ライフサイエンスコーパス: left ventricle

1 ad an intraaortic balloon pump to unload the left ventricle).

2 nal distribution (predominating in the basal left ventricle).

3 The Impella unloads the left ventricle.

4 s encoding the 3-dimensional geometry of the left ventricle.

5 creased cardiomyocyte nuclear density in the left ventricle.

6 volving the aorta, aortic valve annulus, and left ventricle.

7 us 21+/-12 ms; P=0.0005) compared with RV or left ventricle.

8 an Heart Association 17-segment model of the left ventricle.

9 y intervals prolongation in the RVOT, RV, or left ventricle.

10 involvement at the inferolateral wall of the left ventricle.

11 le or posteromedial papillary muscles of the left ventricle.

12 ded right ventricle and good function of the left ventricle.

13 y a typical diffuse spongy appearance of the left ventricle.

14 life-threatening CHD primarily affecting the left ventricle.

15 d HiC chromatin interaction data from LA and left ventricle.

16 VH, and these derangements often involve the left ventricle.

17 vered randomly on the basal, mid and lateral left ventricle.

18 vortex ring from the surrounding flow in the left ventricle.

19 lly greater than previously reported for the left ventricle.

20 We studied single myocytes from pig left ventricle.

21 dies, this results in an adversely remodeled left ventricle.

22 an fetal heart tissue with an underdeveloped left ventricle.

23 fy volumes of elevated velocity (EVV) in the left ventricle.

24 culum (ER) stress, and apoptosis in diabetic left ventricle.

25 =8 using a standard, 17-segment model of the left ventricle.

26 ated from human donor and heart failure (HF) left ventricle.

27 protein were unchanged in MR versus control left ventricles.

28 ventricles and its marked upregulation in MR left ventricles.

29 romoters and ~5000 enhancers active in human left ventricles.

30 entify promoter and enhancer regions used in left ventricles.

31 heavy chain 7 mRNA expression is detected in left ventricles.

32 patients with SCD had severe dilation of the left ventricle (124+/-27 vs 79+/-12 mL/m(2)), right vent

33 ring the matched breathing control (end film left ventricle 199.8 ms [SD, 16] versus control 201.6 ms

34 62 +/- 1.06 vs 15.88 +/- 0.81; p < 0.05) and left ventricles (20.14 +/- 1.40 vs 14.17 +/- 1.53; p < 0

35 in significantly worse pump function of the left ventricle 28 days after MI.

36 ds, 17 mL/min irrigation) and 3 sites in the left ventricle (40 W, 60 seconds, 30 mL/min irrigation)

37 cortex, renal outer medulla, liver, cardiac left ventricle, adrenal gland, and hypothalamus.

38 t of MI than placebo-control animals (15.7 g left ventricle and 12.0 g left ventricle versus 22.8 g l

39 GE yields good to moderate quality in 74% of left ventricle and 84% of right ventricle acquisitions a

40 delity pressure catheters were placed in the left ventricle and aortic root.

41 c was safe and improved contractility of the left ventricle and atrium in a large animal model of non

42 sociated with higher cell death rates in the left ventricle and deteriorated cardiac function.

43 (RV) function has lagged behind that of the left ventricle and historically, the RV has even been re

44 often been viewed as less important than the left ventricle and in contemporary literature received t

45 was increased in the infarcted region of the left ventricle and in the circulation of wild-type mice

46 art tube (HT), with the FHF contributing the left ventricle and part of the atria, and the SHF the re

47 dependent effects of circulating histones on left ventricle and right ventricle function at clinicall

48 otoxicity and the functional consequences on left ventricle and right ventricle remain unclear.

49 hibited discordant reverse remodeling in the left ventricle and the left atrium.

50 y mimics pressure-volume changes seen in the left ventricle and to use this system to achieve cardiac

51 onfirmed the absence of sarcolipin in normal left ventricles and its marked upregulation in MR left v

52 f the pulmonary artery, aorta, and right and left ventricles and the severity of reflux of contrast m

53 ements were made at 222 sites (excluding the left ventricle) and compared with measurements from intr

54 Global (left ventricle) and regional (left anterior descending,

55 ith an ischemic pathogenesis, a more dilated left ventricle, and a detectable hs-troponin had lower o

56 ion were evaluated, as well as the systemic, left ventricle, and adipose tissue inflammatory profile.

