ライフサイエンスコーパス: left ventricular pressure

1 pressure and maximal rate of development of left ventricular pressure).

2 , and decreased myocardial contractility and left ventricular pressure.

3 inhibited TdP, but caused a 15+/-8% drop of left ventricular pressure.

4 pressure product and the first derivative of left ventricular pressure.

5 A lower dose of verapamil without effects on left ventricular pressure (0.06 mg/kg) was not antiarrhy

6 contrast, have normal heart function despite left ventricular pressures 25% higher than wild type.

7 On the basis of end-diastolic left ventricular pressure, 34 MI rats were classified as

8 hearts transduced with Ad.PL had lower peak left ventricular pressure (58.3 +/- 12.9 mmHg, n = 8) co

9 ed later (30+/-11 ms, P<0.015) and at higher left-ventricular pressure (61+/-9 mm Hg, P<0.001) than i

10 BKPC significantly improved the recovery of left ventricular pressure (73+/-5 versus 51+/-4 mm Hg; P

11 Left ventricular pressure, action potential duration, an

12 astolic pressure and a decrease in developed left ventricular pressure (all P<0.01 versus baseline) i

13 pressure and reduced peak and end-diastolic left ventricular pressures (all P<0.05).

14 affeine restored CcOX activity and increased left ventricular pressure and +/-dP/dt toward sham value

15 Left ventricular pressure and cardiac efficiency improve

16 ght dogs chronically instrumented to measure left ventricular pressure and dimension.

17 This increased left ventricular pressure and increased pressure develop

18 ine increased mean heart rate, peak positive left ventricular pressure and its first time-derivative,

19 Left ventricular pressure and maximal change in pressure

20 y blood flow and the maximum rate of rise in left ventricular pressure and reduced peak and end-diast

21 EA-0400 (0.4 and 0.8 mg/kg) had no effect on left ventricular pressure and suppressed dofetilide-indu

22 h high Ppl demonstrated unchanged transmural left ventricular pressure and systemic blood pressure af

23 We measured the left ventricular pressure and volume continuously with a

24 Left ventricular pressure and volume data were determine

25 function by mapping the relationship between left ventricular pressure and volume throughout the card

26 Characteristics of left ventricular pressure and volume unloading between t

27 ular diastolic filling pressures (pre-A wave left ventricular pressure) and Doppler mitral inflow at

28 of forearm blood flow, coronary blood flow, left ventricular pressure, and cardiac output were made

29 Aortic pressure, electrocardiogram, left ventricular pressure, and left ventricular pressure

30 yocardial CcOX activity, oxygen consumption, left ventricular pressure, and pressure developed during

31 trumented to measure aortic, left atrial and left ventricular pressures, and regional myocardial func

32 e on overall valve opening-closing dynamics, left ventricular pressure, aortic pressure, blood flow r

33 Because BNP reflects both elevated left ventricular pressure as well as neurohormonal modul

34 ejection fraction, analog differentiation of left ventricular pressure at 40 mm Hg, and rate of maxim

35 esistance index, the first derivative of the left ventricular pressure at a left ventricular pressure

36 idates in humans an equation relating tau to left ventricular pressure at peak -dP/dt (P0), pressure

37 minutes of reperfusion, maintaining CPP and left ventricular pressure at preischemic values.

38 Larger left heart structures and higher left ventricular pressure at the time of intervention we

39 n of maximum of the first time derivative of left ventricular pressure by dobutamine was blunted by i

40 artery (LAD) bypass were instrumented with a left ventricular pressure catheter and 2 subepicardial c

41 A left ventricular pressure catheter and continuous ECGs w

42 ced at 270 beats per minute, and the rate of left ventricular pressure change (LV dP/dt) was monitore

43 ed area produced minimal changes in systolic left ventricular pressure compared with baseline sinus r

44 the exponential time constant of isovolumic left ventricular pressure decay (Tau) and the "stiffness

45 ular pressure of 40 mm Hg (dP/dt40), rate of left ventricular pressure decline (-dP/dt), and a lower

46 ular pressure of 40 mm Hg (dP/dt40), rate of left ventricular pressure decline (-dP/dt), and end-tida

47 ar pressure at 40 mm Hg, and rate of maximal left ventricular pressure decline were continuously meas

48 ressure of 40 mm Hg, and the maximum rate of left ventricular pressure decline were significantly les

49 The rate of left ventricular pressure decrease was unchanged.

50 Peak left ventricular pressure decreased after TAVR (186 +/-

51 phy) and then cardiac catheterization, where left ventricular pressure development (+dP/dt) and decli

