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1 e promote their rapid interactions only upon ligand stimulation.
2  recruited independently to IL-1R1 following ligand stimulation.
3 to induce Erk phosphorylation in response to ligand stimulation.
4 ted in dissociated sympathetic neurons after ligand stimulation.
5 lated on tyrosine residues in the absence of ligand stimulation.
6 tle colocalization was observed after 4 h of ligand stimulation.
7 NF receptor-associated factor 2 (TRAF2) upon ligand stimulation.
8 h components of the ERK1/2 cascade following ligand stimulation.
9  cAMP response and Erk phosphorylation after ligand stimulation.
10  enhanced activation of EGFR signalling upon ligand stimulation.
11 stitutive complexes that are not affected by ligand stimulation.
12 ty to produce IL-12p70 after subsequent CD40 ligand stimulation.
13 recruited to TLR2 signalling complexes after ligand stimulation.
14 a (TGF-beta) receptors are internalized upon ligand stimulation.
15 m endogenous arachidonic acid in response to ligand stimulation.
16 ore than doubled the sensitivity of cells to ligand stimulation.
17 ne proteins translocate to the nucleus after ligand stimulation.
18 ptors and translocated into the nucleus upon ligand stimulation.
19  blot analysis and immunoprecipitation after ligand stimulation.
20 and recruited to the CD40 receptor upon CD40 ligand stimulation.
21 pathways can be conditionally activated upon ligand stimulation.
22 d degradation of the receptor in response to ligand stimulation.
23 th the C-terminal Src kinase (CSK) following ligand stimulation.
24 ated with Stat1 but not Stat2 or Stat3 after ligand stimulation.
25 ors of transcription 5 (STAT5) activation on ligand stimulation.
26  localization, and reduced responsiveness to ligand stimulation.
27 e activated in normal neurons in response to ligand stimulation.
28 tional mode of signal transduction following ligand stimulation.
29 n and are differentially regulated by ephrin ligand stimulation.
30 eceptor Smoothened in the primary cilia upon ligand stimulation.
31  activities, particularly in the presence of ligand stimulation.
32 GF-B signaling that is rapidly degraded upon ligand stimulation.
33 otch1 in presence of myocardin or by Jagged1 ligand stimulation.
34 gulate their signaling output in response to ligand stimulation.
35 ce of ephrin-A ligands and is disrupted upon ligand stimulation.
36 that initiate signal cascades in response to ligand stimulation.
37  as variable responsiveness to exogenous Wnt ligand stimulation.
38 amily receptor signaling in conjunction with ligand stimulation.
39 ion of type I IFNs and IL12p40 following TLR ligand stimulation.
40  and independent of adaptation to persistent ligand stimulation.
41 ownstream cytokine expression in response to ligand stimulation.
42 re defective in cytokine production upon RLR ligand stimulation.
43 phorylated, ubiquitinated, and degraded upon ligand stimulation.
44 somes and lysosome-mediated degradation upon ligand stimulation.
45 elective and undergoes dynamic regulation by ligand stimulation.
46 the activation of T cells via specific notch ligand stimulation.
47  and degraded in the lysosome upon long-term ligand stimulation.
48 erleukin-8 and MCP-1 in response to specific ligand stimulation.
49 d intranuclear mobility especially following ligand stimulation.
50 ivation events in response to membrane-bound ligand stimulation.
51 th an activated TGFbeta receptor and TGFbeta ligand stimulation.
52 ignal-regulated kinase ERK1 and -2 following ligand stimulation.
53 esses and inhibits the activity of MAPK upon ligand stimulation.
54  nuclear Smad3 was a more precise outcome of ligand stimulation.
55 ed protein kinase activation is sustained on ligand stimulation.
56 lation in response to constant extracellular ligand stimulation.
57 epidermal growth factor or heregulin (a HER3 ligand) stimulation.
