ライフサイエンスコーパス: lavage fluid

1 t protein A concentration in bronchoalveolar lavage fluid.

2 ng epithelium as detected in bronchoalveolar lavage fluid.

3 and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

4 hemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

5 increase in the levels of CCL4 in peritoneal lavage fluid.

6 s, and lower cytokine levels in the alveolar lavage fluid.

7 and KLK14, were assessed in bronchoalveolar lavage fluid.

8 tion and after culture with broncho-alveolar lavage fluid.

9 on of TNF-alpha and MIP-2 in bronchoalveolar lavage fluid.

10 cylcarnitines are detectable in normal human lavage fluid.

11 -associated cytokines in the bronchoalveolar lavage fluid.

12 crophages and eosinophils in bronchoalveolar lavage fluid.

13 ers of inflammatory cells in bronchoalveolar lavage fluid.

14 racts, nasal secretions, and bronchoalveolar lavage fluid.

15 ells and readily detected in bronchoalveolar lavage fluid.

16 entage of eosinophils in the bronchoalveolar lavage fluid.

17 triene (cysLT) levels were measured in nasal lavage fluid.

18 ssociated with increased IL-17 production in lavage fluid.

19 nd cytokine concentration in bronchoalveolar lavage fluid.

20 nfiltration and interleukin-5 (IL-5) in lung lavage fluid.

21 rophages were detected in bronchial alveolar lavage fluid.

22 orrelated with IFN levels in bronchoalveolar lavage fluid.

23 inflammatory cells into the bronchoalveolar lavage fluid.

24 -1beta accumulation into the bronchoalveolar lavage fluid.

25 mucosa and cytokine levels in cervicovaginal lavage fluid.

26 ion, iron overload, and secretion of IL-6 in lavage fluid.

27 carrier proteins in cells or bronchoalveolar lavage fluid.

28 lony forming units per mL of bronchoalveolar lavage fluid.

29 nd inflammatory cells in the bronchoalveolar lavage fluid.

30 sinophils were enumerated in bronchoalveolar lavage fluid.

31 reducing LPA 18:2 content in bronchoalveolar lavage fluid.

32 nia were observed in blood, dermis, and lung lavage fluid.

33 ion using RNA extracted from bronchoalveolar lavage fluid.

34 imilar results were found in bronchoalveolar lavage fluids.

35 3 measured by ELISA in their bronchoalveolar lavage fluids.

36 pups: 1) the 5-HT content in bronchoalveolar lavage fluid, 2) the apneic response to the right atrial

37 gher bacterial burden in the bronchoalveolar lavage fluid 3 h following infection.

38 gnificantly increased in the bronchoalveolar lavage fluid 48 hours after segmental allergen challenge

39 ns (5 cerebrospinal fluid, 7 bronchoalveolar lavage fluid, 5 plasma, 2 serum, and 1 nasopharyngeal as

40 2/3 and DC5) are expanded in bronchoalveolar lavage fluid 8 h after lipopolysaccharide inhalation.

41 We found that relative bronchioalveolar lavage fluid adenosine levels are progressively elevated

42 nted the release of IL-33 in bronchoalveolar lavage fluid after Alternaria challenge, suggesting that

43 nusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen challenge in alle

44 rreactivity to methacholine, bronchoalveolar lavage fluid albumin, and serum IgE levels.

45 een recovered from cell-free bronchoalveolar lavage fluid, alveolar macrophages, and intrapulmonary l

46 s also induced in the serum, bronchoalveolar lavage fluid, alveolar type II epithelial cells, and alv

47 ammatory cytokines and eosinophil numbers in lavage fluid and augmented the histopathologic evidence

48 rs and examine the impact on bronchoalveolar lavage fluid and blood MP repertoire.

49 was evaluated by microscopy; bronchoalveolar lavage fluid and blood were assessed by 10-color flow cy

50 neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronchiolar inflammat

51 Th2 cytokines were found in bronchoalveolar lavage fluid and draining lymph node cells of Nur77-KO m

52 nt inflammation in blood and bronchoalveolar lavage fluid and exacerbations despite high systemic cor

53 monocytes and neutrophils in bronchoalveolar lavage fluid and increased neutrophils in the lung compa

54 DM, the levels of PGP in the bronchoalveolar lavage fluid and infiltration of neutrophils into the lu

55 le reproductive tract tissue, cervicovaginal lavage fluid and its intracellular metabolite (TFV dipho

56 r white blood cell counts in bronchoalveolar lavage fluid and less pronounced peribronchiolar inflamm

57 numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inflammatory chemok

58 ly reduced viral load in the bronchoalveolar lavage fluid and lower respiratory tract tissue of vacci

59 n ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assessed.

