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1 ween other relevant brain regions (i.e., the limbic system).
2 izophrenia relates to overstimulation of the limbic system.
3 relin may act at a range of sites within the limbic system.
4 ge glutamatergic neurons innervating the rat limbic system.
5  of the core circuitry of imitation with the limbic system.
6 tween the temporal lobes, frontal lobes, and limbic system.
7 uted structures, most of which belong to the limbic system.
8 rmalities implicating the insular cortex and limbic system.
9 eceptor before delivery to the neocortex and limbic system.
10 d on emotional and situational cues from the limbic system.
11  the balance of excitation-inhibition in the limbic system.
12 changes in the tissue levels of M-Enk in the limbic system.
13  MRI studies had signal abnormalities in the limbic system.
14 t of schizophrenia is on the hippocampus and limbic system.
15 n specific nuclei of the basal forebrain and limbic system.
16 ted within the temporal lobe regions and the limbic system.
17 discrete regions of the ram hypothalamus and limbic system.
18 s expressed in several discrete areas in the limbic system.
19 h emotional behavior, such as the cortex and limbic system.
20 tly expressed in neurons in the thalamus and limbic system.
21 ions of the preoptic area, hypothalamus, and limbic system.
22 n and inhibition of different targets in the limbic system.
23 oth cortical and subcortical portions of the limbic system.
24 d in cortical and subcortical regions of the limbic system.
25 rved in choroid plexus and components of the limbic system.
26  has been localized to distinct parts of the limbic system.
27 er time, rewire the circuitry throughout the limbic system.
28 l, as well as of the hypothalamus and of the limbic system.
29 ceive input from different structures of the limbic system.
30 tified abnormal activation in the prefrontal-limbic system.
31 y as inhibitory components of the prefrontal-limbic system.
32 bnormal volumes are densely connected to the limbic system.
33 on the mechanisms of ketamine on a disrupted limbic system.
34 aminergic and glutamatergic functions in the limbic system.
35 ed fear, and neurochemical parameters in the limbic system.
36 inter-hemispheric integration and within the limbic system.
37  animals, highlighting the importance of the limbic system.
38 TLE is associated with reorganisation of the limbic system.
39 ce implicating other networks, including the limbic system.
40 rk information to spatial signals within the limbic system.
41 arget neurons in subcortical stations of the limbic system.
42 ities in frontal lobe, basal ganglia and the limbic system.
43 pallidum (VP) is a major component of the BG limbic system.
44 eding centers and mood regulators within the limbic system.
45 sly unknown progenitor pool dedicated to the limbic system.
46 s with a theoretical model of the prefrontal-limbic system.
47  and basal ganglia connections linked to the limbic system.
48 es, since they are two key structures of the limbic system.
49 -myoclonus, and encephalitides affecting the limbic system.
50 ula (LHb) which is often associated with the limbic system.
51 very subtle finding but may be a marker of a limbic system abnormality.
52 , monoamine neurotransmitter metabolism, and limbic system activity.
53 role in regulating neuronal functions of the limbic system, although the cellular targets and molecul
54 aventricular and centromedian thalamus), and limbic system (amygdala and nucleus accumbens).
55 tylcholine is a robust neuromodulator of the limbic system and a critical regulator of arousal and em
56 ificant signal increases within the affected limbic system and additionally in areas connected to the
57 n 5-HT(2A) receptor mRNA expression occur in limbic system and associated areas following chronic D-A
58 rcuit models to explain how key nodes in the limbic system and beyond interact to produce persistent
59 ew DMRs distinguished regions of the cortex, limbic system and brain stem.
60 action signals are widely distributed in the limbic system and can be decoded from the network activi
61                    The increased gradient in limbic system and decreased gradient in thalamic and str
62 r disorder, with most notable changes in the limbic system and emotion regulation circuitry.
