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1 ve, potentially sparing the esophagus during left atrial ablation.
2 d for potential confounders and incident AF, left atrial abnormality was associated with incident isc
3     Abnormal P-wave axis-an ECG correlate of left atrial abnormality- improves ischemic stroke predic
4 l venous approach was used to gain right and left atrial access under general anesthesia in healthy s
5                              LGE imaging and left atrial activation mapping were performed during sin
6 ngs add to the hypothesis that the posterior left atrial adipose tissue mass contributes to structura
7                                    Posterior left atrial adipose tissue mass is significantly larger
8                                The posterior left atrial adipose tissue mass was quantified on comput
9 ry disease), each gram increase of posterior left atrial adipose tissue was associated with 1.32 odds
10 atrial fibrillation, ultrasound M-mode-based left atrial anatomies were successfully created, and abl
11                        Moreover, the role of left atrial and annular dynamics in provoking MR is ofte
12    ESI >3.7 g/day was associated with larger left atrial and left ventricular dimensions (p < 0.05).
13                                         Both left atrial and right atrial emptying fraction were lowe
14                         AIT improved o2peak, left atrial and ventricular ejection fraction, quality-o
15 nificant improvement in AF symptoms, o2peak, left atrial and ventricular function, lipid levels, and
16  associated with reductions in inflammation, left atrial and ventricular remodeling, sleep apnea, blo
17 x, left ventricular volume index and maximal left atrial anterior-posterior diameter while RGS3 can i
18                         Randomized trials of left atrial appendage (LAA) closure with the Watchman de
19 er the empirical electrical isolation of the left atrial appendage (LAA) could improve success at fol
20                                              Left atrial appendage (LAA) electric isolation is report
21  of this study was to evaluate the impact of left atrial appendage (LAA) exclusion on short-term outc
22 the characterization of left atrial (LA) and left atrial appendage (LAA) flow dynamics in patients wi
23 formed in sinus rhythm at 6 months to assess left atrial appendage (LAA) function were included in th
24                Prophylactic exclusion of the left atrial appendage (LAA) is often performed during ca
25                                     Electric left atrial appendage (LAA) isolation (LAAI) may occur d
26                                              Left atrial appendage (LAA) procedures have been develop
27 ning can produce 4-dimensional images of the left atrial appendage (LAA).
28                                              Left atrial appendage closure (LAAC) and nonwarfarin ora
29 Long-term data on the safety and efficacy of left atrial appendage closure (LAAC) for stroke preventi
30                                 Percutaneous left atrial appendage closure (LAAC) is noninferior to v
31 erapy on coagulation system activation after left atrial appendage closure (LAAC) remains unknown.
32                    The risk-benefit ratio of left atrial appendage closure (LAAC) versus systemic the
33                                              Left atrial appendage closure (LAAC) was approved by the
34 Atrial Fibrillation) trial demonstrated that left atrial appendage closure (LAAC) with the Watchman d
35                          The implantation of left atrial appendage closure device (WATCHMAN, Boston S
36 an updated overview of current transcatheter left atrial appendage closure devices and review the res
37           Over the past decade, percutaneous left atrial appendage closure has emerged as a valid alt
38 fibrillation patients receiving the WATCHMAN left atrial appendage closure technology was designed to
39 Fibrillation Patients Receiving the WATCHMAN Left Atrial Appendage Closure Technology, patients with
40                                              Left Atrial Appendage Closure vs. Novel Anticoagulation
41                                             (Left Atrial Appendage Closure vs. Novel Anticoagulation
42 [PST] or patient's self-management [PSM] and left atrial appendage closure) are based on the concept
43 rocedure (mitral and tricuspid valve repair, left atrial appendage closure, and paravalvular leak clo
44 vices and review the results associated with left atrial appendage closure, focusing on procedural an
45                                       During left atrial appendage closure, the estimated dose absorb
46                   In the study, we performed left atrial appendage closure.
47 ty leading to stasis of blood flow following left atrial appendage electrical isolation (LAAEI) could
48  ventricular tachycardia ablation and Lariat left atrial appendage exclusion.
