ライフサイエンスコーパス: left

1 in the right eye and counting fingers on the left.

2 hs (26.7%) were also non-interpretable, that left 44 interpretable radiographs in the study.

3 eriod and found that standard categorization left 46% of patient records unclassified.

4 of vascular loop was type II (right: 69.14%; left: 58.75%).

5 The data reveal that one ('left') 601 DNA end is well ordered whereas the other ('r

6 ent decreased left frontal inhibition of the left amygdala, and larger decreases were associated with

7 excitability of BLA neurons in vitro in the left and right amygdala of postnatal days 22-28 male and

8 iciency: 1) Electrical heterogeneity between left and right atria increases; and 2) Increased fibrosi

9 Speech perception is mediated by both left and right auditory cortices but with differential s

10 associated with modifications of FC between left and right inferior parietal lobes and right insular

11 orm and middle temporal gyri, as well as the left and right insula, for those with MDD relative to HC

12 or depression symptoms, the PTSD findings in left and right LOFG remained significant.

13 imilar bimodal spectral response profiles in left and right primary and secondary regions, with evoke

14 The left and right SBRs for caudate, putamen, and striatum w

15 On a track with cues on left and right sides, most cue cells only responded to c

16 nal and structural hyperconnectivity between left and right temporoparietal regions was positively re

17 ked to the early and late remodelling of the left and right ventricle over the course of monocrotalin

18 on of left-footedness to 23.7% including all left- and mixed-footers as a single non-right category.

19 were substantiated by analyses of FC between left- and right-hemispheric ROIs, which revealed that de

20 ed to either a control site (vertex) or to a left angular gyrus target region.

21 The source activity of the MMN from the left anterior cingulate cortex, inferior frontal gyrus,

22 eeks old male C57BL/6 mice (n = 9) underwent left anterior descending (LAD) coronary artery ligation

23 e native state and 4 after infarction of the left anterior descending artery.

24 g the left circumflex artery followed by the left anterior descending coronary artery branch.

25 al node connected by the white matter of the left arcuate fasciculus.

26 intravenously in a peripheral vessel in the left arm.

27 urvivors were dominated by the rare few that left at intermediate sizes and times, coinciding with ma

28 arises from structural abnormalities in the left atria (LA).

29 (Left Atrial Appendage Closure vs. Novel Anticoagulation

30 [PST] or patient's self-management [PSM] and left atrial appendage closure) are based on the concept

31 ventricular tachycardia ablation and Lariat left atrial appendage exclusion.

32 Left atrial appendage occlusion (LAAO) to prevent stroke

33 oing trials are addressing the usefulness of left atrial appendage occlusion in both primary and seco

34 to the first bifurcation and thrombus in the left atrial appendage.

35 fice area, vena contracta, color Doppler jet/left atrial area, left atrial volume index, left ventric

36 cellular components, and ultrastructure) in left atrial biopsies from 121 patients with persistent/l

37 ds ratio, 0.35 [95% CI, 0.16-0.79], P=0.01), left atrial diameter (odds ratio, 0.52 per 1 cm increase

38 ventricular posterior wall diameter z score, left atrial diameter z score, peak left ventricular outf

39 The left atrial end-systolic volume index (LAESVI) is a pred

40 he 26 patients (77%) with a normal predicted left atrial pressure (grade I diastolic dysfunction) had

41 Evaluation of left atrial pressure is frequently required for mechanic

42 occlusion pressure (a frequent surrogate of left atrial pressure) in this population.

43 tcomes and is the recommended measurement of left atrial size.

44 ntracta, color Doppler jet/left atrial area, left atrial volume index, left ventricular end-diastolic

45 electrical and structural remodeling in the left atrium (LA) that begets atrial myopathy and arrhyth

46 n the normal position and normal size of the left atrium and left ventricle with a normal ejection fr

47 , mitral annulus disjunction (MAD), a larger left atrium and left ventricular end-systolic diameter,

48 cally isolating the pulmonary veins from the left atrium by catheter ablation is superior to antiarrh

49 The left auditory system is specialized for processing of ph

50 ynaptic plasticity in the right, but not the left BLA of males exclusively.

51 US of the left breast (Fig 1), bilateral breast MRI (Fig 2), and f

52 nic for assessment of a palpable mass in her left breast that developed quickly in 2 weeks.

