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1 days in the 7 days following bronchoalveolar lavage.
2 us and total fungus in their bronchoalveolar lavage.
3 mbers of cells by whole-lung bronchoalveolar lavage.
4  patients of whom 159 underwent laparoscopic lavage.
5 and E. coli colony counts in bronchoalveolar lavage.
6 esistances were tested in 10 pigs after lung lavage.
7 ing blood, plasma, urine and bronchoalveolar lavages.
8 s could be confirmed in EVs from human nasal lavages.
9 9%) participants: 13/253 (5%) cervicovaginal lavages, 20/322 (6%) seminal plasmas, and 6/85 (7%) rect
10     Moreover lnc-IL7R levels were reduced in lavaged alveolar macrophages and primary human small air
11          We interrogated the bronchoalveolar lavage and blood viromes during the peritransplant perio
12 at can be recovered from the bronchoalveolar lavage and cerebrospinal fluids of critically ill patien
13 ated after lung injury induced by whole-lung lavage and injurious ventilation in four rabbits.
14  responses in blood, spleen, bronchoalveolar lavage and lung lymph nodes.
15 -producing CD4(+) T cells in bronchoalveolar lavage and lung were significantly decreased in mice tha
16 ons in median viral loads in bronchoalveolar lavage and nasal mucosa compared with after the primary
17 ons in median viral loads in bronchoalveolar lavage and nasal mucosa, respectively, as compared with
18  near-complete protection in bronchoalveolar lavage and nasal swabs after SARS-CoV-2 challenge.
19 es to the offending antigen, bronchoalveolar lavage and pathological features.
20 obiomes were characterized in cervicovaginal lavage and penile meatal swab through high-throughput 16
21 d transcriptomic analyses of bronchoalveolar lavage and peripheral blood and proteomic analyses of se
22 operational taxonomic units, as well as with lavage and peripheral blood neutrophilia.
23 proinflammatory cytokines in both peritoneal lavage and serum.
24      To compare the outcomes of laparoscopic lavage and sigmoid resection in perforated diverticuliti
25 utrophil infiltration to the bronchoalveolar lavage and tissue and simultaneously decreases PC relaxa
26                              Bronchoalveolar lavage and tissues were sampled for mycobacterial persis
27 stress syndrome was induced by repeated lung lavages and injurious mechanical ventilation.
28 her S. maltophilia counts in bronchoalveolar lavages and lung tissue homogenates.
29 eritonitis" AND "therapeutic irrigation" or "lavage" AND randomized controlled trial and any derivati
30 issue compartment (blood and bronchoalveolar lavage) and temporal proteomic signatures that different
31 ng practices, reluctance for bronchoalveolar lavage, and dependence on a chain of trial-related proce
32 hils were examined in blood, bronchoalveolar lavage, and endobronchial biopsies 48 hours after challe
33 sion (MEE) samples, external ear canal (EEC) lavages, and nasopharynx (NPH) samples from children wit
34 from nasal and throat swabs, bronchoalveolar lavages, and tissues was not reduced with baricitinib.
35 arrow (BM), blood, lung, and bronchoalveolar lavage as well as airway hyperresponsiveness and goblet
36 red) ), thus lower sEV-miRNA levels in nasal lavages associated with airway obstruction.
37 was detected in 6/13 (46%) DS cervicovaginal lavages at low levels.
38 f human small airway tissue, bronchoalveolar lavage (BAL) and an experimental murine model of COPD.
39                      TTR for bronchoalveolar lavage (BAL) and endotracheal aspirate (ETA) specimens f
40 ed numbers of eosinophils in bronchoalveolar lavage (BAL) and increased collagen content in the lung
41 test, airway inflammation in bronchoalveolar lavage (BAL) and lung tissue, and total free IgE in seru
42 r study comparing levels of bronchioalveolar lavage (BAL) and serum HA and the HA immobilizer LYVE-1
43             We characterized bronchoalveolar lavage (BAL) cells and fluid (BALF) proteins in recently
44 ived from mRNA isolated from bronchoalveolar lavage (BAL) cells and leukocytes of sarcoidosis patient
45 te the expression of iNOS in bronchoalveolar lavage (BAL) cells and tissue from central and periphera
46 ng, the proportions of blood bronchoalveolar lavage (BAL) classical monocytes peak in adulthood and d
47 ophilia and interleukin-5 in bronchoalveolar lavage (BAL) compared to sham-OVA mice.
