ライフサイエンスコーパス: leukocyte

1 immunomodulatory drug on brain-infiltrating leukocytes.

2 mouse disease model with brain-infiltrating leukocytes.

3 indeed cross-dressed cells and not passenger leukocytes.

4 t and stromal cells, as well as infiltrating leukocytes.

5 howed >80% GFP marking in tumor-infiltrating leukocytes.

6 gnals that modulate the function of effector leukocytes.

7 actions on both the ischemic myocardium and leukocytes.

8 could noninvasively image brain-infiltrating leukocytes.

9 RR) on resident tissue cells and circulating leukocytes.

10 cancer cells and crosstalk with infiltrating leukocytes.

11 larly endothelium, stromal cells, and innate leukocytes.

12 ociated composition and transcriptome of CSF leukocytes.

13 of additional T cells independently of other leukocytes.

14 s, which activate chemokine receptors on the leukocytes.

15 st cells unable to differentiate into mature leukocytes.

16 i-VCAM/liposomes bind to endothelium, not to leukocytes.

17 The recruitment of leukocytes, a hallmark of inflammation, is regulated by

18 e injured nerve, macrophages 'eat' apoptotic leukocytes, a process called efferocytosis, and thereby

19 hibition of this receptor in mouse and human leukocytes abrogated the pro-resolving actions of RvD5n-

20 ause of myocardial infarction and stroke, is leukocyte accumulation in the arterial wall.

21 gnificant reductions in vascular disruption, leukocyte accumulation, and demyelination.

22 ttracting chemokines, and enhanced pulmonary leukocyte accumulation.

23 te adhesion to endothelial cells and guiding leukocytes across the vascular wall.

24 heir pathway marker, 17-HDHA, are markers of leukocyte activation and inflammation in morbid obesity

25 ent, type I and II interferon signaling, and leukocyte activation pathways.

26 op of organ damage, due to vasoconstriction, leukocyte adherence, and activation of the immune respon

27 Functionally, leukocyte adhesion and integrin activation are abrogated

28 Intravital microscopy showed decrease in leukocyte adhesion and rolling after ethanol consumption

29 ound LYVE-1 molecules and thereby facilitate leukocyte adhesion and transmigration.

30 Immune cells from patients with leukocyte adhesion deficiency type 1 (LAD-1) had reduced

31 ition of KDM7A and UTX significantly reduces leukocyte adhesion in mice, establishing the biological

32 rm during inflammatory processes and mediate leukocyte adhesion in the synovial fluids of arthritis p

33 tation of ICAM and VCAM expression, elevated leukocyte adhesion to and migration across BMVEC monolay

34 AM-1 is best known for its role in mediating leukocyte adhesion to endothelial cells and guiding leuk

35 in atherosclerotic plaques, blocks arterial leukocyte adhesion, and inhibits atherosclerosis and inf

36 scular permeability, tissue edema, augmented leukocyte adhesion, platelet aggregation, and dysregulat

37 ith SFA or inhibition of autophagy increased leukocyte adhesion, whereas treatment with metformin dec

38 ent with metformin decreased the SFA-induced leukocyte adhesion.

39 reased with stress, yet maternal whole blood leukocyte analysis indicated monocytosis and classical M

40 t study was to ascertain if a combination of leukocyte and platelet-rich fibrin (L-PRF) + autogenous

41 We compared pan-leukocyte and sympathetic innervation markers in pancrea

42 gamma receptors (FcgammaRs) are expressed by leukocytes and activate or suppress a cellular response

43 pha(L)beta(2) integrin LFA-1 (CD11a/CD18) of leukocytes and can promiscuously bind and also permeabil

44 fast amoeboid mode, observed exclusively for leukocytes and cancer cells, is characterized by weak ad

45 Patient leukocytes and CD34(+) cells were then isolated, phenoty

46 Tissue damage induces rapid recruitment of leukocytes and changes in the transcriptional landscape

47 b-E430G in the presence of polymorphonuclear leukocytes and complement.

