ライフサイエンスコーパス: large-cell

1 spindle placement within disproportionately large cells.

2 ze increases in small cells and decreases in large cells.

3 etworks even prevent granule condensation in large cells.

4 heterogeneous phenotype with the presence of large cells.

5 for small cells, but decrease with size for large cells.

6 of Ccr4-Not complexes results in abnormally large cells.

7 generate action potentials (APs) than medium-large cells.

8 Of these large cells, 20 to 37% were apoptotic, according to both

9 , 19.7% squamous cell, 6.1% undifferentiated large cell, 7.2% non-small cell not otherwise specified,

10 imilar staining patterns for cytokeratins in large cell acanthoma and normal conjunctiva, except for

11 a low Ki-67 proliferation index (PI) in the large cell acanthoma compared with high indices in dyspl

12 Conjunctival large cell acanthoma is a morphologically distinctive cl

13 The large cell acanthoma recurred 4 times but retained its b

14 One large cell acanthoma, 7 epidermoid dysplasias, and 4 squ

15 Evidence for large cell aggregation was only found in N. Pacific samp

16 independently of SHH and displays classic or large cell anaplastic (LCA) pathology and poor prognosis

17 d the diagnosis of breast implant-associated large cell anaplastic lymphoma for their repercussions.

18 the implants; and breast implant-associated large cell anaplastic lymphoma.

19 -induced PNETs are histologically similar to large cell/anaplastic medulloblastomas and can occur in

20 t that sPNET with varied differentiation and large cell/anaplastic medulloblastomas may be two tumor

21 Medulloblastomas that display a large cell/anaplastic morphology and overexpress the cel

22 Metastatic disease and large-cell/anaplastic (LC/A) phenotype were the clinicop

23 % CI, 0 to 55) and 40% (95% CI, 0 to 83) for large-cell/anaplastic (n = 5) medulloblastoma ( P < .001

24 ural progenitors induced tumors exhibiting a large-cell/anaplastic histopathology adjacent to the fou

25 tients with classic medulloblastoma (CMB) or large-cell/anaplastic medulloblastoma (LCA).

26 0.19 for desmoplastic/nodular and 45.97 for large-cell/anaplastic medulloblastoma) and nonresponse t

27 lps identify preneoplastic lesions including large cell and small cell change.

28 uited to convey morphogenetic information in large cells and tissues and provides a versatile model s

29 ell lines, including mantle cell, anaplastic large cell, and Hodgkin lymphoma cell lines.

30 Captured melanoma CTCs were large cells, and consisted of 2 subpopulations, based on

31 pe illumination optical path that produces a large cell- and beam-angle invariant stray excitation th

32 otherwise specified; 2 (13%) had anaplastic large cell; and 1 each had extranodal natural killer/T c

33 creases with increasing size for excessively large cells; and (2) cell division occurs as per the Add

34 ls (decay time, approximately 25 ms) than in large cells ( approximately 2 ms), and repetitive stimul

35 compared with the distribution of all cells, large cells are more dense in the vicinity of veins and

36 Large cells are produced by increasing DNA content or pl

37 Other large cells arise more peripherally by initiating endocy

38 rhabditis elegans aggregates are observed in large cells/blastomeres (oocytes, embryos) and in smalle

39 nd R. minor Grell, another with a relatively large cell body forming lacunae, and another that has si

40 ffect is more pronounced for bacteria with a large cell body.

41 tant in maintaining arousal, we first placed large cell-body-specific lesions in these targets.

42 cellular proliferation is low for small and large cells, but high at intermediate sizes.

43 ding rates of its slowest proteins, and--for large cells--by the rates of its protein diffusion.

44 tological, and functional characteristics of large-cell calcifying Sertoli cell tumors of the testes

45 ) and irregular margin (n = 3, 60%); and for large cell carcinoma (n = 1), solid attenuation and spic

46 adenocarcinoma, squamous cell carcinoma, and large cell carcinoma) caused up-regulation of CCR2/CCL2

47 adenocarcinoma, squamous-cell carcinoma, and large-cell carcinoma.