48 etermine the relationship of bronchoalveolar lavage (BAL) cytokine/chemokine expression patterns in c
49 ication and viral genomes in bronchoalveolar-lavage (BAL) fluid and nasal swab specimens were assesse
50  of allergic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were collected from three
51 rmance has been validated on bronchoalveolar lavage (BAL) fluid and serum specimens, but recent evide
52 were isolated from blood and bronchoalveolar lavage (BAL) fluid before and after segmental allergen c
53 ryngeal aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.
54 s made in favor of obtaining bronchoalveolar lavage (BAL) fluid for lymphocyte cellular analysis, and
55 alue, 4.2), secretory IgA in bronchoalveolar lavage (BAL) fluid from immunized mice, and Yersinia-spe
56    Detection of toxicants in bronchoalveolar-lavage (BAL) fluid from patients with EVALI can provide
57          Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic patients were re
58                  We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines, mediators, and
59 nd in the sputum, serum, and bronchoalveolar lavage (BAL) fluid of asthma patients and are known to c
60 f viruses collected from the bronchoalveolar lavage (BAL) fluid of the animals.
61 oteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observational prospecti
62 ysis of lung homogenates and bronchoalveolar lavage (BAL) fluid showed that LPS-induced lung inflamma
63        Inflammatory cells in bronchoalveolar lavage (BAL) fluid were assessed by flow cytometry.
64 above-named transport media, bronchoalveolar lavage (BAL) fluid, and sputum.
65  in whole blood, spleen, and bronchoalveolar lavage (BAL) fluid, but not in the lung interstitium.
66 ines KC, IP-10, and IL-33 in bronchoalveolar lavage (BAL) fluid.
67 pha, IL-6, and leukocytes in bronchoalveolar lavage (BAL) fluids.
68  in rhesus macaque blood and bronchoalveolar lavage (BAL) following mucosal adenovirus (Ad)-SIV recom
69       This analysis included bronchoalveolar lavage (BAL) for cell and fluid analysis, and tissue for
70 r airspace infiltration, the bronchoalveolar lavage (BAL) neutrophil proteome is further characterize
71                              Bronchoalveolar lavage (BAL) NK cells were immunophenotyped for NKG2C, m
72 way immune cells obtained by bronchoalveolar lavage (BAL) of individuals with latent TB infection (LT
73 2C NK cells in the blood and bronchoalveolar lavage (BAL) of lung transplant recipients and stratifie
74 endobronchial brush (EB) and bronchoalveolar lavage (BAL) samples from 39 asthmatic patients and 19 h
75 , we prospectively collected bronchoalveolar lavage (BAL) samples in ICI-treated patients with (n=12)
76           Twenty consecutive bronchoalveolar lavage (BAL) samples were plated to standard and additio
77 + T cell counts in blood and bronchoalveolar lavage (BAL) samples, it did not reduce the relative ris
78 ounts and protein content in bronchoalveolar lavage (BAL) samples.
79 y and a multiplex assay with bronchoalveolar lavage (BAL) specimens (n = 68) from 52 highly character
80  adult inpatients submitting bronchoalveolar lavage (BAL) specimens for laboratory analysis.
81 he diagnosis of PJP requires bronchoalveolar lavage (BAL) specimens or a decision to treat the patien
82  CMV DNA threshold exists in bronchoalveolar lavage (BAL) to differentiate pneumonia from pulmonary s
83 d B cells in the spleens and bronchoalveolar lavage (BAL) were also observed.
