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1 detected a single hyperintense lesion in the left temporal lobe.
2 se had atrophy that included portions of the left temporal lobe.
3 ion cortex in ventral-medial portions of the left temporal lobe.
4 ominantly or exclusively within the anterior left temporal lobe.
5 F volume, especially in the frontal lobe and left temporal lobe.
6 in anisotropy within the white matter of the left temporal lobe.
7 rocess, at least for verbal material, in the left temporal lobe.
8 confluent enhancing nodules and edema in the left temporal lobe.
9 ions that included the prefrontal cortex and left temporal lobe.
10 nderwent temporal lobe resective surgery (49 left temporal lobe, 36 right temporal lobe) for whom nam
11 sentation, was distributed along the ventral left temporal lobe and along the extent of both superior
12 uptake of both tracers was increased in the left temporal lobe and in the right temporal pole, as we
13 cortical thickness correlations between the left temporal lobe and left occipital cortex not showing
14 the frontal lobes, anterior cingulate gyrus, left temporal lobe, and of white matter bilaterally in t
15 semantic dementia patients with predominant left temporal lobe atrophy showed better recognition of
16 revealed a significant hypometabolism in the left temporal lobe (BAs 20, 36 and 38), in the right fro
17 , revealed significant hypometabolism in the left temporal lobe (Brodmann areas [BAs] 20, 36, and 38)
19 tion, are relatively common after unilateral left temporal lobe damage, and they clarify the lesion c
22 uency tasks in 12 right-handed patients with left temporal lobe epilepsy (LTLE) and 12 right-handed h
24 ypical dominance is greater in patients with left temporal lobe epilepsy and has been shown to be ass
25 agnetic resonance images of 21 patients with left temporal lobe epilepsy and medically intractable co
26 th improved verbal learning in patients with left temporal lobe epilepsy from preoperatively to 12 mo
27 related analysis revealed that patients with left temporal lobe epilepsy had greater activation in th
29 tients and the group effect in patients with left temporal lobe epilepsy suggest that minor structura
30 resonance imaging analysis revealed that in left temporal lobe epilepsy, greater left hippocampal ac
33 ht into a functional dissociation within the left temporal lobe for compositional and distributional
34 oral regions for posterior cortical atrophy, left temporal lobe for logopenic progressive aphasia and
35 surgical resection from either the right or left temporal lobe for the relief of intractable epileps
36 of the anterior, inferolateral parts of the left temporal lobe for the representation of names and t
41 bally corrected ROI means indicated that the left temporal lobes of individuals with schizophrenia we
42 30 min, patients with epileptic foci in the left temporal lobe performed disproportionately poorly o
43 age was associated with reduced activity in left temporal lobe regions from 250 to 350 ms and in lef
44 e left hippocampal remnant at 3 months after left temporal lobe resection compared to control subject
46 o significant differences in total cerebral, left temporal lobe, right temporal lobe, or total tempor
47 y, and visual episodic memory improved after left temporal lobe surgery, indicating a functional rele
48 renics displayed smaller amplitudes over the left temporal lobe than first-episode affective psychoti
49 cts of music perception/reproduction and the left temporal lobe to language semantics; both these reg
54 ss of the right ventral frontal area and the left temporal lobe, which represented a close mirror ima
55 s showed that volumes of cortex areas in the left temporal lobe, which were associated with postopera
56 phasia to ventral and medial portions of the left temporal lobe, while regression analyses in Alzheim