ライフサイエンスコーパス: left ventricular function

1 to experimental MI had significantly reduced left ventricular function.

2 t, in terms of clinical events or changes in left ventricular function.

3 lecting both improved systolic and diastolic left ventricular function.

4 mal experimental studies metformin preserves left ventricular function.

5 reverse remodeling without adverse effect on left ventricular function.

6 luate predictors of recurrence and impact on left ventricular function.

7 al teaching of cardiac physiology focuses on left ventricular function.

8 independent of the concurrent improvement in left ventricular function.

9 ich improved in parallel with improvement in left ventricular function.

10 patients with symptoms at rest and impaired left ventricular function.

11 ism type 1, but rather an improved diastolic left ventricular function.

12 xtensive coronary artery disease or impaired left ventricular function.

13 h image acquisition plane, demographics, and left ventricular function.

14 ts are correlated with sensitive measures of left ventricular function.

15 upling, decreases infarct size, and improves left ventricular function.

16 sis in the noninfarcted region, and improved left ventricular function.

17 (201)Tl along with contrast cineCT to assess left ventricular function.

18 reat acute myocardial infarction and improve left ventricular function.

19 who had stable heart failure with depressed left ventricular function.

20 creased risk for sudden death from depressed left ventricular function.

21 yocardial infarction patients with preserved left ventricular function.

22 l ventricular remodeling and improved global left ventricular function.

23 ischemia and ventricular arrhythmias, and on left ventricular function.

24 from AC6 deletion underlies abnormalities in left ventricular function.

25 anges curbed infarct expansion and preserved left ventricular function.

26 esolution of pulmonary edema and recovery of left ventricular function.

27 coronary artery disease, angina, and normal left ventricular function.

28 elated to the severity of CAD and diminished left ventricular function.

29 ng/body weight ratios, but no improvement in left ventricular function.

30 % of patients with in-hospital assessment of left ventricular function.

31 unction assessed, 21,149 (51%) had preserved left ventricular function.

32 ntricular ectopy results in normalization of left ventricular function.

33 a previous myocardial infarction and reduced left ventricular function.

34 sepsis-related lactic acidosis, and improved left ventricular function.

35 ly decreased perinatal survival and impaired left ventricular function.

36 ata on the subset of patients with preserved left ventricular function.

37 oad reduces adverse remodeling and preserves left ventricular function.

38 ffect blood pressure, systolic, or diastolic left ventricular function.

39 dity and mortality in patients with impaired left ventricular function.

40 ICD implantation on the basis of symptoms or left ventricular function.

41 tion (AMI) has led to limited improvement in left ventricular function.

42 ystemic shunt (TIPS) creation, regardless of left ventricular function.

43 ntricular remodeling and regional and global left ventricular function.

44 not adequately address the effect of PVCs on left ventricular function.

45 r relationship with age, sex, body size, and left ventricular function.

46 e to sudden death, and in patients with poor left ventricular function.

47 r interstitial fibrosis, leading to improved left ventricular function.

48 he degradation pattern differed depending on left ventricular function.

49 te mortality, imparting a negative impact on left ventricular function.

50 with recent-onset heart failure and reduced left ventricular function.

51 response to post-acute myocardial infarction left ventricular functions.

52 is, enhances neovascularization and improves left ventricular functions.

53 hypertrophy, fibrosis and restoration of the left ventricular functions.

54 ed as the gold standard in the evaluation of left ventricular functions.

55 nt MR software programs were used to compare left ventricular functions.

56 the left ventricle and reduced impairment of left ventricular function 2 weeks after I/R injury.

57 much weaker in severe CHF compared to normal left ventricular function; 2) increasing HRR using rate-

58 found reduced myocardial death, and improved left ventricular function 24 hours after I/R injury in M

59 n in cultured endothelial cells, and improve left ventricular function after global ischemia and repe

60 n scar formation, thereby augmenting cardiac left ventricular function after ischemia/reperfusion.