48 o measure arylsulfatase A (ARSA) activity in leukocytes and dried blood spots (DBS) using deuterated

49 ession of immune related genes in peripheral leukocytes and immune-related tissues of dairy calves.

50 of mice led to increased numbers of CD45(+) leukocytes and mitotic markers (phosphorylated histone H

51 and a decrease in the diurnal activation of leukocytes and platelets, as measured by a reduction in

52 that help from the mother, upon transfer of leukocytes and their products via breast milk feeding, g

53 Bacteria, cytokines, leukocytes, and hematopoietic precursors were quantified

54 ized for gas exchange and the trafficking of leukocytes, and is unique to the lung.

55 or deposition was observed mainly in stroma, leukocytes, and tumor vasculature, corresponding to a ro

56 ial biological implant surface disinfectant, leukocyte- and platelet-rich fibrin (L-PRF), on a mature

57 that the critical source of antigen for anti-leukocyte antibodies is in fact the endothelium, which r

58 ear which cell types are bound by these anti-leukocyte antibodies to initiate an immunologic cascade

59 nalyzed humoral immune responses to nonhuman leukocyte antigen (HLA) after cardiac transplantation to

60 MHC (chr6: 29.6-33.1 Mb), imputing 216 human leukocyte antigen (HLA) alleles and 4 complement compone

61 de Polymorphisms (SNPs) and 38 imputed Human Leukocyte Antigen (HLA) alleles were analyzed through a

62 edically important regions such as the human leukocyte antigen (HLA) and killer cell immunoglobulin-l

63 e to determine the titer of individual human leukocyte antigen (HLA) antibody specificities.

64 Efforts to precisely identify tumor human leukocyte antigen (HLA) bound peptides capable of mediat

65 namer and a decamer, all restricted by human leukocyte antigen (HLA) C*08:02.

66 While the relationship of protective human leukocyte antigen (HLA) class I alleles and HIV progress

67 In all human cells, human leukocyte antigen (HLA) class I glycoproteins assemble w

68 eases pathogen resistance is linked to human leukocyte antigen (HLA) class I/II variants and their in

69 signal in the class III region of the human leukocyte antigen (HLA) complex in the South Asian datas

70 ein that are likely to be presented in human leukocyte antigen (HLA) complexes, and discuss the role

71 Chemokine receptor 5 (CCR5), human leukocyte antigen (HLA) DR isotope, and cluster of diffe

72 Human leukocyte antigen (HLA) gene variation is associated wit

73 n thought to arise from alleles of the human leukocyte antigen (HLA) genes at that locus(3-6).

74 uencing studies have demonstrated that human leukocyte antigen (HLA) genes may be expressed in a cell

75 The human leukocyte antigen (HLA) haplotype reference panel used f

76 DSA) directed against mismatched donor human leukocyte antigen (HLA) is a major risk factor for graft

77 ction between inhibitory receptors and human leukocyte antigen (HLA) ligands and bound peptide.

78 However, loss of heterozygosity at the human leukocyte antigen (HLA) locus and loss of chromosome 8p

79 ct ASI to patients expressing specific Human Leukocyte Antigen (HLA) molecules, thus stratifying the

80 These include transcripts encoding human leukocyte antigen (HLA) receptors as well as B cell and

81 et virus-derived peptides presented by human leukocyte antigen (HLA).

82 Antibodies targeting human leukocyte antigen (HLA)/major histocompatibility complex

83 receptor (TCR) recognition of peptide-human leukocyte antigen (pHLA) complexes and is essential for

84 atory T-cell frequency, activated CD38+Human Leukocyte Antigen - DR isotype (HLA-DR)+ CD4 and CD8 T c

85 atibility complex (MHC) class I genes (human leukocyte antigen A [HLA-A], -B, and -C genes) may affec

86 No donor-specific human leukocyte antigen Abs or rejection episodes were noted,

87 art technology employed to assess anti-human leukocyte antigen antibodies (Anti-HLA Ab) for donor-rec

88 use of everolimus, and available anti-human leukocyte antigen antibody data.