49             In this setting, thrombus in the left atrial appendage has been found to be the source of
50  AF the intramyocardial blood vessels of the left atrial appendage have an increased CML presence and
51 tion in alcohol intake, and occlusion of the left atrial appendage in patients with atrial fibrillati
52 mly assigned to undergo empirical electrical left atrial appendage isolation along with extensive abl
53 ng the effectiveness of empirical electrical left atrial appendage isolation for the treatment of LSP
54                         (Effect of Empirical Left Atrial Appendage Isolation on Long-term Procedure O
55 ring repeat procedures, empirical electrical left atrial appendage isolation was performed in all pat
56  ventricular tachycardia ablation and Lariat left atrial appendage ligation that involve the epicardi
57  safety of DCCV in patients with endocardial left atrial appendage occlusion (LAAO) devices.
58  Of the 90 patients with stroke, 84 received left atrial appendage occlusion (LAAO) devices.
59                                              Left atrial appendage occlusion (LAAO) to prevent stroke
60 reviewed 301 consecutive patients undergoing left atrial appendage occlusion at Aarhus University Hos
61 Closure Technology, patients with a WATCHMAN left atrial appendage occlusion device had consistently
62 of stroke, site-specific therapy directed at left atrial appendage occlusion has been now studied for
63 oing trials are addressing the usefulness of left atrial appendage occlusion in both primary and seco
64                                              Left atrial appendage occlusion indication was based on
65    Device-related thrombosis (DRT) following left atrial appendage occlusion is a rare but feared com
66                                              Left atrial appendage occlusion with WATCHMAN has emerge
67 cological, percutaneous therapies, including left atrial appendage occlusion, for stroke prevention h
68 w of stroke prevention strategies, including left atrial appendage occlusion, in patients with atrial
69 X2c RNAs were highly correlated in 233 human left atrial appendage samples.
70                     The PROTECT AF (WATCHMAN Left Atrial Appendage System for Embolic Protection in P
71 omized clinical trials, PROTECT-AF (Watchman Left Atrial Appendage System for Embolic PROTECTion in P
72 premature AT termination, noninducibility or left atrial appendage thrombus.
73               To address this subject, human left atrial appendage tissues were obtained from 10 pati
74 ar) follow-up of Percutaneous Closure of the Left Atrial Appendage Versus Warfarin Therapy for Preven
75 tral annulus along the posterior base of the left atrial appendage visualized by selective angiograph
76 ctural parameters of the left atrium and the left atrial appendage which have been shown to be associ
77 to the first bifurcation and thrombus in the left atrial appendage.
78 al ligament disruption, and exclusion of the left atrial appendage.
79 s (8), septal bags (6) and 1 thrombus in the left atrial appendage.
80 nary sinus activation during pacing from the left atrial appendage.
81 g were performed on DNA from lymphocytes and left atrial appendages of 34 patients (25 with AF).
82 itus had a greater left ventricular mass and left atrial area than patients without diabetes mellitus
83 adjustments for left ventricular mass index, left atrial area, and interim heart failure events parti
84 fice area, vena contracta, color Doppler jet/left atrial area, left atrial volume index, left ventric
85  cellular components, and ultrastructure) in left atrial biopsies from 121 patients with persistent/l
86 ial LGE signal intensity divided by the mean left atrial blood pool intensity was calculated for each
87 t device 5.5%, pulmonary vein or transseptal left atrial cannulation 2.8%).
88 s were analyzed using left ventricle cavity, left atrial cavity, or inferior vena cava as the IDIF.
89 We sought to examine the association between left atrial conduction velocity and LGE in patients with
90 tude, consistent with its positive impact on left atrial contraction.
