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1 veloping zones to one another and the mature leaf blade.
2 t is critical to their ability to generate a leaf blade.
3 ntly reduces or eliminates macrohairs in the leaf blade.
4 cessary for lateral growth of the developing leaf blade.
5 that specify macrohair initiation within the leaf blade.
6 tes macrohair initiation specifically in the leaf blade.
7 re of periclinal division suppression in the leaf blade.
8 synthetic deficiencies throughout the entire leaf blade.
9 oliar margins and in minute hot spots in the leaf blade.
10 ceeds toward the expanded green cells of the leaf blade.
11  was increased up to 32.5-fold in 8-week-old leaf blades.
12 level in sorghum and Johnsongrass but not in leaf blades.
13 ne and sometimes glutamine compared to older leaf blades.
14 hesis genes was higher in younger than older leaf blades.
15 ed the amino acid levels in center and outer leaf blades.
16 ntally regulated auxin gradient in expanding leaf blades.
17  lateral organs results in upward curling of leaf blades.
18 ntly downregulated in affected areas of sxd1 leaf blades.
19 een tissues, with highest levels in maturing leaf blades.
20       The adaxialized leaves fail to develop leaf blades.
21                Awns are likely homologous to leaf blades.
22                  In growing maize (Zea mays) leaf blades, a defined developmental gradient facilitate
23 treatment caused cell death in B. distachyon leaf blades, an effect that was reversed by the addition
24 does, and can substitute for STF function in leaf blade and flower development if expressed under the
25 data suggests that PpSCR1 is involved in the leaf blade and mid-vein development of moss and that its
26                                 Thickness of leaf blade and midrib were recorded separately.
27 ddish-brown vascular tissue in the stem, the leaf blade and sheath.
28 odulate cell proliferation in the developing leaf blade and specific floral tissues; a role that was
29 oped slender leaves with four times narrower leaf blade and three times thicker mid-vein.
30 ally dissimilar to native SAPs, having wider leaf blades and greater leaf area, dense and evenly dist
31 ler rosettes because of shorter petioles and leaf blades and often acquired a twisted leaf morphology
32  developmental trajectories in Kranz (foliar leaf blade) and non-Kranz (husk leaf sheath) leaves of t
33 tly overrepresented among 25 E- > E+ DEGs in leaf blade, and a number of other DEGs were associated w
34 xial cells is important for formation of the leaf blade, and the MYB transcription factor gene PHANTA
35  plants had slightly longer petioles, larger leaf blades, and larger cells than controls.
36 observed in sepal and petal development, but leaf blades are apparently normal.
37 tem; however, add3 prevents the expansion of leaf blades at high temperature.
38         Liguleless3-O (Lg3-O) transforms the leaf blade, auricle and ligule into sheath around the mi
39 is required for EODFR-mediated constraint of leaf blade cell division, while EODFR messenger RNA sequ
40 s prominent at the mid-vein and the flanking leaf blade cells pointing towards its role in leaf devel
41 related with a similar reduction in expanded leaf blade chlorophyll levels.
42 equency of polyploid cells in basal zones of leaf blades, consistent with the disruption of cytokines
43 roxy-beta-diketones in the peduncle and flag leaf blade cuticles.
44 tant bladekiller1-R (blk1-R) is defective in leaf blade development and meristem maintenance and exhi
45       To help understand regulation of maize leaf blade development, including sink-source transition
46  rice, WOX3 homologs are major regulators of leaf blade development.
47 antagonistically to STF and LAM1 to regulate leaf blade development.
48 tion as a transcriptional coactivator during leaf blade development.
49  control points in gene expression along the leaf blade developmental gradient.
50  have evolved repeatedly because a conserved leaf blade developmental program is continuously activat
51 s diversity is achieved by the modulation of leaf blade dissection to form lobes or leaflets.
52  N. sylvestris resulted in a range of severe leaf blade distortions, indicating important role in bla
53 queous auxin application inhibited long-term leaf blade elongation.
54 ptoms, including shoestring leaves that lack leaf blade expansion.
55 apical hook maintenance, and abaxial/adaxial leaf-blade expansion.
56 n biosynthesis and auxin biosynthesis in the leaf blade followed by auxin long-distance transport to
57                         Comparisons included leaf blades from apple, grape, corn, and tomato and leaf
58 pecific stages along the developmental maize leaf blade gradient.
59 pd33 mutants also exhibited chlorosis in the leaf blades, greatly diminished plant growth, and reduce
60 normal adaxial/abaxial polarity and generate leaf blades in the normal position, but the adaxial meso
61 s of four other species with narrow and thin leaf blades, including wheat (Triticum aestivum L.), mai
62 plants can be described as simple, where the leaf blade is entire, or dissected, where the blade is d
63              In particular, the width of the leaf blade is greatly reduced, and each leaflet in the m
64                     Initiation and growth of leaf blades is oriented by an adaxial/abaxial axis align
65 nd (2) annotation of partial sections of the leaf blade (LB).
66 ablished along the developmental axis of the leaf blade, leading from an undifferentiated leaf base j
67 transcriptionally repress its targets during leaf blade morphogenesis.
68 types differed at two major loci controlling leaf blade Na(+) accumulation.
69 rate genetic traits that interact to control leaf blade Na(+).
70                                      Younger leaf blades of aposymbiotic plants (no endophyte present
71 ent was 2-fold elevated in BdWRI1-expressing leaf blades of B. distachyon.
72 hondrial transcripts in stage 2 semi-emerged leaf blades of one month-old maize plants.
73 hanges in the cell wall composition of csld1 leaf blades or epidermal peels, yet a greater abundance
74 a, and Nicotiana sylvestris are required for leaf blade outgrowth and floral organ development as dem
75 iptional coactivator play important roles in leaf blade outgrowth and flower development, but how the
76  gene, STENOFOLIA (STF), plays a key role in leaf blade outgrowth by promoting cell proliferation at
77  development, but LFL has no obvious role in leaf blade outgrowth in M. truncatula on its own or in c
78 STF and LAM1 are WOX1 orthologs required for leaf blade outgrowth in Medicago truncatula and Nicotian
79 a WOX gene, STENOFOLIA (STF), in controlling leaf blade outgrowth.
80 ades from apple, grape, corn, and tomato and leaf blade, petiole, stem, and pod tissues from soybean
81 biotic (E+) vs endophyte-free (E-) clones in leaf blades, pseudostems, crowns and roots.
82 oduct, which is involved in establishing the leaf blade-sheath boundary.
83 tions in young (center) versus older (outer) leaf blades, so LOL gene expression was compared in thes
84 ted developmental time, and is restricted to leaf blade tissue.
85  acceptable results for cyclitol analysis in leaf blade tissue.
86 criptomic data from seedling leaf and mature leaf blade tissues of maize hybrids and their inbred par
87 1A6, which occurred predominantly within the leaf blade tissues.
88  by changing gene expression from the distal leaf blade to its base.
89    The auricles act as a hinge, allowing the leaf blade to project at an angle from the stem, while t
90 in dorsoventrality and lateral growth of the leaf blade was investigated in the 'bladeless' lam1 muta
91  cell division and expansion at the bases of leaf blades, where cytokinesis and cross-wall formation
92  expression was similar in younger and older leaf blades, whereas expression of N. uncinatum LOL gene
93 reased cell size and reduced cell number per leaf blade with incrementing ploidy.
94       A lam1-lam1-glauca chimera generated a leaf blade with lam1 cells in the L1-derived epidermis a