84 nderwent SAC, and cells from bronchoalveolar lavage (BAL) were collected after 24 hours.
85             Bronchoscopy and bronchoalveolar lavage (BAL) were performed 7 days post-vaccination.
86 percentage of lymphocytes in bronchoalveolar lavage (BAL), a decreased transfer factor of the lung fo
87           Subjects underwent bronchoalveolar lavage (BAL), and peripheral whole blood was collected i
88  by increased cellularity in bronchoalveolar lavage (BAL), pulmonary transcriptomic profile and histo
89 inflammatory cell numbers in bronchoalveolar lavage (BAL).
90 raphy, and bronchoscopy with bronchoalveolar lavage (BAL).
91 ) underwent bronchoscopy and bronchoalveolar lavage (BAL).
92  of lung cells obtained from bronchoalveolar lavages (BAL) than TAC pre-treatment alone.
93  tract specimens (sputum and bronchoalveolar lavage [BAL] fluid).
94 tion end products (sRAGE) in bronchoalveolar lavage(BAL) and plasma were performed.
95                              Bronchoalveolar lavages (BALs) from coinfected mice showed rapid bacteri
96 ptom scores, spirometry, and bronchoalveolar lavage before and after rhinovirus-induced asthma exacer
97                           Bronchial alveolar lavage, blood, and lung samples were analyzed.
98                              Bronchoalveolar lavage CD8 T cells showed significantly less in vitro de
99 ed on serum antibody levels, bronchoalveolar lavage cell counts, lung histology, lung cytokine levels
100                              Bronchoalveolar lavage cell mRNA levels of iNOS or iNOS expression in ce
101 itment and activation within bronchoalveolar lavage cells of ACE2-high subjects was reciprocally incr
102 ed <50 copies/mL had sperm or cervicovaginal lavage collected between Weeks 24 and 48.
103  from murine reproductive tracts and vaginal lavages collected 24 and 72 h post-administration were s
104 ofiling of cells isolated by bronchoalveolar lavage confirmed that influenza infection inhibits NFkap
105 d CD4+ T cell frequencies in bronchoalveolar lavage correlated strongly with local C-X-C motif chemok
106 r testing was performed with bronchoalveolar lavage culture and PCR intraoperatively.
107 th HIV-1 genomes from matched cervicovaginal lavage (CVL) samples and plasma.
108 ing symptoms and quality of life; whole-lung lavage effectively removes excessive surfactant.
109 in a significant decrease in bronchoalveolar lavage eosinophil counts, lung interleukin-13 and interl
110 ation phenotype indicated by bronchoalveolar lavage eosinophil surface markers, as well as the releas
111  in circulating eosinophils, bronchoalveolar lavage eosinophilia, and eosinophil peroxidase depositio
112 howed an increased number of bronchoalveolar lavage eosinophils and increased expression of IL-13 mRN
113 owed complete suppression of bronchoalveolar lavage eosinophils and mucous metaplasia.
114 efore mepolizumab treatment, bronchoalveolar lavage eosinophils had more surface IL-3 and granulocyte
115  was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicting factors of se
116 ics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumigatus-infected c
117 ncluded cellular profiles in bronchoalveolar lavage fluid (BALF) and serum IgG and IgE antibody level
118  the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients with sarcoidosi
119 xpression data from cells in bronchoalveolar lavage fluid (BALF) from COVID-19 patients that were use
120 NA is frequently detected in bronchoalveolar lavage fluid (BALF) from immunocompromised subjects with
121  when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with ARDS.
122 ectrometry) were measured in bronchoalveolar lavage fluid (BALF) from patients with N-ERD (n = 22), p
123 -gradient fractionation from bronchoalveolar lavage fluid (BALF) in vivo.