57 FGE acquisitions, respectively, covering the left ventricle, and in 69% and 84%, respectively, coveri

58 position of the great arteries, subpulmonary left ventricle, and left ventricular outflow tract (LVOT

59 rom the release source, down the axis of the left ventricle, and selectively toward the left heart fo

60 favorable energetic effect of unloading the left ventricle, and thus reduction of wall stress, could

61 Logistic regression identified the left ventricle/aorta angle as an indicator of indexed ao

62 ft ventricular outflow axis and aortic root (left ventricle/aorta angle) in both groups (BAV group: r

63 ntractility or an improved relaxation of the left ventricle as assessed by wave intensity analysis.

64 creted in acute stages of inflammation after left ventricle assisted device (LVAD) implantation for p

65 isease characterized by abnormalities in the left ventricle, associated valves, and ascending aorta.

66 dial glucose transporter 4 expression in the left ventricle at 8 d after TAC, indicating altered gluc

67 d component of therapy for HF with a dilated left ventricle because of its effectiveness in inhibitin

68 ale ranging from 0 (uptake less than that in left ventricle blood pool) to 4 (diffuse uptake greater

69 ted well with lesion size for lesions in the left ventricle but less well for lesions in the right ve

70 nriched fractions from syndecan-4(-/-) mouse left ventricles but increased nuclear MLP when syndecan-

71 s no change to the number of ICDs within the left ventricle, but there is an almost doubling of the n

72 ), we observed no significant differences in left ventricle capillary density between wild-type and S

73 ved exosome treatment significantly improved left ventricle cardiac function, inhibited cell apoptosi

74 e dysfunction in myocardial repair, improved left ventricle cardiac function, reduced MI scar size, a

75 Images were analyzed using left ventricle cavity, left atrial cavity, or inferior v

76 nt of the right ventricle in addition to the left ventricle classically studied.

77 was 8-fold more highly expressed in the male left ventricle compared with females in young adult C57B

78 raction (P = 0.08) and surrogate measures of left ventricle compliance did not reach significance.

79 y exhibited decreased survival with impaired left ventricle contractility and decreased serum adipone

80 rosis, leukocyte infiltration, angiogenesis, left ventricle contractility, and inflammatory gene expr

81 e result in severe cardiac hypertrophy, poor left ventricle contraction and death by postnatal day 16

82 g VT ablation and 5 with structurally normal left ventricles (controls) undergoing premature ventricu

83 e allograft (right and left atria, right and left ventricles, coronary arteries) compared to the nati

84 BrS, conduction delay in RVOT, but not RV or left ventricle, correlated to the degree of J-ST point e

85 Stress MBF of the left ventricle decreased by 10% +/- 6% (P = 0.005) after

86 These data support that first unloading the left ventricle despite delaying coronary reperfusion dur

87 volume (SV) at 12 weeks of age and decreased left ventricle diastolic volume with subsequent reduced

88 Vessel density in the left ventricle did not change during and after compactio

89 expression, direct targets of FXR1 in human left ventricle dilated cardiomyopathy (DCM) biopsy sampl

90 Thereafter, the left ventricle dilates and restores the baseline mass-to

91 mitral valve disease and may develop as the left ventricle dilates or remodels or as a result of lea

92 n inhibit TGF-beta signaling associated with left ventricle dilation and systolic dysfunction.

93 essive deterioration of cardiac function and left ventricle dilation.

94 unctionally, rAAV.hHO-1 and hHO-1 transgenic left ventricles displayed a smaller loss of ejection fra

95 As the left-ventricle does not have a uniform shape and functio

96 terminant factor in how much blood fills the left ventricle during diastole and thus in the etiology

97 partially mediated by diseases affecting the left ventricle during follow-up (myocardial infarction [

98 CTRP9-KO mice had exacerbated contractile left ventricle dysfunction following intraperitoneal inj

99 Compared with the left ventricle, E12 values were lower in the right ventr

100 s the function of the proximal aorta and the left ventricle (eg, aortic arch pulse wave velocity and

101 Left ventricle ejection fraction <55% was strongly assoc

102 ume, left ventricle end-systolic volume, and left ventricle ejection fraction (LVEF) were evaluated.