48 rcinoma, squamous cell, and undifferentiated large cell carcinomas comparing the highest with the low

49 In contrast, the wild type formed large cell clumps of dead and live cells, indicating the

50 pective of the age of the donor hepatocytes, large cell clusters appeared in juvenile, but only small

51 proliferate within distinct islands, forming large cell clusters that eventually fuse during metamorp

52 Deletion of the enzyme results in large cell clusters with disordered division patterns, i

53 oil, chromoplasts, small cell clusters, and large cell clusters) were isolated from different types

54 w strategy for the mechanical stimulation of large cell clusters, taking advantage of dielectrophores

55 and may prove advantageous for the study of large cell collectives, such as the spheroid models prev

56 f small groups of cells and the expansion of large cell colonies.

57 y functional theory (DFT) calculation with a large cell containing 1024 atoms (614 Ni and 410 Nb).

58 Large cell deposits first appeared at 1 month in 40% of

59 so inhibition potentially prevents damage of large cell envelope components, but not host digestion.

60 To accommodate the large cells following zygote formation, early blastomere

61 Common to all succulents is the presence of large cells for water storage.

62 Large cell formation is functionally important because w

63 We propose that large cells generated by polyploidization or cell fusion

64 The most successful cryoprotectant for "large cell" (> 9 mum) viability (72.6% +/- 10.5%) compri

65 istently, several studies have revealed that large cells have lower mass accumulation rates per unit

66 ve disease (86% v 60%; P < .001), anaplastic large-cell histology (53% v 40%; P = .04), and two or fe

67 PreBII large cells in adults, but not in children, showed highl

68 cting to VPM; and 7) approximately 30-50% of large cells in PrV send collaterals to ZI and VPM.

69 l cells were 10-100 times more abundant than large cells in the N. Pacific, whereas in the S. Pacific

70 , replication-competent virus measurement by large cell input quantitative viral outgrowth assay, and

71 macrophages had similar numbers of small and large cells; interstitial macrophages were mainly small.

72 ctivity for neurofilament was predominant in large cells led us to directly examine the relationship

73 In large cells like neurons, how is microtubule polymerizat

74 This study reinforces the utility of large cell line panels for the study of cancer and ident

75 th senescence should be useful for screening large cell line panels to identify genomic biomarkers of

76 n therapy monitoring, 2 murine tumor models (large cell lung cancer cell line H460 and prostate cance

77 g adenocarcinomas, squamous cell carcinomas, large cell lung cancer, adenoid cystic carcinomas and ce

78 ose with pancreatic tumours or small-cell or large-cell lung cancer, as well as those without complet

79 contributes to the oncogenesis of anaplastic large cell lymphoma (ALCL) are not completely understood

80 uxotrophy, particularly in ALK(+) anaplastic large cell lymphoma (ALCL) cell lines and primary tumour

81 We used various ALK-positive anaplastic large cell lymphoma (ALCL) cell lines to evaluate two in

82 malignant lymphocytes, including anaplastic large cell lymphoma (ALCL) cells.

83 IP is frequently low or absent in anaplastic large cell lymphoma (ALCL) compared to other T cell lymp

84 ic lymphoma kinase (ALK)-positive anaplastic large cell lymphoma (ALCL) constitutes an ideal model di

85 dies in Hodgkin lymphoma (HL) and anaplastic large cell lymphoma (ALCL) has had profound clinical suc

86 ic lymphoma kinase (ALK)-negative anaplastic large cell lymphoma (ALCL) is a CD30-positive T-cell non

87 Anaplastic large cell lymphoma (ALCL) is a distinct entity of T-cel

88 Anaplastic large cell lymphoma (ALCL) is a mature T cell neoplasm t

89 Anaplastic large cell lymphoma (ALCL) is a mature T-cell lymphoma t

90 Anaplastic large cell lymphoma (ALCL) is a peripheral T-cell lympho

91 Systemic anaplastic large cell lymphoma (ALCL) is an aggressive CD30(+) non-

92 Systemic anaplastic large cell lymphoma (ALCL) is an aggressive T-cell lymph

93 Anaplastic large cell lymphoma (ALCL) is the most common type of pe

94 a subset of T-cell lymphomas with anaplastic large cell lymphoma (ALCL) morphology (ALK+ ALCL), the v

95 lymphoma kinase-positive (ALK(+)) anaplastic large cell lymphoma (ALCL) patients.