89 Human leukocyte antigen class I (HLA-I) molecules are encoded

90 between the T-cell receptor (TCR) and human leukocyte antigen class II (HLA-II).

91 dyslipidemia (P=0.009), class II anti-human leukocyte antigen donor-specific antibodies (P=0.004), a

92 id, which suppresses the expression of human leukocyte antigen E (HLA-E) in cancer cells, thus activa

93 ts associated with DHRs are located in human leukocyte antigen genes and genes involved in drug metab

94 SCCHN risk highlight the importance of human leukocyte antigen loci for oropharyngeal cancer risk, su

95 usceptibility has been associated with human leukocyte antigen locus.

96 r T-cell-mediated rejection and BKVAN, human leukocyte antigen mismatch, cyclosporine therapy, and in

97 characteristics, the identification of human leukocyte antigen risk alleles, and drug-induced prolife

98 icroproteins that are presented by the human leukocyte antigen system.

99 creates a neoepitope presented by the human leukocyte antigen with the A2 serotype (HLA-A2) that has

100 Human leukocyte antigen-DM (HLA-DM) is an integral component o

101 septic shock patients stratified using human leukocyte antigen-DR expression on monocytes (mHLA-DR).

102 lity complex (MHC), a mouse homolog of human leukocyte antigen-E (HLA-E), inhibits antibody-mediated

103 ere compared to CD4(+) responses in 10 human leukocyte antigen-matched persons with HCV spontaneous r

104 n of hematopoietic stem cells from his human leukocyte antigen-matched sister 1 year prior to admissi

105 n of hematopoietic stem cells from his human leukocyte antigen-matched sister 1 year prior to admissi

106 transplantation at age 49 with a 12/12 human leukocyte antigen-matched unrelated donor.

107 evasion of innate immunity.IMPORTANCE Human leukocyte antigens (HLAs) are cell surface proteins that

108 The immunosuppressive human leukocyte antigens HLA-G and HLA-F are expressed on trop

109 Swine leukocyte antigens play indispensable roles in immune re

110 In axotomized DRGs, few hematogenous leukocytes are detected and resident macrophages acquire

111 Understanding how leukocytes are produced and how they contribute to ather

112 -cell activation via the inhibitory receptor leukocyte-associated immunoglobulin-like receptor 1 (LAI

113 s because their use prevented the arrival of leukocytes at sites of acute inflammation.

114 g lipid peroxidation gradients that promoted leukocyte attraction.

115 Leukocytes, B-cell subsets, and DSA were measured using

116 overed from the mouse PJI model and in vitro leukocyte-biofilm co-cultures, we show that bacterial-de

117 ) similarly reduced the level of circulating leukocytes by 12+/-5% (all P<5.0x10(-2)).

118 er regression models, adjusted for age, sex, leukocyte cell composition, and other potential confound

119 immune parameters, such as quantification of leukocyte cell subsets, cytokine and chemokine secretion

120 The LPS + PM group showed increased BALF leukocytes, characterized by increased macrophages, incr

121 II interferon signaling, cell-cell adhesion, leukocyte chemotaxis, and angiogenesis.

122 powerful novel approach to accurately define leukocyte chimerism in a complex organ such as a transpl

123 genitors and erythroid-, megakaryocyte-, and leukocyte-committed progenitors, and we identified CD44,

124 ution of nACD-affected skin identified major leukocyte compositional changes at 48 and 96 hours, incl

125 .15 [1.04-1.27] per mmol/L decrease), higher leukocyte count >=13.0 x 103/muL (2.35 [1.17-4.72]) and

126 luded younger age (0.77 [.69-.85] per year), leukocyte count >=13.0 x 103/muL (2.54 [1.42-4.54]), hig