58 mplexes of Lyn-Cbl and Cbl-p85 exist without ligand stimulation, (2) upon ligand binding, Lyn becomes
59 R) can lead to cell transformation, and with ligand stimulation, a broader spectrum of phosphorylated
60                                         Upon ligand stimulation, a fragment of Notch is released prot
61 pathway activity specifically in response to ligand stimulation--a process that involves endocytic tr
62 re transcription sites, indicating that upon ligand stimulation, AhR is recruited to active transcrip
63 accumulate at or are depleted from points of ligand stimulation along the axons.
64 ion by desensitizing Met signaling following ligand stimulation and by eliminating potentially oncoge
65 ithin an embryonic epithelial sheet by local ligand stimulation and coordinates a long-range contract
66 l maturation of LSECs can be achieved by TLR ligand stimulation and elucidated the mechanisms involve
67 sought to characterize pathway targets using ligand stimulation and genetic models of activation.
68        Also, severe oxidative stress (unlike ligand stimulation and moderate oxidative stress, both o
69 orms, with PKCtheta being more responsive to ligand stimulation and PKCalpha/beta to growth-factor av
70 maturation exclusively in response to TLR1/2 ligand stimulation and that the immunological status of
71 lice variant of NRP2 becomes sumoylated upon ligand stimulation and translocates to the inner nuclear
72 ncreasing ESR1 protein cellular content upon ligand stimulation and upregulated the expression of est
73 -derived growth factor (PDGF) receptor after ligand stimulation, and binding of SHPTP2 to this recept
74 ate microtiter plates, one for cell culture, ligand stimulation, and cell lysis/receptor solubilizati
75 and dephosphorylated receptor in response to ligand stimulation, and that this may be a general mecha
76 gammaR each form discrete nanoclusters after ligand stimulation, and their synergistic co-activation
77                In response to membrane-bound ligand stimulation, antigen aggregation occurs in B cell
78                                        After ligand stimulation, approximately 60-70% of transfected
79 e plasma membrane and is overactivated after ligand stimulation because of destabilization and degrad
80 ion and release in response to TLR4 and TLR2 ligand stimulation but not for TLR-independent stimuli.
81  growth, and slow their cell cycle following ligand stimulation but show increased cellular migration
82 creases in effector activity after 15 min of ligand stimulation, but only the serine phosphorylation
83  is not phosphorylated by MET in response to ligand stimulation, but rather to increasing levels of M
84 diated innate immunity due to inadequate Wnt ligand stimulation by monocytes provides an additional m
85 ution of the AhR/ARNT complex in response to ligand stimulation, by using live-cell confocal and high
86  which rapidly attenuate signals elicited by ligand stimulation, causing desensitization.
87          At low levels of receptor and UCP2, ligand stimulation creates a distinct temporal response
88                                         Upon ligand stimulation, epidermal growth factor receptor (EG
89 to MyD88 in mouse macrophages following TLR7 ligand stimulation, highlighting species-specific differ
90                              After PPARgamma ligand stimulation, HMGA1 and PPARgamma were recruited t
91 ative stability of IGF-IR in the presence of ligand stimulation, IGF-IR ubiquitination sites have yet
92     This mutant receptor fails to respond to ligand stimulation, implicating the GREAT gene in the et
93 f the HERP family, HERP1, was not induced by ligand stimulation in any cells tested, leading to the s
94 rmal growth factor receptor (EGFR) following ligand stimulation in breast and lung cancer cells.
95 dings indicate that EGFR does not respond to ligand stimulation in M phase and suggest that a negativ
96 g, conditional deletion, mosaic analysis and ligand stimulation in mice to determine that both villou
97 e studies suggest a role for continuous self-ligand stimulation in the periphery for the maintenance
98 eraction partners of endogenous NOTCH2 after ligand stimulation in the presence of a gamma-secretase
99 s were hyper-responsive to anti-IgM and TLR7 ligand stimulation in vitro and produced high concentrat
100 he ALK1 pathway and respond robustly to ALK1 ligand stimulation in vitro.
