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1 days in the 7 days following bronchoalveolar lavage.
2 us and total fungus in their bronchoalveolar lavage.
3 mbers of cells by whole-lung bronchoalveolar lavage.
4 patients of whom 159 underwent laparoscopic lavage.
5 and E. coli colony counts in bronchoalveolar lavage.
6 esistances were tested in 10 pigs after lung lavage.
7 ing blood, plasma, urine and bronchoalveolar lavages.
8 s could be confirmed in EVs from human nasal lavages.
9 9%) participants: 13/253 (5%) cervicovaginal lavages, 20/322 (6%) seminal plasmas, and 6/85 (7%) rect
10 Moreover lnc-IL7R levels were reduced in lavaged alveolar macrophages and primary human small air
12 at can be recovered from the bronchoalveolar lavage and cerebrospinal fluids of critically ill patien
15 -producing CD4(+) T cells in bronchoalveolar lavage and lung were significantly decreased in mice tha
16 ons in median viral loads in bronchoalveolar lavage and nasal mucosa compared with after the primary
17 ons in median viral loads in bronchoalveolar lavage and nasal mucosa, respectively, as compared with
20 obiomes were characterized in cervicovaginal lavage and penile meatal swab through high-throughput 16
21 d transcriptomic analyses of bronchoalveolar lavage and peripheral blood and proteomic analyses of se
25 utrophil infiltration to the bronchoalveolar lavage and tissue and simultaneously decreases PC relaxa
29 eritonitis" AND "therapeutic irrigation" or "lavage" AND randomized controlled trial and any derivati
30 issue compartment (blood and bronchoalveolar lavage) and temporal proteomic signatures that different
31 ng practices, reluctance for bronchoalveolar lavage, and dependence on a chain of trial-related proce
32 hils were examined in blood, bronchoalveolar lavage, and endobronchial biopsies 48 hours after challe
33 sion (MEE) samples, external ear canal (EEC) lavages, and nasopharynx (NPH) samples from children wit
34 from nasal and throat swabs, bronchoalveolar lavages, and tissues was not reduced with baricitinib.
35 arrow (BM), blood, lung, and bronchoalveolar lavage as well as airway hyperresponsiveness and goblet
38 f human small airway tissue, bronchoalveolar lavage (BAL) and an experimental murine model of COPD.
40 ed numbers of eosinophils in bronchoalveolar lavage (BAL) and increased collagen content in the lung
41 test, airway inflammation in bronchoalveolar lavage (BAL) and lung tissue, and total free IgE in seru
42 r study comparing levels of bronchioalveolar lavage (BAL) and serum HA and the HA immobilizer LYVE-1
44 ived from mRNA isolated from bronchoalveolar lavage (BAL) cells and leukocytes of sarcoidosis patient
45 te the expression of iNOS in bronchoalveolar lavage (BAL) cells and tissue from central and periphera
46 ng, the proportions of blood bronchoalveolar lavage (BAL) classical monocytes peak in adulthood and d
48 etermine the relationship of bronchoalveolar lavage (BAL) cytokine/chemokine expression patterns in c
49 ication and viral genomes in bronchoalveolar-lavage (BAL) fluid and nasal swab specimens were assesse
50 of allergic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were collected from three
51 rmance has been validated on bronchoalveolar lavage (BAL) fluid and serum specimens, but recent evide
52 were isolated from blood and bronchoalveolar lavage (BAL) fluid before and after segmental allergen c
54 s made in favor of obtaining bronchoalveolar lavage (BAL) fluid for lymphocyte cellular analysis, and
55 alue, 4.2), secretory IgA in bronchoalveolar lavage (BAL) fluid from immunized mice, and Yersinia-spe
56 Detection of toxicants in bronchoalveolar-lavage (BAL) fluid from patients with EVALI can provide
59 nd in the sputum, serum, and bronchoalveolar lavage (BAL) fluid of asthma patients and are known to c
61 oteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observational prospecti
62 ysis of lung homogenates and bronchoalveolar lavage (BAL) fluid showed that LPS-induced lung inflamma
65 in whole blood, spleen, and bronchoalveolar lavage (BAL) fluid, but not in the lung interstitium.
