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1 s not affect the nuclear localization of the liganded receptor.
2 he activating and inhibitory activity of the liganded receptor.
3 tified that associated specifically with the liganded receptor.
4 -containing fractions did Pol II bind to the liganded receptor.
5 rates of desensitization, even for partially liganded receptors.
6 r, with a reduced open probability for fully liganded receptors.
7  and C parameters allowed us to evaluate the liganded receptor according to the motional characterist
8      In protease protection experiments, the liganded receptor adopted a typical agonist conformation
9 ol II holoenzyme, did not associate with the liganded receptor, and only in certain of the holoenzyme
10 centrations of receptor, both unliganded and liganded receptors are monomers.
11        Our results suggest that NRC binds to liganded receptors as a dimer and this association leads
12                The results argue that doubly liganded receptors can rapidly desensitize before openin
13 ity upon multiprotein complexes recruited by liganded receptors during transcriptional activation.
14 such a way that the "conformational wave" of liganded-receptor gating takes place in the ECD and the
15                                              Liganded receptor interacts with the membrane-bound comp
16       Although transcriptional activation by liganded receptors involves chromatin remodeling, the ro
17  artificially activated, indicating that the liganded receptor is not required as a substrate for ind
18 is thought to target the entire complex to a liganded receptor through a domain containing two of the
19                  RAC3 interacts with several liganded receptors through a mechanism which requires th
20  of NRIF3 directly mediates interaction with liganded receptors through an LXXIL (a variant of the ca
21 he role of co-activator is to associate with liganded receptors whereas the KRE-receptor interaction