53 Left bundle branch block may also develop following aort

54 Left bundle branch block may be due to conduction system

55 t failure with reduced ejection fraction and left bundle branch block may respond positively to cardi

56 ent pathophysiologic and clinical aspects of left bundle branch block, as well as current and future

57 ay have the capacity to exploit scent trails left by prey which can be tracked to a final source, tho

58 : 116 anomalous right CA (71%), 25 anomalous left CA (15%), 17 single CA (10%), and 5 anomalous circu

59 excitatory fronto-temporal projection in the left cerebral hemisphere.

60 rperfusion was found in APOE epsilon4+group (left cingulate and lateral frontal and parietal regions

61 , and higher nodal centrality metrics in the left cingulate cortex and left thalamus.

62 Recurrent MI was induced by ligating the left circumflex artery followed by the left anterior des

63 The presence of a left common trunk was significantly associated with the

64 or, and retromode deviated to right (DR) and left (DL).

65 nt three separate sessions of HD-tDCS (sham, left DLPFC and right DLPFC) for 20 min.

66 Given that left DLPFC has been proposed as an explicit target of de

67 glutamate concentration and WM activation in left DLPFC, with a positive association in unmedicated p

68 r the word "left" or "right" in the right or left ear, eliciting slower responses when the word and t

69 e interval, 0.07-1.08 dB; mean change in the left eye 0.84 dB; 95% confidence interval, 0.22-1.47 dB)

70 with a diagnosis of anterior uveitis in his left eye due to varicella-zoster virus and Toxoplasma go

71 d with a complaint of increasing pain in the left eye more than the right, along with decreasing visu

72 Left eye was unremarkable.

73 nd tractional retinal detachment (RD) in her left eye, 3 years after the last intra-arterial chemothe

74 ative training methods (right eye, P = 0.19; left eye, P = 0.10).

75 10% using the most conservative criterion of left-footedness to 23.7% including all left- and mixed-f

76 FSTA surgery when the mucosal flap margin is left free and unsutured when compared with leaving the f

77 subgroups by pathway location for right and left free wall AP.

78 evidence suggested that treatment decreased left frontal inhibition of the left amygdala, and larger

79 Specifically, left frontal regions and their underlying white matter t

80 the rATL (active and sham condition) or the left frontopolar region while participants attempted to

81 ral activity, was correlated with GMV in the left fusiform gyrus (r = -0.19, P(uncorrected) = 0.049)

82 ized 1:1 to either sham or TBE targeting the left gastric artery using an occlusion balloon microcath

83 ound a significantly higher (i.e. feminized) left-hand 2D:4D in the male-to-female transgender (MtF)

84 History A 36-year-old left-handed woman with a history of developmental delay

85 life on Earth, that is, the predominance of "left-handed" or l-amino acids and "right-handed" or d-su

86 double helix around microtubules, which are left-handed.

87 s (8 women and 17 men; 23 right-handed and 2 left-handed; age range, 16-64 years), who did language,

88 und 41 loci associated (P < 5 x 10(-8)) with left-handedness and 7 associated with ambidexterity.

89 Hypoplastic left heart syndrome (HLHS) is a complex congenital heart

90 groups: "HLHS/TGA" fetuses with hypoplastic left heart syndrome (HLHS) or transposition of the great

91 e recruited 8 adults with chronic stroke and left hemiplegia/paresis and 22 healthy adults.

92 in antipsychotic-treated vs HC's in a larger left hemisphere cluster (100 voxels, CCLAV = 0.01).

93 h in 52 speakers during the acute stage of a left hemisphere stroke.

94 participants with aphasia due to unilateral left hemisphere stroke.

95 In the left hemisphere, we find that CA1-entorhinal connectivit

96 We observed the same relationship in left hemispheric amygdala (p = 0.010), caudate (p = 0.00

97 We studied tau propagation in the left hemispheric syntactic network, which comprises an a

98 igher dichotic listening LQs indicating more left-hemispheric language processing.