96 In ALK-rearranged anaplastic large cell lymphoma (ALCL), a specific subtype of T-cell

97 sed Hodgkin lymphoma and systemic anaplastic large cell lymphoma (ALCL), the single agent response ra

98 sed Hodgkin lymphoma and systemic anaplastic large cell lymphoma (ALCL).

99 y efficacious in animal models of anaplastic large cell lymphoma (ALCL).

100 ic lymphoma kinase (ALK)-positive anaplastic large cell lymphoma (ALCL).

101 psed or refractory (R/R) systemic anaplastic large cell lymphoma (ALCL).

102 ic lymphoma kinase (ALK)-negative anaplastic large cell lymphoma (ALK-negative), despite their unique

103 lved in breast implant-associated anaplastic large cell lymphoma (BI-ALCL) remain elusive.

104 rate of breast implant-associated anaplastic large cell lymphoma (BIA-ALCL) at a high-volume single i

105 Breast implant-associated anaplastic large cell lymphoma (BIA-ALCL) is a very rare type of T-

106 h rare, breast implant-associated anaplastic large cell lymphoma (BIA-ALCL), a CD30+ T-cell lymphoma

107 Breast implant-associated anaplastic large cell lymphoma (BIA-ALCL), a rare peripheral T-cell

108 Breast implant-associated anaplastic large cell lymphoma (breast implant ALCL) is an uncommon

109 as robust enough to differentiate anaplastic large cell lymphoma (n = 32) from other PTCLs.

110 a in patients with secondary CL were diffuse large cell lymphoma (n = 5, 28%), chronic lymphocytic le

111 phoma kinase (NPM-ALK) expressing anaplastic large cell lymphoma are not completely understood.

112 breaks and translocations in the anaplastic large cell lymphoma breakpoint regions of NPM1 and ALK.

113 ase (ALK)-positive and -negative, anaplastic large cell lymphoma cell lines and primary patient tumou

114 plastic lymphoma kinase-positive, anaplastic large cell lymphoma cell lines and that ectopically expr

115 Breast implant-associated anaplastic large cell lymphoma is a rare cancer in patients with br

116 target different receptors on the anaplastic large cell lymphoma line L-82, but delivered the same cy

117 ation detected in the majority of anaplastic large cell lymphoma patients, and has recently been impl

118 s fungoides (T-MF), and cutaneous anaplastic large cell lymphoma were studied in parallel with corres

119 morphologically more high grade (50% diffuse large cell lymphoma) compared with primary CL (37% low-g

120 16 relapsed CLL and 9 RT (all proven diffuse large cell lymphoma) patients were enrolled, and 60% rec

121 omas (targeting 75% with systemic anaplastic large cell lymphoma) were randomly assigned 1:1 to recei

122 aplastic lymphoma kinase-positive anaplastic large cell lymphoma), upfront auto-SCT was associated wi

123 ALK)-positive and 48 ALK-negative anaplastic large cell lymphoma, 14 adult T-cell leukemia/lymphoma a

124 to 3% of patients per year into aggressive, large cell lymphoma, a critical event in the course of t

125 apulosis (LyP), primary cutaneous anaplastic large cell lymphoma, and borderline cases.