61 e in humans that myocardial infarct size and left ventricular function after STEMI have a circadian d

62 n and cardiac fibrosis, as well as decreased left ventricular function, after myocardial infarction.

63 diographic assessment of myocardial mass and left ventricular function also did not suggest myocardia

64 FOG-2(R3K5A) mice leads to an improvement in left ventricular function and a partial rescue of left v

65 use a state of lipotoxicity that compromises left ventricular function and aggravate heart failure.

66 ouse myocardial infarction model can improve left ventricular function and attenuate cardiac remodeli

67 k region were accompanied by improvements in left ventricular function and attenuation of left ventri

68 ls, cardiopoietic stem cell therapy improves left ventricular function and blunts pathological remode

69 nts with recent-onset heart failure, reduced left ventricular function and clinically suspected myoca

70 CVB3-infected mice developed an impaired left ventricular function and displayed an increased lef

71 of left ventricular volumes, mass, regional left ventricular function and dyssynchrony.

72 cts of iC-BCP were associated with preserved left ventricular function and endothelium-dependent rela

73 timated dopamine pharmacokinetics as well as left ventricular function and global hemodynamics after

74 n limited myocardial infarct size, preserved left ventricular function and had no influence on haemod

75 myocardial infarction complicated by reduced left ventricular function and HF.

76 ited more adverse cardiac remodeling, poorer left ventricular function and higher mortality by increa

77 2 years vs. 52.3 years), had worse right and left ventricular function and higher pulmonary artery pr

78 at significant cardiovascular alterations in left ventricular function and in aortic stiffness occur

79 s and is associated with early impairment of left ventricular function and increased myocardial oxida

80 been beneficial for adult patients with poor left ventricular function and intraventricular conductio

81 Regional left ventricular function and late-gadolinium enhancemen

82 genic mice, Tg-GSK-3beta-DN exhibited better left ventricular function and less fibrosis and apoptosi

83 on deficit in human HCM with normal systolic left ventricular function and may contribute to HCM dise

84 Left ventricular function and New York Heart Association

85 mbolic complications; and (5) stable, normal left ventricular function and New York Heart Association

86 mouse IL-10 administration markedly improved left ventricular function and not only inhibited but als

87 In isolated perfused heart experiments, left ventricular function and oxygen consumption were si

88 ents with unexplained cardiac arrest (normal left ventricular function and QT interval) and selected

89 t failure (HF) therapy documented to improve left ventricular function and reduce mortality.

90 ion, reduce its hemodynamic repercussions on left ventricular function and remodeling, and improve cl

91 ssessed by echocardiographic measurements of left ventricular function and structure.

92 er time, whereas other risk factors, such as left ventricular function and surgery on the thoracic ao

93 It also improves left ventricular function and survival in mice (n=20-22

94 trates that metformin significantly improves left ventricular function and survival via activation of

95 duced cardiomyocyte hyperplasia had improved left ventricular function and survival.

96 rdial infarction is associated with improved left ventricular function and survival.

97 ardiovascular events in women with preserved left ventricular function and suspected myocardial ische

98 th a particular focus on those with improved left ventricular function and those who have not experie

99 Secondary end points were left ventricular function and volumes by magnetic resona

100 bdominal, liver, and myocardial fat content, left ventricular function, and (31)P magnetic resonance

101 vascularization priority, diabetes mellitus, left ventricular function, and 3-vessel disease.

102 clinical variables, perfusion defect scores, left ventricular function, and absolute myocardial flow

103 k Heart Association functional class 3, poor left ventricular function, and age (all p < 0.001) along

104 died, and the remaining patients had normal left ventricular function, and all but 1 have New York H

105 icity, sex, comorbidities, insurance status, left ventricular function, and aortic stenosis severity

106 ssion attenuates cardiac fibrosis, preserves left ventricular function, and improves survival in a mo

107 d similar improvements in clinical symptoms, left ventricular function, and left ventricular reverse

108 , cardiovascular parameters (carotid plaque, left ventricular function, and myocardial late gadoliniu

109 hyperlipidemia, chronic renal insufficiency, left ventricular function, and plasma F11R levels.