127 83] x 10(9) cells/L, p = 2.32 x 10(-60)) and leukocyte count (0.218 [0.198, 0.239] x 10(9) cells/L, p

128 -11 cells x 10(9)/L]), a normal differential leukocyte count (74% neutrophils [normal range, 40%-80%]

129 on, ICU admission was predicted by increased leukocyte count (P < .0001), alanine aminotransferase (P

130 AL overexpression was associated with a high leukocyte count (P = 0.007) and was independently associ

131 sely proportional to baseline QTc levels and leukocyte count and directly to basal heart rates(p<0.01

132 ggested an inverse association between total leukocyte count and not only aerobic fitness but also pa

133 e investigated the association between total leukocyte count and physical fitness in a military cohor

134 10 g/dL; normal range, 12-15 g/dL), a total leukocyte count of 14 000 cells per microliter (14 cells

135 Subsequent laboratory analyses comprised leukocyte counting and differentiation, platelet countin

136 (6.50-7.49 x 10(3)/mm(3)) and highest normal leukocyte counts (7.50-9.99 x 10(3)/mm(3)) were correlat

137 otypes were also observed, including reduced leukocyte counts and increased presence of autoimmune di

138 pot, gut-homing CD8 T cells, intraepithelial leukocyte counts, and HLA-DQ2-restricted gluten-specific

139 These analyses and leukocyte deconvolution highlighted neutrophil-mediated

140 Leukocyte deconvolution of nACD-affected skin identified

141 interface, density of placental mononuclear leukocytes decreased with stress, yet maternal whole blo

142 Quantitative analyses of leukocytes demonstrated an increase of proinflammatory L

143 de association analyses for diet quality and leukocyte-derived DNA methylation at over 400 000 CpGs (

144 These mediators exert potent leukocyte directed actions and control vascular inflamma

145 The leukocyte donor/recipient ratio varied with rejection st

146 H started), apoptotic removal of infiltrated leukocytes during the remission phase was markedly accel

147 As endothelial cell-surface EMCN attenuates leukocyte-EC interactions during inflammation, we propos

148 CTLA-4), respectively, on tumor-infiltrating leukocytes eliciting immunosuppression.

149 The negative depletion of antibody-tagged leukocytes enables isolation of potentially viable CTCs

150 croscopy to study endothelial activation and leukocyte-endothelial interactions.

151 Leukocyte entry into the central nervous system (CNS) is

152 d threshold of >=1 malignant cell per 10,000 leukocyte equivalents.

153 ed for the determination of infection enzyme leukocyte esterase (LE) in human synovial (joint) fluid

154 ng quantitative polymerase chain reaction in leukocytes extracted from cord blood shortly after birth

155 Leukocyte extravasation and vascular leakage are both pr

156 ductions of TREM1 signaling, complement, and leukocyte extravasation with stress were reversed by nIL

157 mic inflammatory response (concentrations of leukocytes), feasibility (evaluated by abdominal pain re

158 Neuro-COVID CSF leukocytes featured an enriched interferon signature; ho

159 analysed gene expression in peripheral blood leukocytes for a subset of individuals (n = 390).

160 d 2.0% ripasudil reduced CD45(+)-infiltrated leukocyte frequency, Cd11b and Cd11c mRNA levels, and th

161 Recruitment of leukocytes from blood vessels to inflamed zones is guide

162 Relative to healthy controls, leukocytes from patients with FMF harboring homozygous o

163 omplementary single-cell approaches to study leukocytes from PsA joints.

164 release of stem and progenitor cells and of leukocytes from the bone marrow.

165 n, adhesion to the extracellular matrix, and leukocyte function.

166 A single transcriptomic metric of blood leukocyte gene expression can be used in blunt trauma co

167 However, unlike the related leukocyte HA receptor CD44, which uses ERM and ankyrin m

168 Adipose-resident leukocytes have non-traditional immunological functions

169 w distinct trajectories with regard to their leukocyte, hematocrit, and platelet counts over time.