99 (a) broadly distributed delta networks, (b) left-hemispheric theta networks with a local integrating

100 RIC consisted of 4 x 5 min cycles of left hind limb ischemia.

101 network increased activity of the targeted (left) hippocampus during scene encoding and increased su

102 The left internal mammary artery graft is done by sternal-sp

103 stocervical trunk pseudoaneurysm (n = 1/24), left internal mammillary artery pseudoaneurysm (n = 1/24

104 ological and immunohistochemical, and on the left kidney biochemical (malonyl-aldehyde [MDA], glutath

105 results revealed that most participants with left language dominance display the prototypical pattern

106 proach, we identified a small cluster in the left lateral frontal lobe where children with greater up

107 ateral NW striking fault and then ruptured a left-lateral fault to the surface.

108 These timing maps were partially left lateralized and widely spread, from occipital visua

109 ive languages, while language production was left lateralized, lateralization for language comprehens

110 uction showed negligible change and remained left lateralized.

111 aning were less likely than white or liberal/left-leaning members to have high self-initiated partici

112 l (BGCX, n = 13), or right (RGCX, n = 13) or left (LGCX, n = 9) unilateral GC lesions and sham-operat

113 delivered 10 ms before the end of TUS to the left M1 hotspot of the first dorsal interosseous muscle.

114 0 ms) receptor activation were recorded from left M1.

115 1,800 patients with de novo 3-vessel and/or left main coronary artery disease randomized to treatmen

116 In revascularisation of left main coronary artery disease, PCI was associated wi

117 yocardial infarction, and had multivessel or left main disease.

118 erator volume and survival after unprotected left main stem percutaneous coronary intervention (uLMS-

119 ub-paramarginally around the cervix of right-left mandibular first molars and maintaining the sutures

120 Single pulse TMS was administered over the left motor cortex, using anatomical scans of each subjec

121 isparity in thermo-mechanical parameters has left much debate as to which factors dominate fabricatio

122 tric breath conditions between the right and left nostril have been recorded and analyzed for potenti

123 erson-years) for women in whom they had been left open.

124 th different actions (i.e., licking either a left or a right spout).

125 ies not passing through over 2 clusters in a left or downward direction, the specificity for detectin

126 epsy patients with electrode implantation in left or right primary, secondary, and/or association aud

127 n chicks could choose between two identical, left or right, stimuli both representing either 2, 8, or

128 ric movements, including the ability to move left or right.

129 Simon task where participants hear the word "left" or "right" in the right or left ear, eliciting slo

130 eft: p < 0.001), thalamus (right: p < 0.001; left: p < 0.001), putamen (right: p = 0.001; left: p = 0

131 ral volume of hippocampus (right: p = 0.001; left: p < 0.001), thalamus (right: p < 0.001; left: p <

132 left: p < 0.001), putamen (right: p = 0.001; left: p = 0.001), and angular gyrus (right: p = 0.011; l

133 0.001), and angular gyrus (right: p = 0.011; left: p = 0.001).

134 A genotype by condition interaction in left parahippocampus indicated that in e4 carriers activ

135 Connectivity between left PCC and right posterior cerebellum (Crus I and II)

136 ork were sensitive to semantic features: the left pMTG/ITG was sensitive to haptic perception and the

137 in the spatial network, associations between left posterior cingulate (PCC) and right retrosplenial c

138 ers showed CBF increases in right middle and left posterior hippocampus.

139 STN-DBS lead, placement impacting fibers to left prefrontal areas should be avoided to maximize impr

140 Left prefrontal piTBS monotherapy is effective for the t

141 independent cohorts of patients who received left prefrontal transcranial magnetic stimulation (TMS)

142 For DaTQUANT and MIMneuro, the left, right, total, and average SBRs and z scores for wh

143 Many organs develop left-right asymmetric shapes and positions that are cruc

144 a key regulator of mesoderm development and left-right axis specification; components for nervous sy

145 fy the descending motor system that commands left-right locomotor asymmetries in mammals.

146 y, and malfunctional motile monocilia of the left-right organizer during early embryonic development

147 ver, showed pervasive reductions in 22q11DS (left/right hemispheres: d = -1.01/-1.02).

148 ow), flat on the right side, and flat on the left side.

149 We computed the percentage of Left-sided Choice (LC).

150 ere counted on CT from 22 right-sided and 64 left-sided colon tumours.

151 ssive conduit' of lesser importance than its left-sided counterpart.

