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1 er neurologic deterioration in patients with leptomeningeal metastasis.
2 arding methods of diagnosis and treatment of leptomeningeal metastasis.
3 F-liberating proteases that could facilitate leptomeningeal metastasis.
4 ion, perivascular niche micrometastasis, and leptomeningeal metastasis.
5 upting the blood-brain barrier and promoting leptomeningeal metastasis.
6 nd patient-derived humanized mouse models of leptomeningeal metastasis.
7 er neurologic deterioration in patients with leptomeningeal metastasis.
8 e systemic therapy may benefit patients with leptomeningeal metastasis and obviate the need for intra
9 include central nervous system prophylaxis, leptomeningeal metastasis, and common hematologic compli
10 ts increasingly utilized in the treatment of leptomeningeal metastasis are targeted mAbs such as ritu
11 tilized intra-CSF agents in the treatment of leptomeningeal metastasis are targeted monoclonal antibo
13 lide cerebrospinal fluid (CSF) flow study if leptomeningeal metastasis-directed therapy is being cons
14 ed therapeutically beneficial in suppressing leptomeningeal metastasis in these preclinical models.
16 single most important aspect to diagnosis of leptomeningeal metastasis is considering and pursuing th
27 cerebrospinal-fluid-filled leptomeninges, or leptomeningeal metastasis, represents a fatal complicati
29 survival of 2-3 months (15% of patients with leptomeningeal metastasis survive 1 year), treatment may