124                              Bronchoalveolar lavage fluid (BALF) levels of SOCS3 were reduced in asth
125 ed a significant increase in bronchoalveolar lavage fluid (BALF) macrophages and neutrophils and whol
126 otein E (APOE) expression by bronchoalveolar lavage fluid (BALF) macrophages from asthmatic subjects
127  We measured 13 cytokines in bronchoalveolar lavage fluid (BALF) of 88 children with MPP and 26 child
128 n and ferritin levels in the bronchoalveolar lavage fluid (BALF) of participants enrolled in the SubP
129 njury marked by increases in bronchoalveolar lavage fluid (BALF) protein and histochemical evidence o
130 pective multicenter study on bronchoalveolar lavage fluid (BALF) samples obtained from 296 patients w
131                 All archived bronchoalveolar lavage fluid (BALF) samples that had previously tested p
132               Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of inflammation.
133                Rhinoceros bronchial alveolar lavage fluid (BALF) was found to have an inhibitory effe
134 g function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs were collected on
135 d in increased OPN levels in bronchoalveolar lavage fluid (BALF).
136 gnificantly increased in the bronchoalveolar lavage fluid 48 hours after segmental allergen challenge
137 2/3 and DC5) are expanded in bronchoalveolar lavage fluid 8 h after lipopolysaccharide inhalation.
138 nted the release of IL-33 in bronchoalveolar lavage fluid after Alternaria challenge, suggesting that
139 rreactivity to methacholine, bronchoalveolar lavage fluid albumin, and serum IgE levels.
140 rs and examine the impact on bronchoalveolar lavage fluid and blood MP repertoire.
141 was evaluated by microscopy; bronchoalveolar lavage fluid and blood were assessed by 10-color flow cy
142 monocytes and neutrophils in bronchoalveolar lavage fluid and increased neutrophils in the lung compa
143 le reproductive tract tissue, cervicovaginal lavage fluid and its intracellular metabolite (TFV dipho
144 r white blood cell counts in bronchoalveolar lavage fluid and less pronounced peribronchiolar inflamm
145 numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inflammatory chemok
146 ly reduced viral load in the bronchoalveolar lavage fluid and lower respiratory tract tissue of vacci
147 n ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assessed.
148 and cytokine measurements in bronchoalveolar lavage fluid and lung tissue were followed by in vitro T
149 ponses were assessed in both bronchoalveolar lavage fluid and lung tissue.
150                              Bronchoalveolar lavage fluid and matched blood were obtained from asympt
151 , we also isolated macrophages from alveolar lavage fluid and used the transcriptome to identify sign
152             Viral load was measured in nasal lavage fluid at day 3, 6 and 14.
153 vasculitis, and increases in bronchoalveolar lavage fluid cell numbers were detected a lower doses an
154                                        Nasal lavage fluid cells were analyzed for inflammatory gene e
155                  Analysis of bronchoalveolar lavage fluid collected from human patients with P. aerug
156 nophil and T cell numbers in bronchoalveolar lavage fluid compared with those in diluent-treated or c
157 d persistent eosinophilia in bronchoalveolar lavage fluid compared with wild-type and SP-A2 223Q/Q mi
158 ith significant reduction in bronchoalveolar lavage fluid concentration of IL-5, a cytokine associate
159 eumonia demonstrated cytotoxic activity, and lavage fluid contained amyloid molecules, including olig
160                              Bronchoalveolar lavage fluid demonstrates greater than 20% lymphocytes i
161 mice had complete absence of bronchoalveolar lavage fluid eosinophilia, accompanied with significant
162                              Bronchoalveolar lavage fluid from 23 lean, 12 overweight, and 20 obese s
163     Microbiology was done on bronchoalveolar lavage fluid from all patients and ventilator-associated
164 significantly upregulated in bronchoalveolar lavage fluid from HIV-infected smokers, and increased CH
165 vere asthma compared with in bronchoalveolar lavage fluid from individuals without asthma.