68 in rhesus macaque blood and bronchoalveolar lavage (BAL) following mucosal adenovirus (Ad)-SIV recom
70 r airspace infiltration, the bronchoalveolar lavage (BAL) neutrophil proteome is further characterize
72 way immune cells obtained by bronchoalveolar lavage (BAL) of individuals with latent TB infection (LT
73 2C NK cells in the blood and bronchoalveolar lavage (BAL) of lung transplant recipients and stratifie
74 endobronchial brush (EB) and bronchoalveolar lavage (BAL) samples from 39 asthmatic patients and 19 h
75 , we prospectively collected bronchoalveolar lavage (BAL) samples in ICI-treated patients with (n=12)
77 + T cell counts in blood and bronchoalveolar lavage (BAL) samples, it did not reduce the relative ris
79 y and a multiplex assay with bronchoalveolar lavage (BAL) specimens (n = 68) from 52 highly character
81 he diagnosis of PJP requires bronchoalveolar lavage (BAL) specimens or a decision to treat the patien
82 CMV DNA threshold exists in bronchoalveolar lavage (BAL) to differentiate pneumonia from pulmonary s
86 percentage of lymphocytes in bronchoalveolar lavage (BAL), a decreased transfer factor of the lung fo
88 by increased cellularity in bronchoalveolar lavage (BAL), pulmonary transcriptomic profile and histo
96 ptom scores, spirometry, and bronchoalveolar lavage before and after rhinovirus-induced asthma exacer
99 ed on serum antibody levels, bronchoalveolar lavage cell counts, lung histology, lung cytokine levels
101 itment and activation within bronchoalveolar lavage cells of ACE2-high subjects was reciprocally incr
103 from murine reproductive tracts and vaginal lavages collected 24 and 72 h post-administration were s
104 ofiling of cells isolated by bronchoalveolar lavage confirmed that influenza infection inhibits NFkap
105 d CD4+ T cell frequencies in bronchoalveolar lavage correlated strongly with local C-X-C motif chemok
109 in a significant decrease in bronchoalveolar lavage eosinophil counts, lung interleukin-13 and interl
110 ation phenotype indicated by bronchoalveolar lavage eosinophil surface markers, as well as the releas
111 in circulating eosinophils, bronchoalveolar lavage eosinophilia, and eosinophil peroxidase depositio
112 howed an increased number of bronchoalveolar lavage eosinophils and increased expression of IL-13 mRN
114 efore mepolizumab treatment, bronchoalveolar lavage eosinophils had more surface IL-3 and granulocyte
115 was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicting factors of se
116 ics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumigatus-infected c
117 ncluded cellular profiles in bronchoalveolar lavage fluid (BALF) and serum IgG and IgE antibody level
118 the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients with sarcoidosi
119 xpression data from cells in bronchoalveolar lavage fluid (BALF) from COVID-19 patients that were use
120 NA is frequently detected in bronchoalveolar lavage fluid (BALF) from immunocompromised subjects with
122 ectrometry) were measured in bronchoalveolar lavage fluid (BALF) from patients with N-ERD (n = 22), p
125 ed a significant increase in bronchoalveolar lavage fluid (BALF) macrophages and neutrophils and whol
126 otein E (APOE) expression by bronchoalveolar lavage fluid (BALF) macrophages from asthmatic subjects
127 We measured 13 cytokines in bronchoalveolar lavage fluid (BALF) of 88 children with MPP and 26 child
128 n and ferritin levels in the bronchoalveolar lavage fluid (BALF) of participants enrolled in the SubP
129 njury marked by increases in bronchoalveolar lavage fluid (BALF) protein and histochemical evidence o
130 pective multicenter study on bronchoalveolar lavage fluid (BALF) samples obtained from 296 patients w
134 g function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs were collected on
136 gnificantly increased in the bronchoalveolar lavage fluid 48 hours after segmental allergen challenge
137 2/3 and DC5) are expanded in bronchoalveolar lavage fluid 8 h after lipopolysaccharide inhalation.
