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1 F patients had larger and more dysfunctional left atria.
2  during acute fibrillation in ovine isolated left atria.
3 otential durations (APDs) in isolated canine left atria.
4 t into increased the arrhythmogenesis of the left atria.
5 vigation targets and 13 of 13 targets in the left atria.
6 entially regulates PRRX1 expression in human left atria.
7 hy, with occasional thrombi formation in the left atria.
8 g the conduction delay between the right and left atria.
9  cycle length were localized to parts of the left atria (5/7), the right atria (1/7), or both atria (
10 een, PANCR was expressed specifically in the left atria and eye and in no other chambers of the heart
11 ad hypertension and diabetes, and had larger left atria and higher left ventricular mass.
12 rogeneous myofiber architecture of right and left atria and interatrial septum provide a means of inv
13 dpc, they demonstrated dilation of right and left atria and ventricles.
14 tives with HCM had less hypertrophy, smaller left atria, and less systolic and diastolic dysfunction
15  and, in addition, showed that the right and left atria arise from the right and left heart fields.
16 cted, patients also had dilated and impaired left atria compared with control subjects (all P<0.001).
17 sis was performed from 10 right atria and 10 left atria data sets, including 86 axial LGE CMR planes
18  Heart Association class II to IV), enlarged left atria, dilated left ventricles with reduced systoli
19 vale (PFO), an opening between the right and left atria during normal fetal development that fails to
20 stolic (shorter deceleration time and larger left atria) function compared with SR.
21               Patients in Group 1 had larger left atria, higher incidence of AFL pre-PVAI, and lower
22 resence of excitable tissue in the right and left atria in type I AF.
23 ivation frequency in right atria (RA) versus left atria (LA) in patients, but the underlying molecula
24                                Sixteen donor left atria (LA) were used as controls (CNs).
25  arises from structural abnormalities in the left atria (LA).
26 rdially mapped from the right atria (RA) and left atria (LA).
27 1+/-17.5131.1+/-22.7Total thrombus volume in left atria, mm(3)22.8+/-5.311.8+/-3.317.0+/-3.723.3+/-6.
28 212.2+/-2.622.5+/-5.6Mean thrombus volume in left atria, mm(3)8.2+/-1.54.0+/-0.95.5+/-1.68.1+/-3.3Mea
29 as were placed in the pulmonary arteries and left atria of eight excised rabbit lungs.
30      Direct contact mapping was performed in left atria of goats with acute AF (n=6) or persistent AF
31 cyte apoptosis were present in the right and left atria of MHC-TGFcys33ser hearts, the extent of fibr
32 imultaneous optical mapping of the right and left atria (RA and LA) and frequency sampling of optical
33  atrial wires attached to both the right and left atria, respectively, at the conclusion of surgery.
34 cting the SVC and RUPV flow to the right and left atria, respectively, by implantation of a covered s
35 tes from the sinoatrial (SA) node, right and left atria, right and left ventricles, and interatrial a
36 y in all regions of the allograft (right and left atria, right and left ventricles, coronary arteries
37                                     Isolated left atria studied on a multielectrode array revealed mo
38                           In isolated canine left atria, the effects of vernakalant and ranolazine we
39                       MR patients had larger left atria (volume index: 32 cm(3)/m(2) vs. 26 cm(3)/m(2
40                                              Left atria were harvested 6 hrs (n = 20) or 16 hrs (n =
41  Patients with HOCM frequently have enlarged left atria, which predisposes them to atrial fibrillatio