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1 rooted in a downregulation of stress-induced limbic activations and concomitant strengthening of top-
5 les influenced by physical abuse occurred in limbic (amygdalar-hippocampal), paralimbic (cingulo-insu
6 m robustly recapitulates established sensory-limbic and anterior-posterior dimensions of brain organi
10 he motivational drive, hunger, and, finally, limbic and cognitive processes that bring about hunger-m
11 l effects by modulating monoamine release in limbic and cortical areas, which was investigated herein
12 pes receive converging glutamate inputs from limbic and cortical regions, encoding contextual and emo
13 between dorsal prefrontal and regions of the limbic and default-mode networks serves as a significant
20 that it is possible to infer activity in the limbic and paralimbic systems from pre-frontal EEG asymm
21 asymmetry reflects activity in parts of the limbic and paralimbic systems, the entropy of that asymm
25 ibe findings with respect to frontocortical, limbic and striatal brain volume, functional activation
26 in 12 brain regions, including neocortical, limbic and subcortical areas from Alzheimer's disease ca
27 preferentially engaged by EL_INSTs were the Limbic and the Frontoparietal Networks (Mean VOR > 1.5).
28 actional anisotropy in left thalamocortical, limbic, and association fibers, as well as greater conne
29 integrate inputs from diverse sensorimotor, limbic, and associative regions to guide action-selectio
30 occipito-temporal, temporal, parahippocampal-limbic, and inferior fronto-temporal), confirming our hy
32 between the habenula and the basal ganglia, limbic, and sensory systems was already present in the c
34 tly reduced in the striatum (p = 0.0012) and limbic archicortex (p = 0.004) in freezers compared to n
36 current study we show that this classically limbic area directly controls motor behavior on multiple
38 ndings emphasize the role of the frontal and limbic areas in the aetiology of psychotic symptoms, in
39 stress management, targeting deeply located limbic areas involved in stress processing(2) has paved
40 ar responses across each of the cortical and limbic areas signal movement during the TST and open fie
42 ore in specific brain areas, especially some limbic areas, while full-length amyloid-beta42 tended to
47 band oscillations in the vStr and associated limbic areas.SIGNIFICANCE STATEMENT The ventral striatum
48 of immunoreactivity for TH and DARPP-32 in "limbic" areas such as the hippocampus, septum, and exten
49 conducted in non-human primates dissociating limbic, associative and motor frontal hyper-direct conne
50 he whole striatum and its subdivisions (i.e. limbic, associative, and sensorimotor), averaged regiona
51 are positioned to influence outputs to the 'limbic-associative' striatum, which is distinct from str
52 nd somatomotor networks, the DMN and visual, limbic, auditory and ventral attention networks, and bet
53 They further suggest that mesolimbic and non-limbic basal ganglia dopamine circuits are functionally
55 ated that kisspeptin administration enhanced limbic brain activity specifically in response to sexual
56 to induce tinnitus and measured auditory and limbic brain activity using manganese-enhanced MRI (MEMR
57 neuronal excitability have been observed in limbic brain areas after associative learning, but littl
58 onal interactions between frontoparietal and limbic brain circuitry at rest, which may reflect a pote
61 uit in vivo activity from eight cortical and limbic brain regions as mice were subjected to the tail
62 gene expression data in four interconnected limbic brain regions implicated in depression and its tr
63 ure cause atrophy of neurons in cortical and limbic brain regions implicated in depression, and brain
64 lts indicate that VP, unlike other connected limbic brain regions, is essential for resumption of dru
70 Furthermore, kisspeptin's enhancement of limbic brain structures correlated with psychometric mea
75 ion and microbiome alterations on the fronto-limbic circuitry as a convergence hub for emotional dysr
76 he "uncinate circuit," a component of fronto-limbic circuitry that is connected by the uncinate fasci
78 dopamine receptors, is expressed throughout limbic circuits affected in neuropsychiatric disorders,
79 is necessary to understand how plasticity of limbic circuits is associated with the pathophysiology o
81 reased functional connectivity in prefrontal-limbic circuits), cognitive deficits (e.g., executive fu
83 n the structure and function of critical PFC-limbic circuits, linking alterations in the processing o
85 tion network) and cytoarchitectonic classes (limbic class), with an epicenter in the anterior cingula
87 e is a vulnerable period of development when limbic connection of the prefrontal cortex (PFC) involve
88 increased task-induced deactivation, altered limbic connectivity and increased cortical thickness.