91 entricular damage), Stage 2 (mitral valve or left atrial damage), Stage 3 (tricuspid valve or pulmona
92 -up of 976.5 days between patients with LAE (left atrial diameter > 45 mm; LAE group) and those witho
93 ajor exclusions were ejection fraction <35%, left atrial diameter >60 mm, ventricular pacing dependen
94 (-0.24 mm [95% CI, -0.39 to -0.10]), smaller left atrial diameter (-0.75 mm [95% CI, -0.95 to -0.56])
95 root diameter (0.40 mm [95% CI, 0.08-0.73]), left atrial diameter (0.34 mm [95% CI, -0.09 to 0.78]),
96                 In atrial fibrillation (AF), left atrial diameter (LAD) and low voltage area (LVA) ar
97 ds ratio, 0.35 [95% CI, 0.16-0.79], P=0.01), left atrial diameter (odds ratio, 0.52 per 1 cm increase
98  diastolic and systolic diameters and larger left atrial diameter (P<0.05).
99 mprising 1643 patients (31.3% paroxysmal AF, left atrial diameter 41+/-3.1 mm) were included in the f
100 ears; mean CHA2DS2-VASc score was 4.1+/-1.4; left atrial diameter averaged 4.7+/-0.8 cm; and 48% had
101 ventricular posterior wall diameter z score, left atrial diameter z score, peak left ventricular outf
102  confidence interval, 2.070-7.143; P<0.001), left atrial diameter>/=50 mm (hazard ratio 2.083; 95% co
103 egnancy have a greater aortic root diameter, left atrial diameter, and left ventricular mass and high
104 es, cardiac structure (aortic root diameter, left atrial diameter, left ventricular mass, and fractio
105 s; and 1.15 (1.02-1.30) per 1 SD increase in left atrial diameter.
106 entricular diastolic dysfunction and reduced left atrial diameter.
107  and E/e' ratio (11 vs. 7; P < 0.001), and a left atrial dilatation (40 vs. 29 mL/m(2) ; P = 0.011).
108  mm), severe basal LVOTO (70-120 mm Hg), and left atrial dilatation (44-57 mm).
109                                              Left atrial dilatation in the population is more common
110 c dysfunction, left ventricular hypertrophy, left atrial dilatation, and interstitial fibrosis.
111 o [HR] per decade, 1.55; 95% CI, 1.11-2.15), left atrial dimension (HR per centimeter diameter, 1.43;
112  wall thickness, LV diastolic dimension, and left atrial dimension (P<0.01 for all; n=2392; mean age,
113  had associated cardiovascular disease, mean left atrial dimension was 46+/-6 mm, and median CHA2DS2-
114  age at diagnosis, female sex, and increased left atrial dimension.
115 s also exhibited a signi fi cant increase in left atrial ejection fraction at 2 months after gene del
116     Left ventricular adaptation was similar; left atrial ejection fraction improved by +3.17% (P < 0.
117                                              Left atrial electroanatomic high-density mapping was per
118 ng fraction: -6.2% [-10.2 to -2.1], P=0.003; left atrial emptying fraction:-3.5% [-6.9 to -0.1], P=0.
119                                          The left atrial end-diastolic volume index (LAEDVI), represe
120 s, including LV ejection fraction (LVEF) and left atrial end-diastolic volume indexed to body surface
121                                          The left atrial end-systolic volume index (LAESVI) is a pred
122           Left ventricular end-diastolic and left atrial end-systolic volumes increased by 3.63 ml/m(
123             Eligibility also required either left atrial enlargement (>/=4.4 cm or volume >/=58 mL) o
124         Controversy exists regarding whether left atrial enlargement (LAE) is a predictor of stroke/s
125 ive physiology, characterized by progressive left atrial enlargement and diastolic dysfunction with p
126 d age, female sex, greater right atrial than left atrial enlargement and lower systolic pulmonary art
127                              Mutant mice had left atrial enlargement and Micu2(-/-) cardiomyocytes ha
128                                              Left atrial enlargement is frequent in degenerative mitr
129                                 The frequent left atrial enlargement of DMR as measured by LAVI in ro
130 nce of LV hypertrophy was 21%, prevalence of left atrial enlargement was 83%, prevalence of elevated
131  traditionally have been used as markers for left atrial enlargement, and both have been associated w
132                       Diastolic dysfunction, left atrial enlargement, and pulmonary hypertension were
133 ic Csk knockout in mice led to increased AF, left atrial enlargement, fibrosis, and inflammation, phe
134 rutinib for 4 weeks results in inducible AF, left atrial enlargement, myocardial fibrosis, and inflam
135 r hypercontractility, diastolic dysfunction, left-atrial enlargement and left ventricular fibrosis, a
136 to 0.039+/-0.005 cm(-2) s(-)(1) (P<0.001) in left atrial epicardium (LA(epi)), and prolonged AF cycle
137  patients with heart failure, we showed that left atrial ETV1 expression is downregulated at the RNA
138         Our goal is to find and characterize left atrial-expressed transcripts in the chromosome 4q25
139 ent, were predominantly found in the lateral left atrial free wall, and likely acted as drivers.
