ライフサイエンスコーパス: light stimulation

1 ements were performed, both with and without light stimulation.

2 rements were performed both with and without light stimulation.

3 corneal electrode and ganzfeld (full-field) light stimulation.

4 sing the same time-course paradigm as visual light stimulation.

5 etained the ability to die after ultraviolet light stimulation.

6 orphology at hatching but fail to respond to light stimulation.

7 Circadian clocks can be reset by light stimulation.

8 he synthesis of proteins for about 4 h after light stimulation.

9 correlated activity in response to repeated light stimulation.

10 rential release of phosphorylated hTau after light stimulation.

11 atiotemporal control of OGT activity through light stimulation.

12 ut their oscillations became synchronized by light stimulation.

13 in a desynchronized wake-like state prior to light stimulation.

14 (ChR2) directly release HMGB1 in response to light stimulation.

15 clusively in endomembrane regions using blue light stimulation.

16 nd at room temperature without requiring any light stimulation.

17 ation frequencies during a single flickering light stimulation.

18 s generated action potentials in response to light stimulation.

19 , including several with sustained firing to light stimulation.

20 x) of retinal vessels in response to flicker light stimulation.

21 and the second to implant an opto-probe for light stimulation.

22 ll as the ability to induce rapid gravity or light stimulation.

23 DOPAC/DA ratio is observed following in vivo light stimulation.

24 use retina, activated by proximal and distal light stimulation.

25 unction that can be triggered to twitch upon light stimulation.

26 s and neurogenesis at time points beyond the light stimulation.

27 (NO) synthesis in the retina is triggered by light stimulation.

28 old change in binding affinity for SspB with light stimulation.

29 s its affinity for SspB by over 50-fold with light stimulation.

30 ctivity will not be recorded simultaneous to light stimulation.

31 in the dark and became phosphorylated after light stimulation.

32 required for recycling endocytosed Rh1 upon light stimulation.

33 and responded with sustained fashion to step-light stimulation.

34 retinal layers and decrease by 10% following light stimulation.

35 ated direct sun compass based on unpolarized light stimulation.

36 n modulate the kinetics of recovery from dim light stimulation.

37 ficantly less inactivation during persistent light stimulation.

38 ive in dissociation from the membrane during light stimulation.

39 s its hydrogen bonding pattern with FMN upon light stimulation.

40 undus reflectance changes induced by visible light stimulation.

41 eripheral healthy retina responds to natural light stimulation.

42 Ca2+ that enters photoreceptor cells during light stimulation.

43 rs in the isolated rat retina in response to light stimulation.

44 are phosphorylated by protein kinase C upon light stimulation.

45 play strong phototropic responses to lateral light stimulation.

46 thelium by becoming activated upon TLR or UV light stimulations.

47 al fibers (TOFs) to deliver highly localized light stimulations.

48 e surround during both stationary and moving light stimulations.

49 y acoustic or optoacoustic (induced by laser light) stimulations.

50 In addition, pulsed light stimulation (10 Hz) elicited a 1:1 repetitive depo

51 Following light stimulation, 73% more cis-retinyl esters were stor

52 Moreover, upon light stimulation, a matter flux of Pt(2) L escaping fro

53 with foot shock in mice induces freezing to light stimulation alone during fear retrieval.

54 arely engaged when neurons were activated by light stimulation alone, pairing norepinephrine release

55 ive in the ability to recover from prolonged light stimulation and caused photoreceptor degeneration

56 Daily blue-light stimulation and ChR2-dependent activation control

57 Cry2/CIB-BAX system requiring less frequent light stimulation and established a timeline of critical

58 We found that light stimulation and glial cell stimulation can both ev

59 n cell temporal properties using ultraviolet light stimulation and linear systems analysis.