170 In subgroups, leukocyte homing was blocked by integrin antibodies (n =

171 and stalk antibody titers; peripheral blood leukocyte host gene expression response profiles; daily

172 e, I discuss the newest advances on how milk leukocytes impact early life immunity, with an emphasis

173 with genomic expression patterns from blood leukocytes in humans.

174 eted enrichment of the most potent migratory leukocytes in immunotherapies.

175 may influence levels of mtDNA copy number in leukocytes in Mexican Americans.

176 , the co-existence of these cells with donor leukocytes in the absence of rejection.

177 matopoietic niche via Mcp1 silencing reduced leukocytes in the diseased heart, improved healing after

178 o excessive exudation of plasma proteins and leukocytes in the interstitium, which characterizes seve

179 sive care unit (ICU) and increased number of leukocytes in the pancreatic tissue.

180 fined entry and exit routes, the movement of leukocytes in the peritoneal cavity is largely unknown.

181 disease characterised by the accumulation of leukocytes in the synovium, cartilage destruction and bo

182 (18)F-FAC accumulates in brain-infiltrating leukocytes in this model.

183 rgeted gene expression in tumor-infiltrating leukocytes, including antigen-presenting dendritic cells

184 qPCR further analyzed ex vivo the glomerular leukocyte infiltrate during NTN.

185 The diverse leukocyte infiltrate in atherosclerotic mouse aortas was

186 onal degeneration, necrosis, and mononuclear leukocyte infiltrates, was observed in the dorsal root,

187 ody receptor Notch3 deficiency protects from leukocyte infiltration and organ fibrosis.

188 Notch3 signaling independently orchestrates leukocyte infiltration and organ fibrosis.

189 uced endothelial barrier permeability, lower leukocyte infiltration and reduced activation of the end

190 aining identified the extent of inflammatory leukocyte infiltration early in pressure overload.

191 (+)Ly6c(low)Ly6g(high) cells, but only minor leukocyte infiltration into acutely ischemic-reperfused

192 ucing extravascular albumin leakage or major leukocyte infiltration into the brain.

193 Leukocyte infiltration into the CNS was inhibited in cor

194 Leukocyte infiltration was elevated in the prostates har

195 e tightly controlled to prevent overwhelming leukocyte infiltration, activation, and, consequently, o

196 formation is characterized by inflammation, leukocyte infiltration, and vascular remodeling.

197 n of STAT3 in myeloid cells is essential for leukocyte infiltration, neuroinflammation, and demyelina

198 ue from damage, LPS-induced weight loss, and leukocyte infiltration.

199 tration, PAS800-IL-1Ra significantly reduced leukocyte influx and inflammatory markers in MSU-induced

200 ies have revealed key roles for platelet and leukocyte integrins in adhesion and migration and, there

201 -Gly-Asp (RGD)-binding, laminin-binding, and leukocyte integrins.

202 endogenous fibrinolysis, whereas blockade of leukocyte interaction with the vessel wall is being stud

203 lucidate the mechanisms involved in platelet-leukocyte interaction.

204 nciple function is in trafficking of various leukocytes into sites of bacterial infection and inflamm

205 disorder, based on the entry of inflammatory leukocytes into the CNS where these cells cause demyelin

206 chemotaxis as a mechanism for recruitment of leukocytes into the heart.

207 ent pore-forming toxins that target and kill leukocytes, known collectively as the leukocidins.

208 Finally, subacutely reduced levels of the leukocyte marker CD45 and even greater reduction of the

209 nd in LPS-stimulated whole blood samples and leukocyte membrane fatty acid profiles.

