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1 reported almost exclusively in patients with lepromatous leprosy.
2 d human polymorphism that is associated with lepromatous leprosy.
3 tana Roo, Mexico, was diagnosed with diffuse lepromatous leprosy.
4             In Brazil, most patients develop lepromatous leprosy, a clinical form characterized by po
5 tured human pathogen associated with diffuse lepromatous leprosy and a reactional state known as Luci
6 eprosum (ENL), which occurs in patients with lepromatous leprosy and is characterized by neutrophil i
7 oor cellular immune response associated with lepromatous leprosy and may have important implications
8                                Patients with lepromatous leprosy are unresponsive to lepromin skin-te
9                                   Lesions in lepromatous leprosy contained macrophages with a regulat
10 in skin lesions of patients with progressive lepromatous leprosy, correlating and colocalizing with I
11 d in a rare form of leprosy known as diffuse lepromatous leprosy (DLL).
12 d in a rare form of leprosy known as diffuse lepromatous leprosy (DLL).
13                                          The lepromatous leprosy granuloma is a dynamic entity requir
14 ymorphonuclear leukocytes from patients with lepromatous leprosy iodinate ingested bacteria normally.
15 cess whereby some patients with disseminated lepromatous leprosy (L-lep) transition toward self-limit
16 in Winchester, UK, showing skeletal signs of lepromatous leprosy (LL) have been studied using a multi
17 alterations were most frequently observed in lepromatous leprosy (LL) patients.
18         In histological sections (n = 10), 1 lepromatous leprosy (LL), 1 DLL, and 3 Lucio reactions c
19         In histological sections (n=10), one lepromatous leprosy (LL), one DLL, and three Lucio react
20                        The chronic course of lepromatous leprosy may be interrupted by acute inflamma
21 ular exhaustion to the hyporesponsiveness of lepromatous leprosy patients by evaluating the classical
22 ytes from the blood of either tuberculoid or lepromatous leprosy patients.
23 0-CD40L interaction, which is not evident in lepromatous leprosy, probably participates in the cell-m
24  immune defect leading to the development of lepromatous leprosy resides in the lymphocyte or in the
25             The striking finding was that in lepromatous leprosy, T cells did not efficiently recogni
26   Multibacillary disease is similar to human lepromatous leprosy, with variable/high levels of antibo