89 from adults suggests that altered prefrontal-limbic connectivity is a pathophysiological feature of a
91 tive control network, dysfunctional auditory-limbic connectivity, and deafferentation-induced atrophy
96 and by activation of PV interneurons in pre-limbic cortex and ventral subiculum of the hippocampus.
98 ntral to the claustrum, has connections with limbic cortical areas and should be considered part of a
99 reported using Fos labeling that only a few limbic cortical structures including the retrosplenial c
100 perience of control over drug seeking from a limbic cortical-ventral striatal circuit underlying goal
105 otion perception by suggesting engagement of limbic effective connectivity in recognizing the lack of
108 ium channel (VGKC) complex antibody-mediated limbic encephalitis with generalized hippocampal atrophy
110 cohort of patients (n = 38) after autoimmune limbic encephalitis, a syndrome associated with focal st
111 otein-2 (CASPR2), are found in patients with limbic encephalitis, faciobrachial dystonic seizures, Mo
112 patients: (1) developmental abnormalities in limbic fibers, (2) accelerated aging and abnormal matura
113 ) is a brain structure receiving inputs from limbic forebrain areas and innervating major midbrain mo
114 ssion in central autonomic components of the limbic forebrain, the locus coeruleus and cerebellar Pur
116 parisons with other disorders) was found for limbic, frontoparietal executive, default mode, and sali
118 showed increased connectivity in frontal and limbic hubs over time, children with PME showed increase
119 In contrast, exposure to UPS induced fronto-limbic hyper-connectivity in males, but either no change
120 motor and affective disturbances, along with limbic hyper-reactivity and enhanced motor-limbic connec
121 ced tinnitus may play a role in auditory and limbic hyperactivity, the non-auditory effects of blast
122 f the stria terminalis (BNST) is part of the limbic-hypothalamic system important for behavioral resp
123 f the stria terminalis (BNST) is part of the limbic-hypothalamic system important for behavioral resp
124 sive protection of the newborn decreases the limbic-hypothalamic-pituitary-adrenal (LHPA) axis activi
125 nt for the temporal integration of motor and limbic information during flexible goal-directed behavio
127 amus (PVT), a hub for cortical, sensory, and limbic information, to the nucleus accumbens shell (an a
129 ens (NAc) core, is typically associated with limbic input but was historically linked to high-level m
130 naptic organization revealed a predominantly limbic input onto LHA Vglut2 neurons, while sensorimotor
131 ts control of the NAc shell from cortical to limbic input, likely contributing to cognitive and psych
132 ated processing, we propose that this fronto-limbic interaction might be related to the involvement o
134 reporting using the Pennebaker Inventory of Limbic Languidness questionnaire from a case-control coh
135 matter volume increases in the temporal and limbic lobes and decrease in the superior temporal gyrus
136 g fMRI in mice, we mapped a discrete cortico-limbic loop between insular cortex (IC), central amygdal
138 tomical basis for the impairment in aversive limbic memory observed during withdrawal in alcohol depe
139 s 32, 24, 14, and 25 (mPFC) form part of the limbic memory system, but little is known about their fu
140 t forms of emotionally salient memory, i.e., limbic memory, that promote escalated alcohol consumptio
141 that chronic alcohol use leads to decreased limbic mGluR5 availability, which was associated with le
142 d with an enhanced neural response in fronto-limbic, midline, and occipitotemporal regions to a learn
143 sal ganglia function to include the study of limbic-motor cross-territory interactions in both health
144 te that the dCA1->NAc pathway instantiates a limbic-motor interface for neuronal representations of s
147 The results stress the importance of the limbic network's increased response to neutral facial st
148 development and plasticity of the prefrontal-limbic network, which therefore may increase the vulnera
149 Network, Ventral Attention Network, and the Limbic Network-with percentage volume overlap of 19.5%,
153 or globus pallidus internus, connectivity to limbic networks, associative networks, caudate, thalamus
159 five structural covariance networks (insular-limbic, occipito-temporal, temporal, parahippocampal-lim
160 the classically defined auditory system, in limbic or association neocortical regions involved in co
163 hogenesis and synaptic transmission genes in limbic/paralimbic areas; (ii) locomotory behavior and ne