60 and cytokine measurements in bronchoalveolar lavage fluid and lung tissue were followed by in vitro T

61 biochemical measurements in bronchoalveolar lavage fluid and lung tissue.

62 ponses were assessed in both bronchoalveolar lavage fluid and lung tissue.

63 monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR in mice.

64 Bronchoalveolar lavage fluid and matched blood were obtained from asympt

65 NETs were also measured in bronchoalveolar lavage fluid and plasma from lung transplant patients wi

66 d in BOS versus normal lung tissues and both lavage fluid and plasma HA concentrations were increased

67 to extracellular fluid (eg, bronchoalveolar lavage fluid and plasma).

68 mmatory cell accumulation in bronchoalveolar lavage fluid and proinflammatory cytokines levels in lun

69 IL-8 levels were measured in nasal lavage fluid and serum on randomization, day 8, and day

70 e presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can be found in alveo

71 rophilic inflammation in the bronchoalveolar lavage fluid and Th1, Th2, and Th17 inflammation in the

72 , we also isolated macrophages from alveolar lavage fluid and used the transcriptome to identify sign

73 s the major MMP found in the bronchoalveolar lavage fluids and bronchial biopsies from patients with

74 e detected in nasal wash and bronchoalveolar lavage fluids and in sera from mice intranasally inocula

75 ly the wild-type virus was found in tracheal lavage fluids and urine.

76 morphonuclear recruitment in bronchoalveolar lavage fluid), and lethality were evaluated in a pneumon

77 trophil predominance in the bronchioalveolar lavage fluid, and enhanced airway mucus production.

78 ll counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of macrophages.

79 C in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased significantly in

80 mmation, cytokine levels in broncho-alveolar lavage fluid, and mucus production were determined.

81 and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collected for assessing

82 No changes in bronchoalveolar lavage fluid angiotensin-converting enzyme 2 activity we

83 creased extracellular DNA in bronchoalveolar lavage fluid as well as in lung tissue, confirming the p

84 tissues, including lung and bronchoalveolar lavage fluid, as measured by H2-Db NP366 and PA224 tetra

85 Viral load was measured in nasal lavage fluid at day 3, 6 and 14.

86 ecific memory CD8 T cells in bronchoalveolar lavage fluid (BAL), lungs, and spleen.

87 njury was evaluated by using bronchoalveolar lavage fluid (BALF) analysis and pathology.

88 cles (EVs) are detectable in bronchoalveolar lavage fluid (BALF) and culture medium of lung epithelia

89 Lung tissue, bronchoalveolar lavage fluid (BALF) and draining lymph node cells were a

90 was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicting factors of se

91 treptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values were determined.

92 ics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumigatus-infected c

93 metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifies the degree of

94 ncluded cellular profiles in bronchoalveolar lavage fluid (BALF) and serum IgG and IgE antibody level

95 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophils and inflammat

96 s were assessed in lungs and bronchoalveolar lavage fluid (BALF) by multiparametric flow cytometry.

97 Bacterial burdens, bronchoalveolar lavage fluid (BALF) cell counts, cell types, and cytokin

98 characterized by increased broncho-alveolar lavage fluid (BALF) cells and cytokines (IL-6 and TNF-al

99 ity decreased by 80% in COPD bronchoalveolar lavage fluid (BALF) due to serine protease cleavage, pri

100 the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients with sarcoidosi

101 We studied bronchoalveolar lavage fluid (BALF) from 36 patients with ARDS (20 survi

102 ng of serum, lung tissue and bronchoalveolar lavage fluid (BALF) from a non-lethal mouse model with i

103 of two biomarkers present in bronchoalveolar lavage fluid (BALF) from chlorine gas exposed mice.

104 xpression data from cells in bronchoalveolar lavage fluid (BALF) from COVID-19 patients that were use

105 NA is frequently detected in bronchoalveolar lavage fluid (BALF) from immunocompromised subjects with

106 on neutrophils in blood and bronchoalveolar lavage fluid (BALF) from mechanically ventilated patient

107 when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with ARDS.