60 sed by monitoring c-Fos expression following light stimulation and pattern-reversal visual evoked pot

61 SACY activity, bPAC restored motility after light-stimulation and, thereby, enabled sperm to fertili

62 retinal vessel analysis (DVA) during flicker light stimulation, and functional near infrared spectros

63 ations in pupil size occurred independent of light stimulation, and spontaneous eye movements made it

64 the genetic pathways activated by asymmetric light stimulation, and their time course, we exposed emb

65 opus tadpoles showed that repetitive dimming-light stimulation applied to the contralateral eye resul

66 cone and rod visual pigments in response to light stimulation, but also as a chaperone for normal tr

67 eceptor cells depolarized normally following light stimulation, but failed to activate postsynaptic n

68 We propose that upon light stimulation, Crag is required for trafficking of R

69 Using patterned light stimulation delivered by a spatial light modulator

70 Finally, light stimulation did not change the Mg2+ concentration

71 The present study suggests that paired light stimulation differently modulates On and Off EPSPs

72 Light stimulation evoked in all ipRGCs both synaptically

73 Bright light stimulation evoked ON and OFF L-IPSCs in axotomize

74 In adult retinas, light stimulation evoked rapid decreases in ECS alpha.

75 Light stimulation evokes neuronal activity in the retina

76 ecordings in brain slices showed that CTZ or light stimulation facilitated synaptic transmission and

77 Light stimulation for 24 hr selectively increased dendri

78 f cognitive-motor functions, optimization of light stimulation for Alzheimer's therapy, and effects o

79 h precisely follow the ChR2 activation up to light stimulation frequencies of 20 Hz.

80 n to fire with high spike fidelity with blue-light stimulation frequencies up to 40 Hz for periods of

81 a mechanism by which the mouse distinguishes light stimulation from developmental patterns of spontan

82 can be rapidly released under near-infrared light stimulation from nanovesicles implanted in the bra

83 s (>40%), maintain high cell viability under light stimulation (>96%), and negligibly affect the elec

84 uman white adipocytes respond to direct blue light stimulation in a cell-autonomous manner, highlight

85 suprachiasmatic nucleus (SCN) to respond to light stimulation in a phase-specific manner constitutes

86 tering, pattern, and switch in-between under light stimulation in an electric field.

87 terations in the pupillary response to white light stimulation in patients with Fabry disease and the

88 ls in multielectrode array recordings during light stimulation in retinas of adult guinea pigs of eit

89 recordings demonstrate impaired responses to light stimulation in SynCAM 1 knockout (KO) mice.

90 ted in the presence of D-serine, followed by light stimulation in the presence of dichlorokynurenic a

91 vgf mRNA levels are induced as a response to light stimulation in the suprachiasmatic nuclei (SCN), t

92 The mice showed increased freezing only upon light stimulation, indicating light-induced fear memory

93 Light stimulation induced translocation of PKCalpha immu

94 Upon light stimulation, interdomain interactions weaken to fa

95 Recent studies showed that light stimulation is required for the maturational segre

96 ly reproduce the temporal sequence even when light stimulation is turned off.

97 Here, we show that morphogen light-stimulation is a scalable tool that induces self-o

98 show a large change in binding affinity upon light stimulation, it should not cross-react with other

99 With paired light stimulation, latencies of ON L-IPSCs increased at

100 n alone, pairing norepinephrine release with light stimulation markedly enhanced astrocyte Ca(2+) sig

101 We present a visible light stimulation method for modulating the firing patte

102 In the absence of light stimulation, mice showed a conditioned place avers

103 ucible promoters and that, after exposure to light stimulation, NGF-1-associated HAT activity leads t

104 Light stimulation of both genotypes during the dark peri

105 ical activation of IPN GABAergic neurons via light stimulation of channelrhodopsin elicited physical

106 ablation of DVC or activation of DVC through light stimulation of channelrhodopsin-2 specifically exp

107 In contrast, light stimulation of corticothalamic terminals induced s

108 oride analog benzamil, responded robustly to light stimulation of GAD65(+) TBCs on the anterior tongu

109 Using focal light stimulation of GCs that express optogenetic light-

110 Furthermore, light stimulation of hESC-derived neurons transplanted t

111 Pulsed light stimulation of human cardiomyocytes showed that th

112 Direct light stimulation of ipRGCs can regulate many nonimage-f

113 In loss of function studies, yellow light stimulation of keratinocytes that express halorhod

114 It is a commonly accepted view that light stimulation of mammalian photoreceptors causes a g

115 In awake mice, light stimulation of Opn7b expressed in pyramidal cells

116 Moreover, light stimulation of optoFGFR1 partially occluded LTP in

117 y within organoids could be controlled using light stimulation of photosensitive cells, which may off