110 goal becomes limiting remodeling, preserving left ventricular function, and preventing heart failure.

111 istensibility by high-resolution ultrasound, left ventricular function, and quality of life were meas

112 n, improved stability of the circulation and left ventricular function, and reduced plasma levels of

113 assessment reveals normal coronary arteries, left ventricular function, and resting ECG.

114 ll evolution, thromboembolic risk, preserved left ventricular function, and severely decreased levels

115 actors in women, gender-specific measures of left ventricular function, and the relationship between

116 mptoms, quality of life, exercise tolerance, left ventricular function, and the survival of patients

117 Hemodynamic variables, indices of left ventricular function, and tumor necrosis factor-alp

118 P-2 precommitted mES cells, improved post-MI left ventricular functions, and enhanced capillary densi

119 iac magnetic resonance imaging and change in left ventricular function as affected by timing of treat

120 HIV-infected patients showed alterations in left ventricular function as demonstrated by a lower eje

121 ed animals recovered 85.4% of their baseline left ventricular function as measured by 2-dimensional t

122 The left ventricular function as measured by two-dimensional

123 BMCs; secondary end points included regional left ventricular function assessed by magnetic resonance

124 Of the 41,267 patients with left ventricular function assessed, 21,149 (51%) had pre

125 versus 10.9+/-2.3 cm/s; P=0.01) and regional left ventricular function, assessed by peak systolic vel

126 converting enzyme inhibitor prescription and left ventricular function assessment; no other HF qualit

127 IGF-1/HGF reduced infarct size and improved left ventricular function at 2 months after AMI.

128 Symptoms and left ventricular function at 2 years did not differ sign

129 and iPSC-EV-treated mice exhibited improved left ventricular function at 35 d after myocardial infar

130 m to determine the effects of this system on left ventricular function at 4 months after AMI.

131 ll), which translated into impaired systolic left ventricular function at 6 to 12 months post TAVR (p

132 s postreperfusion and significantly improved left ventricular function at 7 days after myocardial isc

133 ith new LE feature a significant decrease in left ventricular function at discharge.

134 artery disease, normal regional, and global left ventricular function at rest and during stress (exe

135 We evaluated left ventricular function at rest, maximal aerobic capac

136 me to return of spontaneous circulation, and left ventricular function at return of spontaneous circu

137 ventricular dilatation and better-preserved left ventricular function at up to 8 weeks post-MI despi

138 The present study aimed to evaluate left ventricular function, baroreflex sensitivity, auton

139 /-0.8 mm Hg/mL/m(2)), and worse longitudinal left ventricular function (basal longitudinal strain=-11

140 significant difference in global or regional left ventricular function between groups.

141 )Lin(-) cell-treated rats exhibited improved left ventricular function, blunted left ventricular hype

142 sonance imaging data showed no difference in left ventricular function but more RV dilation among PVR

143 myocardial infarction with severely impaired left ventricular function but no inducible ventricular t

144 et for 12 weeks did not alter body weight or left ventricular function but reduced maximal oxygen upt

145 3D-ST) has been used extensively to quantify left ventricular function, but its value for RV assessme

146 ere was an increase in frequency of impaired left ventricular function, but there was no change in Lo

147 termined physical activity level, as well as left ventricular function by cardiac magnetic resonance

148 nt in DM2 is already detectable in preserved left ventricular function by cardiovascular magnetic res

149 d by Doppler-derived pulse-wave velocity and left ventricular function by echocardiography.