108 ectrometry) were measured in bronchoalveolar lavage fluid (BALF) from patients with N-ERD (n = 22), p

109 -gradient fractionation from bronchoalveolar lavage fluid (BALF) in vivo.

110 (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV replication in the pu

111 Bronchoalveolar lavage fluid (BALF) levels of SOCS3 were reduced in asth

112 ed a significant increase in bronchoalveolar lavage fluid (BALF) macrophages and neutrophils and whol

113 otein E (APOE) expression by bronchoalveolar lavage fluid (BALF) macrophages from asthmatic subjects

114 We measured 13 cytokines in bronchoalveolar lavage fluid (BALF) of 88 children with MPP and 26 child

115 CD4(+) T cells in the bronchoalveolar lavage fluid (BALF) of Af5517-aspirated mice displayed d

116 n and ferritin levels in the bronchoalveolar lavage fluid (BALF) of participants enrolled in the SubP

117 njury marked by increases in bronchoalveolar lavage fluid (BALF) protein and histochemical evidence o

118 ed controls, ozone increased bronchoalveolar lavage fluid (BALF) protein, a marker of lung permeabili

119 pective multicenter study on bronchoalveolar lavage fluid (BALF) samples obtained from 296 patients w

120 All archived bronchoalveolar lavage fluid (BALF) samples that had previously tested p

121 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of inflammation.

122 Bronchoalveolar lavage fluid (BALF) was analysed for total protein, lact

123 Rhinoceros bronchial alveolar lavage fluid (BALF) was found to have an inhibitory effe

124 arallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by approximately 3-fo

125 of Th2 and Th17 cytokines in bronchoalveolar lavage fluid (BALF), accompanied by an increment in tran

126 Bronchoalveolar lavage fluid (BALF), airway inflammation and hyperrespon

127 r 33 d after exposure, SpO2, bronchoalveolar lavage fluid (BALF), and histologic analyses were perfor

128 mber of cells and macrophages in bronchiolar lavage fluid (BALF), as well infiltrating inflammatory c

129 ice, with fewer cells in Wsh bronchoalveolar lavage fluid (BALF), despite similar levels of cytokines

130 g function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs were collected on

131 d in increased OPN levels in bronchoalveolar lavage fluid (BALF).

132 ntration of total protein in bronchoalveolar lavage fluids (BALF) from patients with sepsis-related a

133 activation, was detected in bronchoalveolar lavage fluids (BALF) in a macrophage- and neutrophil-dep

134 itative bacterial culture of bronchoalveolar lavage fluids (BALF) is labor-intensive, and the delay i

135 ic response were assessed in bronchoalveolar lavage fluids (BALFs) after allergen challenge.

136 spiratory distress syndrome broncho-alveolar lavage fluid by 60%, whereas serum amyloid P replenishme

137 eased lung injury scores and bronchoalveolar lavage fluid cell numbers in control but not TREK-1-defi

138 vasculitis, and increases in bronchoalveolar lavage fluid cell numbers were detected a lower doses an

139 rease in lung injury scores, bronchoalveolar lavage fluid cell numbers, and cellular apoptosis and a

140 Nasal lavage fluid cells were analyzed for inflammatory gene e

141 munosuppressive functions of bronchoalveolar lavage fluid cells, inhibited bone marrow cell transendo

142 evere asthma whole blood and bronchoalveolar lavage fluid cells.

143 endobronchial brushings and bronchoalveolar lavage fluid collected from 39 asthmatic patients and 19

144 Analysis of bronchoalveolar lavage fluid collected from human patients with P. aerug

145 nophil and T cell numbers in bronchoalveolar lavage fluid compared with those in diluent-treated or c

146 d persistent eosinophilia in bronchoalveolar lavage fluid compared with wild-type and SP-A2 223Q/Q mi

147 F production in the lung and bronchoalveolar lavage fluid compared with wild-type mice, without chang

148 inflammatory cytokines in perfusate and lung lavage fluid, compared to control.

149 ith significant reduction in bronchoalveolar lavage fluid concentration of IL-5, a cytokine associate

150 eumonia demonstrated cytotoxic activity, and lavage fluid contained amyloid molecules, including olig

151 Outcome measures included: bronchoalveolar lavage fluid cytology to assess airway eosinophilia, pul

152 kines interleukin-5 (IL-5) and IL-13 in lung lavage fluid, decreased regulatory T cell-associated FOX

153 g cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expression and NF-kappaB

154 Bronchoalveolar lavage fluid demonstrates greater than 20% lymphocytes i

155 use, human CD49d(+) PMNs isolated from nasal lavage fluid during a viral respiratory tract infection

156 d dramatically increased in broncho-alveolar lavage fluid during acute respiratory distress syndrome.