138 nted the release of IL-33 in bronchoalveolar lavage fluid after Alternaria challenge, suggesting that
141 was evaluated by microscopy; bronchoalveolar lavage fluid and blood were assessed by 10-color flow cy
142 monocytes and neutrophils in bronchoalveolar lavage fluid and increased neutrophils in the lung compa
143 le reproductive tract tissue, cervicovaginal lavage fluid and its intracellular metabolite (TFV dipho
144 r white blood cell counts in bronchoalveolar lavage fluid and less pronounced peribronchiolar inflamm
145 numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inflammatory chemok
146 ly reduced viral load in the bronchoalveolar lavage fluid and lower respiratory tract tissue of vacci
148 and cytokine measurements in bronchoalveolar lavage fluid and lung tissue were followed by in vitro T
151 , we also isolated macrophages from alveolar lavage fluid and used the transcriptome to identify sign
153 vasculitis, and increases in bronchoalveolar lavage fluid cell numbers were detected a lower doses an
156 nophil and T cell numbers in bronchoalveolar lavage fluid compared with those in diluent-treated or c
157 d persistent eosinophilia in bronchoalveolar lavage fluid compared with wild-type and SP-A2 223Q/Q mi
158 ith significant reduction in bronchoalveolar lavage fluid concentration of IL-5, a cytokine associate
159 eumonia demonstrated cytotoxic activity, and lavage fluid contained amyloid molecules, including olig
161 mice had complete absence of bronchoalveolar lavage fluid eosinophilia, accompanied with significant
163 Microbiology was done on bronchoalveolar lavage fluid from all patients and ventilator-associated
164 significantly upregulated in bronchoalveolar lavage fluid from HIV-infected smokers, and increased CH
166 t1 and Rorc Additionally, bronchial alveolar lavage fluid from infected IL-8R2-deficient mice contain
167 d IL-8 rapidly determined in bronchoalveolar lavage fluid from patients randomised to the biomarker-b
168 H complexes were detected in bronchoalveolar lavage fluid from patients with acute respiratory distre
170 e our method to characterize bronchoalveolar lavage fluid from patients with asthma, and find elevate
172 mide species were altered in bronchoalveolar lavage fluid from patients with severe asthma compared w
174 the 27 cytokines analyzed in cervicovaginal lavage fluid from women in this village, the level of in
175 ions of IL-1beta and IL-8 in bronchoalveolar lavage fluid have been validated as effective markers fo
176 rmine whether measurement of bronchoalveolar lavage fluid IL-1beta and IL-8 could effectively and saf
177 eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adoptive transfer of
178 d fewer DEP exposure-induced bronchoalveolar lavage fluid immune cells and proinflammatory cytokines
179 petitive O3 exposure, higher bronchoalveolar lavage fluid inflammatory cells were observed in all mic
