1 herapy could be shown in those who developed left ventricular enlargement.

2 as left ventricular systolic dysfunction and left ventricular enlargement after excluding usual clini

3 as left ventricular systolic dysfunction and left ventricular enlargement after excluding usual clini

4 14 weeks, the phenotype progressed to marked left ventricular enlargement and severely depressed syst

5 function; dilated cardiomyopathy (DCM), with left ventricular enlargement and systolic dysfunction; a

6 increased lung uptake chi2=9.6, P=0.002; and left ventricular enlargement chi2=8.3, P=0.004.

7                                  Progressive left ventricular enlargement, hypocontractility, left at

8                               HF rabbits had left ventricular enlargement (left ventricular end-diast

9 White FDRs, or for DCM partial phenotypes of left ventricular enlargement (LVE) or left ventricular s

10                  Twenty percent (n = 45) had left ventricular enlargement (LVE), defined as LV end-di

11  of asymptomatic family members was based on left ventricular enlargement (LVE).

12 y, calsequestrin mice at 7 weeks showed mild left ventricular enlargement, mild decreased fractional

13 ers who underwent rescreening, two (one with left ventricular enlargement only, one with a left bundl

14 DCM defined by the presence of DCM or either left ventricular enlargement or left ventricular systoli

15 ith men, women had surgery rarely for severe left ventricular enlargement (systolic diameter > or = 5

16 ovement in ejection fraction, but persistent left ventricular enlargement was more frequent in group