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1 limiting the spread of HER2(+) LC beyond the leptomeninges.
2 as well as those that restrict its growth to leptomeninges.
3  molecular landscape of the developing human leptomeninges.
4 sfully attempted to eliminate B cells in the leptomeninges.
5  risk of melanoma within the skin, brain, or leptomeninges.
6 llaries of the cerebral cortex and overlying leptomeninges.
7 ection of cortex, white matter and overlying leptomeninges.
8 d to the implant pocket and to adjacent host leptomeninges.
9 n regions, and in white and gray matter, and leptomeninges.
10 four mGATs was present to some degree in the leptomeninges.
11  areas of contrast enhancement involving the leptomeninges.
12  dispersed across the surface of the brain's leptomeninges.
13 ecular properties of cell types within human leptomeninges.
14 r cells to control cancer cell growth in the leptomeninges.
15 unique arachnoid clusters specific to ageing leptomeninges.
16 king of effector T cells from the gut to the leptomeninges after stroke.
17 rogeneity as medulloblastoma metastasizes to leptomeninges and as it evolves in the face of radiation
18  arachnoid and pia mater cells that form the leptomeninges and border the subarachnoid space (SAS) fi
19 tia of small arteries and capillaries of the leptomeninges and cerebral cortex and is a major cause o
20 ulates predominantly in the small vessels of leptomeninges and cerebral cortex, leading to fatal stro
21  dissemination of malignant cells within the leptomeninges and cerebrospinal fluid.
22 d certain intracranial malignancies into the leptomeninges and cerebrospinal fluid.
23 lso was detected in the lung, as well as the leptomeninges and choroid plexus of the fetal brain.
24 nt strategies that can target B cells in the leptomeninges and CNS parenchyma.
25 econd most common finding was involvement of leptomeninges and cranial nerves, which manifested as ab
26  adherence of different Neisseria species to leptomeninges and cultured cells.
27        Macrophages are widely present in the leptomeninges and dura mater of meninges.
28                  Flatmounts of the dissected leptomeninges and dura were used to facilitiate high-qua
29            It was, however, expressed in the leptomeninges and in a subpopulation of brain blood vess
30 c cells (DCs) accumulated in the dorsal root leptomeninges and led to an increase in kynurenine level
31  She had extensive amyloid deposition in the leptomeninges and liver as well as the involvement of th
32 d a specific predilection for binding to the leptomeninges and meningeal blood vessels in human brain
33 ly more serotype A than B spirochetes in the leptomeninges and more serotype B than A spirochetes in
34     This Review describes the anatomy of the leptomeninges and routes of cancer spread to the CSF.
35  increased Fos immunoreactivity in the basal leptomeninges and several regions implicated in autonomi
36 ers was then compared to cells cultured from leptomeninges and to two other types of endothelial cell
37 cells produce metastatic lesions only in the leptomeninges and ventricles.
38 ne patient developed a relapse in the spinal leptomeninges and was rendered free of disease with spin
39 ll lymphoma in which the brain, spinal cord, leptomeninges and/or eyes are exclusive sites of disease
40 l anomalies, and abnormalities of the brain, leptomeninges, and eye.
41 opment of murine BAMs in the choroid plexus, leptomeninges, and perivascular spaces required CSF-1, w
42 lary-associated pericytes, astroglial cells, leptomeninges, and the choroid plexus.
43  in the brain localized predominantly to the leptomeninges, and those in peripheral tissues localized
44 resident within the parenchyma of the brain, leptomeninges, and vascular beds, as well as through sec
45 Thus, the data demonstrate that cells of the leptomeninges are not inert but are active participants
46  of Neisseria meningitidis with cells of the leptomeninges are pivotal events in the progression of b
47                    They were detected in the leptomeninges around the neural tube, and only rarely we
48  inflammation in the cerebrospinal fluid and leptomeninges before the parenchyma.
49  in multiple tissues, including the cerebral leptomeninges, brainstem, peripheral nerves from both fo
50    Although any malignancy can spread to the leptomeninges, breast cancer, particularly the HER2(+) s
51 ated with the cerebral blood vessels and the leptomeninges by immune stimuli such as intravenous admi
52 ith MB-MAFs enhances the colonization of the leptomeninges by medulloblastoma cells and promotes the
53                                              Leptomeninges, consisting of the pia mater and arachnoid
54 or cells, as well as explant cultures of the leptomeninges, cortical slice cultures and brain tumor c
55 formed on mRNA isolated from the human fetal leptomeninges detected expression of the G protein-coupl
56 ly, TTR amyloid deposits were present in the leptomeninges, especially the leptomeningeal vessels, an
57  within the cerebrospinal fluid (CSF)-filled leptomeninges face substantial microenvironmental challe
58            NPs were well retained within the leptomeninges for over 3 weeks, showing preference for v
59 uR2/3, mGluR4a, and mGluR7 were expressed in leptomeninges from adult rats.
60 gle nuclei profiling of dissected postmortem leptomeninges from aged individuals.
