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1 mmatory hyperpigmentation, melasma and solar lentigines.
2  the germline RASopathy Noonan syndrome with lentigines.
3 c diagnosis of Noonan syndrome with multiple lentigines and hypertrophic cardiomyopathy between 2002
4  patients with Noonan syndrome with multiple lentigines and hypertrophic cardiomyopathy, with a media
5  patients with Noonan syndrome with multiple lentigines and hypertrophic cardiomyopathy.
6  patients with Noonan syndrome with multiple lentigines and hypertrophic cardiomyopathy.
7                       Poikiloderma, atypical lentigines, and skin cancers, the primary cutaneous feat
8                                           In lentigines- and blue nevi-prone areas from mutant mice a
9 n manifestation of Carney complex, including lentigines, blue nevi, and cutaneous myxomas in unusual
10 orphic developmental abnormalities including lentigines, cardiac defects, short stature and deafness.
11 s with postinflammatory hyperpigmentation or lentigines displayed decreased skin NNT levels, suggesti
12 autosomal dominant disorder characterized by Lentigines, Electrocardiogram abnormalities, Ocular hype
13 onym formed from its main features; that is, lentigines, electrocardiographic abnormalities, ocular h
14                   LEOPARD syndrome (multiple Lentigines, Electrocardiographic conduction abnormalitie
15  reverse transcriptase in skin aging-related lentigines formation.
16 ns of photoaging other than rhytids, such as lentigines, hyperpigmentation, and telangiectasias, shou
17          The differential diagnosis for oral lentigines includes several syndromes with important ass
18 solar exposure, and moderate to severe solar lentigines; it was the most prevalent confocal type in f
19                                     Multiple lentigines/LEOPARD syndrome (LS) is a rare, autosomal do
20   In addition to evidence of multiple GISTs, lentigines, malignant melanoma, and an angioleiomyoma we
21    Noonan syndrome (NS) and NS with multiple lentigines (NSML) cognitive dysfunction are linked to SH
22                Noonan syndrome with multiple lentigines (NSML) is a rare autosomal dominant disorder
23                Noonan Syndrome with Multiple Lentigines (NSML) patients, SHP2 knock-in mice, and ACK1
24  CHD and cause Noonan syndrome with multiple lentigines (NSML), a condition that often presents with
25 ), also called Noonan syndrome with multiple lentigines (NSML), is a rare autosomal dominant disorder
26 s: Noonan syndrome (NS) and NS with multiple lentigines (NSML), respectively.
27  dioxide levels in outdoor air and number of lentigines on the cheek.
28 melanomas, 15 basal cell carcinomas, 7 solar lentigines or seborrheic keratoses, and 3 actinic kerato
29                                        Solar lentigines (SLs) are a hallmark of human skin aging.
30  combination of DMBA and UVB developed solar lentigines within 5 to 10 months of treatment.