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1 ng) was quantified by autoradiography in DRN-lesioned animals.
2 rats and in the intact hemispheres of 6-OHDA-lesioned animals.
3 m or in the unlesioned hemispheres of 6-OHDA-lesioned animals.
4 ess, a small day/night rhythm was present in lesioned animals.
5 ic activity were observed only in the 6-OHDA lesioned animals.
6  patterns in forebrain regions of intact and lesioned animals.
7 ion patterns in the forebrains of intact and lesioned animals.
8 lent levels of True Blue label in intact and lesioned animals.
9  putamen, thalamus, and amygdala of the MPTP-lesioned animals.
10 nimals, and CDP improving the performance of lesioned animals.
11 nding appeared by day 15 in both control and lesioned animals.
12 into the lateral ventricle of fimbria-fornix lesioned animals.
13 ffect on impaired performance in hippocampal lesioned animals.
14 lls, and ultimately improved the behavior of lesioned animals.
15 on in REM sleep time was observed in the SOM-lesioned animals.
16  of correlations at large time scales in SCN-lesioned animals.
17 uppression of learning-induced plasticity in lesioned animals.
18 t as severe as those shown by the dorsal CA1 lesioned animals.
19 ocations relative to control and ventral CA1 lesioned animals.
20 both D2R and D3R relative to vehicle-treated lesioned animals.
21 y was not observed in either the LMN- or DTN-lesioned animals.
22 ree M. bovis strains were recovered from non-lesioned animals.
23 trast, LTD was preserved in nicotine-treated lesioned animals.
24 duced in BLA-lesioned animals but not in CeA-lesioned animals.
25 de was collected from a random sample of non-lesioned animals.
26 s mild compared with cells recorded from PoS-lesioned animals.
27 re likely to choose the LR arm than the sham-lesioned animals.
28 essed relative to that in the shell- or sham-lesioned animals.
29 mRNA, but NPY mRNA was reduced in the ARC of lesioned animals.
30 anges in DA similar to those seen in the non-lesioned animals.
31 al root ganglion (DRG) neurons in intact and lesioned animals.
32 iate in low-lesioned, and smallest in highly lesioned animals.
33 atic induction of c-Fos in the CPu in 6-OHDA-lesioned animals.
34 representations in PoS-lesioned, but not ADN-lesioned, animals.
35       At 1 month after surgery, successfully lesioned animals (3 or less forelimb akinesia score, and
36 n throughout the dorsal striatum of neonatal lesioned animals, a response not observed within the int
37                                              Lesioned animals also showed diminished activity at base
38                                 Only 2 of 11 lesioned animals and 6% of slices from cortex exposed to
39  control groups consisted of a group of sham lesioned animals and a group of animals that had unilate
40 mmatory responses were similar in normal and lesioned animals and the acetylcholinesterase inhibitor,
41 formation relative to control and dorsal CA1 lesioned animals, and a mild deficit for the temporal or
42  binding was evident in the striatum of MPTP lesioned animals as compared with the control group.
43 oxylase mRNA increased in 2 and 12-month-old lesioned animals both 3 and 10 days post-treatment.
44  VC lesions produced behavior similar to VLF-lesioned animals but did not significantly affect tcMMEP
45  after training were severely reduced in BLA-lesioned animals but not in CeA-lesioned animals.
46 oral arrest could be reconstituted in fornix-lesioned animals by inducing synchronous activity in the
47                                           In lesioned animals, chronic amitriptyline (AMI; 5 mg/kg) t
48  in neocortical areas of both hemispheres of lesioned animals compared with protein levels of sham-op
49 e, was significantly impaired in hippocampus-lesioned animals compared with sham control animals.
50                                     Some DTN-lesioned animals contained cells whose firing rates were
51                               Unlike acutely lesioned animals, continuously infused rats revealed no
52 ntly altered, suggesting that smaller CPs in lesioned animals could be largely attributable to greate
53                             Nonetheless, OFC-lesioned animals could still approximate choice-outcome
54 ransplanting fetal SCN into the brain of the lesioned animal, demonstrating the first two of the esse
55                    Although place cells from lesioned animals did not differ from controls on many pl
56                           The performance of lesioned animals did not differ significantly from sham-
57                             By contrast, ACC-lesioned animals did not show this additional budget eff
58  pattern to avoid the satiated food, whereas lesioned animals did not, suggesting that neonatal hippo
59  nucleus (VPL) in thalamic brain slices from lesioned animals displayed an increased probability of b
60                     In contrast, hippocampal-lesioned animals displayed impairments across all spatia
61                                      Hearing-lesioned animals displayed tinnitus with a pitch in the
62  administration of apomorphine to the 6-OHDA lesioned animal evoked a robust and long-lasting excitat
63        When re-tested four weeks later, sham-lesioned animals exhibited long-term memory; in contrast
64 mpaired in BLA-lesioned animals, whereas CeA-lesioned animals exhibited only mild deficits.