210 Consequently, fewer leukocytes migrated to the ischemic myocardium.

211 Chemokines mediate leukocyte migration and homeostasis and are key targets

212 ing Csf-1 and Lgals3bp, directly involved in leukocyte migration and invasion, were significantly upr

213 toxicity in ALS, and stimulating peripheral leukocyte migration into the brain in inflammatory condi

214 ple components of the built environment with leukocyte mtDNA copy number among 5,502 Mexican American

215 r, BMI, etc.) significantly affect levels of leukocyte mtDNA copy number in Mexican Americans.

216 track the systemic behaviour and dynamics of leukocytes non-invasively in vivo are lacking.

217 ssociated with human platelet number but not leukocyte or hemoglobin levels.

218 c depletion of P2X7Rs in bone-marrow derived leukocytes or pharmacological block of P2X7Rs primarily

219 s how an imbalance in vascular activation by leukocytes outside the airways and lungs may contribute

220 ificant increases in antigen-presenting cell:leukocyte pairings, FOXP3(+) /CD4(+) T cells, Tbet(+) /C

221 hemogram determination and peripheral blood leukocytes (PBLs) isolation.

222 Whether donor leukocytes persist within kidney transplants or play any

223 had poorer survival and experienced loss of leukocytes, platelets, and red blood cells.

224 Neutrophils, polymorphonuclear (PMN) leukocytes, play an important role in the early innate i

225 Notch3 expression on CD45(+) leukocytes plays a prominent role in efficient cell tran

226 ere subjected to histologic changes, hepatic leukocyte population, hepatic transcripts level related

227 lator of a growing number of tissue-resident leukocyte populations.

228 involved in the time-dependent regulation of leukocyte postadhesion in sterile inflammation.

229 of autoantibodies to the neutrophil proteins leukocyte proteinase 3 (PR3-ANCA) or myeloperoxidase (MP

230 ation by reducing endothelial activation and leukocyte recruitment along with limiting proinflammator

231 Leukocyte recruitment and heterocellular aggregate forma

232 TNF deficiency did not affect viral load or leukocyte recruitment but caused severe lung pathology a

233 regulates Mac-1, but not LFA-1, and affects leukocyte recruitment by controlling postadhesion streng

234 m conservation of the receptor signaling and leukocyte recruitment capacities of human MIF by its pla

235 ted signaling pathways are also critical for leukocyte recruitment following wounding in larval zebra

236 During inflammation, leukocyte recruitment has to be tightly controlled to pr

237 Leukocyte recruitment is a universal feature of tissue i

238 A central regulator of leukocyte recruitment is Rac1.

239 nd enhanced proliferation without triggering leukocyte recruitment or overt neuropathology.

240 oding Galpha(i2)), consistent with a reduced leukocyte recruitment previously observed in Gnai2 (-/-)

241 ell spread by L. monocytogenes, 2) defective leukocyte recruitment to infection foci, and 3) producti

242 As in other respiratory infections, leukocyte recruitment to the respiratory system in peopl

243 e Pez ortholog (PTPN21) exhibit a failure in leukocyte recruitment to wounds.

244 lation, hepatic transcripts level related to leukocyte recruitment, and hepatic RNA-seq analysis.

245 CrmD dampened pathology, leukocyte recruitment, and inflammatory cytokine product

246 alyzed the effects of the RacGAP ArhGAP15 on leukocyte recruitment.

247 Leukocyte reduced NADP (NADPH) oxidase plays a key role

248 We demonstrate infiltration of recipient leukocytes, regardless of rejection status, and in toler

249 provide a benchmark dissection of the blood leukocyte 'regulome' that can facilitate prioritization

250 f CNS diseases, but our understanding of CSF leukocytes remains superficial.

251 In this study, we investigated leukocyte responses in a mouse model of AKI and observed

252 allenge through the regulation of antifungal leukocyte responses in mice.

253 Ex vivo leukocyte responses to unrelated stimuli and pathogens w

254 entrations and peripheral blood platelet and leukocyte responses.

255 ta and/or caspase-1 secretion and attenuated leukocyte-smooth muscle cell interactions under high glu

256 sed on transcriptomic studies of circulating leukocytes, specifically patients with a Sepsis Response

257 ls of the innate immune system and the major leukocyte subpopulation in blood.