52 ignificant reduction in the maximum rates of left ventricular pressure development and pressure decli

53 ll acceleration of cross-bridge kinetics and left ventricular pressure development cannot be achieved

54 rload, fractional shortening and the rate of left ventricular pressure development decreased by 36% a

55 ate of cardiac myosin, and reduces force and left ventricular pressure development in isolated myofil

56 d as a percentage of the zone at risk; ZAR), left ventricular pressure development, and coronary flow

57 l tissue and showed electrical conductivity, left ventricular pressure development, and metabolic fun

58 tamine stress, VS attenuated the increase in left ventricular pressure-diameter area from 235.9 +/- 7

59 conscious dogs chronically instrumented for left ventricular pressure-dimension analysis, PDE5A inhi

60 nd-diastolic pressure (LVEDP), and developed left ventricular pressure (dLVP=LVSP-LVEDP), ischemia-re

61 n consumption (MVO(2)), peak rate of rise of left ventricular pressure (dP/dt(max)), stroke work (SW)

62 ve maximal values of the first derivative of left ventricular pressure (dP/dt) were significantly imp

63 wall stress and positive first derivative of left ventricular pressure (dP/dt).

64 ak positive and negative first derivative of left ventricular pressure (dP/dt).

65 sure (LVSP), the maximum first derivative of left ventricular pressure (dp/dtmax ), and the slope of

66 obutamine increased the peak rate of rise of left ventricular pressure (+dP/dt) by 49 +/- 8% (p < 0.0

67 on) revealed no differences in heart weight, left ventricular pressure, dP/dt, cardiac index, time co

68 Left ventricular pressure, dP/dt40, negative dP/dt and c

69 of discordance between right ventricular and left ventricular pressures during inspiration, a sign of

70 mic contracture as indicated by increases in left ventricular pressure from 9+/-3 to 33+/-6 mm Hg (p

71 ed animal weight, mean impact velocity, mean left ventricular pressure generated by the blow, mean QR

72 In multivariable analysis, mean left ventricular pressure generated by the blow, mean QR

73 Peak left ventricular pressure generated by the chest blow wa

74 poxia neuromodulates the heart and increases left ventricular pressure generating capacity.

75 ealed that a single-session of AIH increased left ventricular pressure generation and arterial blood

76 decreases in heart rate and rate of rise in left ventricular pressure, improvement of regional coron

77 These previous studies, however, used left ventricular pressure in formulas that assumed the a

78 Left ventricular pressures in vivo and cardiomyocyte sar

79 cardial function, as measured by the rate of left ventricular pressure increase at 40 mm Hg, left ven

80 was significantly less depression in rate of left ventricular pressure increase measured at a left ve

81 Left ventricular pressure, rate of left ventricular pressure increase measured at a left ve

82 tricular end-diastolic pressure, the rate of left ventricular pressure increase measured at a left ve

83 Upon administration of NA, heart rate and left ventricular pressure increased, and activation reco

84 Left ventricular pressure is 2-fold higher than wild typ

85 We demonstrate that left ventricular pressure is closely linked to KATP chan

86 The AHR (maximum rate of left ventricular pressure [LV-dP/dt(max)]) was assessed

87 Left ventricular pressure (LVP) and ECG were monitored d

88 In Cohort 2, we assessed left ventricular pressure (LVP) during stimulation and r

89 modynamic testing to determine the change in left ventricular pressure maximal first derivative (LV d

90 rbital-anesthetized intact dogs arterial and left ventricular pressure (Millar) and left ventricular

91 stimulation parameters, Pt was calculated as left ventricular pressure minus balloon luminal pressure

92 ardiography (n = 4 per group), and right and left ventricular pressure (n = 5 and n = 4 per group, re

93 Significant reductions in left ventricular pressures occurred in vivo and in cardi

94 The volume intercept at left ventricular pressure of 100 mm Hg increased from 43

95 cular end-systolic volume at an end-systolic left ventricular pressure of 100 mm Hg.