65                       ANFs from successfully lesioned animals exhibited significant pathophysiology c
66         An alternative hypothesis is that AM-lesioned animals fail to acquire certain fear CRs simply
67  patterns showed that, unlike sham rats, ACC-lesioned animals failed to maximize session-bundle utili
68 ippocampal deafferentation in both groups of lesioned animals failed to prevent the accumulation of G
69          Additional cells were recorded from lesioned animals for up to 3 weeks after the lesion.
70                                          UNI lesioned animals from 6-36 days post-lesion showed no ne
71                                     Thus, in lesioned animals, Galpha(olf) upregulation is critical f
72   As compared with control animals, amygdala-lesioned animals had blunted responding to both positive
73                                              Lesioned animals had increased food intake in light and
74                                              Lesioned animals had reductions in 5-HT in the NA, and D
75                                HD cells from lesioned animals had similar firing properties compared
76 tion of nerve growth factor (NGF) to aged or lesioned animals has been shown to reverse the atrophy o
77              The absence of sensitization in lesioned animals implies that, before physiological func
78 iorate a characteristic phenotype of the DAN-lesioned animals in a marble burying task demanding high
79 i.p.) improved passive avoidance behavior in lesioned animals in a mecamylamine-sensitive manner.
80               Residual Fos induction seen in lesioned animals in response to higher doses of LPS prov
81 b in the food-restricted, methyl bromide gas-lesioned animals indicates that the mechanisms that guid
82                                       In NBM-lesioned animals, k* was elevated significantly (by up t
83                                              Lesioned animals learned normally after multiple shocks,
84  nerve fiber (ANF) recordings were made from lesioned animals, lesion shams, and normal controls.
85 a high degree of recovery can be observed in lesioned animals, like in human hemicord patients.
86                          Further, cells from lesioned animals maintained a stable preferred firing di
87 spatial correlates of complex spike cells in lesioned animals may rely on a more limited set of senso
88 e of CS preexposure), Novelty in hippocampal lesioned animals might be larger, equal, or smaller (cor
89                     In the 6-hydroxydopamine-lesioned animal model of Parkinson's disease, 5 altered
90 uced by apomorphine in the 6-hydroxydopamine-lesioned animal models of Parkinson's disease were studi
91 ibited long-term memory; in contrast, the TA-lesioned animals no longer showed target quadrant prefer
92                                              Lesioned animals not receiving Fluoro-Gold exhibited the
93 ion of the task was significantly delayed in lesioned animals of either lesion direction.
94 animals with mPFC lesions compared with sham-lesioned animals on the first day of retesting in all fi
95                                  Hippocampal lesioned animals performed poorly on the task and inject
96 DP in the septal lesioned subjects, with the lesioned animals performing worse than control animals,
97 ral asymmetries (compared to nonimmobilized, lesioned animals), presumably attributable to mild disus
98  to the increased locomotion observed in non-lesioned animals, rats pretreated with 6-OHDA showed no
99                  Separate groups of sham and lesioned animals received either 50% or 25% reinforcemen
100  FGF-2 and compared it with that observed in lesioned animals receiving infusate controls.
101 elay-discounting curves was observed in wOFC-lesioned animals relative to shams--differences that dis
102                                           In lesioned animals, retrograde tracing revealed MT-project
103 resentation of CS alone), onset responses in lesioned animals returned to their preconditioning firin
104  was identical to that of the experimentally lesioned animals revealed a bilateral ischemic lesion re
105                   However, the same amygdala-lesioned animals showed a complete blockade of fear-pote
106                                          The lesioned animals showed a delay in the onset of desensit
107 -term memory was examined, both sham- and TA-lesioned animals showed a significant preference for the
108  following conditioned fear stress, habenula-lesioned animals showed decreased PPI which normalized w
109                               Pretrained HCX-lesioned animals showed deficits similar to those of HIP
110                                      The nBM-lesioned animals showed initial acquisition impairment i
111 by damage to the auditory nerve, because the lesioned animals showed intact compound action potential