150 d infarct size, microvascular perfusion, and left ventricular function by histopathology, microsphere

151 the ability of echocardiography to evaluate left-ventricular function by improving delineation of th

152 ve also been observed in the manner in which left ventricular function changes across the lifespan, w

153 ) subjects underwent assessment of diastolic left ventricular function (cine magnetic resonance imagi

154 ds that is incremental to clinical, ECG, and left ventricular function combined.

155 xhibited a significantly greater increase in left ventricular function compared with all the other gr

156 reased valve-related morbidity, and improved left ventricular function compared with MVR.

157 ant effect on recovery of global or regional left ventricular function compared with placebo.

158 ymptomatic severe aortic stenosis and normal left ventricular function, current practice guidelines e

159 Although systolic right and left ventricular function declined, peak exercise capaci

160 nsplantation may be associated with improved left ventricular function, decreased N-terminal pro B-ty

161 ation functional class 3 or 30-d death), and left ventricular function did not differ between two gro

162 Left-ventricular function did not change in any group.

163 turn plays an essential role in maintaining left ventricular function during starvation.

164 Left ventricular function (echocardiography), clinical s

165 Left ventricular function, edema, vascular permeability,

166 Importantly, SIM improved left ventricular function (EF, 32.4+/-4.1% to 51.7+/-3.2

167 moderate aortic regurgitation and preserved left ventricular function (ejection fraction >/=55%).

168 ase, and a nontransfemoral approach; whereas left ventricular function (ejection fraction <30%), the

169 Patients with new LE had a decreased left ventricular function (ejection fraction: pre, 55.5

170 raphics, risk factors, coronary anatomy, and left ventricular function, end-systolic volume index and

171 s like acute cor pulmonale determination and left ventricular function estimation.

172 to rupture and the significant reduction in left ventricular function evident as early as 1 week pos

173 peremia was associated with reduced regional left ventricular function expressed as lower Ecc in the

174 species generation, myocardial necrosis, and left ventricular function following ischemia/reperfusion

175 significant differences in change in global left ventricular function for patients treated at day 3

176 al and demographic predictors of recovery of left ventricular function for subjects with recent onset

177 End.LepR-KO was associated with improved left ventricular function (fractional shortening, 28.4%

178 The ability to record changes in left ventricular function from rest to peak stress and t

179 Improvements in left ventricular function, functional status, and qualit

180 the timing of cell delivery on the effect on left ventricular function has not been analyzed.

181 fferences between these mice with regards to left ventricular function, heart ultrastructure or elect

182 composition, health-related quality of life, left ventricular function, hemodynamics, endothelial fun

183 patients with heart failure (HF) who recover left ventricular function (HF-Recovered) have a distinct

184 Left ventricular function, histochemical injury, and dif

185 However, activity reduced left ventricular function (ie, ejection and shortening f

186 -flow ischemia and reperfusion, postischemic left ventricular function improved commensurately with a

187 ic resonance imaging was performed to assess left ventricular function in 20 women with signs and sym

188 y, decreases apoptosis, and greatly enhances left ventricular function in a model of ischemic cardiom

189 of endothelial progenitor cells and improves left ventricular function in a rat model of myocardial i

190 relationship between Hcy levels and regional left ventricular function in an asymptomatic population.

191 ically managed aortic stenosis and preserved left ventricular function in an era where surgical aorti

192 This resulted in preservation of left ventricular function in both acute and chronic myoc

193 T cells was correlated with the decrease in left ventricular function in CHD patients (r=0.629, P=0.

194 eukin-1 receptor antagonist, on coronary and left ventricular function in coronary artery disease (CA

195 In particular, stress exercise disturbs left ventricular function in dysferlin-null mice and inc

196 in global (LVEF) and regional (wall motion) left ventricular function in infarct and border zones at

197 ynchronization reduces symptoms and improves left ventricular function in many patients with heart fa

198 Tissue Doppler showed better diastolic left ventricular function in mutation carriers (e', P=0.

199 ession and CVB3 copy number, and an improved left ventricular function in NOD2(-/-) CVB3 mice compare

200 hese data also demonstrate that SIM improves left ventricular function in pacing-induced CHF rabbits.