118 al TRP (TRPC) channels that are regulated by light stimulation of rhodopsin and engagement of Galpha(

119 n in the transduction of brief environmental light stimulation of the retina into molecular changes i

120 e membrane-proximal domain, and in addition, light stimulation of the SRII inhibition of HtrII cross-

121 ctrophysiological recordings of responses to light stimulation of the transplanted cells were made fr

122 and potential occipital cortical response to light stimulation of the transplanted eye was demonstrat

123 white-opsin in response to repetitive white light stimulation of varying pulse width was observed.

124 The inhibition is released upon light-stimulation of photoreceptor cells.

125 aration that allows for specific optogenetic light stimulation on GABAergic synaptic terminals across

126 we pharmacologically mimicked the effects of light stimulation on mouse On bipolar cells, thus avoidi

127 of a subset of visual inputs evoked by local light stimulation on the retina.

128 Responding with an unusually long latency to light stimulation, OND RGCs respond earlier as the visua

129 However, bright light stimulation or application of the D1 agonist SKF38

130 Visible light stimulation over a 200-fold intensity range caused

131 l ganglion cells (RGCs) typically respond to light stimulation over their spatially restricted recept

132 del, we investigated the effect of different light stimulation paradigms on cells expressing select v

133 es valuable information for the selection of light stimulation paradigms that elicit desired astrocyt

134 ed upon short (17-25 ms) and long (48-76 ms) light stimulation paradigms.

135 m transcoding the visual input into tailored light-stimulation patterns which drive in situ the optog

136 is required for motility and fertilization: light-stimulation rapidly elevates cAMP, accelerates the

137 However, it is not clear to what extent light stimulation regulates the maturation of RGC dendri

138 these receptors in the intact circuit during light stimulation remains unclear.

139 time and that disrupting sleep via constant light stimulation renders D. mojavensis more sensitive t

140 Paired-pulse light stimulation resulted in a depression of On-, and a

141 e statistically compared between (1) tPBM vs light-stimulation sham, (2) thermo_stim vs heat-stimulat

142 Although light stimulation showed that FLI expression was very si

143 the minimal responses observed following UV light stimulation suggest only a limited role for the no

144 lfhydryls were available for MTSES following light stimulation, suggesting that light itself could no

145 gs indicate that ChR2 expression methods and light stimulation techniques influence synaptic response

146 For UV light stimulation, the ON pathway inputs to the OFF path

147 Upon light stimulation, these cells exhibit calcium/cyclic AM

148 ed TRPV1-Ai32 optogenetic mice and cutaneous light stimulation to activate cutaneous neurons in the a

149 eceptors in diaphragm myofibers would enable light stimulation to evoke functional diaphragm activity

150 mes faster than HEI-OC1 cells, which allowed light stimulation up to rates of 10/s to elicit correspo

151 ociated viral vector encoding ChrimsonR with light stimulation via engineered goggles.

152 ils tethered to esophageal fibroblasts after LIGHT stimulation via intercellular adhesion molecule-1.

153 We demonstrate that 380-nm light stimulation via OPN5 and VGAT (the vesicular GABA/

154 Light stimulation was most likely to induce behavioral a

155 d by low-intensity blue light, and this blue-light stimulation was suppressed in three different RNAi

156 Light stimulation was used to trigger the assembly of na

157 multiple conditions precisely controlled by light stimulation, we identify a series of signaling cir

158 ipolar cell (BC) terminals with paired-pulse light stimulation, we isolated and quantified the short-

159 With near-infrared light stimulation, we show that activating single excita

160 ulate nitrergic myenteric neurons using blue light stimulation with a goal of understanding how inhib

161 pyramidal neurons in vitro and in vivo upon light stimulation, with ChR2-EYFP and Arch-ER2 demonstra

162 l parameters necessary to produce pinpointed light stimulation within a single nerve, we employed a s