96 he rate of left ventricular pressure rise at left ventricular pressure of 40 mm Hg (dP/dt40) and fall

97 ventricular pressure increase measured at a left ventricular pressure of 40 mm Hg (dP/dt40), rate of

98 ventricular pressure increase measured at a left ventricular pressure of 40 mm Hg (dP/dt40), rate of

99 ventricular pressure increase measured at a left ventricular pressure of 40 mm Hg, and the maximum r

100 vative of the left ventricular pressure at a left ventricular pressure of 50 mm Hg, rate-pressure pro

101 rease in LVdP/dtmax (maximal rate of rise of left ventricular pressure) of >/=90% of the maximum LVdP

102 orbidities, such as hypertension, leading to left ventricular pressure overload and adverse remodelin

103 linical models of aortic stenosis can induce left ventricular pressure overload and coarsely control

104 sphorylation increased to 23% in response to left ventricular pressure overload as compared with 7% p

105 normal postnatal cardiac growth, concurrent left ventricular pressure overload hypertrophy did not s

106 ntricular function despite persistent severe left ventricular pressure overload in ascending aortic-b

107 Left ventricular pressure overload in mouse working hear

108 We induced persistent left ventricular pressure overload in rats by ascending

109 ardiac fibroblast cell cycle and fibrosis in left ventricular pressure overload induced by transaorti

110 Transient activation of AMPK preceding left ventricular pressure overload reduces adverse remod

111 A prolonged state of left ventricular pressure overload, commonly caused by h

112 improved cardiac dysfunction in animals with left ventricular pressure overload.

113 governs the timing and extent of fibrosis in left ventricular pressure overload.

114 l oxidative stress in response to persistent left ventricular pressure overload.

115 d precipitated a robust fibrotic response to left ventricular pressure overload.

116 icular (LV) inotropic effects (adjusted peak left ventricular pressure rate of rise (dP/dt)max/P, 21.

117 Left ventricular pressure, rate of left ventricular pres

118 ary bypass, the average intraoperative right/left ventricular pressure ratio was 55% +/- 13%.

119 High-fidelity left atrial and left ventricular pressure recordings with simultaneous D

120 ll thickening as well as the maximal rate of left ventricular pressure rise (+dP/dt) and ventricular

121 he onset of sepsis, the maximal rates of the left ventricular pressure rise (+dP/dtmax) and fall (-dP

122 to LVP and BiVP (% change in maximal rate of left ventricular pressure rise [LVdP/dtmax]) was measure

123 ll thickening as well as the maximal rate of left ventricular pressure rise and fall (+dP/dt and -dP/

124 Cardiac index and the rate of left ventricular pressure rise at left ventricular press

125 ntricular systolic pressure, maximal rate of left-ventricular pressure rise and decline (+dP/dt, -dP/

126 alues of key parameters such as arterial and left ventricular pressures, serum lipoprotein, and other

127 ted with intracardiac transducers to measure left ventricular pressure, sonomicrometer crystals in th

128 Direct curve fitting to the left ventricular pressure trace by Levenberg-Marquardt r

129 rocardiogram, left ventricular pressure, and left ventricular pressure value of 40 mm Hg were continu

130 weeks followed by a terminal measurement of left ventricular pressure volume loops.

131 Left ventricular pressure-volume (PV) loop measurement p

132 mechanical function was characterized using left ventricular pressure-volume compliance measurements

133 kit(-/-) mice, and that the leftward shifted left ventricular pressure-volume curve in the MHCsTNF/c-

134 rtery catheterization to define Starling and left ventricular pressure-volume curves.

135 ion without significant myocardial necrosis (left ventricular pressure-volume curves; 1% triphenyltet

136 (n=9), and Wistar-Kyoto rats (n=9) underwent left ventricular pressure-volume loop evaluation and syn

137 iovascular function in the form of right and left ventricular pressure-volume loops and ventricular p

138 Cardiac function was assessed with left ventricular pressure-volume loops during implantati

139 Left ventricular pressure-volume relations were measured

140 -induced myocardial dysfunction by improving left ventricular pressure-volume relationship.

141 Left ventricular pressure-volume relationships utilizing

142 Left ventricular pressure-volume relationships were asse

143 Left-ventricular pressure-volume analyses in adult homoz

144 lenge (dobutamine 0.3-10 mug/kg/min) using a left ventricular pressure/volume catheter.

145 ronically instrumented to measure aortic and left ventricular pressures, wall thickness, and left cir

146 Left ventricular pressure was monitored.

147 ith either endotoxin or saline, systemic and left ventricular pressures were measured, and the first

148 High fidelity measures of left atrial and left ventricular pressures were obtained simultaneously

149 ed rat model of full thickness scald injury, left ventricular pressures were recorded in vivo followe

150 nce imaging, the rats were catheterized, and left ventricular pressures were recorded.

151 racic echo Doppler studies, and closed-chest left ventricular pressures with direct left ventricular