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1 ese lesions approached those of pre-existing leukoaraiosis.
2 at tiny silent acute infarcts are a cause of leukoaraiosis.
3 SIVD), multiple infarcts, mixed dementia and leukoaraiosis.
4 ventricle width depending on the severity of leukoaraiosis.
5 st of potential poor outcomes in people with leukoaraiosis.
6 n and more diffuse ischaemia, referred to as leukoaraiosis.
7 tter changes, with the imaging appearance of leukoaraiosis.
8 s: isolated lacunar infarction and ischaemic leukoaraiosis.
9 stronger risk factor in those with ischaemic leukoaraiosis [12.92 (95% CI 4.40-37.98), P < 0.0005) pe
15 is the relationship between the extension of leukoaraiosis and severity of ischaemic stroke and brain
17 risk factor for SVD, particularly ischaemic leukoaraiosis, and this effect may be mediated via endot
18 MRI scans were visually scored for degree of leukoaraiosis, central atrophy, and cortical atrophy.
19 ed lacunar infarction (n = 47) and ischaemic leukoaraiosis, defined as a clinical lacunar stroke and
20 ions that stroke outcomes may be affected by leukoaraiosis differentially depending on stroke subtype
21 es present an association between the degree leukoaraiosis extension and brain atrophy, but no associ
22 lele was a risk factor only in the ischaemic leukoaraiosis group [odds ratio (OR) 2.02 (95% CI 1.31-3
25 (MRI) white matter hyperintensities (WMH; or leukoaraiosis) in patients with high vascular amyloid de
31 althy elderly participants with an aggregate leukoaraiosis lesion volume of more than 25 cm(3) and 18
39 th the number of lacunes (P = 0.008) and the leukoaraiosis score (P = 0.03), but TF levels and the TF
42 ntrol participants with less than 5 cm(3) of leukoaraiosis underwent functional MR imaging to allow c
45 g confirmed lacunar stroke with radiological leukoaraiosis were recruited and completed cognitive tes
46 e patients with lacunar stroke and confluent leukoaraiosis were recruited into the ongoing SCANS stud