103 athy, there was a significant improvement in left ventricle ejection fraction from 30+/-11% to 42+/-1

104 Median left ventricle ejection fraction was 24% (10%-36%).

105 early, cardiac remodeling, deterioration of left ventricle ejection fraction, and cardiac arrhythmia

106 CMR showed higher left ventricle end-diastolic volume (mean difference: 43

107 Left ventricle end-diastolic volume index and end-systol

108 Patients experiencing MACE showed higher left ventricle end-diastolic volume, higher left ventric

109 All patients underwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-

110 lume (mean difference: 43+/-22.5 mL), higher left ventricle end-systolic volume (mean difference: 34+

111 MR, and left ventricle end-diastolic volume, left ventricle end-systolic volume, and left ventricle e

112 left ventricle end-diastolic volume, higher left ventricle end-systolic volume, and lower LVEF with

113 remodeling characterized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 17

114 using a 64-electrode basket catheter on the left ventricle endocardium and 54 6-electrode plunge nee

115 monary disease, a reduction in the estimated left ventricle epicardial volume correlated with a loss

116 erts in the critical tasks of segmenting the left ventricle, estimating ejection fraction and assessi

117 Underdeveloped left ventricle exerts biomechanical stress on the right

118 rovement of its volumes and function; 2) the left ventricle experiences improvement of its function;

119 ure work is warranted to investigate whether left ventricle fibrosis affects clinical outcomes.

120 s used to quantify the edema-based AAR (% of left ventricle) following ischemic preconditioning (IPC)

121 ive, active, or anatomical properties of the left ventricle for reproducing measured patient phenotyp

122 valve plane (VP) during segmentation of the left ventricle for SPECT myocardial perfusion imaging (M

123 old; P<0.05), which coincided with decreased left ventricle fractional shortening (-Delta11%; P<0.05)

124 rk to create 3D finite element models of the left ventricle from cardiac ultrasound or magnetic reson

125 adult mouse hearts and was also elevated in left ventricles from patients with dilated cardiomyopath

126 ccording to standard 17-segment model of the left ventricle (fully automatic analysis).

127 uced cardiac fibrosis response and preserves left ventricle function as compared to control-exosome a

128 heart disease are more likely to experience left ventricle function recovery with successful AF abla

129 (55/107), of which 51% (28/55) had preserved left ventricle function.

130 le eccentric myocardial hypertrophy, altered left ventricle geometry, perturbed systolic and diastoli

131 ion patterns of a healthy and ischemic human left ventricle geometry.

132 ariations in longitudinal deformation of the left ventricle have been suggested to be useful for diff

133 rs for pacemaker placement included systemic left ventricle (hazard ratio [HR], 2.2; P=0.006) and lat

134 ed insulin levels, cardiac systole deficits, left ventricle hypertrophy, a predictor of a later onset

135 dative stress allowed myocardial energetics, left ventricle hypertrophy, and diastolic dysfunction to

136 f the reduced inflammatory parameters in the left ventricle (IFN-gamma, IL-6, and IL-1beta), as well

137 or linear (38%, nine of 24) and involved the left ventricle in 13 patients and both ventricles in 24

138 s (63%) and in the endocardial inferolateral left ventricle in 3 patients (37%).

139 y showed a hypodense area in the apex of the left ventricle in a 57-year-old man with a history of an

140 ed for native T1 and ECV measurements of the left ventricle in health adult study participants.

141 ferential and longitudinal strain within the left ventricle in healthy Chinese subjects.

142 tive physiology leading to the growth of the left ventricle in parallel with somatic growth.

143 of life, the number of cardiomyocytes in the left ventricle increased 3.4-fold, which was consistent

144 Whether the left ventricle is also affected by VIP gene deletion is

145 Flow entering the left ventricle is reversed toward the outflow tract thro

146 ss disrupts insulin signaling in the cardiac left ventricle leading to adverse cardiac programming.

147 tage on the body surface, coronary sinus and left ventricle leads, requires a delivered charge of 0.0

148 In guinea-pig left ventricle, left atrium, and right atrium, carbenoxo

149 termined for the investigated organs (brain, left ventricle, liver, and muscle) due to different anim

150 abdomen including, at least partly, cardiac left ventricle, liver, spleen, and kidney (n = 2) or thr

151 lacrimal, parotid, and submandibular glands; left ventricle; liver; spleen; kidneys; bowel; urinary b

152 egeneration after surgical amputation of the left ventricle (LV) (apical resection) and coronary arte

153 se To prospectively evaluate the accuracy of left ventricle (LV) analysis with a two-dimensional real

154 enosis (AS) leads to variable stress for the left ventricle (LV) and consequently a broad range of LV

155 as due to enhanced myofiber work of both the left ventricle (LV) and RV.