157 frequency was significantly higher in nasal lavage fluid during acute respiratory symptoms in all su

158 n important mediator in resolving tissue and lavage fluid eosinophilia in allergic mouse models.

159 mice had complete absence of bronchoalveolar lavage fluid eosinophilia, accompanied with significant

160 ic fibrosis underwent annual bronchoalveolar lavage fluid examination, and chest computed tomography.

161 Our findings validate bronchoalveolar lavage fluid exosomal shuttle RNA as a source for unders

162 counts were performed on the bronchoalveolar lavage fluid, followed by histological analysis of lung

163 mucosal IgA was detected in bronchioalveolar lavage fluid for up to 6 weeks.

164 Mutations originally only detected in lavage fluid fractions were later confirmed to be presen

165 Bronchoalveolar lavage fluid from 23 lean, 12 overweight, and 20 obese s

166 counts, and total protein in bronchoalveolar lavage fluid from acute respiratory distress syndrome 1.

167 Bronchoalveolar lavage fluid from Ad-MD-2s mice transferred into lungs o

168 Microbiology was done on bronchoalveolar lavage fluid from all patients and ventilator-associated

169 CG and NE in bronchoalveolar lavage fluid from CF patients both contributed to C5aR c

170 significantly upregulated in bronchoalveolar lavage fluid from HIV-infected smokers, and increased CH

171 In bronchoalveolar lavage fluid from human lung transplant recipients, NETs

172 In bronchoalveolar lavage fluid from humans with ARDS, gut-specific bacteri

173 vere asthma compared with in bronchoalveolar lavage fluid from individuals without asthma.

174 t1 and Rorc Additionally, bronchial alveolar lavage fluid from infected IL-8R2-deficient mice contain

175 inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kinase beta transact

176 d IL-8 rapidly determined in bronchoalveolar lavage fluid from patients randomised to the biomarker-b

177 H complexes were detected in bronchoalveolar lavage fluid from patients with acute respiratory distre

178 icant elevation of IL-17A in bronchoalveolar lavage fluid from patients with ARDS, and rIL-17A direct

179 ssion patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

180 e our method to characterize bronchoalveolar lavage fluid from patients with asthma, and find elevate

181 were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asthmatic patients,

182 NA composition of EVs in the bronchoalveolar lavage fluid from patients with IAV-induced ARDS.

183 ophilia were measured in the bronchoalveolar lavage fluid from patients with mild asthma 48 hours aft

184 mide species were altered in bronchoalveolar lavage fluid from patients with severe asthma compared w

185 s elevated in both serum and bronchoalveolar lavage fluid from Rasgrp1-deficient mice; 4) GM-CSF-spec

186 Endotoxin concentrations in vaginal lavage fluid from SPI dams were significantly higher tha

187 Additionally, bronchoalveolar lavage fluid from this group of hepSTAT3(-/-) mice allow

188 the 27 cytokines analyzed in cervicovaginal lavage fluid from women in this village, the level of in

189 tokines were compared in the bronchoalveolar lavage fluid from WT and SP-D(-/-) mice after C. neoform

190 veolar epithelial cells with bronchoalveolar lavage fluids from ARDS patients drove betaENaC internal

191 Peritoneal lavage fluids from CREB-inhibited tumor-bearing mice sho

192 Bronchoalveolar lavage fluids from ozone-treated rats reproduced this ef

193 s and the surfactant proteins A, B, and C in lavage fluids from patients with proteinosis of differen

194 as non-ventilated controls' broncho-alveolar lavage fluid had no effect on fibrocyte differentiation.

195 ions of IL-1beta and IL-8 in bronchoalveolar lavage fluid have been validated as effective markers fo

196 y elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tissue parasite bur

197 lammatory mediator levels in bronchoalveolar lavage fluid, ii) lung parenchymal leukocyte counts and

198 rmine whether measurement of bronchoalveolar lavage fluid IL-1beta and IL-8 could effectively and saf

199 eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adoptive transfer of

200 but resulted in a greater decrease in nasal lavage fluid IL-8 levels by day 15 (P = .03).

