.17) for developm
33 2; intellectual disorder 1.00, 0.93 to 1.07; language disorder 1.05, 1.02 to 1.09; and epilepsy 1.03,
34 ellectual disorder (1.07, 0.98 to 1.15), and language disorder (1.04, 1.00 to 1.08) in the sibling an
35 disability (DD/ID, 17/21), epilepsy (10/21), language disorders (16/21), movement disorders (12/21),
36 rocessing disorder, expressive and receptive language disorders (71% of individuals >3 years old with
37 earning disability, 1.2% and 0.6%; speech or language disorder, 8.4% and 4.5%; developmental coordina
38 n factors--age, cognitive level, and type of language disorder--account for variability in the report
39 enotyping Tool for Identifying Developmental Language Disorder algorithm of the Vanderbilt University
40 ing our understanding of factors that signal language disorder and delay has the potential to improve
41  memory impairment, personality alterations, language disorder and facial paralysis have all been app
42    Dyslexia is the most common developmental language disorder and is marked by deficits in reading a
43 aired in some individuals with developmental language disorder and/or dyslexia.
44                 A link between developmental language disorders and atypical cerebral lateralization
45  understanding of the etiology of speech and language disorders and enable us to better understand th
46 that has been implicated in human speech and language disorders and plays important roles in the plas
47  one's differential diagnosis for speech and language disorders and understanding the link between ea
48 sm spectrum disorder, intellectual disorder, language disorder, and epilepsy in children.
49 nvolvement in autoimmune disease, speech and language disorders, and lung development.
50 g autism, intellectual disability, epilepsy, language disorders, and movement disorders.
51  a transcription factor implicated in speech/language disorders, and that this interaction is disrupt
52 ealed significant differences (speech and/or language disorder: AOR, 5.83 [95% CI, 4.31-7.89]; behavi
53                                Developmental language disorders are highly heritable with substantial
54  idea that some individuals with reading and language disorders are impaired in their perception of n
55                                   Speech and language disorders are known to have a substantial genet
56 y progressive aphasia (PPA) is a progressive language disorder associated with atrophy of the dominan
57 of this gene causes a rare severe speech and language disorder but does not appear to be involved in
58 ription factor cause an inherited speech and language disorder, but how FoxP2 contributes to learning
59  neurological pathways underlying speech and language disorders, but such investigations are only jus
60 neralizable neural prostheses for speech and language disorders by leveraging large-scale intracrania
61                     ATP2C2 was implicated in language disorders by linkage and association studies, a
62 rs of the KE family suffer from a speech and language disorder caused by a mutation in the FOXP2 gene
63 ded if they had another premorbid speech and language disorder caused by a neurological deficit other
64                            Are developmental language disorders caused by poor auditory discriminatio
65  special senses, conduct disorder, receptive language disorder, chronic serous otitis media, and expr
66 ded oversimplified accounts of the nature of language disorders, classifying patients into syndromes
67 d a significant 37% increased risk of speech/language disorders compared with offspring in the unmedi
68 lines the landscape of paediatric speech and language disorders, confirming the phenotypic complexity
69                     Developmental speech and language disorders cover a wide range of childhood condi
70 ity disorder, learning disability, speech or language disorder, developmental coordination disorder,
71 y disorder, learning disabilities, speech or language disorders, developmental coordination disorders
72           Subgroups of paediatric speech and language disorders differ significantly with respect to
73 gh learning disorders (LD) and developmental language disorder (DLD) can be linked to overlapping psy
74                                Developmental language disorder (DLD) is a common (with up to 7% preva
75                                Developmental language disorder (DLD) is a common neurodevelopmental d
76                                Developmental language disorder (DLD) is a common neurodevelopmental d
77                                Developmental language disorder (DLD) is a neurodevelopmental disorder
78                             In developmental language disorder (DLD), learning to comprehend and expr
79 ported a similar phenomenon in developmental language disorder (DLD).
80 gh-functioning autism and with developmental language disorder (DLD).
81 the lives of the millions with developmental language disorder (DLD).
82  disorders of language include developmental language disorder, dyslexia, and motor-speech disorders
83                                Developmental language disorders exist in 5% to 10% of preschoolers an
84 o be implicated in the cause of a speech and language disorder (FOXP2).
85 l prenatal cannabis use and child speech and language disorders (HR, 0.93; 95% CI, 0.84-1.03), global
86 truncation mutation, segregating with speech/language disorder in a second family, yields an unstable
87 spectrum disorder, intellectual disorder, or language disorder in children.
88 d an in-depth characterization of speech and language disorders in 52 143 individuals, reconstructing
89 only known cause of developmental speech and language disorders in humans.
90  found significant comorbidity of speech and language disorders in neurodevelopmental conditions (30.
91 ould be a significant risk factor for speech-language disorders in the general adult population.
92 , intellectual disabilities (IDs) and speech/language disorder-in the ethnic minority of African Amer
93 nderlying biological nature of listening and language disorders, inform treatment strategies, and pro
94 uptions of the FOXP2 gene cause a speech and language disorder involving difficulties in sequencing o
95 n pedigree, KE, in which a severe speech and language disorder is transmitted as an autosomal-dominan
96 2, the first gene causally linked to a human language disorder, is implicated in song acquisition, pr
97 me 7 which co-segregates with the speech and language disorder (maximum lod score = 6.62 at theta = 0
98       Early developmental delays (speech and language disorders, motor delays, global delays) in chil
99                                     Although language disorders occur in about 50% of patients with a
100 ctrum of childhood epileptic, cognitive, and language disorders of unknown etiology.
101                                 Prepsychotic language disorder predicted abnormal planum temporale as
102 ses of brain structure in genetic speech and language disorders provide an opportunity to identify ne
103                           Genetic speech and language disorders provide the opportunity to investigat
104             The protean nature of speech and language disorders requires careful consideration of sev
105         A popular view is that developmental language disorders result from a poorly lateralized brai
106 a unique family in which a severe speech and language disorder segregates in a monogenic fashion, at
107                       A common developmental language disorder, specific language impairment (SLI), a
108 ons between auditory processing deficits and language disorders such as dyslexia; but whether the for
109 Ms, enabling us to refine models of existing language disorders, such as aphasia.
110 fied as the basis of an inherited speech and language disorder suffered by members of the family know
111 imer's disease (AD) display a characteristic language disorder that resembles LPA, or if LPA is a cli
112              The cumulative hazard of speech/language disorders was 0.0087 in the SSRI-exposed group
113 subjects with a neurodegenerative speech and language disorder were prospectively recruited and under
114 embers are affected with a severe speech and language disorder which appears to be transmitted as an
115 ncoded by SLC6A8), which leads to speech and language disorders with severe cognitive impairment.
116  framework for the examination of speech and language disorders, with additional implications for the

 
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