156 progenitor populations that give rise to the left ventricle (LV) and sinus venosus (SV) are still amb

157 r pulmonary circulation after birth when the left ventricle (LV) becomes the systemic ventricle.

158 eperfusion alone, mechanically unloading the left ventricle (LV) before reperfusion reduces infarct s

159 When remodeling and enlargement of the left ventricle (LV) cause annular dilatation and tetheri

160 efficient myocardial perfusion and improved left ventricle (LV) diastolic function in HFpEF.

161 ons, palmitate can, at least in part, offset left ventricle (LV) dysfunction in hearts from diabetic

162 Although contributors to remodeling of the left ventricle (LV) have been well studied in general po

163 Pathological left ventricle (LV) hypertrophy (LVH) results in reactiv

164 severe cardiac malformation characterized by left ventricle (LV) hypoplasia and abnormal LV perfusion

165 ardiac MRI data included segmentation of the left ventricle (LV) in cardiac MRI perfusion and cardiac

166 Remodeling of the left ventricle (LV) in response to pressure overload lea

167 n (MI), overactive inflammation remodels the left ventricle (LV) leading to heart failure coinciding

168 An understanding of left ventricle (LV) mechanics is fundamental for designi

169 Whether T-cell recruitment to the left ventricle (LV) participates in the development of H

170 ary artery diameter and right ventricle (RV)/left ventricle (LV) ratio.

171 of direct parasympathetic innervation of the left ventricle (LV) remain controversial.

172 s this problem and better understand how the left ventricle (LV) remodels post-MI at both the molecul

173 Sex differences in the left ventricle (LV) size could provide an explanation fo

174 with (18)F-flurpiridaz PET MPI according to left ventricle (LV) size.

175 ined the reference values of left atrium and left ventricle (LV) structure in a large ethnically dive

176 =0.047) was significantly higher in the male left ventricle (LV) than in the female LV.

177 ght ventricle (RV) does not respond like the left ventricle (LV) to guideline-directed medical therap

178 Purpose To determine if excess greater left ventricle (LV) trabeculation is associated with dec

179 left ventricular hypertrophy, a thick-walled left ventricle (LV) ultimately transitions to a dilated

180 tively and quantitatively in order to assess left ventricle (LV) wall thickness (full width at half m

181 ardiac MR imaging analyses of the RV and the left ventricle (LV) were performed to determine cardiac

182 rse aortic constriction activated FYN in the left ventricle (LV), and FYN-deficient mice displayed ex

183 nificant volume and pressure overload on the left ventricle (LV), but such patients typically remain

184 were placed around the epicardium and in the left ventricle (LV), respectively.

185 ining maintains a youthful compliance of the left ventricle (LV), whereas a year of exercise training

186 n time was determined along 4 anatomic axes: left ventricle (LV)-right ventricle (RV), LV:apico-basal

187 ed in T2DM patients because of dysfunctional left ventricle (LV).

188 rated post-MI that mediate remodeling of the left ventricle (LV).

189 activation waves and optimal filling of the left ventricle (LV).

190 cyte hypertrophy were present throughout the left ventricle (LV).

191 udy the transcriptional profile of the human left ventricle (LV, n=4) and right ventricle (RV, n=4) a

192 The right (RV) and left ventricles (LV) do not function in isolation, shari

193 Gene array on left ventricles (LV) showed increased fractalkine, a che

194 Infarct volume (22 +/- 7% vs. 19 +/- 9% of left ventricle [LV]) and LVEF (24 +/- 8% vs. 28 +/- 9%)

195 Infarct size (percent of left ventricle [LV]) by CMR did not differ between the m

196 enting did not reduce final infarct size (9% left ventricle [LV]; interquartile range [IQR]: 3% to 18

197 uctive pulmonary disease (COPD) have smaller left ventricles (LVs) due to reduced preload.

198 timulation did not increase cAMP in USP20-KO left ventricles (LVs), whereas NKH477-induced adenylyl c

199 rtners bind differently to syndecan-4 in the left ventricle lysate from aortic-banded heart failure (

200 ipitated with NCX1 in rat cardiomyocytes and left ventricle lysate.

201 recipitated with NCX1 in rat cardiomyocytes, left ventricle lysates, and HEK293 cells.