166 t1 and Rorc Additionally, bronchial alveolar lavage fluid from infected IL-8R2-deficient mice contain
167 d IL-8 rapidly determined in bronchoalveolar lavage fluid from patients randomised to the biomarker-b
168 H complexes were detected in bronchoalveolar lavage fluid from patients with acute respiratory distre
169 ssion patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.
170 e our method to characterize bronchoalveolar lavage fluid from patients with asthma, and find elevate
171 NA composition of EVs in the bronchoalveolar lavage fluid from patients with IAV-induced ARDS.
172 mide species were altered in bronchoalveolar lavage fluid from patients with severe asthma compared w
173          Endotoxin concentrations in vaginal lavage fluid from SPI dams were significantly higher tha
174  the 27 cytokines analyzed in cervicovaginal lavage fluid from women in this village, the level of in
175 ions of IL-1beta and IL-8 in bronchoalveolar lavage fluid have been validated as effective markers fo
176 rmine whether measurement of bronchoalveolar lavage fluid IL-1beta and IL-8 could effectively and saf
177  eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adoptive transfer of
178 d fewer DEP exposure-induced bronchoalveolar lavage fluid immune cells and proinflammatory cytokines
179 petitive O3 exposure, higher bronchoalveolar lavage fluid inflammatory cells were observed in all mic
180                              Bronchoalveolar lavage fluid LT levels were increased in neonatal and ad
181 ECs, in association with low bronchoalveolar lavage fluid mitochondrial DNA and more severe disease.
182    Macrophages can be isolated from alveolar lavage fluid obtained during routine care and used for R
183 nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques (in which CD4 d
184 iene levels are increased in bronchoalveolar lavage fluid of HIV-infected patients.
185 g ferritin are higher in the lung tissue and lavage fluid of individuals with chronic obstructive pul
186 e frequently detected in the bronchoalveolar lavage fluid of lung transplant patients diagnosed with
187 the total number of cells in bronchoalveolar lavage fluid of mice challenged with house dust mite ext
188 he levels of UDP detected in bronchoalveolar lavage fluid of mice.
189 CXCL1, CCL2, and CCL5 in the bronchoalveolar lavage fluid of RSV-infected mice, without increasing vi
190                  Percent neutrophils in lung lavage fluid post-infection are significantly higher in
191 RSV-specific CD8+ T cells in bronchoalveolar lavage fluid preinfection (subjects with higher levels h
192 hicle-treated rats increased bronchoalveolar lavage fluid protein, albumin, neutrophils, IL-6 and TNF
193  from eyelid skin and peritoneal and uterine lavage fluid provide unprecedented opportunities to diss
194 mbers of Asm(-/-) T cells in bronchoalveolar lavage fluid released lower levels of IL-4 and IL-5, and
195 A3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of the TH17 transcr
196 is of Aspergillus conidia in bronchoalveolar lavage fluid using the combination of transient isotacho
197 ole-body plethysmography and bronchoalveolar lavage fluid was analyzed for cellular composition and c
198                              Bronchoalveolar lavage fluid was evaluated by microscopy; bronchoalveola
199 tionally, VEGF levels measured in peritoneal lavage fluid were 300-fold lower compared to PTX-solutio
200    Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower levels of 15-epi
201 pups: 1) the 5-HT content in bronchoalveolar lavage fluid, 2) the apneic response to the right atrial
202 nusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen challenge in alle
203 een recovered from cell-free bronchoalveolar lavage fluid, alveolar macrophages, and intrapulmonary l
204 s also induced in the serum, bronchoalveolar lavage fluid, alveolar type II epithelial cells, and alv
205 g cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expression and NF-kappaB
206 counts were performed on the bronchoalveolar lavage fluid, followed by histological analysis of lung
207 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasal polyp tissue w
208 of the injury markers in the bronchoalveolar lavage fluid, respectively.
209  or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflammatory end point
210  inflammatory cells into the bronchoalveolar lavage fluid.
211 -1beta accumulation into the bronchoalveolar lavage fluid.
212 mucosa and cytokine levels in cervicovaginal lavage fluid.
