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1 ed primary brain endothelial cells inhibited leukocyte transmigration.
2 eby controlling endothelial permeability and leukocyte transmigration.
3 E-cadherin's role as a negative regulator of leukocyte transmigration.
4 at Esm1(KO) mice displayed a 40% decrease in leukocyte transmigration.
5 for tissue fluid homeostasis and normalizing leukocyte transmigration.
6 impairs both docking structure formation and leukocyte transmigration.
7 howed that endothelial RhoG and SGEF control leukocyte transmigration.
8 the involvement of VLDLR in fibrin-dependent leukocyte transmigration.
9 o the effects of shear flow and cytokines on leukocyte transmigration.
10 phology and expression patterns of ICAM-1 in leukocyte transmigration.
11 nvergences are optimally equipped to support leukocyte transmigration.
12 the opening of gaps before the initiation of leukocyte transmigration.
13 uld in turn have functional consequences for leukocyte transmigration.
14 ars capable of mediating PECAM-1-independent leukocyte transmigration.
15 iation is required for ICAM-1 clustering and leukocyte transmigration.
16  transmit outside-in signals that facilitate leukocyte transmigration.
17 bling ICAM-1 to form clusters and facilitate leukocyte transmigration.
18 chemokines and other potential regulators of leukocyte transmigration.
19 on quiescent endothelial cells only mediates leukocyte transmigration.
20  behavior of these adhesion molecules during leukocyte transmigration.
21 othelial cell interactions that occur during leukocyte transmigration.
22 ted in the regulation of tight junctions and leukocyte transmigration.
23 iated with, but TspanC8s remain unstudied in leukocyte transmigration.
24 al cells with anti-PECAM-1 antibody inhibits leukocyte transmigration.
25     Gaps rapidly resealed within 5 min after leukocyte transmigration.
26 onoclonal antibodies to JAM failed to reduce leukocyte transmigration.
27 netheless, be a physiological process during leukocyte transmigration.
28 ecules act as counter-receptors in mediating leukocyte transmigration.
29 helial cell migration, and polymorphonuclear leukocyte transmigration.
30 ion of the VE-cadherin complex occurs during leukocyte transmigration.
31 orting a role for astrocytes in facilitating leukocyte transmigration.
32 understanding of the role of EC junctions in leukocyte transmigration.
33 ession of endothelial adhesion molecules and leukocyte transmigration.
34 reases endothelial P-selectin expression and leukocyte transmigration.
35                                              Leukocyte transmigration across endothelial and extracel
36 ral cell adhesion molecule, implicates it in leukocyte transmigration across the blood-brain barrier.
37 sion molecule (ALCAM) has been implicated in leukocyte transmigration across the endothelium.
38  that was previously shown to be involved in leukocyte transmigration across the endothelium.
39  levels of MMP-9 activity, which facilitates leukocyte transmigration across vascular barriers in hep
40 inase-9 (MMP-9) synthesis, which facilitates leukocyte transmigration across vascular barriers in liv
41                                              Leukocyte transmigration across vessel walls is a critic
42 ing studies have implicated it in modulating leukocyte transmigration and angiogenesis.
43 ce that VE-cad gap formation is required for leukocyte transmigration and identify p120 as a critical
44 on to show that endothelial Nogo-B regulates leukocyte transmigration and intercellular adhesion mole
45 astin and collagen and regulates blood-borne leukocyte transmigration and lesion progression.
46 ng at the cellular level in order to predict leukocyte transmigration and plaque evolution.
47 or for fibrin that promotes fibrin-dependent leukocyte transmigration and thereby inflammation.
48 ting that they contribute to control of host leukocyte transmigration and trafficking during viral in
49                Oxidative endothelial stress, leukocyte transmigration, and pulmonary thrombosis are i
50 g extracellular domain of endoglin, enhanced leukocyte transmigration, and this increased motility wa
51  blockade using probenecid markedly inhibits leukocyte transmigration, aortic inflammation and remode
52  in disease was paralleled by a reduction in leukocyte transmigration, as demonstrated by intravital
53 unohistochemistry, and in the authors' novel leukocyte transmigration assay.