202 osition of the immune cell population in the left ventricle manifested by lowered abundance of proinf

203 th a measure of inflammation and with higher left ventricle mass and volumes.

204 Lower MFR was associated with higher left ventricle mass index and higher left ventricle volu

205 ife was independently associated with higher left ventricle mass index only among women (P=0.001).

206 edian Society of Thoracic Surgeons score and left ventricle mass.

207 odel was then implemented in a 3-dimensional left ventricle model, demonstrating that such early afte

208 racterised by abnormal trabeculations in the left ventricle, most frequently at the apex.

209 aging measurements, we show that the healthy left ventricle moves in tandem with the expanding vortex

210 urs of reoxygenation, plasma troponin level, left ventricle myocardial levels of lipid hydroperoxides

211 P < .001) and ratio of scar volume to total left ventricle myocardial volume (%LGE) (r = 0.91, P < .

212 ons of IKr and IKs to repolarizing the human left ventricle (n = 18).

213 og of MIB2, have been found in patients with left ventricle non-compaction (LVNC), we investigated me

214 ortic flow reversal and ejection flow in the left ventricle occurs at optimal AVD.

215 lity were greater for right ventricle versus left ventricle (odds ratio, 1.8; 95% confidence interval

216 IFNgamma) were significantly elevated in the left ventricle of heart failure patients carrying the MI

217 rsible cysteine oxidation of proteins in the left ventricle of hearts from mice with metabolic syndro

218 atrial pulmonary vein junction, and freewall left ventricle of intact animals.

219 ated serelaxin's antifibrotic actions in the left ventricle of mice with cardiomyopathy, indicating t

220 c collagen I and titin n2b expression in the left ventricle of mice with HFpEF.

221 1 expression was significantly suppressed in left ventricle of mice with transverse aortic constricti

222 ion, CaM-M37Q, into the anterior wall of the left ventricle of RyR2 wild type or mutant mouse hearts.

223 Fibroblasts were isolated from the left ventricle of the explanted hearts of transplant rec

224 from xenon dissolved in the blood inside the left ventricle of the heart, it is possible to directly

225 6-electrode plunge needles inserted into the left ventricles of 6 dogs.

226 were implanted epicardially on the right and left ventricles of a porcine model and were inductively

227 xpression and increased Wisp-1 levels in the left ventricles of patients with ischemic dilated cardio

228 a-adrenergic signaling and sarcolipin in the left ventricles of patients with isolated MR and LV ejec

229 ependently up-regulated in the atria and the left ventricles of RacET mice on mRNA and protein levels

230 mining the cellular growth mechanisms of the left ventricle on a set of healthy hearts from humans ag

231 ated adaptive atlas algorithm to segment the left ventricle on CAC-CT, extracting 107 radiomics featu

232 In patients referred for left ventricle outflow tract premature ventricular contr

233 S AND Twenty-eight consecutive patients with left ventricle outflow tract premature ventricular contr

234 ventricular contractions originating in the left ventricle outflow tract represent a significant sub

235 rium and atrioventricular node compared with left ventricle (P=5.6x10(-6)).

236 in all animals, 42 +/- 12 vs. 35% +/- 12% of left ventricle, P < 0.001).

237 than epinephrine (41 +/- 8 vs. 47% +/- 6% of left ventricle, P = 0.004), whereas defect of a third ca

238 onsistent with origin from scar in the basal left ventricle, particularly the septum, but also basal

239 Left ventricle performance was improved in double transg

240 high P(PL) demonstrated unchanged transmural left ventricle pressure and systemic blood pressure afte

241 Methods and Results We used a mouse model of left ventricle pressure overload coupled to in vitro stu

242 The posterior-superior process of the left ventricle (PSP-LV) is the most inferior and posteri

243 rameter to predict lesion depth in right and left ventricle (r=0.47; P<0.0001; multiple regression P=

244 Analysis showed that the volume of the left ventricle receiving 5 Gy (LV-V5) was the most impor

245 rigins of parasympathetic innervation of the left ventricle remain controversial.

246 Left ventricle remodeling after anterior wall myocardial

247 (MMPs), have been identified in all forms of left ventricle remodeling and can be a contributory fact

248 more efficiently, as determined by improved left ventricle remodeling and ejection fraction.

249 impairs infarct healing, and contributes to left ventricle remodeling and heart failure.