140 ass, lower LV systolic function, and reduced left atrial function over long-term follow-up.
141 al contraction (MV A Peak) indicating poorer left atrial function was associated with lower retinal v
142  in patients with heart failure (HF) reduces left atrial hypertension by shunting oxygenated blood to
143 sure was </=15 mm Hg in 54%, indicating that left atrial hypertension was absent in a majority of pat
144 eficial in this cohort but could also worsen left atrial hypertension, exacerbating lung congestion.
145                                    Moreover, left atrial hypertrophy led to AT proliferation, with a
146 ntricular (LV) dysfunction, ischemic MR, and left atrial infarction (LAI); and 2) to analyze how LA r
147 n FEV1/FVC ratio was associated with smaller left atrial internal dimension (beta = -0.038 cm per SD
148 luded pulmonary vein isolation in 50 (100%), left atrial isthmus line in 47 (94%), anterior line in 4
149 imaging was used for the characterization of left atrial (LA) and left atrial appendage (LAA) flow dy
150                                              Left atrial (LA) compliance and contractility influence
151 ence of dual muscular coronary sinus (CS) to left atrial (LA) connections, coupled with rate-dependen
152      We aimed to evaluate the feasibility of left atrial (LA) deformations and its prognostic values
153                                 The value of left atrial (LA) diameter, volume, and strain to risk st
154                                              Left atrial (LA) dysfunction and stiffness contribute to
155                                     Although left atrial (LA) dysfunction is common in heart failure
156                            We sought whether left atrial (LA) electromechanical conduction time (EMT)
157 oncentric left ventricular (LV) hypertrophy, left atrial (LA) enlargement and dysfunction, and LV dia
158                                     Although left atrial (LA) enlargement is a recognized risk factor
159                                              Left atrial (LA) enlargement is associated with adverse
160                                  Severity of left atrial (LA) fibrosis is a strong predictor of atria
161                                     Defining left atrial (LA) function has recently emerged as a powe
162                                              Left atrial (LA) function is tightly linked to several c
163                  Data on the clinical use of left atrial (LA) hemodynamic monitoring during MitraClip
164 ata have been reported on the association of left atrial (LA) late gadolinium enhancement (LGE) with
165 /- 10; 14 men) obtained using a reference 3D left atrial (LA) LGE sequence with 1.3 mm x 1.3 mm x 2.5
166 onths of high intensity exercise training on left atrial (LA) mechanical and electric remodeling in s
167 tudy sought to determine the implications of left atrial (LA) myopathy and dysrhythmia across the spe
168 d, we investigated left ventricular (LV) and left atrial (LA) pathophysiological changes and their pr
169                       Background The role of left atrial (LA) performance in acute myocardial infarct
170 ious studies showed that the quantity of the left atrial (LA) periatrial fat tissue predicts recurren
171                                              Left atrial (LA) remodeling after an acute myocardial in
172                                              Left atrial (LA) remodeling is an important underlying s
173                                              Left atrial (LA) size is a marker of diastolic function
174                                              Left atrial (LA) size is an established marker of risk f
175                 Purpose To determine whether left atrial (LA) strain quantification with cardiac magn
176 le describing factors influencing changes in left atrial (LA) structure.