213 ion, iron overload, and secretion of IL-6 in lavage fluid.
214 carrier proteins in cells or bronchoalveolar lavage fluid.
215 lony forming units per mL of bronchoalveolar lavage fluid.
216 nd inflammatory cells in the bronchoalveolar lavage fluid.
217 -associated cytokines in the bronchoalveolar lavage fluid.
218 sinophils were enumerated in bronchoalveolar lavage fluid.
219 reducing LPA 18:2 content in bronchoalveolar lavage fluid.
220 nia were observed in blood, dermis, and lung lavage fluid.
221 t protein A concentration in bronchoalveolar lavage fluid.
222 ion using RNA extracted from bronchoalveolar lavage fluid.
223 ng epithelium as detected in bronchoalveolar lavage fluid.
224  and IL-13, but not IL-5, in bronchoalveolar lavage fluid.
225 orrelated with IFN levels in bronchoalveolar lavage fluid.
226 itative bacterial culture of bronchoalveolar lavage fluids (BALF) is labor-intensive, and the delay i
227 ught to compare MP types and levels in nasal lavage fluids (NLFs) from controls and patients with chr
228                    Ethmoid tissues and nasal lavage fluids (NLFs) were obtained from control patients
229 s and the surfactant proteins A, B, and C in lavage fluids from patients with proteinosis of differen
230 L-33 protein was detected in bronchoalveolar lavage fluids without any exogenous stimuli, and patholo
231 ound (EUS)-guided chemoablation with ethanol lavage followed by infusion of paclitaxel is effective f
232     Methods: ARDS was induced by saline lung lavage followed by injurious mechanical ventilation in 1
233     Lung injury was induced with saline lung lavage followed by injurious ventilation in supine posit
234 y a double-hit consisting of repeated saline lavages followed by 2 hours of injurious ventilation.
235                                        Nasal lavage for flow cytometry and nasal swabs for viral PCR
236                     Positive bronchoalveolar lavage for GM or Aspergillus cultures was reported for 1
237 o perform aerosol-generating bronchoalveolar lavages for galactomannan testing and microscopic and cu
238       AMs were obtained from bronchoalveolar lavage from healthy donors or patients with COPD and cha
239 properties and is reduced in bronchoalveolar lavage from patients with IPF.
240  IL-1beta levels were detected in peritoneal lavages from Cd44(-/-) mice (p < 0.01).
241 of hydrosalpinx, we compared the proteome of lavages from disease vs. healthy fallopian tubes.
242 nd neutrophil populations in bronchoalveolar lavages from mice with asthma.
243 dole, and IFN-gamma levels in cervicovaginal lavages from women with either naturally cleared or pers
244 pulmonary aspergillosis with bronchoalveolar lavage galactomannan and cultures performed on admission
245 ic plaques with reduction in bronchoalveolar lavage galactomannan values (7.48-2.15 ng/mL).
246 ys postoperatively was 45/159 (28.3%) in the lavage group and 13/148 (8.8%) in the resection group (r
247 monocyte recruitment and resulted in reduced lavage IL-1beta levels in the same model.
248  total organ homogenates vs.broncho alveolar lavages implicated tight association of these bacteria w
249 ntly cultured from blood and bronchoalveolar lavage in humans and lungs in animals.
250 days in the 7 days following bronchoalveolar lavage in the intention-to-treat analysis (p=0.58).
251  the macaques, as well as in bronchoalveolar lavages; in one macaque, we observed prolonged rectal sh
252                             ECP in the nasal lavage increased after the NAC-P in the group A (P = .00
253 lly in five mice ventilated before and after lavage injury, and computationally using a viscoelastic
254            Bronchoscopy with bronchoalveolar lavage is very useful in its diagnosing.
255                       Blood, bronchoalveolar lavage, large proximal, and small distal airway brushing
256 sulted in elevated serum and bronchoalveolar lavage levels of anti-P6-specific IgG and IgA that were
257     OVA-induced increases in bronchoalveolar lavage lymphocytes, eosinophils, IL-13, IL-5, and eotaxi
258 ripheral blood, and elevated bronchoalveolar lavage lymphocytes.