250 g and unpredicted changes in MMP profiles in left ventricle remodeling processes, such as with pressu

251 left ventricular function and attenuation of left ventricle remodeling that were sustained during 6 m

252 inflammation, arteriosclerotic lesions, and left ventricle remodeling, we performed a cross-sectiona

253 tiation; both are critical events in adverse left ventricle remodeling.

254 ries to the aorta, aortic valve annulus, and left ventricle require open surgical repair.

255 icle and 12.0 g left ventricle versus 22.8 g left ventricle, respectively).

256 Contrary to conventional thinking, the left ventricle responds to exercise with initial concent

257 Transcriptomic profiling of left ventricles revealed three specific genes [natriuret

258 icular outflow tract (P<0.001) and higher in left ventricle-right ventricle pairs (P=0.021) and left

259 hanges characterized by echocardiography and left ventricle/right ventricle catheter-derived variable

260 ntricular arrhythmias (VAs) arising from the left ventricle's papillary muscles has been associated w

261 ndertook transcriptome analysis of human DMD left ventricle samples and found that DMD hiPSC-derived

262 RV/(left ventricle + septum) did not rise directly in propor

263 Overall, 24+/-19% of left ventricle showed HED uptake levels comparable with

264 ft structures, exemplified here by an active left ventricle simulator.

265 Cardiovascular risk factors, CAC scores, left ventricle size and function, and carotid intima-med

266 d Performance of Electrodes implanted in the Left Ventricle) study is a prospective multicenter non-r

267 anges compared to our previous report of the left ventricle, suggesting there is likely to be a compo

268 In patients with left ventricle systolic dysfunction, 37% (10/27) showed

269 erential strain gradient was observed in the left ventricle that showed universal increment from the

270 itral regurgitation, mainly a disease of the left ventricle, the vision for the next 5 years is not n

271 l direction from the base to the apex of the left ventricle, there was a trend of decreasing peak sys

272 islocalization was also evident in autopsied left ventricle tissue from HGPS patients, suggesting int

273 g, and we also find that LBBB will cause the left ventricle to contract later than the right ventricl

274 which oxygenated blood is delivered from the left ventricle to end organs with each cardiac cycle (20

275 After myocardial infarction, the left ventricle undergoes a wound healing response that i

276 ction in left ventricular (LV) function, the left ventricle undergoes structural remodelling under th

277 rough imaging of tissue taken from the sheep left ventricle using serial block face scanning electron

278 The close relationship between the RV and left ventricle (ventricular interdependence) and its cou

279 ol animals (15.7 g left ventricle and 12.0 g left ventricle versus 22.8 g left ventricle, respectivel

280 higher left ventricle mass index and higher left ventricle volumes but not with ejection fraction or

281 all myocardial infarction leads to increased left ventricle volumes, myocardial stress, and ultimatel

282 The left ventricle was accessed via transseptal, retrograde

283 n of parametric and functional measures, the left ventricle was analyzed over 200 sectors.

284 lysis of cine multidetector CT images of the left ventricle was optimized and analyzed with feature-t

285 The left ventricle was segmented with standard clinical soft

286 hearts, ischemic zone territory (34+/-1% of left ventricle) was selected so that ischemia evoked VF

287 our-chamber sections that covered the entire left ventricle were acquired by using simultaneous multi

288 adial, circumferential, longitudinal) of the left ventricle were analyzed using DRA on steady-state f

289 ntricle (RV) body, outflow tract (RVOT), and left ventricle were calculated and analyzed at baseline

290 Regions of interest for TA encompassing the left ventricle were drawn by two blinded, independent re

291 Peak strain and strain rate values of the left ventricle were not significantly different; however

292 omically and functionally different from the left ventricle, which precludes direct extrapolation of

293 njected subepicardially into the anterobasal left ventricle with 40 to 75 rhythmically contracting em

294 am videos, our model accurately segments the left ventricle with a Dice similarity coefficient of 0.9

295 ition and normal size of the left atrium and left ventricle with a normal ejection fraction.

296 farction underwent sequential mapping of the left ventricle with both catheters.

297 tion of VA from the papillary muscles of the left ventricle with either cryoenergy or radiofrequency.

298 Three-dimensional tractograms of the left ventricle with no SMS and rate 2 and rate 3 SMS exc

299 plained by secondary causes and a nondilated left ventricle with preserved or increased ejection frac

300 Preterm-born individuals had short left ventricles with small internal diameters and a disp