177 associated with significant abnormalities of left atrial (LA) systolic and diastolic function.
178 s were stratified into 2 groups according to left atrial (LA) volume index >/=35 mL/m(2).
179  sought to (1) identify reference values for left atrial (LA) volumes and phasic function indices by
180 ional area, vs. 3.0 +/- 0.6% in control) and left atrial (LA: 11.8 +/- 0.5% vs. 5.4 +/- 0.8% control)
181                At terminal open-chest study, left-atrial (LA) effective refractory period was reduced
182     Prior studies have demonstrated regional left atrial late gadolinium enhancement (LGE) heterogene
183 eems to be sufficient to treat patients with left atrial low voltage < 10%.
184                                       During left atrial mapping, optimal contact parameters minimizi
185 ) with measures of left ventricular (LV) and left atrial mechanical function on cardiac magnetic reso
186 ickness and image intensity ratio defined as left atrial myocardial LGE signal intensity divided by t
187                             Samples of human left atrial myocardium showed a positive correlation bet
188  clinical in vivo study, we demonstrate that left atrial myocardium with increased gadolinium uptake
189                These effects were greater in left atrial myocytes from Ang II-treated NPR-C(-/-) mice
190      In vitro I-1c gene transfer in isolated left atrial myocytes from both pigs and rats increased c
191 Stage 0), left ventricular damage (Stage 1), left atrial or mitral valve damage (Stage 2), pulmonary
192 rial triggers and substrate most commonly of left atrial origin.
193 l voltage amplitude (right atrial, P=0.0005; left atrial, P=0.0001), slower conduction velocities (ri
194 conduction velocities (right atrial, P=0.02; left atrial, P=0.0002), and higher prevalence of electro
195 on Feature tracking of cardiac MRI to derive left atrial peak reservoir strain provided incremental p
196            Intraoperatively a small infected left atrial perforation was oversewn and a fistula to th
197  key nonclinical factors (arrhythmia burden, left atrial physiology and anatomy, chemical and electro
198                   Adjunctive ablation of the left atrial posterior wall (LAPW) may improve outcomes,
199 e relationship between the esophagus and the left atrial posterior wall is variable, and the esophagu
200      The close proximity of esophagus to the left atrial posterior wall predisposes esophagus to ther
201  +/- 44 mm Hg to 12 +/- 6 mm Hg; p = 0.007), left atrial pressure (29 +/- 11 mm Hg to 20 +/- 8 mm Hg;
202  hypertension (PH), with or without elevated left atrial pressure (eLAP), and mortality in candidates
203 iations between PH, with or without elevated left atrial pressure (eLAP), and mortality in candidates
204 he 26 patients (77%) with a normal predicted left atrial pressure (grade I diastolic dysfunction) had
205 f the 24 patients with an elevated predicted left atrial pressure (grade II/III diastolic dysfunction
206 systemic hypotension occurred with a fall in left atrial pressure and little change in left ventricul
207                            Ageing, increased left atrial pressure and stiffness, mitral regurgitation
208 evice is feasible, seems to be safe, reduces left atrial pressure during exercise, and could be a new
209 rt failure, interventions to reduce elevated left atrial pressure improve symptoms and reduce the ris
210 nd we describe the design of REDUCE Elevated Left Atrial Pressure in Heart Failure (REDUCE LAP-HF I),
211 ed trial of a device-based therapy to reduce left atrial pressure in HFpEF.
212                          The REDUCe Elevated Left Atrial Pressure in Patients with Heart Failure (RED
213             REDUCE LAP-HF I (Reduce Elevated Left Atrial Pressure in Patients With Heart Failure) was
214                                Evaluation of left atrial pressure is frequently required for mechanic
215 y was that a mechanical approach to reducing left atrial pressure might be effective in HFPEF.
216 sel pressure was increased either by raising left atrial pressure or by aortic constriction.
217 nt device that allows shunting to reduce the left atrial pressure provides clinical and hemodynamic b
218  occlusion pressure (a frequent surrogate of left atrial pressure) in this population.