112               Most notably, HD cells in dLGN-lesioned animals showed little ability to distinguish hi
113                              Pretrained HIPP-lesioned animals showed marked delay-dependent deficits
114                                   The OBs of lesioned animals showed marked expansions of 2A4(+)ORN b
115                             Surprisingly, AN-lesioned animals showed no difference in hearing perform
116                     In contrast, hippocampal-lesioned animals showed normal (slowed) learning.
117                   In addition, HD cells from lesioned animals showed normal responses to two environm
118                                     However, lesioned animals showed reduced cocaine-seeking during c
119                              In contrast, BI lesioned animals showed severe spatial learning deficits
120                                           TA-lesioned animals still exhibited a deficit in long-term
121                                       In the lesioned animals, striatal DA ipsilateral to 6-OHDA infu
122                                   In the non-lesioned animals, striatal DA was reduced and striatal D
123 l dopamine release from synaptosomes of MPTP-lesioned animals, suggesting that nicotine treatment had
124                     Although fields from ADN-lesioned animals tended to have less landmark control th
125 r-expression of both D2R and D3R compared to lesioned animals that received blank vector.
126                                          The lesioned animals that received BMP7 pretreatment, as com
127 alysis experiments indicated that only those lesioned animals that received the mixture of MD-TH and
128 ies, revealed important factors operating in lesioned animals that were either absent or insignifican
129 tral forelimb area compared with that in the lesioned animals that were not rehabilitated.
130                            A subset of these lesioned animals then had a cast applied for 7 d to the
131                                           In lesioned animals there were several differences in the a
132 alamic nucleus HD cells was still present in lesioned animals; thus, this property cannot be attribut
133 of ventrolateral PFC and the failure of LPFC-lesioned animals to disengage from the immediately prece
134               Another group of HIPP- and HCX-lesioned animals trained on the tasks after the lesion s
135 2 activation was confirmed in vivo in 6-OHDA-lesioned animals treated systemically with SKF38393.
136 tic assumptions of decision theory, the mOFC-lesioned animals' value comparisons were no longer indep
137 rection remained stable across days when the lesioned animal was placed into a novel environment.
138 halamic nucleus, c-fos mRNA induction in the lesioned animals was abolished in the bed nucleus of the
139 read of visual corticopontine connections in lesioned animals was greatly increased relative to unles
140 he performance of both the lesioned and sham-lesioned animals was impaired by the presentation of a v
141      In addition, the responding of amygdala-lesioned animals was insensitive to the omission of the
142 g followed by surgery, it was found that all lesioned animals were able to remember the sequence.
143                The responses of the amygdala-lesioned animals were compared with a group of age-match
144 quisition and overall response rates in core-lesioned animals were depressed relative to that in the
145  simultaneous olfactory discrimination; PRER-lesioned animals were dramatically and persistently impa
146 roximately 3 weeks of age, lesioned and sham-lesioned animals were either tested for the display of p
147                                              Lesioned animals were exposed to moderate doses of ethan
148                                              Lesioned animals were highly impaired in recall and acqu
149 ment with our prediction, postsurgery, VLPFC lesioned animals were impaired in performing a series of
150                             In contrast, OFC lesioned animals were impaired regardless of whether the
151               Surprisingly, place cells from lesioned animals were more likely modulated by the direc
152 ndmark cue showed that place fields from PoS-lesioned animals were not controlled by the cue and shif
153 e third and fourth days of retraining, these lesioned animals were performing at a level comparable t
154 preferred direction drifted when HD cells in lesioned animals were recorded in the dark.
155 e assessed for neuronal differentiation, and lesioned animals were tested for amphetamine-induced rot
156 ition) in unlesioned rats and in cortices of lesioned animals were unaffected by priming.
157                                 In contrast, lesioned animals were unimpaired when responding on a pr
158 responses were significantly impaired in BLA-lesioned animals, whereas CeA-lesioned animals exhibited
159 tions of muscimol was markedly attenuated in lesioned animals, whereas the inhibition by VTA injectio
160 ase was similar to control in unlesioned and lesioned animals with chronic oral nicotine.
161 nition or motor function in symptomatic MPTP-lesioned animals with deficits in both of these areas.
162                   When both non-lesioned and lesioned animals with different ages were pooled for lin
163                                              Lesioned animals with ectopic grafts or sham surgery as
164 ditioned response (CR) more slowly than sham-lesioned animals with either the 1.5-s ISI or with the 2
165 R and D3R binding in the ventral striatum of lesioned animals with lentiviral over-expression of both
166 irole generated only modest motor effects in lesioned animals with sole over-expression of D2R or D3R
167 imb and produced robust circling behavior in lesioned animals with striatal over-expression of both D
168 in expression was significantly increased in lesioned animals within proximal and distal regions of p
169 ute and transient working memory deficits in lesioned animals without effect in unlesioned controls.
170                                              Lesioned animals without transplants maintained higher t
171 ar reduction of trkC probed hybridization in lesioned animals without transplants.

 
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