63 ortical thickness/subcortical volumes in the limbic system and frontal lobe; reported childhood histo
64 ojections connecting the hypothalamus to the limbic system and higher-order brain centers in the cort
65 eptide cholecystokinin (CCK) is expressed in limbic system and hypothalamic nuclei that form a circui
66 ndogenous opioids that activate mu-OR in the limbic system and hypothalamus providing a "neurochemica
67 ions throughout reproductive circuits of the limbic system and hypothalamus.
68 ral cortex, occipital cortex, basal ganglia, limbic system and hypothalamus.
69 tion in steroid-sensitive cell groups of the limbic system and hypothalamus.
70            The hippocampus forms part of the limbic system and is involved in the learning process; i
71 ology that was substantially enhanced in the limbic system and olfactory cortex.
72 orpus callosum, is recognised as part of the limbic system and plays numerous vital roles.
73 atest differences occurred in regions of the limbic system and primary sensory and posterior parietal
74  subpallial areas strongly implicated in the limbic system and show robust intrahypothalamic connecti
75 e find MVP expression highly specific to the limbic system and show that Mvp regulates neuronal morph
76 ongs to the amygdalocentric component of the limbic system and stands to integrate exteroceptive info
77 ynaptotagmin-9 is primarily expressed in the limbic system and striatum.
78 hest levels found in specific regions of the limbic system and the brainstem where NPVF-producing neu
79 as a pivotal point that mediates between the limbic system and the frontal cortex in reward-related p
80  in extensive neuronal damage throughout the limbic system and the loss of selectively vulnerable neu
81 imuli, in particular regions of the extended limbic system and the ventral striatum.
82  via tractography of the main bundles in the limbic system and using Tract-Based Spatial Statistics,
83   Region of interest analyses focused on the limbic system and ventral PFC - basal ganglia - thalamoc
84 ylogenetically more primitive brain regions (limbic system) and newer ones (prefrontal cortex).
85 dopamine, endogenous opioids), neuroanatomy (limbic system), and self-medication behaviors.
86 directly damaged: the (i) premotor loop (ii) limbic system, and (iii) ventral attentional network.
87 idely distributed in many regions of cortex, limbic system, and basal ganglia, appear to play a key i
88 ave been described in the prefrontal cortex, limbic system, and cerebellum in individuals with schizo
89 interconnecting the basal ganglia, thalamus, limbic system, and higher cortical centre are implicated
90                           Disruptions in the limbic system, and in emotion regulation circuitry that
91 of the serotonergic afferents along with the limbic system, and it was shown that serotonin was signi
92 ve degeneration of neurons in the neocortex, limbic system, and nucleus basalis, among others.
93  found increased Fos in the cerebral cortex, limbic system, and parts of the arousal and autonomic sy
94 filtrates, encephalitis, particularly of the limbic system, and spinal cord white matter inflammation
95  neural system involving the sensory cortex, limbic system, and striatum.
96 ajor excitatory pathways linking the cortex, limbic system, and thalamus, regions that have been impl
97 the nuclei in the main olfactory system, the limbic system, and the olivocortical projection.
98 hological process in schizophrenia is on the limbic system, and there have been several postmortem re
99 were located in subcortical areas and in the limbic system, and to a lesser degree in the frontal, te
100 teractions between the default mode network, limbic systems, and the rest of the brain.
101      Head direction (HD) cells in the rodent limbic system are believed to correspond to a cognitive
102 omeostatic function, areas in the cortex and limbic system are important for processing information r
103                          Areas of the rodent limbic system are important for solving spatial tasks an
104 on cells, grid cells, and place cells in the limbic system, are thought to underlie navigation by pat
105  These results and demonstrations that other limbic system areas make time-limited contributions to l
106                         Cells within several limbic system areas of the mammalian brain discharge all
107 xtensive transneuronal labeling was found in limbic system areas: (1) extended amygdaloid complex, (2
108 tress and neuropathology produced in the rat limbic system as a result of seizure activity elicited b
109  changes were localized mainly to within the limbic system, as well as between the limbic and nocicep
110               We designated AH9 antigen as a limbic system associated protein-1, lap-1.
111                                 Parts of the limbic system associated with the midbrain dopamine syst
112                   The Lsamp gene encodes the limbic system-associated membrane protein (LAMP) an immu
113          Neurotrimin (Ntm) together with the limbic system-associated membrane protein (LAMP) and the
114                                          The limbic system-associated membrane protein (LAMP) is a 64
115                                          The limbic system-associated membrane protein (LAMP) is an a
116 specific fates, assayed by expression of the limbic system-associated membrane protein.