181 ECs, in association with low bronchoalveolar lavage fluid mitochondrial DNA and more severe disease.
182 Macrophages can be isolated from alveolar lavage fluid obtained during routine care and used for R
183 nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques (in which CD4 d
185 g ferritin are higher in the lung tissue and lavage fluid of individuals with chronic obstructive pul
186 e frequently detected in the bronchoalveolar lavage fluid of lung transplant patients diagnosed with
187 the total number of cells in bronchoalveolar lavage fluid of mice challenged with house dust mite ext
189 CXCL1, CCL2, and CCL5 in the bronchoalveolar lavage fluid of RSV-infected mice, without increasing vi
191 RSV-specific CD8+ T cells in bronchoalveolar lavage fluid preinfection (subjects with higher levels h
192 hicle-treated rats increased bronchoalveolar lavage fluid protein, albumin, neutrophils, IL-6 and TNF
193 from eyelid skin and peritoneal and uterine lavage fluid provide unprecedented opportunities to diss
194 mbers of Asm(-/-) T cells in bronchoalveolar lavage fluid released lower levels of IL-4 and IL-5, and
195 A3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of the TH17 transcr
196 is of Aspergillus conidia in bronchoalveolar lavage fluid using the combination of transient isotacho
197 ole-body plethysmography and bronchoalveolar lavage fluid was analyzed for cellular composition and c
199 tionally, VEGF levels measured in peritoneal lavage fluid were 300-fold lower compared to PTX-solutio
200 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower levels of 15-epi
201 pups: 1) the 5-HT content in bronchoalveolar lavage fluid, 2) the apneic response to the right atrial
202 nusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen challenge in alle
203 een recovered from cell-free bronchoalveolar lavage fluid, alveolar macrophages, and intrapulmonary l
204 s also induced in the serum, bronchoalveolar lavage fluid, alveolar type II epithelial cells, and alv
205 g cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expression and NF-kappaB
206 counts were performed on the bronchoalveolar lavage fluid, followed by histological analysis of lung
207 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasal polyp tissue w
209 or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflammatory end point
226 itative bacterial culture of bronchoalveolar lavage fluids (BALF) is labor-intensive, and the delay i
227 ught to compare MP types and levels in nasal lavage fluids (NLFs) from controls and patients with chr
229 s and the surfactant proteins A, B, and C in lavage fluids from patients with proteinosis of differen
230 L-33 protein was detected in bronchoalveolar lavage fluids without any exogenous stimuli, and patholo
231 ound (EUS)-guided chemoablation with ethanol lavage followed by infusion of paclitaxel is effective f
232 Methods: ARDS was induced by saline lung lavage followed by injurious mechanical ventilation in 1
233 Lung injury was induced with saline lung lavage followed by injurious ventilation in supine posit
234 y a double-hit consisting of repeated saline lavages followed by 2 hours of injurious ventilation.
237 o perform aerosol-generating bronchoalveolar lavages for galactomannan testing and microscopic and cu
243 dole, and IFN-gamma levels in cervicovaginal lavages from women with either naturally cleared or pers
244 pulmonary aspergillosis with bronchoalveolar lavage galactomannan and cultures performed on admission
246 ys postoperatively was 45/159 (28.3%) in the lavage group and 13/148 (8.8%) in the resection group (r
248 total organ homogenates vs.broncho alveolar lavages implicated tight association of these bacteria w
250 days in the 7 days following bronchoalveolar lavage in the intention-to-treat analysis (p=0.58).
251 the macaques, as well as in bronchoalveolar lavages; in one macaque, we observed prolonged rectal sh
253 lly in five mice ventilated before and after lavage injury, and computationally using a viscoelastic
256 sulted in elevated serum and bronchoalveolar lavage levels of anti-P6-specific IgG and IgA that were
257 OVA-induced increases in bronchoalveolar lavage lymphocytes, eosinophils, IL-13, IL-5, and eotaxi
262 mal RNA sequencing data from bronchoalveolar lavage obtained as part of the COMET-IPF (Correlating Ou
263 l RNA were isolated from 189 bronchoalveolar lavages obtained from 116 patients post lung transplanta
267 erize macrophages from human bronchoalveolar lavage of HIV-infected and -uninfected smokers and nonsm
270 ected AMs were isolated from bronchoalveolar lavage of young (3 mo) and old (18 mo) C57BL/6 mice.
271 by size-exclusion chromatography from nasal lavages of children with mild-to-moderate (n = 8) or sev
274 logical lung injury scores, broncho-alveolar lavage protein levels and cell counts, and IL-6 and IP-1
276 ents can be safely managed with laparoscopic lavage, resulting in reduced mortality and stoma formati
277 nd interleukin-6 pathways in bronchoalveolar lavage samples and repression of natural killer cell- an
281 on pooled nasopharyngeal and bronchoalveolar lavage samples taken from patients who had negative resu
286 ents, respiratory specimens (bronchoalveolar lavage specimens, tracheal aspirates, and sputum samples
287 imental step, but the lowest bronchoalveolar lavage sRAGE levels were obtained at minimal driving pre
291 equate in patients with CF, and that colonic lavage using modified CF bowel preparation is required t
298 coli colony-forming units in bronchoalveolar lavage were reduced in both cell therapy groups, despite
300 ps that underwent EUS-guided pancreatic cyst lavage with either 80% ethanol (control) or normal salin