201 d fewer DEP exposure-induced bronchoalveolar lavage fluid immune cells and proinflammatory cytokines

202 uction was attenuated in the bronchoalveolar lavage fluid in all factor-deficient mice compared with

203 solute neutrophil numbers in bronchoalveolar lavage fluid in GSTM1+ but not GSTM1null asthmatics.

204 onverting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elderly when compared

205 terleukin-1beta levels obtained from vaginal lavage fluid increased by day 3 onward.

206 petitive O3 exposure, higher bronchoalveolar lavage fluid inflammatory cells were observed in all mic

207 spiratory distress syndrome broncho-alveolar lavage fluid inhibited by 71% (55-94) fibrocyte differen

208 delayed (6-8 hr) increase in bronchoalveolar lavage fluid interleukin-6 concentration (p < 0.001) and

209 as associated with increased bronchoalveolar lavage fluid levels of IL-10 and TGF-beta and decreased

210 elial cell proliferation and bronchoalveolar lavage fluid levels of keratinocyte growth factor were o

211 Bronchoalveolar lavage fluid LT levels were increased in neonatal and ad

212 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasal polyp tissue w

213 ells were detected in blood, bronchoalveolar lavage fluid, lungs, spleen, and brain, demonstrating th

214 he study day, and plasma and bronchoalveolar lavage fluid markers of inflammation were measured.

215 ECs, in association with low bronchoalveolar lavage fluid mitochondrial DNA and more severe disease.

216 on, including cell counts in bronchoalveolar lavage fluid, mucin production, ASM mass, and subepithel

217 tracheal aspirates (n = 71), bronchoalveolar lavage fluid (n = 152), pleural fluid (n = 76), cerebral

218 We analysed cytokine levels in nasal lavage fluid (NLF) in 59 subjects (46 with asthma) with

219 ught to compare MP types and levels in nasal lavage fluids (NLFs) from controls and patients with chr

220 Ethmoid tissues and nasal lavage fluids (NLFs) were obtained from control patients

221 Macrophages can be isolated from alveolar lavage fluid obtained during routine care and used for R

222 Similarly, bronchoalveolar lavage fluid obtained from human volunteers exposed to O

223 control lung tissue, (ii) in bronchoalveolar lavage fluid obtained from non-severe and severe asthmat

224 ng could detect somatic mutations in uterine lavage fluid obtained from women undergoing hysteroscopy

225 kines, and growth factors in bronchoalveolar lavage fluid of 20 stable patients, 20 patients sufferin

226 rnithine) were isolated from bronchoalveolar lavage fluid of a patient with idiopathic pulmonary fibr

227 nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques (in which CD4 d

228 ignificantly elevated in the bronchoalveolar lavage fluid of all mice infected with C. neoformans Del

229 evels were also increased in bronchoalveolar lavage fluid of allergic asthmatic patients after segmen

230 tion driver mutations were identified in the lavage fluid of approximately half of the women without

231 OCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria-infected mice,

232 ned that cells collected from the peritoneal lavage fluid of E. faecalis-infected mice showed reduced

233 iene levels are increased in bronchoalveolar lavage fluid of HIV-infected patients.

234 found ILC3-like cells in the bronchoalveolar lavage fluid of individuals with asthma, we suggest that

235 g ferritin are higher in the lung tissue and lavage fluid of individuals with chronic obstructive pul

236 ines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

237 e frequently detected in the bronchoalveolar lavage fluid of lung transplant patients diagnosed with

238 MUC1-ED were elevated in the bronchoalveolar lavage fluid of mechanically ventilated patients with P.

239 mined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum of human donors

240 the total number of cells in bronchoalveolar lavage fluid of mice challenged with house dust mite ext

241 ophils, and cytokines in the bronchoalveolar lavage fluid of mice than its mutant counterpart 7 days

242 roteins contained within the bronchoalveolar lavage fluid of mice that are cleaved and/or processed b

243 he levels of UDP detected in bronchoalveolar lavage fluid of mice.

244 CXCL1, CCL2, and CCL5 in the bronchoalveolar lavage fluid of RSV-infected mice, without increasing vi

245 ophils were increased in the bronchoalveolar lavage fluid of wild-type mice.

246 he levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

247 centrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

248 onuclear cell recruitment in bronchoalveolar lavage fluid (p<0.05 for both).