152 Removing waveform-related phase shifts left significant residual phase shifts.

153 -circle targets and the largest (of right or left) single-Maddox rod values were similar to double-Ma

154 ment: the crossed right hand is localized in left space, and this conflict presumably provokes hand a

155 Bacterial ribosomes never left stasis.

156 ation supports that reductions in Glx in the left STG may be critical to the pathophysiology of schiz

157 Our results suggest that for the left STN-DBS lead, placement impacting fibers to left pr

158 Schizophrenia subjects had lower Glx in the left superior (STG) and middle temporal gyri (16 voxels,

159 cluster enhancement, TFCE) and in FH +group (left temporal and parietal regions p<0.01, TFCE).

160 oral regions for posterior cortical atrophy, left temporal lobe for logopenic progressive aphasia and

161 ss of the right ventral frontal area and the left temporal lobe, which represented a close mirror ima

162 increased creatine in two clusters involving left temporal, parietal and occipital regions (32, and 1

163 ity metrics in the left cingulate cortex and left thalamus.

164 cortex (ACC) and glutamate and Glx levels in left thalamus.

165 The bats swerved sideways and left the pathway on more flights in the hoop tunnel comp

166 History A 70-year-old man had a posterior left thigh lesion confirmed to be biopsy-proven melanoma

167 These patches were then left to be colonized by ovipositing bromeliad insects.

168 iency virus (HIV), and then examined who was left to be treated.

169 ries to fulfil their promises; countries are left to choose their conditional and unconditional emiss

170 kin models were inoculated with bacteria and left to incubate.

171 (QUBIC2), which is empowered by: (i) a novel left-truncated mixture of Gaussian model for an accurate

172 risk after ICU admission was assessed using left-truncation with increasing minimum survival period.

173 Posterior wounds were left undisturbed to clinically evaluate healing (using p

174 om three distinct genotypic backgrounds were left unmated or singly mated in a fully reciprocal desig

175 If left unrepaired, meiotic DSBs can cause aneuploidy in ga

176 181C, and Y181V HIV-1 were performed in mice left untreated or after RPV PrEP.

177 The disease is considered fatal if left untreated.

178 atic animals were either treated with 3HP or left untreated.

179 biotic, and asymptomatic bacteriuria is best left untreated.

180 e BP measured at the four most common sites (left upper arm, left wrist, right upper arm, right wrist

181 cluded prior left upper lobectomy for remote left upper lobe stage IIIA adenocarcinoma and prior bila

182 Her surgical history included prior left upper lobectomy for remote left upper lobe stage II

183 This left us with a set of physically identical choices that

184 mixed model whole-brain analysis identified left V3/V3A as the area with the most specific connectiv

185 G was sensitive to haptic perception and the left ventral temporal cortex (VTC) to size.

186 ons, palmitate can, at least in part, offset left ventricle (LV) dysfunction in hearts from diabetic

187 udy the transcriptional profile of the human left ventricle (LV, n=4) and right ventricle (RV, n=4) a

188 (RV) function has lagged behind that of the left ventricle and historically, the RV has even been re

189 ved exosome treatment significantly improved left ventricle cardiac function, inhibited cell apoptosi

190 y showed a hypodense area in the apex of the left ventricle in a 57-year-old man with a history of an

191 osition of the immune cell population in the left ventricle manifested by lowered abundance of proinf

192 ion, CaM-M37Q, into the anterior wall of the left ventricle of RyR2 wild type or mutant mouse hearts.

193 ated adaptive atlas algorithm to segment the left ventricle on CAC-CT, extracting 107 radiomics featu

194 ition and normal size of the left atrium and left ventricle with a normal ejection fraction.

195 isease characterized by abnormalities in the left ventricle, associated valves, and ascending aorta.

196 The Impella unloads the left ventricle.

197 creased cardiomyocyte nuclear density in the left ventricle.

198 s encoding the 3-dimensional geometry of the left ventricle.

199 PET imaging identified significantly higher left ventricular (18)F-FDG accumulation in TAC mice than

200 erate aortic stenosis (ModAS) (n=13), SevAS, left ventricular (LV) ejection fraction >=55% (SevAS-pre

201 pathy (oHCM) is characterized by unexplained left ventricular (LV) hypertrophy associated with dynami

202 nters continue CRT while others turn off the left ventricular (LV) lead at LVAD implant.

203 tors for changes in prognostically important left ventricular (LV) parameters.