117                                          The limbic-system-associated membrane protein (LAMP) is a 64
118 l protein that is relatively specific to the limbic system, at least in the rat brain.
119  serotonergic pontine nuclei, basal ganglia, limbic system, basal forebrain and prefrontal cortex, wh
120  have long considered it part of an extended limbic system based on its location posterior to the orb
121 of cognitive and reward functions in cortico-limbic systems (bottom-up) and then on pathways by which
122 y a paraneoplastic disorder that affects the limbic system, brain stem, and cerebellum.
123 limbic" striatum.SIGNIFICANCE STATEMENT The "limbic" system broadly refers to brain circuits that coo
124 ly on ancient 'hard-wired' components of the limbic system, but also use sensory processing to determ
125 renaline and GABA levels in key areas of the limbic system, but not with functional alterations in th
126 l plasticity and seizures, is induced in the limbic system by chronic ethanol consumption, temporally
127 m and additionally in areas connected to the limbic system by neural pathways, including the lingual
128 interface between the visual pathway and the limbic system, by which increasingly refined and specifi
129         Head direction (HD) cells in the rat limbic system carry information about the direction the
130 volving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontoparietal cortices,
131 volving the occipital lobes, temporal lobes, limbic system, cerebellum, and frontoparietal cortices,
132 iety was enriched for genes expressed in the limbic system, cerebral cortex, cerebellum, metencephalo
133 d self-reported mood, reduced attention, MRI limbic system changes and the absence of conventional ep
134 d self-reported mood, reduced attention, MRI limbic system changes and the absence of conventional ep
135 r hand, age-related DNA methylation in these limbic system components commonly and sensitively applie
136  comparing mouse hypothalamus with two other limbic system components, we show that the hypothalamus
137                                      The rat limbic system contains head direction (HD) cells that fi
138          The hippocampus, a component of the limbic system, contains the highest concentration of cor
139   The patient, R.M., had extensive bilateral limbic system damage, affecting orbitofrontal cortex, te
140 elieved to be impaired by basal ganglia, not limbic system damage, but there is uncertainty whether l
141  including heteromodal association areas and limbic system, demonstrate the high variability.
142                    Theta oscillations in the limbic system depend on the integrity of the medial sept
143 ration throughout the association cortex and limbic system, deposition of amyloid-beta peptide (Abeta
144 ructures, LAMP may play an important role in limbic system development and function, as suggested by
145 at emphasize stress pathways and accelerated limbic system development.
146 ew rat SRT model clarifies the basal ganglia-limbic system dichotomy suggested by human work.
147 estigated functional connectivity within the limbic system during face identification; a primary comp
148  cortex modulates excitatory activity in the limbic system during seizures.
149  (in layers 3 and 5), several members of the limbic system (e.g., hippocampus, central amygdala, and
150 es and loss of synapses in the neocortex and limbic system each correlate strongly with cognitive imp
151 l prefrontal cortex (vmPFC) is a part of the limbic system engaged in the regulation of social, emoti
152 correction in dMRI studies and highlight the limbic system, especially the cingulum and fornix, as ke
153 al network measures in structures within the limbic system, especially the left hippocampus, the left
154         Head direction (HD) cells in the rat limbic system fire according to the animal's orientation
155 ment across populations; for example, in the limbic system for BMI, the per-standardized-annotation e
156 ive analogues to components of the mammalian limbic system (for example, hippocampus)(2-4).
157 ent increase in neuronal excitability in the limbic system has not been unequivocally determined.