101 be up-regulated by chemokine CXCL13 and CD40 ligand stimulation in wild-type B cells, elevation of ly
102 ulations of receptors as soon as 1 min after ligand stimulation, indicating early diversification of
103 Rgamma, increased activation was observed by ligand stimulation, indicating that both PPARgamma-depen
104                                              Ligand stimulation induced phosphorylation of cortactin
105 oughout receptor internalization, direct EGF ligand stimulation initiates the internalization of EGFR
106 - and hetero-oligomers both before and after ligand stimulation is controversial.
107 oop may contribute to the mechanism by which ligand stimulation is coupled to discrete biological res
108 way by which arachidonate released following ligand stimulation is made available only to prostagland
109 at tonic T cell receptor signaling from self-ligand stimulation is required to maintain a basal state
110 let-derived growth factor receptor ECD, upon ligand stimulation, is coupled to its intracellular doma
111  with lipid rafts in the apparent absence of ligand stimulation, it has been proposed that raft-assoc
112            Our results provide evidence that ligand stimulation leads to internalization of the insul
113                                        After ligand stimulation, many G protein-coupled receptors (GP
114  induced by both mechanical LIS and chemical ligand stimulation may determine downstream signaling ch
115                      In agreement with this, ligand stimulation of ALK7 suppressed POX and KLF15 expr
116 t was less responsive to H3 relaxin based on ligand stimulation of cAMP production.
117                        Data herein show that ligand stimulation of cells that express both the EGFR a
118                            In contrast, CD95 ligand stimulation of cells unable to internalize CD95 r
119 ponses to provocative stimulation, including ligand stimulation of cultured cardiomyocytes, pressure
120 1R) inhibitors had a synergistic effect, and ligand stimulation of EGFR and MET rescued DDR2-mutant l
121                  Herein, we demonstrate that ligand stimulation of EphA2 promotes the nucleus translo
122                        The results show that ligand stimulation of Flt3 can induce association of SOC
123                                              Ligand stimulation of fTie1 resulted in Tie1 autophospho
124 f Cl- flux reflected selective disruption of ligand stimulation of GCC rather than the chloride chann
125                                              Ligand stimulation of IGF1R promotes its clathrin-depend
126 taurosporine treatment of HeLa cells and Fas ligand stimulation of Jurkat cells.
127 ucts is not reduced by DEP or PAHs following ligand stimulation of macrophages or fibroblasts.
128             It is also well established that ligand stimulation of many plasma membrane receptors lea
129                                     Instead, ligand stimulation of mGluR5 caused a dynamic transactiv
130                                 By contrast, ligand stimulation of non-overexpressed ERBB2 transientl
131 e mechanism of signal transmission following ligand stimulation of receptor tyrosine kinases in livin
132 o differed between AngII and beta-arr-biased ligand stimulation of receptors and between azF-labeled
133 s EGFR-dependent phosphorylation of RON, but ligand stimulation of RON does not trigger EGFR phosphor
134             These observations indicate that ligand stimulation of RTKs is not generic, and point to
135 xpressing RAGEv1 in tumor cells altered RAGE ligand stimulation of several novel classes of genes tha
136  in 293T and NIH 3T3 cells demonstrated that ligand stimulation of several RPTKs (epidermal growth fa
137 tic cell, G proteins in the Gq family couple ligand stimulation of the m1 muscarinic receptor to acti
138 timulates a similar Ca2+ transient as native ligand stimulation of the naive precursors, consistent w
139                                         Upon ligand stimulation of the receptors, Jaks are activated
140                    Moreover, altered peptide ligand stimulation of the Th1 line stimulates a similar
141                      In particular, ephrin-A ligand stimulation of tumor cells induces EphA2 receptor
142 ivation of signaling pathways in response to ligand stimulation of upstream cell surface receptors.