126 angioimmunoblastic, ALK-negative anaplastic large cell lymphoma, and enteropathy-associated T-cell l

127 rge B-cell lymphoma, ALK-positive anaplastic large cell lymphoma, chronic myelogenous leukemia, and a

128 actory Hodgkin lymphoma, systemic anaplastic large cell lymphoma, relapsed or refractory B-cell precu

129 rhabdomyosarcoma, neuroblastoma, anaplastic large cell lymphoma, renal cell carcinoma, and inflammat

130 a randomized, prospective clinical trial in large cell lymphoma, we conducted serial fluorodeoxygluc

131 ry syndrome, or primary cutaneous anaplastic large cell lymphoma, with disease progression after >/=

132 e for patients with CD30-positive anaplastic large cell lymphoma.

133 als in CD30+ Hodgkin lymphoma and anaplastic large cell lymphoma.

134 ng classical Hodgkin lymphoma and anaplastic large cell lymphoma.

135 of MALT lymphoma to activated B-cell diffuse large-cell lymphoma (ABC-DLBCL).

136 -derived neoplasias, most notably anaplastic large-cell lymphoma (ALCL) and Hodgkin's lymphoma.

137 ents in several tumors, including anaplastic large-cell lymphoma (ALCL) and non-small cell lung carci

138 homa kinase-positive (NPM-ALK(+)) anaplastic large-cell lymphoma (ALCL) as model system, we found in

139 Most of the anaplastic large-cell lymphoma (ALCL) cases carry the t(2;5; p23;q3

140 Anaplastic large-cell lymphoma (ALCL) frequently carries the t(2;5)

141 Anaplastic large-cell lymphoma (ALCL) is a clinical and biological

142 Breast implant-associated anaplastic large-cell lymphoma (ALCL) is a recently described clini

143 Systemic anaplastic large-cell lymphoma (ALCL) is a T-cell lymphoma, whose a

144 Systemic anaplastic large-cell lymphoma (ALCL) is an aggressive subtype of T

145 c proteins in the pathogenesis of anaplastic large-cell lymphoma (ALCL) is well established.

146 matologic malignancy ALK-positive anaplastic large-cell lymphoma (ALCL) resistant to ALK-specific tyr

147 regulated in T-lymphomas, such as anaplastic large-cell lymphoma (ALCL) tumors.

148 c lymphoma kinase (ALK) -positive anaplastic large-cell lymphoma (ALCL) was excluded.

149 Anaplastic large-cell lymphoma (ALCL) was initially recognized on t

150 lymphoma kinase-positive (ALK(+)) anaplastic large-cell lymphoma (ALCL), and adult T-cell leukemia/ly

151 ts with Hodgkin lymphoma (HL) and anaplastic large-cell lymphoma (ALCL), the study by Jacobsen and co

152 ic lymphoma kinase (ALK)-positive anaplastic large-cell lymphoma (ALCL).

153 aplastic lymphoma kinase-positive anaplastic large-cell lymphoma (ALK(+) ALCL) is a unique type of T-

154 Breast implant-associated anaplastic large-cell lymphoma (BIA-ALCL) is a CD30-positive, anapl

155 In patients with diffuse large-cell lymphoma (DLBCL), we observed higher levels o

156 NHL and of two predominant subtypes, diffuse large-cell lymphoma (DLCL) (n = 233) and follicular lymp

157 T-cell lymphoma/leukemia (n = 4), anaplastic large-cell lymphoma (n = 2), and extranodal natural kill

158 astic T-cell lymphoma [AITL], and anaplastic large-cell lymphoma [ALCL]) is difficult, with the morph

159 All seven patients without anaplastic large-cell lymphoma achieved CR.

160 ALK translocations, particularly anaplastic large-cell lymphoma and inflammatory myofibroblastic tum

161 y expressed in a subset of T-cell anaplastic large-cell lymphoma and non-small-cell lung cancer, resp

162 Hodgkin's lymphoma and anaplastic large-cell lymphoma are the two most common tumors expre

163 of relapsed Hodgkin lymphoma and anaplastic large-cell lymphoma by the Food and Drug Administration.