158 h include the basal ganglia and parts of the limbic system, have key roles in learning, motor control
159                   A detailed analysis of the limbic system highlight clear expression boundaries betw
160 is reveals convergence on the basal ganglia, limbic system, hippocampus and cerebellum.
161        Head direction cells in the mammalian limbic system implement an allocentric neuronal compass.
162   The head-direction (HD) cells found in the limbic system in freely mov ing rats represent the insta
163 he medial prefrontal cortex and the extended limbic system in methadone maintenance patients with a h
164 dies in almost all of the key regions of the limbic system in the forebrain, including the midline an
165 atter connections involving the thalamus and limbic system in the HEU(pre) group compared to HUU.
166      Our findings suggest involvement of the limbic system in the pathophysiology of ADHD.
167 e of dysregulation of our main analgesic and limbic systems in chronic TMD pain.
168  noradrenergic, serotonergic, olfactory, and limbic systems in the modified behavioral response to qu
169 ations in areas of the visual, salience, and limbic systems in VSS.
170 nt nuclei associated with motor, sensory, or limbic systems; in the caudate nucleus, Thy-1 expression
171 lar, cingulate and somatomotor cortices, the limbic system including amygdaloid nuclei, hippocampus a
172 HD cells have been identified throughout the limbic system including the anterodorsal thalamus (ADN)
173 ithin the neocortex and in structures of the limbic system, including the amygdala, hippocampus, and
174 y pinpointed selective cell types within the limbic system, including the hippocampal dentate gyrus g
175 ctral and temporal alterations in the fronto-limbic system, including the ventromedial prefrontal cor
176 rior cingulate cortex (ACC), a region of the limbic system involved in processing the emotional compo
177 monstrated that DA release in regions of the limbic system is important in the promotion of REM sleep
178                                          The limbic system is necessary to associate stimuli with the
179 thology diagnosis.SIGNIFICANCE STATEMENT The limbic system is often compromised in diverse clinical c
180                 Our results suggest that the limbic system is one of the important loci for the neuro
181                                          The limbic system is thought to underlie dysfunctional affec
182 gdaloid complex (AC), a key component of the limbic system, is a brain region critical for the detect
183   LAMP is expressed by neurons composing the limbic system, is highly conserved between rodents and h
184 ), which provides top-down modulation of the limbic system, is unknown.
185 ressant efficacy have largely focused on the limbic system, leaving it unclear whether this signaling
186 r results suggest that alpha-activity in the limbic system may be a signature of symptom severity in
187  that abnormal neural connections within the limbic system may contribute to the social impairments o
188  findings extend previous reports of altered limbic system microstructural disorganization as a trait
189 ttern of progression of the lesion along the limbic system network and an associated memory impairmen
190 eceptor subunit Grin2b, were modified in the limbic system of adult animals, in a region-specific, se
191 enia tecta and other regions of the anterior limbic system of male weanling rats 3 days after four do
192 d in producing degeneration in the posterior limbic system of saline controls.
193 d in cortical and subcortical regions of the limbic system of the adult and developing rat central ne
194 is preferentially expressed in the olfactory-limbic system of the adult rat brain.
195 vior, the neural activity of neocortical and limbic systems often reflects dynamic combinations of se
196                Lesions induced by CHI in the limbic system on the right may inhibit subsequent manife
197 ection to the ventral pallidum (VP), a major limbic system output region.
198    Abnormal accumulation of alpha-syn in the limbic system, particularly in the hippocampus, correlat
199 rized by recurrent seizures generated in the limbic system, particularly in the hippocampus.
200 trajectory of white matter tracts within the limbic system, particularly the uncinate fasciculus.
201 s in glucose utilization concentrated in the limbic system, particularly those areas mediating motiva
202 tem damage, but there is uncertainty whether limbic system pathology does in fact leave the SRT unimp
203 o the occipital lobe, cortical spinal tract, limbic system, piriform lobe, and entorhinal pathway was
204                                          The limbic system plays a critical role in motivation, emoti
205 recent imaging studies that suggest that the limbic system plays an important role in the mediation o
206  field potential (LFP) activity in the human limbic system reflect disease state and symptom severity
207 eptide Y Y1R differentially expressed in the limbic system regulates anxiety and stress responses via
208   The mechanism by which dlPFC regulates the limbic system remains largely unclear.
209  of the amygdala, a central structure of the limbic system, remains poorly understood.