201 free fatty acids and insulin sensitivity on left ventricular function in patients with clinically se

202 lead to further improvement in management of left ventricular function in patients with diabetes.

203 (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Inter

204 The EXPLORE (Evaluating Xience and Left Ventricular Function in Percutaneous Coronary Inter

205 ted information exists regarding the role of left ventricular function in predicting exercise capacit

206 (CPCs) 4 hours after reperfusion ameliorates left ventricular function in rats with acute myocardial

207 The full scorpion venom increased left ventricular function in sedated mice in vivo, prolo

208 t orifice area, 38+/-18 mm(2)) and preserved left ventricular function in whom initial management was

209 used loss of myocardial tissue and depressed left ventricular function in WT mice.

210 tandard volumetric MRI demonstrated improved left ventricular functioning in hydrogel treated mice fo

211 rospective study, we investigated changes of left ventricular functions including speckle tracking in

212 Indices of left ventricular function, including ejection fraction a

213 chocardiography revealed improved indices of left ventricular function, including ejection fraction a

214 hase I clinical trials, MSC therapy improved left ventricular function, induced reverse remodeling, a

215 In vivo secretoneurin improved left ventricular function, inhibited remodeling, and red

216 Baseline left ventricular function is a marker for prognosis rega

217 Left ventricular function is a principal determinant of

218 Impaired left ventricular function is associated with worse progn

219 Furthermore, although improvement in left ventricular function is one of the goals of revascu

220 Of note, measurement of baseline left ventricular function is the single most powerful pr

221 rtrophy but exhibited significantly improved left ventricular function, less cardiac fibrosis and car

222 tration of NaHS improved survival, preserved left ventricular function, limited infarct size, and imp

223 of life and exercise capacity and to improve left ventricular function long-term.

224 33% vs. 14%; p = 0.0027, respectively), and left ventricular function </=35% (26% vs. 10%; p = 0.007

225 Left ventricular function <30% was observed in 44.1% of

226 for ablation before ICD implantation because left ventricular function may improve, consequently decr

227 In nonsurgical candidates with compromised left ventricular function, MitraClip therapy could be co

228 cell transplantation significantly improved left ventricular function, myocardial metabolism, and ar

229 l infarction results in divergent effects on left ventricular function, myocardial remodeling, and mo

230 picardial biopsies from patients with normal left ventricular function (n=2) and from donor hearts (n

231 ents with coronary artery disease and normal left ventricular function (n=79).

232 All animals demonstrated vigorous left ventricular function, no outflow tract obstruction,

233 >50% of peripartum cardiomyopathy patients, left ventricular function normalizes with pharmacologic

234 hes (e.g., beta-blockers) partially preserve left ventricular function, novel strategies are needed t

235 no significant treatment effect on regional left ventricular function observed in either infarct or

236 Whether the improved left ventricular function observed in the Aldo-DHF trial

237 (HCRTR2), were less likely to have improved left ventricular function (odds ratio: 0.40 per minor al

238 al status or that the deleterious effects on left ventricular function offset any salutary vascular e

239 he only significant predictor of recovery of left ventricular function on the basis of the percent ch

240 E) inhibitor therapy in patients with normal left ventricular function on the progression of myocardi

241 t data reported no effect of cell therapy on left ventricular function or clinical outcome.

242 jection of autologous BMNCs does not improve left ventricular function or quality of life in patients

243 essel, cardiogenic shock, severely depressed left ventricular function, or ST segment elevation myoca

244 wever, new PPI did have a negative effect on left ventricular function over time.

245 s was the most important factor in improving left ventricular function (p < 0.001) and 30-day combine

246 Worse left ventricular function (P<0.0001), diabetes (P<0.0001

247 less myocardial salvage (P<0.001), impaired left ventricular function (P<0.001), and more pronounced

248 listing status (P<0.0001), severely impaired left ventricular function (P<0.01), pulmonary hypertensi

249 reas placebo pigs had persistently depressed left ventricular function (P<0.05).