164 set of acute myeloid leukemia and anaplastic large-cell lymphoma cell lines by inducing cell-cycle ar

165 patients, particularly those with anaplastic large-cell lymphoma histology.

166 In T-cell lymphomas, anaplastic large-cell lymphoma is defined by mutually exclusive rea

167 is fungoides or primary cutaneous anaplastic large-cell lymphoma who had been previously treated.

168 pression of CD30 (the hallmark of anaplastic large-cell lymphoma) and of immunosuppressive cytokine I

169 's transformation (CLL that has evolved into large-cell lymphoma) and only one case of CLL progressio

170 pression of CD30 (the hallmark of anaplastic large-cell lymphoma).

171 Outcomes of 241 patients (112 anaplastic large-cell lymphoma, 102 peripheral T-cell lymphoma not

172 iated with an improved outcome in anaplastic large-cell lymphoma, ALK positive.

173 t otherwise specified (PTCL-NOS), anaplastic large-cell lymphoma, and adult T-cell leukemia/lymphoma

174 amine T-cell lymphomas, including anaplastic large-cell lymphoma, angioimmunoblastic T-cell lymphoma,

175 to Hodgkin lymphoma and systemic anaplastic large-cell lymphoma, CD30 expression of malignant lympho

176 , or oncogenic mutations, such as anaplastic large-cell lymphoma, inflammatory myofibroblastic tumour

177 K aberrations (eight of nine with anaplastic large-cell lymphoma, one of 11 with neuroblastoma, three

178 valuable solid or CNS tumours, or anaplastic large-cell lymphoma, refractory to therapy and for whom

179 atients with Hodgkin lymphoma and anaplastic large-cell lymphoma.

180 with refractory solid tumours and anaplastic large-cell lymphoma.

181 nd blood samples of patients with anaplastic large-cell lymphoma.

182 primarily Hodgkin's lymphoma and anaplastic large-cell lymphoma.

183 and ATLL and a lower incidence of anaplastic large-cell lymphoma; Hispanics had a higher incidence of

184 netic lesion underlying pediatric anaplastic large cell lymphomas (ALCL) and inflammatory myofibrobla

185 (NSCLC) and approximately 70% of anaplastic large cell lymphomas (ALCL).

186 ccurring in approximately half of anaplastic large cell lymphomas (ALCLs).

187 event in the pathogenesis of most anaplastic large cell lymphomas (ALCLs).

188 aplastic lymphoma kinase (ALK)(-) anaplastic large cell lymphomas (ALCLs).

189 stic lymphoma kinase (ALK)- nodal anaplastic large cell lymphomas (ALCLs; n = 24) were obtained.

190 ubset included lines derived from anaplastic large cell lymphomas, non-small-cell lung cancers, and n

191 of relapsed Hodgkin and systemic anaplastic large cell lymphomas--both characterized by high express

192 ng Classical Hodgkin and systemic anaplastic large cell lymphomas.

193 indistinguishable from patients' anaplastic large cell lymphomas.

194 matory myofibroblastic tumors and anaplastic large cell lymphomas.

195 istics of primary patient-derived anaplastic large cell lymphomas.

196 Anaplastic large-cell lymphomas (ALCLs) are a group of clinically a

197 Anaplastic large-cell lymphomas (ALCLs) bearing the t(2;5) transloc

198 Anaplastic large-cell lymphomas (ALCLs) encompass at least 2 system

199 ts in mice, including Karpas-299 (anaplastic large-cell lymphomas [ALCL]) and H3122 (NSCLC).

200 In addition to 100% anaplastic large-cell lymphomas, 57% of other PTCL entities were CD

201 indistinguishable from patients' anaplastic large-cell lymphomas.

202 characteristic of patient-derived anaplastic large-cell lymphomas.

203 numerical simulations, we predicted that the large cell morphology and the high cellularity of tumor

204 induced inward current densities than medium-large cells, most of which did not respond to capsaicin.