143  stimulation of Eph-B4 signaling, either via ligand stimulation or expression of a constitutively act
144 ibited genotype selection under RTK-targeted ligand stimulation or pharmacologic inhibition in vitro.
145  or ATPgammaS effectively sensitized GC-A to ligand stimulation over prolonged periods of time in eit
146                                      On EGFR ligand stimulation, phosphorylation of PLCgamma-1 increa
147                                    Following ligand stimulation, phosphorylation of specific tyrosyl
148                                              Ligand stimulation promoted recruitment of PELP1 to 17be
149  Furthermore, the mutated receptor was, upon ligand-stimulation, quickly internalized and degraded.
150 d cytokine responses to TLR4 ligand and TLR5 ligand stimulation relative to PB DCs, yet similarly pro
151 MP2A induces a heightened sensitivity to TLR ligand stimulation, resulting in increased proliferation
152      Receptor activator for NF-kappaB (RANK) ligand stimulation results in IFN-beta upregulation, whi
153             A proteolytic cascade induced by ligand stimulation results in release of the intracellul
154     Activation of FGFR3, through mutation or ligand stimulation, results in autophosphorylation of mu
155                                    After Fas ligand stimulation, SB-HCV-infected Molt-4 cells had inc
156              Upon ER Ca(2+) depletion or via ligand stimulation, Sig-1Rs dissociate from BiP, leading
157 to produce large amounts of IL-12 after CD40 ligand stimulation, similar to IL-4 priming of DCs.
158  (InsP3R) channels responding to incremental ligand stimulation, single-channel patch-clamp electroph
159 t down-regulation efficiently in response to ligand stimulation, suggesting that activation of classi
160 ntly delays dephosphorylation of CXCR2 after ligand stimulation, suggesting that clathrin-mediated en
161  and exhibited increased MED1 occupancy upon ligand stimulation, suggesting their involvement in gene
162                                         Upon ligand stimulation, TGFbeta receptors phosphorylate Smad
163  with the IGF-I receptor (IGF-IR), and after ligand stimulation the association of IGF-IR with Galpha
164                                         Upon ligand stimulation, the endoplasmic reticulum (ER) prote
165                                        After ligand stimulation, the internalized CXCR2 colocalized w
166   In this report, we demonstrate that, after ligand stimulation, the PDGF beta receptor (PDGFRbeta) b
167 BR2 and TGFBR1 form a signaling complex upon ligand stimulation, they are expected to be equally impo
168                       In contrast, with TLR2 ligand stimulation, TNF-alpha production was reduced, wh
169  addition to receptor activator of NF-kappaB ligand stimulation to initiate greater bone remodeling.
170  phosphorylation in response to non-androgen ligand stimulation using phospho-specific antibodies.
171 ar cytokine production in the absence of TLR ligand stimulation was elevated in cells from older comp
172                      The initial response to ligand stimulation was increased and sensitization to re
173 okine production in mast cells following TLR ligand stimulation was markedly reduced by HIF-1alpha kn
174 R signals influence responses to agonist TCR ligand stimulation, we analyzed naive CD4(+) cells expre
175 e the cell populations able to respond to Hh ligand stimulation, we expressed an oncogenic allele of
176 ocyte cytokine production in response to TLR ligand stimulation were associated with PTD but not SGA
177 tor (IR)/IGF-1R is augmented upon respective ligand stimulation, whereas association with STAT3 is co
178 pithelial alphaTN4-1 cells in the absence of ligand stimulation, whereas the mutants exhibited signif
179 nderwent rapid cellular internalization upon ligand stimulation, whereas the TRAIL/DR4 complex was no
180  Thus, LLPS of NLRP6 is a common response to ligand stimulation, which serves to direct NLRP6 to dist
181               The effects can be reversed by ligand stimulation, which triggers the intrinsic tumor s
182                                           On ligand stimulation with CB agonists, CB receptors induce
183                                              Ligand stimulation with Sema6A does not change the degre

 
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