259                              Bronchoalveolar lavage macrophages were stimulated in vitro, and iNOS ex
260 oups, despite a reduction in bronchoalveolar lavage neutrophils.
261                                        Nasal lavage (NL) samples from 20 subjects who were exposed to
262 mal RNA sequencing data from bronchoalveolar lavage obtained as part of the COMET-IPF (Correlating Ou
263 l RNA were isolated from 189 bronchoalveolar lavages obtained from 116 patients post lung transplanta
264 asts (LFs) were derived from bronchoalveolar lavage of BOS patients.
265 rescent cells present in the bronchoalveolar lavage of cigarette smokers.
266 rway macrophages obtained by bronchoalveolar lavage of HIV-1-infected individuals.
267 erize macrophages from human bronchoalveolar lavage of HIV-infected and -uninfected smokers and nonsm
268 ture and was also present in bronchoalveolar lavage of lung transplantation patients.
269 eins were recovered during presurgical nasal lavage of patients at a sinus clinic.
270 ected AMs were isolated from bronchoalveolar lavage of young (3 mo) and old (18 mo) C57BL/6 mice.
271  by size-exclusion chromatography from nasal lavages of children with mild-to-moderate (n = 8) or sev
272 ), systemic (p = 0.019), and bronchoalveolar lavage (p = 0.011) quantitative bacterial culture.
273                        After bronchoalveolar lavages, piglets developed respiratory distress syndrome
274 logical lung injury scores, broncho-alveolar lavage protein levels and cell counts, and IL-6 and IP-1
275 rted by normal histology and bronchoalveolar lavage protein levels.
276 ents can be safely managed with laparoscopic lavage, resulting in reduced mortality and stoma formati
277 nd interleukin-6 pathways in bronchoalveolar lavage samples and repression of natural killer cell- an
278                                       Penile lavage samples collected from the coronal sulcus at base
279 wo redondovirus genomes from bronchoalveolar lavage samples from human lung donors.
280          Although sputum and bronchoalveolar lavage samples provide the highest diagnostic sensitivit
281 on pooled nasopharyngeal and bronchoalveolar lavage samples taken from patients who had negative resu
282 tely 1000 kDa was found in all patient nasal lavage samples.
283                              Bronchoalveolar lavage sampling may provide useful diagnostic informatio
284                               Cervicovaginal lavage sequencing (n = 109) resulted in a low-diversity
285                   Plasma and bronchoalveolar lavage soluble receptor for advanced glycation end-produ
286 ents, respiratory specimens (bronchoalveolar lavage specimens, tracheal aspirates, and sputum samples
287 imental step, but the lowest bronchoalveolar lavage sRAGE levels were obtained at minimal driving pre
288                All animals received a saline-lavage surfactant depletion lung injury model.
289  and reduced virus titres in bronchoalveolar lavages twelve hours after the first dose.
290                                 Laparoscopic lavage used in the management of Hinchey grade III diver
291 equate in patients with CF, and that colonic lavage using modified CF bowel preparation is required t
292                              Bronchoalveolar lavage was associated with a small and transient increas
293                                      A nasal lavage was collected before and after the NACs to measur
294                              Bronchoalveolar lavage was done in all patients, with concentrations of
295              Inflammation in bronchoalveolar lavage was increased in old versus young baboons.
296                                        Nasal lavage was performed before exposure (Day 0) and on Days
297                     Bronchoalveolar wash and lavage were obtained 24 hours after exposure.
298 coli colony-forming units in bronchoalveolar lavage were reduced in both cell therapy groups, despite
299                             Bronchioalveolar lavage, whole-lung cellular isolation, and gene expressi
300 ps that underwent EUS-guided pancreatic cyst lavage with either 80% ethanol (control) or normal salin

 
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