249 Percent neutrophils in lung lavage fluid post-infection are significantly higher in

250 RSV-specific CD8+ T cells in bronchoalveolar lavage fluid preinfection (subjects with higher levels h

251 hicle-treated rats increased bronchoalveolar lavage fluid protein, albumin, neutrophils, IL-6 and TNF

252 measured by respiratory system mechanics and lavage fluid protein.

253 from eyelid skin and peritoneal and uterine lavage fluid provide unprecedented opportunities to diss

254 mbers of Asm(-/-) T cells in bronchoalveolar lavage fluid released lower levels of IL-4 and IL-5, and

255 say is validated for testing bronchoalveolar lavage fluids, replacing the requirement for culture and

256 of the injury markers in the bronchoalveolar lavage fluid, respectively.

257 spiratory distress syndrome broncho-alveolar lavage fluid restored their full inhibitory effect.

258 Cytologic examination of bronchial alveolar lavage fluid revealed a high proportion of lipid-laden m

259 virulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosis factor alpha (

260 onventional culture by using bronchoalveolar lavage fluid samples from patients with underlying respi

261 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48 hours of acute r

262 Endobronchial biopsies and bronchoalveolar lavage fluid samples were collected from 13 GSTM1+ and 1

263 Plasma and broncho-alveolar lavage fluid serum amyloid P contents were determined by

264 or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflammatory end point

265 y attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mice challenged wit

266 nt, was significantly increased in LCAD(-/-) lavage fluid, suggesting increased epithelial permeabili

267 A3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of the TH17 transcr

268 a had increased HA levels in bronchoalveolar lavage fluid that correlated with pulmonary function and

269 tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate functions normally a

270 intra-alveolar coagulation (bronchoalveolar lavage fluid thrombin-antithrombin complex levels) and P

271 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA titers and viru

272 .03) and a minor increase in bronchoalveolar lavage fluid tumor necrosis factor-alpha were observed (

273 neutrocytosis and increased bronchoalveolar lavage fluid tumor necrosis factor-alpha, interleukin-6,

274 is of Aspergillus conidia in bronchoalveolar lavage fluid using the combination of transient isotacho

275 Ventilated controls' broncho-alveolar lavage fluid was a less potent inhibitor (51% [23-66%] o

276 ole-body plethysmography and bronchoalveolar lavage fluid was analyzed for cellular composition and c

277 alyzed for immune cell subsets, and alveolar lavage fluid was analyzed for ILC2-derived cytokines.

278 Bronchoalveolar lavage fluid was analyzed for inflammatory cells, and bl

279 Streptococcus pneumoniae) in bronchoalveolar lavage fluid was associated with clinically significant

280 Bronchoalveolar lavage fluid was collected from 23 steroid-free nonsmoki

281 Bronchoalveolar lavage fluid was collected from patients with severe ast

282 Bronchoalveolar lavage fluid was evaluated by microscopy; bronchoalveola

283 Mouse bronchoalveolar lavage fluid was harvested for cell counts and TH2 cytok

284 onor; however, pretransplant bronchoalveolar lavage fluid was only available from the donor for patie

285 The lavage fluid was separated into cellular and acellular f

286 g, the median log10 HIV copies/milliliter of lavage fluid was significantly lower in men with ART-ind

287 tion of CXCL12 in plasma and bronchoalveolar lavage fluids was quantified by ELISA.

288 tionally, VEGF levels measured in peritoneal lavage fluid were 300-fold lower compared to PTX-solutio

289 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower levels of 15-epi

290 examination, eosinophils in bronchoalveolar lavage fluid were counted.

291 nts in homogenized lungs and bronchoalveolar lavage fluid were decreased after cranberry proanthocyan

292 iquots of blood, sputum, and bronchoalveolar lavage fluid were obtained from asthma subjects for medi

293 21, levels of neutrophils and macrophages in lavage fluid were reduced by 49% and increased by 287%,

294 crobial burdens in the organs and peritoneal lavage fluid were similar between mono- and coinfected a

295 differential cell counts in bronchoalveolar lavage fluid were similar between the Syk(flox/flox) and

296 s adenosine levels found in bronchioalveolar lavage fluid, were determined in mouse models of resolva

297 umbers of eosinophils in the bronchoalveolar lavage fluid, while simultaneously decreasing the percen

298 Cytologic examination of bronchial alveolar lavage fluid with oil red O staining is a useful diagnos

299 cytologic examination of bronchial alveolar lavage fluid with oil red O staining.

300 L-33 protein was detected in bronchoalveolar lavage fluids without any exogenous stimuli, and patholo