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1 ment with PD98059 delayed the development of left ventricular dilatation.
2 verity as measured by dimensional indexes of left ventricular dilatation.
3 gression analysis to determine predictors of left ventricular dilatation.
4 ricular arrhythmia, even after adjusting for left ventricular dilatation.
5 left ventricular end-diastolic pressure, and left ventricular dilatation.
6 , respectively), with 26% of patients having left ventricular dilatation and 20% having right ventric
7 0% or less and is accompanied by progressive left ventricular dilatation and adverse cardiac remodeli
8 ing because the GSK-3beta knockouts had less left ventricular dilatation and better-preserved left ve
9 Lmna(H222P/H222P) mice after they developed left ventricular dilatation and decreased ejection fract
10 the p38alpha inhibitor ARRY-371797 prevented left ventricular dilatation and deterioration of fractio
11 larization, inhibit apoptosis, and attenuate left ventricular dilatation and disease progression.
12 nrollment of older patients with less severe left ventricular dilatation and dysfunction during the y
13 In the mice that survived the first 10 days, left ventricular dilatation and dysfunction remained wor
14 week post-MI, KO mice had significantly more left ventricular dilatation and dysfunction than WT mice
15 poptosis, proliferation of nonmyocyte cells, left ventricular dilatation and dysfunction, and slightl
17 of CD4(+) T cells and prevented progressive left ventricular dilatation and hypertrophy, whereas ado
18 n, PLN-ASO treatment prevents progression of left ventricular dilatation and improves left ventricula
19 limus treatment (n=17) significantly reduced left ventricular dilatation and increased contractility
20 ted cardiomyopathy (IDC) is characterized by left ventricular dilatation and systolic dysfunction aft
21 l [(68)Ga]FAPI-46 uptake was associated with left ventricular dilatation and systolic dysfunction.
22 thy with predominantly endocardial fibrosis, left ventricular dilatation, and systolic dysfunction.
24 ompensation in most animals characterized by left ventricular dilatation, depressed systolic function
25 ce, homed to the heart and induced long-term left ventricular dilatation, dysfunction, and fibrosis i
26 strain dyssynchrony >200 ms, lack of severe left ventricular dilatation (end-systolic dimension inde
27 g-/- mice developed severe heart failure and left ventricular dilatation even under baseline conditio
28 I) structural remodeling is characterized by left ventricular dilatation, fibrosis, and hypertrophy o
30 The study establishes the role of very early left ventricular dilatation on outcome in MI and may be
31 ng occurs soon after MI, the effect of early left ventricular dilatation on outcome is not establishe
33 a variant of PPCM in mice, characterized by left ventricular dilatation (postpartum second delivery:
35 unction secondary to increased afterload and left ventricular dilatation secondary to volume overload
36 con-deficient mice showed heart failure with left ventricular dilatation, systolic dysfunction and lu
37 fraction, left ventricular hypertrophy, and left ventricular dilatation were associated with the dev
38 ehicle-treated HF mice displayed progressive left ventricular dilatation with significantly increased