164 y, highlighting the importance of the spinal-limbic pathway and the striking lack of hippocampal conn
170 tration robustly attenuated both the cortico-limbic phMRI response and the fronto-hippocampal hyper-c
171 e classified as either typical AD (n = 100), limbic-predominant (n = 33), or hippocampal-sparing (n =
172 scribe a recently recognized disease entity, limbic-predominant age-related TDP-43 encephalopathy (LA
173 lap between presentations of dementia due to limbic-predominant age-related TDP-43 encephalopathy (LA
174 for common age-related TDP-43 proteinopathy: limbic-predominant, age-related TDP-43 encephalopathy (L
176 ly salient stimuli, associated with elevated limbic reactivity (eg, amygdala), may contribute to the
177 k of psychosis to determine whether aberrant limbic reactivity to neutral stimuli is an early neurofu
179 differences in neuronal activity in one key limbic region, the basolateral amygdala (BLA), whose act
180 rodent claustrum is closely tied to frontal/limbic regions and plays a role in processes, such as at
182 of 5-HT) caused sustained activation in key limbic regions during processing of negative facial emot
183 of the ventral striatum, midbrain, and other limbic regions for neutral cues, neutral outcomes, and n
184 of the ventral striatum, midbrain, and other limbic regions for rewards and positive prediction error
185 P(ND) were also higher in other striatal and limbic regions in patients, and correlated with symptom
186 x differences are focused in deep, ancestral limbic regions involved in the control of reproductive b
187 eostatic control of the temporal dynamics in limbic regions is altered in adults with ASD, and provid
188 sion levels in PFC, as well as other cortico-limbic regions of AUD subjects when compared with contro
189 ty along the auditory pathway and in certain limbic regions of rats with tinnitus compared to age-mat
190 distinct, mainly sensorimotor, cognitive and limbic regions of the cerebral cortex and subcortex.
191 e impairments; smaller grey matter volume in limbic regions such as the amygdala, insula and orbitofr
192 al cortex are functionally connected to deep limbic regions such as the subgenual cingulate cortex.
193 entiation traversing from primary sensory to limbic regions that followed shifts in laminar different
194 istopathology in neocortical and subcortical/limbic regions were compared between groups and related
199 BOLD and CBVw fMRI signals in VTA-innervated limbic regions, including the ventral striatum (nucleus
213 ) by provoking a release of serotonin in key limbic regions; and (2) by increasing the availability o
214 ated differences in the responsivity of key 'limbic' regions (including amygdala, ventromedial prefro
215 y consistent with glutamatergic and cortical-limbic related theories of depression and resulted in re
219 APOE and (ii) AD-PRS on a vulnerable cortico-limbic scene-processing network heavily implicated in AD
220 gnificant resistance to electrically-induced limbic seizures and to pentylenetetrazole induced tonic-
221 a new piece of evidence to the proposal that limbic seizures can be supported by GABAergic hyperactiv
222 ) degradation versus synthetic CB agonist on limbic seizures induced by maximal dentate activation (M
223 vertebrates-takes part in the integration of limbic, sensory, and basal ganglia information to guide
226 act measureable physiological alterations in limbic striatal circuitry that vary as a function of dop
228 tum), 0.7781 (associative striatum), 1.0344 (limbic striatum), and 1.0189 (sensorimotor striatum) in
229 as restricted to D3 selective brain regions (limbic striatum, globus pallidus, and ventral pallidum (
230 y disorder-related alterations in prefrontal-limbic structural connectivity are present early in life
231 ceives inputs from medial pallium-originated limbic structures (e.g., the medial prefrontal cortex [m
232 ical studies have implicated cortico-striato-limbic structures and circuits in the pathophysiology of
234 the expression pattern of Pcdh19 and Ncdh in limbic structures at four postnatal stages of C57BL/6J m
235 to address neuroplasticity features in human limbic structures connected to the defensive survival ci
236 xometry imaging) of tissue properties in two limbic structures implicated in age-related memory decli
237 sing functional neuroanatomy that only a few limbic structures including the retrosplenial cortex (RS
238 X microinjections from recruiting downstream limbic structures into neurobiological activation, and s
239 sed by subregion in the amygdala and insula, limbic structures key to the disorder pathophysiology.