204 Left ventricular (LV) wall thickness and LV mass were gr

205 sociation with incident HF and HF phenotype (left ventricular [LV] ejection fraction [LVEF] >= or < 5

206 mammillary artery pseudoaneurysm (n = 1/24), left ventricular aneurysms (n = 3/24), pulmonary arterio

207 We prospectively obtained paired left ventricular apical myocardial tissue from nonfailin

208 er, CRT management following continuous flow Left Ventricular Assist Device (LVAD) implant vary: some

209 cause of major morbidity and mortality after left ventricular assist device (LVAD) implantation.

210 rt failure (termed responders [R]) following left ventricular assist device (LVAD)-induced mechanical

211 e obtained before and after (median=82 days) left ventricular assist device implantation (stage D; pr

212 from normal, failed and partially recovered (left ventricular assist device treatment) adult human he

213 th heart failure, heart transplantation, and left ventricular assist device.

214 Although intravascular microaxial left ventricular assist devices (LVADs) provide greater

215 advanced heart failure, heart transplant and left ventricular assist devices have been the mainstay o

216 twork (CNN) model was trained to segment the left ventricular cavity, myocardium, and right ventricle

217 dP/dtmax is not a load-independent marker of left ventricular contractility and should be not used to

218 ed that in humans with stable heart failure, left ventricular contractility could be accentuated with

219 with symptomatic PE and right ventricular to left ventricular diameter ratio >=0.9 as documented by c

220 ignificant reduction in right ventricular to left ventricular diameter ratio and thrombus burden.

221 lities, global left ventricular dysfunction, left ventricular diastolic dysfunction grade II or III,

222 calmodulin kinase II (CaMKII) activation and left ventricular dilation in mice one week after myocard

223 ion was associated with oxidative stress and left ventricular dysfunction assessed by electron spin r

224 CD studies had fewer comorbidities, had less left ventricular dysfunction, and received more inapprop

225 entricular wall motion abnormalities, global left ventricular dysfunction, left ventricular diastolic

226 MR left ventricular eccentricity index (EI), main pulmonary

227 were a lateral e'-wave greater than 8 (for a left ventricular ejection fraction >= 45%) or an E/A rat

228 n E/A ratio less than or equal to 1.5 (for a left ventricular ejection fraction < 45%).

229 This were consistent for left ventricular ejection fraction < 50% or >= 50%.

230 ibrillation (HR, 2.6 [95% CI, 1.7-3.5]), and left ventricular ejection fraction <35% (HR, 2.0 [95% CI

231 fraction (HFrEF; heart failure with reduced left ventricular ejection fraction <40%) referred for st

232 iation functional class II or greater with a left ventricular ejection fraction <=40% and a modest el

233 0-mm Hg increase; 95% CI, 1.05 to 1.28), and left ventricular ejection fraction (aOR, 1.07 per 5% inc

234 dmission and on discharge in patients with a left ventricular ejection fraction (LVEF) >= 40%.

235 l fibrillation (AF) in patients with reduced left ventricular ejection fraction (LVEF).

236 ratio, 1.89 [95% CI, 1.04-3.44]; P=0.04) and left ventricular ejection fraction (per 10% decrement fr

237 sistently in men and women and patients with left ventricular ejection fraction above or below the me

238 nts with congestive heart failure or reduced left ventricular ejection fraction had a higher risk of

239 Left ventricular ejection fraction increased >=10% in 46

240 ysfunction (LVSD), defined as occurring when left ventricular ejection fraction is <50%.

241 Of these, 96 correlated with left ventricular ejection fraction measured at 4 months

242 We then correlated plasma proteins with left ventricular ejection fraction measured at 4 months

243 tricular wall thickness was 18 +/- 8 mm, and left ventricular ejection fraction was 61 +/- 12%.

244 ar ejection fraction (per 10% decrement from left ventricular ejection fraction, 50%; hazard ratio, 1

245 Three of 170 patients (2%) had abnormal left ventricular ejection fraction.

246 enefits are most prominent in patients whose left ventricular end-diastolic dimension Z score before

247 systolic volume index threshold of 227% or a left ventricular end-diastolic volume index of 58 ml/m(2

248 /left atrial area, left atrial volume index, left ventricular end-diastolic volume index, peak E wave

249 disjunction (MAD), a larger left atrium and left ventricular end-systolic diameter, and T-wave inver

250 gative remodeling, defined as an increase in left ventricular end-systolic volume index of >15% at 24

251 ic nerve density was reduced in the anterior left ventricular epicardium of DBH-Sap hearts compared t

252 ntuated without an increase in heart rate or left ventricular filling pressures.