205 logic appearance of totally undifferentiated large-cell NBs (LCNs), the most aggressive and deadly fo

206 arcinoid, small cell lung cancer (SCLC), and large cell NE cancer.

207 10, 71%); for small cell or mixed small and large cell neuroendocrine carcinoma (n = 7), solid atten

208 s with small-cell lung cancer and eight with large-cell neuroendocrine carcinoma, all of whom receive

209 using platinum plus etoposide for those with large-cell neuroendocrine carcinoma.

210 onary carcinoids (including 35 atypical), 75 large-cell neuroendocrine carcinomas (LCNEC), and 66 sma

211 High-grade neuroendocrine lung malignancies (large-cell neuroendocrine cell carcinoma, LCNEC, and sma

212 In contrast to small-cell lung cancer and large-cell neuroendocrine lung tumours, TP53 and RB1 mut

213 ogically confirmed small-cell lung cancer or large-cell neuroendocrine tumours with progressive measu

214 transfer represents a promising therapy, the large cell number required to achieve clinical efficacy

215 unit-fibroblasts (CFU-Fs) on plastic and the large cell numbers required for transplantation.

216 ecules is limited by the need for relatively large cell numbers, especially when studying post-transl

217 (23 classic, eight desmoplastic-nodular, two large cell, one anaplastic), 17 ependymomas (13 World He

218 nvironment of bird macrophages, exhibiting a large cell phenotype that rescues growth suppression.

219 nced suppression of mat3 and even to cause a large-cell phenotype that is comparable to that caused b

220 This matrix is averaged over a large cell population, revealing diagonal blocks called

221 revents the application of these assays to a large cell population, which produces information with s

222 dual cells in an organ-scale simulation of a large cell population.

223 ipal neurons, the Kenyon cells (KCs), form a large cell population.

224 deformation of a macroscopic substrate by a large cell population.

225 d to measuring average radiotracer uptake in large cell populations and, as a result, lack the abilit

226 fast and quantitative mechanical sampling of large cell populations becomes feasible.

227 ntermediate-sized clusters to segregation of large cell populations by formation of smooth boundaries

228 matic study of mechanical characteristics of large cell populations complementing conventional tools

229 lication of single-cell genome sequencing to large cell populations has been hindered by technical ch

230 t enhancer hubs impinging on MYC detected in large cell populations likely do not exist in single cel

231 ugh the overall levels of gene expression in large cell populations seem to be precisely controlled,

232 These measurements, performed over large cell populations, show quite generally that sequen

233 ng improved optical and analytical tools and large cell populations, we show that P1 plasmid foci are

234 cannot be obtained by techniques relying on large cell populations.

235 hniques that measure the ensemble average of large cell populations.

236 studying heterogeneity in gene expression in large cell populations.

237 Large cells received short-latency excitatory inputs and

238 ndividual cells from a particular cell line, large cells should be easier to electroporate than small

239 ow via a dedifferentiation process involving large cell size increases driven by Myc.

240 Large cell size is not restricted to a particular bacter

241 ftsZ gene, which may be responsible for the large cell size of members of this species, as well as g

242 With the evolution of extreme polyploidy and large cell size, Epulopiscium has acquired some of the a

243 lastic leukemia-like cells, characterized by large cell size, multiploid nuclei, and expression of CD

244 id, increasing their DNA content to attain a large cell size.

245 We uncover that large cell-size increase is accompanied by downregulatio

246 econdary dendrites of neurons with medium or large cell somas.

247 with cell length in small cells, but in very large cells spindle length approaches an upper limit of

248 is still not well-understood, especially in large cells such as oocytes.

249 Mucin-4 (Muc4) is a large cell surface glycoprotein implicated in the protec

250 CD13 is a large cell surface peptidase expressed on the monocytes

251 erologous cells, PSD-93 induces formation of large cell-surface clusters.