219 EF is complex but characterised by increased left atrial pressure, especially during exertion, which
220                                     Elevated left atrial pressure, particularly during exercise, is a
221  pressure is directly related to an enhanced left atrial pressure, which is common to both heart fail
222  a heterogeneous clinical syndrome, elevated left atrial pressure-either at rest or with exertion-is
223               Two of 27 pigs did not undergo left atrial procedures and were injected with microembol
224 reath-hold to the atrioventricular junction, left atrial pulmonary vein junction, and freewall left v
225  reflect RFC catheter instability in certain left atrial regions, and thus required fewer lesions for
226 on in MR with favorable left ventricular and left atrial reverse remodeling.
227 al-middle coronary sinus and septally at the left atrial ridge.
228  the most common adjunctive lesions included left atrial roof or posterior/inferior lines, and cavotr
229 e performed gene expression profiling in 265 left atrial samples from patients who underwent cardiac
230 antly associated with gene expression in 329 left atrial samples.
231  among patients, most commonly involving the left atrial septum (32/43; 74.4%).
232 Tm-E180G mice had a significant reduction in left atrial size (1.99+/-0.19 [n=7] versus 2.70+/-0.44 [
233 ), C-reactive protein levels (P<0.0001), and left atrial size (P=0.03).
234 io [HR], 1.39 [95% CI, 1.22-1.58]; P<0.001), left atrial size (per cm: HR, 1.32 [95% CI, 1.19-1.46];
235 g clinical predictors with the evaluation of left atrial size by echocardiography serving as the sole
236 and 10 cm/s, respectively), whereas E/e' and left atrial size demonstrated good agreement with guidel
237 ptide levels, C-reactive protein levels, and left atrial size were associated with arrhythmia recurre
238 is composite, while LV ejection fraction and left atrial size were not (p > 0.05 for all).
239                            No differences in left atrial size were observed between patients with POA
240  (tissue Doppler imaging [TDI] e', E/e', and left atrial size) with concomitant N-terminal pro-brain
241 rsistent AF, hypertension, coronary disease, left atrial size, left ventricular ejection fraction, an
242            After adjusting for age, sex, and left atrial size, standard ablation was predictive of re
243 tcomes and is the recommended measurement of left atrial size.
244                             The intensity of left atrial spontaneous echo contrast (LASEC) by transes
245  strain, global circumferential strain, peak left atrial strain, and peak longitudinal strain of righ
246 s (namely segmental left ventricular strain, left atrial strain, and right ventricular strain) are al
247          After ablation of 17 +/- 10% of the left atrial surface and 18 months of follow-up, the atri
248 atients of group I with low amount (< 10% of left atrial surface area) of atrial low voltage.
249 Basket electrodes were within 1 cm of 54% of left atrial surface area, and a mean of 31 electrodes pe
250 res of LV geometry, including LV mass index, left atrial systolic diameter, interventricular septum,
251 ths (IQR: 11.8 to 14.2 months), no device or left atrial thrombosis, device dislodgement, or a new de
252 rs of stroke, CHADS2 score and CHA2DS2-Vasc, left atrial thrombus (LAT), the five-grades of LASEC and
253                                    Right and left atrial tissue was obtained from patients with parox
254 .20 [95% CI, 0.08-0.31], P=0.001), and lower left atrial total emptying fraction (beta coefficient pe
255 s a disease of variable interactions between left atrial triggers and substrate most commonly of left
256 [apnea-hypopnea index <5]), right atrial and left atrial voltage distribution, conduction velocities,
257             CCL+CD+ patients exhibited lower left atrial voltage than the remaining patients (p = 0.0
258 1), Sokolow-Lyon Index ( r=-0.54; P<0.0001), left atrial volume ( r=-0.49; P<0.0002), and Mainz Sever
259 diastolic volume (139 to 107 mL; P=0.03) and left atrial volume (118 to 85 mL; P=0.04).
