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1 nction and a surrogate marker of significant left to right shunts.
2 lar side of a ventricular septal defect with left-to-right shunting.
3 lary wedge pressure reductions and decreased left-to-right shunting.
6 monary overcirculation attributable to large left-to-right shunts and (2) can predict functional clin
7 ); and stretched ASD sizes were 14 +/- 4 mm (left to right shunts) and 10 +/- 3 mm (right to left shu
10 he intervening septal myocardium, leading to left-to-right shunting in the form of ventricular septal
14 may alter pulmonary vascular function, while left-to-right shunting might compromise systemic perfusi
16 A fails to occur after birth, resulting in a left-to-right shunt of blood and subsequently in death.
17 congenital heart disease causing significant left-to-right shunting of blood are at risk of developin
18 device patency at 6 months was confirmed by left-to-right shunting (pulmonary/systemic flow ratio: 1
21 scribes the repair of aortic arch anomalies, left-to-right shunts, valvular disease, tetralogy of Fal
25 cally confirmed PDA (diameter, >1.5 mm, with left-to-right shunting) who were extremely preterm (<28