240 ls recorded from olfactory, sensorimotor and limbic structures of rats performing an olfactory task.
241 to the peripheral circulation and project to limbic structures or parvocellular cells that regulate t
242 y domain and this is considered to depend on limbic structures such as the amygdala or the septal nuc
243 interconnectivity of cortical, striatal, and limbic structures that regulate alcohol intake, it has b
244 uated recruitment of Fos expression in other limbic structures throughout the brain, which was otherw
245 ing.SIGNIFICANCE STATEMENT In the neocortex, limbic structures, and auditory brainstem, glutamatergic
246 In many brain areas, such as the neocortex, limbic structures, and auditory brainstem, synaptic zinc
247 terone, c-Fos activation in hypothalamic and limbic structures, and species-typical behaviors were co
248 ted with a higher gray matter signal in deep limbic structures, as well as connectivity with cortical
249 eticular nucleus, certain cortices and other limbic structures, as well as in some specific nuclei in
250 est connectivity to ARAS structures included limbic structures, thalamus and certain neocortical area
257 tion is associated with anterior associative-limbic subthalamic nucleus and right dorsolateral prefro
258 ight specificity of the anterior associative-limbic subthalamic nucleus in decisional impulsivity.
259 rcuit models to explain how key nodes in the limbic system and beyond interact to produce persistent
260 as a pivotal point that mediates between the limbic system and the frontal cortex in reward-related p
261 d self-reported mood, reduced attention, MRI limbic system changes and the absence of conventional ep
266 ttern of progression of the lesion along the limbic system network and an associated memory impairmen
267 eceptor subunit Grin2b, were modified in the limbic system of adult animals, in a region-specific, se
268 also connections within the networks of the limbic system that are both critical and permissive for
269 both, Ncdh and Pcdh19, in structures of the limbic system with overlapping expression patterns parti
270 directly damaged: the (i) premotor loop (ii) limbic system, and (iii) ventral attentional network.
271 of the serotonergic afferents along with the limbic system, and it was shown that serotonin was signi
272 ly on ancient 'hard-wired' components of the limbic system, but also use sensory processing to determ
273 volving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontoparietal cortices,
274 ctral and temporal alterations in the fronto-limbic system, including the ventromedial prefrontal cor
276 ressant efficacy have largely focused on the limbic system, leaving it unclear whether this signaling
277 trajectory of white matter tracts within the limbic system, particularly the uncinate fasciculus.
278 s, and tuberomammillary nucleus region), the limbic system, sensory processing nuclei, and additional
280 ional perspective of selected regions of the limbic system, the medial temporal lobe structures-the h
281 nucleus, insula, and cortical spread to the limbic system, whereas the occipital lobes and cerebella
282 major depressive disorder were found in the limbic system, which were similar to the differences in
295 , with no significant differences in frontal limbic tracts important for emotional processing and soc
296 were also found traveling through the major limbic tracts, suggesting a role of TRPM8-expressing cen