54                 Functional cell adhesion and leukocyte transmigration assays further demonstrated cen
55 ion of cell adhesion molecules, preferential leukocyte transmigration, association with perivascular
56 ecam1, also known as CD31) deficiency blocks leukocyte transmigration at the level of the outer vesse
57 ribution and clustering appear necessary for leukocyte transmigration, but the mechanisms controlling
58 rane molecular scissor that is implicated in leukocyte transmigration by proteolytically cleaving its
59 udy we compared the pulmonary thrombosis and leukocyte transmigration caused by GOX targeting to the
60 contrast to wild-type mice, fibrin-dependent leukocyte transmigration does not occur in such mice.
61 f the animal, and is likely to contribute to leukocyte transmigration events important to intestinal
62 matrix proteins in vitro implicating them in leukocyte transmigration events.
63 Both dexamethasone and bevacizumab inhibited leukocyte transmigration from angiogenic vessels; howeve
64              Knowledge on viral invasion and leukocyte transmigration has also shed insights into ger
65 JAM and PECAM-1, but did not cause decreased leukocyte transmigration in an in vitro flow assay.
66 al microscopy, there was a 43% inhibition of leukocyte transmigration in mesenteric venules in respon
67  mAb also exhibited a selective reduction in leukocyte transmigration in response to IL-1beta while a
68 AR/nectin molecules mediate virus uptake and leukocyte transmigration in strikingly similar manners.
69 ue from other adhesion molecules involved in leukocyte transmigration in that its adhesiveness appear
70                ICAM-2 has been implicated in leukocyte transmigration in vitro, but there is little i
71               C16 and angiopoietin-1 reduced leukocyte transmigration in vitro.
72 nd CXCL5 and led to a functional increase in leukocyte transmigration in vivo and CXCR2-dependent neu
73 derscore the multifaceted role of sICAM-1 in leukocyte transmigration, inflammation, and endothelial
74 th the CXCR1/2 antagonist SCH527123 inhibits leukocyte transmigration into lung and subsequently reve
75  L2 knockout mice have an inherent defect in leukocyte transmigration into sites of inflammation.
76 scular adhesion molecule-1, which facilitate leukocyte transmigration into the lymphatic vessels.
77         The anti-PECAM-1 Ab markedly blocked leukocyte transmigration into the peritoneal cavity of c
78                                              Leukocyte transmigration is a key event in host defense.
79 glycocalyx participates in the regulation of leukocyte transmigration is not clear.
80 ticularly in the CNS, where inflammation and leukocyte transmigration must be tightly regulated.
81                                              Leukocyte transmigration occurs at specific locations (p
82                                              Leukocyte transmigration occurs through intercellular ga
83 d and secreted) did not enhance HIV-infected leukocyte transmigration or BBB permeability.
84 CAM-1-mediated adhesion to prevent excessive leukocyte transmigration remain unknown.
85 1), a transmembrane glycoprotein involved in leukocyte transmigration, represents a good target for e
86 related endothelial-dependent mechanisms for leukocyte transmigration that involve alterations in lat
87 ause PECAM-1 and JAM have been implicated in leukocyte transmigration, the observed redistribution by
88                                              Leukocyte transmigration through cell monolayers of endo
89  in a number of vascular processes including leukocyte transmigration through endothelium.
90 hat the cytoplasmic tail of ICAM-1 regulates leukocyte transmigration through MT1-MMP interaction.
91 lopment of neural inflammation by supporting leukocyte transmigration through the blood-brain barrier
92 e control of endothelial sheet migration and leukocyte transmigration through the endothelium.
93 ich subsequently leads to firm adherence and leukocyte transmigration through the vessel wall into th
94 tigates blood-brain barrier (BBB) damage and leukocyte transmigration to the brain by attenuating the
95 lood vessels, constitute barriers regulating leukocytes transmigration to the site of inflammation.
96 e operative to finely tune polymorphonuclear leukocytes transmigration to the site of inflammation.
97              To study this, we first blocked leukocyte transmigration using anti-PECAM Ab and then sp
98                                Consequently, leukocyte transmigration was inhibited after 6-MP/6-T-GT
99           As found before, TNF-alpha-induced leukocyte transmigration was unaltered in the KO mice.
100 nolayers, a commonly used model for studying leukocyte transmigration, were characterized using elect
101 n molecules in periodontitis is regulated by leukocyte transmigration, whereas the neutrophilic antim
102 sion, transporter activity, plasma membrane, leukocyte transmigration, Wnt signaling pathways and ang

 
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