61                          Inflammation in the leptomeninges has been identified as a key feature of se
62 cellular heterogeneity within the developing leptomeninges, identifying distinct subpopulations of fi
63 a melatonin receptor expression in the fetal leptomeninges implies that melatonin may play a role in
64 and thus potential barrier properties of the leptomeninges in controlling access of immune mediators
65 esenting cells (APCs) within the spinal cord leptomeninges in experimental autoimmune encephalomyelit
66 has also been observed in choroid plexus and leptomeninges in normal mouse brain.
67 ells was used to investigate the role of the leptomeninges in the inflammatory response.
68 ed it to amyloid in the neuritic plaques and leptomeninges in the same patients.
69 ecrease in the degree of inflammation of the leptomeninges in these mice.
70 NS relapse in the brain parenchyma, eyes, or leptomeninges is an uncommon but devastating complicatio
71 erature shows that amyloid deposition in the leptomeninges is not uncommon in TTR amyloidoses clinica
72  The structural organization of the dura and leptomeninges is reflected in its magnetic resonance (MR
73 1 (SDF1) (CXCL12), which is expressed by the leptomeninges, is necessary and sufficient to cause marg
74 t fibroblast-like transcriptomes in cerebral leptomeninges; link them to anatomically distinct cell t
75          Tumors metastasizing to the CNS and leptomeninges (LM) are associated with significant morta
76 BCC) metastasis and proliferation within the leptomeninges (LM) are poorly understood, which limits t
77              In multiple sclerosis (MS), the leptomeninges (LM) are populated with immune cell aggreg
78       B cells are known to accumulate in the leptomeninges of MS patients and associate with patholog
79 ve, abnormal capillary venous vessels in the leptomeninges of the brain and choroid, glaucoma, seizur
80 Weber Syndrome (SWS), CMs occur in the skin, leptomeninges of the brain, and choroid of the eye.
81 with the exception of the choroid plexus and leptomeninges of the brain, where it is expressed from b
82 cular malformations affecting skin, eyes and leptomeninges of the brain, which can lead to glaucoma,
83 es except adult liver and the choroid plexus/leptomeninges of the central nervous system where IGF-II
84 alisation of Mel1a mRNA transcripts over the leptomeninges of the fetal brain.
85 nstrated enhancement of the cauda equina and leptomeninges of the lower spinal cord.
86            Rare spirochetes were seen in the leptomeninges of two cases by immunohistochemistry.
87 enchyma but did not induce metastasis to the leptomeninges or ventricles.
88 yma, without a decrease in metastasis to the leptomeninges or ventricles.
89 Metastasis to the cerebrospinal-fluid-filled leptomeninges, or leptomeningeal metastasis, represents
90 ignancy that affects the brain, spinal cord, leptomeninges, or vitreoretinal space, without evidence
91  cells of the pia and arachnoid mater of the leptomeninges over large areas of the cerebral hemispher
92 cortex (P<0.001) and seven times more in the leptomeninges (P = 0.013); among the affected blood vess
93 l wall in the cortex (P = 0.001), and in the leptomeninges (P = 0.015).
94 the unimmunized cases (cortex: P = 0.009 and leptomeninges: P = 0.002).
95 ion was explored in blood vessels located in leptomeninges (pial vessels) and brain parenchyma (paren
96                                          The leptomeninges play a pivotal role in the CNS, serving bo
97 the subarachnoid space and indicate that the leptomeninges play an important role in experimental aut
98 ly, severe meningitis with thickening of the leptomeninges, prominent vasculitis, and encephalitis wa
99              Anatomical understanding of the leptomeninges provides a roadmap of corridors for cancer
100                     Additionally, ECs in the leptomeninges redistribute CLDN5 and PECAM1, and leptome
101                This study identifies unique, leptomeninges-specific IFNgamma signalling and suggests
102                                          The leptomeninges, subpial astrocytes and astrocytes ensheat
103 nescence, with rare metastatic spread to the leptomeninges, suggesting roles for MYCN in both progres
104 lls spread to cerebrospinal fluid-containing leptomeninges surrounding the brain and spinal cord, a c
105 spread to the cerebrospinal fluid-containing leptomeninges surrounding the brain and spinal cord.
106 tonin binding sites were identified over the leptomeninges surrounding the human fetal brain using qu
107 ormative landmark for in vivo imaging of the leptomeninges that can be used to visualize the borders
108 sclerosis have extensive inflammation in the leptomeninges that is associated with increased subpial
109  neuropathy have TTR amyloid deposits in the leptomeninges, the brain parenchyma, and the eye.
110 cement of the parenchyma (three of 20; 15%), leptomeninges (three of 20; 15%), cranial nerves (two of
111 expression of Igf2 in the choroid plexus and leptomeninges, tissues where the gene is thought not to
112 onsistent with the barrier properties of the leptomeninges to N. meningitidis observed in vivo.
113 ositum (VI) is a membranous extension of the leptomeninges under the hippocampus.
114  this futile anti-tumour response within the leptomeninges, we developed syngeneic lung cancer, breas
115 hylaxis or extended intrathecal treatment if leptomeninges were involved.
116 at echoviruses specifically replicate in the leptomeninges, where they induce profound inflammation a
117 tic disease to the central nervous system or leptomeninges who met eligibility criteria for (131)I-om
118 macrophages in the dura mater but not in the leptomeninges, without affecting MLVs or CSF drainage.

 
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