258 Our aim was to phenotype leukocyte subpopulations and the cytokine milieu in the

259 X3CR1-CCR2 dependent accumulation of diverse leukocyte subpopulations in the CP without inducing extr

260 inflammatory parameters (cytokine levels and leukocyte subpopulations) compared with the control grou

261 nce that alternatively activated, reparative leukocyte subsets and their products can be deployed to

262 These results highlight key immunological leukocyte subsets as well as associated pathways in nACD

263 ol, we evaluated gene expression of purified leukocyte subsets from IC versus NUC-treated patients, a

264 ed the parameters of activation of different leukocyte subsets in COVID-19-infected patients in relat

265 Leukocyte subsets were assessed in nasal mucosa biopsies

266 ominently time-of-day dependent in epidermal leukocytes, suggesting that these cell types play an imp

267 ion of FcgammaRs isolated from primary human leukocytes, summarizes recent efforts to engineer Fcgamm

268 ive adhesion to its ligand hyaluronan in the leukocyte surface glycocalyx.

269 Leukocyte telomere length (LTL) is a heritable biomarker

270 Leukocyte telomere length (LTL) might be causal in cardi

271 ki et al evaluated the role of pretransplant leukocyte telomere length (LTL) on survival outcomes in

272 Leukocyte telomere length (TL) shortens with age and is

273 Leukocyte telomere length, a marker of immune system fun

274 inophils are circulating and tissue-resident leukocytes that have potent proinflammatory effects in a

275 n wounds, arachidonic acid rapidly attracted leukocytes through dual oxidase (Duox) and 5-lipoxygenas

276 lexia-associated splenic atrophy and loss of leukocytes to dramatically improve the endogenous abilit

277 same donor could be cultured with autologous leukocytes to generate a same donor "vessel in a dish" b

278 hat effectively bind HIV and trigger various leukocytes to kill the virus and restrict viral spread.

279 The directed migration of leukocytes to sites of damage or infection is necessary

280 r responses as well as regulate migration of leukocytes to the CNS during disease.

281 activity, led to enhanced ability to attract leukocytes to the infection site, and was able to contro

282 ascular endothelium, regulates permeability, leukocyte traffic, nitric oxide production, and coagulat

283 CCL22 is a key mediator of leukocyte trafficking in inflammatory immune responses,

284 le with COVID-19 is orchestrated by specific leukocyte trafficking molecules, and when uncontrolled a

285 e review the molecular signals orchestrating leukocyte trafficking to airway and lung compartments du

286 ne cell phenotypes, reviewing hematopoiesis, leukocyte trafficking, and innate immune cell accumulati

287 ariates, we found that persistently elevated leukocyte trajectories were not associated with the haza

288 mice are replicated at the circulating blood leukocyte transcriptome has not been demonstrated.

289 lopment of neural inflammation by supporting leukocyte transmigration through the blood-brain barrier

290 ed primary brain endothelial cells inhibited leukocyte transmigration.

291 row chimeras to test the role of IRF3 within leukocytes versus stroma.

292 Production of cytokines by splenic leukocytes were altered in EE-exposed mice.

293 whereas small round parabasal-like cells and leukocytes were associated with late disease (i.e., high

294 Leukocytes were collected every 4 hours for 24 hours fro

295 (BOECs), smooth muscle cells (BO-SMCs), and leukocytes were obtained from four donors.

296 stingly, while equal numbers of inflammatory leukocytes were present in the spinal cord at peak disea

297 grafts were rapidly infiltrated by recipient leukocytes, which recapitulated the liver myeloid and ly

298 It was initially identified in pig leukocytes with a broad-spectrum antibacterial and antiv

299 the laser site and leading to a reduction of leukocytes within the choroid after CNV induction.

300 uced weight and volume, increased density of leukocytes) within the exocrine pancreas in this disease