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1 , and breastfed infants are also at risk for lead poisoning.
2 e in need of emergency chelation therapy for lead poisoning.
3 s either familial or can be acquired through lead poisoning.
4 een addressed to the prevention of childhood lead poisoning.
5 ilinogen synthase (PBGS) is a main target in lead poisoning.
6 ith differential susceptibility of humans to lead poisoning.
7 ron is routinely prescribed in children with lead poisoning.
8 r appropriate flushing procedures to prevent lead poisoning.
9 d for testing the environment and preventing lead poisoning.
10 oung children are particularly vulnerable to lead poisoning.
11 ly to have >/= 1 child < 5 years of age with lead poisoning (68% vs. 50%, p = 0.17) or death followin
13 subclinical disease; the fact that childhood lead poisoning affected mostly families that were politi
14 sociated with reduced PBGS activity, such as lead poisoning and ALAD porphyria, the latter of which i
18 We aimed to evaluate the association between lead poisoning and liver fibrosis as well as the change
19 the brink of extinction, in part, because of lead poisoning, and lead poisoning remains a significant
21 phrenia, post-traumatic stress disorder, and lead poisoning--conditions characterized by impoverished
22 llages with suspected or confirmed childhood lead poisoning continued to be identified in Zamfara Sta
26 vironmental remediation to address childhood lead poisoning epidemic due to artisanal gold mining in
30 es of L1, L2, L3, and L4 lumbar bones in the lead poisoning group were significantly lower than those
31 We investigated the extent of childhood lead poisoning [>/= 1 child with a blood lead level (BLL
32 s given to children with iron deficiency and lead poisoning have been demonstrated to improve develop
33 s Sans Frontieres (MSF) discovered extensive lead poisoning impacting several thousand children in ru
37 children (R(2) = 0.68) and the incidence of lead poisoning in children under age 1.3 years (R(2) = 0
39 ities are using local policy tools to reduce lead poisoning in high-risk areas, but little is known a
40 made in reducing the incidence of childhood lead poisoning in the United States in the past three de
43 or death in the previous year from suspected lead poisoning included the age of the child, the mother
47 he prevalence of osteoporosis in people with lead poisoning is not different from that in healthy ind
49 recommended frequently to reduce the risk of lead poisoning, its role in preventing lead absorption h
50 ban on leaded gasoline has markedly reduced lead poisoning, many other environmental sources of lead
51 lopmental toxicity associated with childhood lead poisoning may be attributable to interactions of Pb
53 We detected unexpectedly high frequencies of lead poisoning of eagles, both chronic (46 to 47% of bal
55 2010, Medecins Sans Frontieres discovered a lead poisoning outbreak linked to artisanal gold process
57 was to determine the cause of the childhood lead poisoning outbreak, investigate risk factors for ch
59 The condor's recent near-extinction from lead poisoning, poaching, and loss of habitat is well do
60 Rochester experience may inform future local lead poisoning prevention policies in other communities.
64 ts have reduced lead exposure significantly, lead poisoning remains the most common environmental hea
65 matic activity due to genetic aberrations or lead poisoning results in a mild to moderate nonspherocy
66 in blood lead in iron-replete children with lead poisoning secondary to iron supplements have not be
68 hese factors, the use of iron supplements in lead poisoning should be individualized, and the supplem
70 The present study used an animal model of lead poisoning to examine the extent to which different
75 nic abdominal pain is a potential symptom of lead poisoning, which is often challenging to diagnose.