252 nin-like sequence) gene family encodes eight large cell-surface glycoproteins (Als1-Als7 and Als9) th

253 he cytoplasmic portion of plexins, which are large cell-surface semaphorin receptors, and to mediate

254 rator causing high short circuit current and large cell temperature increase; conversely, negligible

255 cterized histologically by the appearance of large cells that are either aneuploid or polyploid.

256 mal rate, approximately 70 spikes/s), unlike large cells that project to premotor areas (maximal rate

257 In addition to forming large cells, the two-molecule-long bendy walls give this

258 This role is not limited to large cells: the stronger checkpoint in germline precurs

259 Gunflint populations that display relatively large cells (thick-walled spheres, filament-forming rods

260 To achieve sensitivity comparable to that of large cells, those that report upon a small region of vi

261 These defects are due to the inability of large cells to scale nucleic acid and protein biosynthes

262 sion and acid transport, helping to overcome large cell-to-capillary distances that are characteristi

263 Due to large cell-to-cell variability and the lack of direct me

264 Interferon-beta (IFNB1) mRNA shows very large cell-to-cell variability in primary human dendriti

265 This corresponded to large cell-to-cell variability in the decay (tau(w) = 3-

266 Pom1p gradients exhibit large cell-to-cell variability, as well as dynamic fluct

267 propensity for calcium oscillations, despite large cell-to-cell variability.

268 present in a mix of oligomeric states with a large cell-to-cell variation.

269 Surprisingly, there were large cell-to-cell variations for all supplied lutropin

270 In the current study, we describe large cell-to-cell variations in AIS length or distance

271 Despite large cell-to-cell variations in the concentrations of i

272 cells marked by emergence of slow kinetics, large cell-to-cell variations of copy numbers, temporall

273 rganization at all length scales, reflecting large cell-to-cell variations.

274 Large cell transformation (LCT) in mycosis fungoides (MF

275 ith advanced-stage disease (ASD, n = 92) and large cell transformation (LCT, n = 22).

276 Age at diagnosis, large cell transformation and secondary bacterial infect

277 Large cell transformation often hallmarks cases with a m

278 the CTCL lymph node lesions with or without large cell transformation.

279 f positive cells present in the cases with a large cell transformation.

280 phocytic leukemia for 29 and 7 months before large-cell transformation in the eye.

281 l hyperplasia, eosinophil infiltration, less large-cell transformation, and favorable prognosis in MF

282 bles tested, four (stage IV, age > 60 years, large-cell transformation, and increased lactate dehydro

283 Overall, 21 patients (3.8%) developed large-cell transformation, with a significantly higher t

284 onstrated tumor-stage MF,with no evidence of large-cell transformation.

285 D30(+) lymphoproliferative disorders from MF large-cell transformation.

286 ary cutaneous anaplastic T-cell lymphoma and large-cell transformed cutaneous T-cell lymphoma.

287 pharmacologic inhibition of EZH2 activity in large-cell transformed cutaneous T-cell lymphoma.

288 rent obstacles including small cell numbers, large cell-type heterogeneity, complex anatomy, and diff

289 had squamous-cell carcinoma, and 7 (2%) had large-cell undifferentiated carcinoma.

290 e applied for the microfluidic enrichment of large cells, up to the 40 microm diameter level examined

291 by the transition from small-cell variant to large-cell variant.

292 discriminative power of chemometric tools on large cell wall-chromatographic data sets to discover no

293 glycoproteins of Gram-positive bacteria are large, cell wall-anchored adhesins that mediate binding

294 nection between protein localization and the large cell-wall curvature at the poles.

295 large and exhibit many of the phenotypes of large cells, we propose that the range of DNA:cytoplasm

296 Large cells, which are found in all subdivisions of the

297 Large cells, which were targeted under two-photon guidan

298 These attributes contrast with large cells, whose brief IPSCs and rapid firing rates ca

299 nges as very young thread cells develop into large cells with fully mature coiled threads.

300 ndant anti-phospho-ribosomal S6 labelling of large cells within the frontal cortex, basal ganglia, hi