253 Left ventricular function and New York Heart Association

254 Symptoms and left ventricular function at 2 years did not differ sign

255 lead to further improvement in management of left ventricular function in patients with diabetes.

256 sease (CAD), atrial fibrillation, or reduced left ventricular function, suggesting shared genetic aet

257 to experimental MI had significantly reduced left ventricular function.

258 Left ventricular functional parameters, especially ventr

259 The left ventricular global longitudinal strain was higher i

260 rial impedance (Z(va)), which reflects total left ventricular hemodynamic burden, was lower with TAVR

261 Individuals with left ventricular hypertrophy (LVH) and elevated cardiac

262 s was evaluated with respect to diagnosis of left ventricular hypertrophy (LVH), eligibility for dise

263 Left ventricular hypertrophy was present in 19 individua

264 Cardiac structural abnormalities, left ventricular hypertrophy, or concentric geometry, we

265 function at submaximal and maximal exercise, left ventricular mass and compliance, and blood volume c

266 Greater early left ventricular mass index (LVMi) regression is associa

267 ncentration of 135 mmol/L did not change the left ventricular mass index, despite significant reducti

268 ialysate sodium of 135 mmol/L did not reduce left ventricular mass relative to control, despite impro

269 crovascular obstruction (MVO) (percentage of left ventricular mass) quantified by cardiac magnetic re

270 Patients with extensive LGE (>=15% of left ventricular mass) were at highest risk (HR, 12; 95%

271 461.9+/-178.3 mm(3); P=0.023) and infarcted left ventricular myocardium (1052.3+/-543.0 versus 340.3

272 n to the atrioventricular junction (n=5) and left ventricular myocardium (n=20) of intact animals.

273 h MPRI < 2) were more likely to have dynamic left ventricular outflow tract (LVOT) obstruction (63.3%

274 myopathy, and a major determinant of dynamic left ventricular outflow tract (LVOT) obstruction.

275 z score, left atrial diameter z score, peak left ventricular outflow tract gradient, and presence of

276 of increase in velocity-time integral of the left ventricular outflow tract greater than or equal to

277 One delayed left ventricular outflow tract obstruction required elec

278 nexplained syncope, septal diameter z-score, left ventricular posterior wall diameter z score, left a

279 sure, pulmonary artery systolic pressure, RV/left ventricular ratio, and RV fractional area change.

280 A clinical score (RAISE) that used left ventricular remodeling (hypertrophy/diastolic dysfu

281 The only predictor of left ventricular remodeling was treatment with sonothrom

282 the control group was more likely to exhibit left ventricular remodeling with an odds ratio of 2.79 (

283 ure, reduce weight, have salutary effects on left ventricular remodeling, and reduce hospitalization

284 erioration in exercise capacity and promotes left ventricular reverse remodeling in asymptomatic or m

285 rences in baseline clinical characteristics, left ventricular reverse remodeling, or outcomes on mult

286 cribe hypertrophic cardiomyopathy (HCM) with left ventricular systolic dysfunction (LVSD), defined as

287 ng regarding the prognosis and management of left ventricular thrombus (LVT).

288 his international, multicenter cohort study, left ventricular unloading was associated with lower mor

289 more severe MR (p = 0.0005) despite smaller left ventricular volumes (p = 0.005) and higher right ve

290 Left ventricular volumes, ejection fraction, risk area (

291 Left ventricular vorticity metrics were observed to be h

292 chocardiographic abnormalities that included left ventricular wall motion abnormalities, global left

293 Maximum left ventricular wall thickness was 18 +/- 8 mm, and lef

294 whether face perception ability would show a left visual field (LeVF) bias due to earlier reports sug

295 ce and longevity of protective immunity have left vulnerable communities fearful that they may become

296 Although left-wing actors operate primarily through "hashtag acti

297 Many people who survive PML are left with neurological sequelae and some with persistent

298 .98) on the right and 4.33 (SD, 0.92) on the left with no significant differences (p = 0.236 and p =

299 However, these patients might be left with severe neurological sequelae.

300 the four most common sites (left upper arm, left wrist, right upper arm, right wrist) had adequate c