250 (MR) imaging findings were used to quantify left ventricular function, perfusion, and infarct size.

251 art markedly limits remodeling and preserves left ventricular function post-MI.

252 ncRNAs did not affect cardiac hypertrophy or left ventricular function post-stress.

253 stheses may allow for greater improvement in left ventricular function postoperatively.

254 Left ventricular function predicts cardiovascular mortal

255 linical (older age, diabetes, renal disease, left ventricular function, recent myocardial damage, and

256 Furthermore, left ventricular function recovered within 72 hours of c

257 atment had no significant effect on regional left ventricular function recovery.

258 L-10-treated mice corroborated with improved left ventricular function, reduced infarct size, and fib

259 l prohormone brain natriuretic peptide), and left ventricular function/remodeling (left ventricular e

260 quantify myocardial triglyceride content and left ventricular function, respectively.

261 AFCA provides long-term benefits on left ventricular function, significantly reducing the nu

262 sease (CAD), atrial fibrillation, or reduced left ventricular function, suggesting shared genetic aet

263 Conclusively, Cfz decreased left ventricular function through increased PP2A activit

264 ronary artery disease and severely depressed left ventricular function to intra-aortic balloon pump (

265 miR-294-treated mice significantly improved left ventricular functions together with decreased infar

266 oncomitant coronary artery surgery, impaired left ventricular function, urgent or emergency status, p

267 onal activation in CHF patients with reduced left-ventricular function using SPECT to precisely quant

268 ft ventriculography is a method of measuring left ventricular function usually performed at the discr

269 perior cardiac repair in vivo with regard to left ventricular function, vascularization, and ameliora

270 ian follow-up of 36 months, normalization of left ventricular function was achieved in >90% of patien

271 Longitudinal left ventricular function was assessed by 2D-strain and

272 Left ventricular function was assessed by conventional a

273 Left ventricular function was assessed by measuring deve

274 In patients +CAN, left ventricular function was decreased in both diastole

275 Left ventricular function was evaluated using echocardio

276 The decline in left ventricular function was further supported by signi

277 Diastolic left ventricular function was graded as normal, mild, mo

278 Left ventricular function was impaired (ejection fractio

279 Consequently, infarct size was increased and left ventricular function was impaired 28 days after myo

280 Left ventricular function was measured by Millar pressur

281 Left ventricular function was measured by the pressure-v

282 Left ventricular function was measured continuously over

283 t time (71%); left atrium was 43+/-6 mm; and left ventricular function was normal (59+/-9%).

284 Left ventricular function was normal in >50% of patients

285 A greater improvement in left ventricular function was observed after intramyocar

286 Left ventricular function was restored in the 25 of 35 o

287 Myocardial perfusion and left ventricular function were also investigated using a

288 aline, and echocardiographic measurements of left ventricular function were greater in the AMD3100-tr

289 lectrolytes, glomerular filtration rate, and left ventricular function were performed on all patients

290 Myocardial blush grade and left ventricular function were significantly lower in pa

291 istics, vascular stiffness measurements, and left ventricular function were similar between participa

292 Left ventricular functions were consistently depressed i

293 Secondary outcomes, including symptoms and left ventricular function, were also evaluated.

294 farct vessel densities, and better preserved left ventricular function when compared with MSCs transf

295 ardiac MRI assessed myocardial viability and left ventricular function, whereas bioluminescence imagi

296 depends on the actual stenosis severity and left ventricular function, which is of prognostic import

297 g a reduced infarct size and preservation of left ventricular function, which suggests a role for thi

298 S100A8 on protein level, showed an improved left ventricular function, which was associated with a r

299 comparison group of 131 patients with normal left ventricular function who underwent coronary artery

300 dictors for fast-track failure were impaired left ventricular function with or without recent acute c