260 astolic dysfunction (P = .003) and increased left atrial volume (57 +/- 11 vs 46 +/- 12 mL/m(2), P =
261 olume (beta=0.350; adjusted P=0.048), higher left atrial volume (beta=0.214; adjusted P=0.009), and h
262 than with enalapril in all others, including left atrial volume (from 30.4 mL/m2 to 28.2 mL/m2 vs fro
263  ventricular end-systolic volume (LVESV) and left atrial volume (LAV), are unknown.
264 amber pacemaker implantation, independent of left atrial volume (LAV).
265 s (odds ratio, 2.61; CI, 1.04-6.57; P=0.04), left atrial volume (odds ratio, 1.24; CI, 1.02-1.51; P=0
266 g transmitral flow, tissue velocity, maximum left atrial volume [LAV], and estimated pulmonary artery
267 tion rate was higher (51.9%/y) in those with left atrial volume above the median value of 73.5 mL.
268                                  Decrease in left atrial volume at follow-up was associated with a lo
269                                 Reduction in left atrial volume at follow-up was associated with a lo
270 ricular global longitudinal strain (GLS) and left atrial volume index (LAVI) have been recently propo
271  purpose of this study was to assess whether left atrial volume index (LAVI) measured in routine clin
272 DVI), LV end-systolic volume index (LVESVI), left atrial volume index (LAVI), and ratio of early tran
273  interval, 1.04-1.43; P=0.015) and increased left atrial volume index (LAVi; adjusted hazard ratio/un
274 and the fibrosis stages correlated with both left atrial volume index and AF duration.
275 d diastolic function (based on e', E/e', and left atrial volume index) were each independently and ad
276 rate, older age, elevated creatinine, larger left atrial volume index, and larger left ventricular en
277 iastolic volume indexes (LVESVI and LVEDVI), left atrial volume index, and ventricular-vascular coupl
278 ntracta, color Doppler jet/left atrial area, left atrial volume index, left ventricular end-diastolic
279 e, including carotid intima-media thickness, left atrial volume index, monocyte count and serum YKL-4
280 with diastolic dysfunction (E/e') and 5 with left atrial volume index.
281  with greater left ventricular (LV) mass and left atrial volume indexed to height(2.7) in both men an
282 nal pro B-type natriuretic peptide; however, left atrial volume reduction varied by baseline level of
283 atriuretic peptide) and secondary (change in left atrial volume) end points.
284 V end-systolic volume, LV ejection fraction, left atrial volume, and LV dyssynchrony at 1-year in CRT
285  volume, left ventricular ejection fraction, left atrial volume, and LV dyssynchrony.
286 of cardiac structure (left ventricular mass, left atrial volume, and mitral annulus e-prime) and dise
287 s, mass, maximal wall thickness, morphology, left atrial volume, and mitral valve leaflet lengths (al
288 ection fraction, relative wall thickness and left atrial volume, and worse New York Heart Association
289 dverse) LV mass, LV end diastolic volume and left atrial volume, but not with other cardiac measures,
290 AEDVI), representing the minimum or residual left atrial volume, has not been fully evaluated as a pr
291 s and heart failure hospitalizations; higher left atrial volume, NT-proBNP (N-terminal pro-B-type nat
292 and <17.8 ng/mL galectin 3) had reduction in left atrial volume, those above median did not.
293 , indexed left ventricular mass, and indexed left atrial volume.
294 al pro B-type natriuretic peptide, E/E', and left atrial volume.
295 tive wall and septal thickness, LV mass, and left atrial volume.
296 ness, and hypertrophy patterns and function; left atrial volume; and aortic root diameter.
297 1 +/- 36 ml to 122 +/- 30 ml; p < 0.001) and left atrial volumes (106 +/- 36 ml to 69 +/- 24 ml; p <
298  systolic circumferential strain (PSCS), and left atrial volumes and function, whereas phosphorus-31
299 sis, clustered by patient, and adjusting for left atrial wall thickness, conduction velocity was asso
300 monary vein (PV) regions and inferoposterior left atrial wall.

 
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