210 h the GPe is disynaptically connected to the limbic system, representing novel access points for the
211 s, and tuberomammillary nucleus region), the limbic system, sensory processing nuclei, and additional
212  associated with worse psychiatric symptoms, limbic system structural abnormalities (hippocampal volu
213  is associated with psychiatric symptoms and limbic system structure in healthy athletes, with increa
214 eexperiencing criterion had fewer lesions in limbic system structures (eg, the cingulum) on the right
215 ibited substantial genetic associations with limbic system structures and neuropsychiatric traits inc
216 nd selective activation of somatosensory and limbic system structures in the brain and brainstem foll
217 d disorders because they connect subcortical limbic system structures such as the amygdala with the p
218          Labelled neurons were also found in limbic system structures such as the bed nucleus of stri
219 ely correlated with fMRI signals measured in limbic system structures, including the anterior hypotha
220  long-lasting alterations in the function of limbic system structures, including the nucleus accumben
221  disorder featured comparable changes in the limbic system, such as the fornix and cingulum.
222  describe how structures supplying NE to the limbic system, such as the nucleus tractus solitarius (N
223  to abnormal structures in the temporal lobe limbic system suggests that p3 may be a contributing fac
224 lls) or head direction (HD cells) in the rat limbic system suggests that these cell types are importa
225 : Although neuropathologic studies show that limbic system tau disease occurs ubiquitously in middle-
226 olve molecules and processes in the immature limbic system that are activated by stressful challenges
227  also connections within the networks of the limbic system that are both critical and permissive for
228 PY is abundantly expressed in regions of the limbic system that are implicated in arousal and in the
229 ntified a population of cells throughout the limbic system that discharge as a function of the animal
230 g a sensitized BOLD response in the extended limbic system that may subserve the process of dependenc
231 specific subcortical forebrain nuclei of the limbic system that project to the thalamus and to cortic
232 the patterns of binding in the olfactory and limbic systems, that PP receptors might be involved in p
233 ogical disorder includes overactivity of the limbic system, the development of an internal symptom mo
234 ional perspective of selected regions of the limbic system, the medial temporal lobe structures-the h
235 , revealed a high level of expression in the limbic system-the hippocampal formation, the amygdala, t
236  in neocortical association areas and in the limbic system throughout life, where the protein might p
237 tex, which modulates activity throughout the limbic system to enable behavioral flexibility.
238  with the ventromedial prefrontal cortex and limbic system, together with dorsal and fronto-striatal
239 limbic compromise, it is unknown whether the limbic system undergoes dynamic regional perfusion netwo
240  has identified key neuronal networks of the limbic system underlying social behavior.
241 tion representation and are connected to the limbic system via the insula.
242 ed a conserved pattern of expression in the "limbic system." We suggest that FnTm2 may be sensitive t
243 ve individuals, volumes of structures in the limbic system were consistently associated with current
244 e longitudinal changes of gradient scores in limbic system were significantly associated with symptom
245 mphetamine can produce notable damage to the limbic system when seizures occur and to the basal gangl
246 d training group showed higher FC within the limbic system, whereas the nonexternally reinforced grou
247  nucleus, insula, and cortical spread to the limbic system, whereas the occipital lobes and cerebella
248  likely increased activation of the extended limbic system, which was dysfunctional, and therefore le
249  major depressive disorder were found in the limbic system, which were similar to the differences in
250 mber of excitatory synaptic loops within the limbic system, which, in addition to being important in
251  both, Ncdh and Pcdh19, in structures of the limbic system with overlapping expression patterns parti
252 h drug-resistant epilepsy, we stimulated the limbic system with single-pulse electrical stimulation.
253 yrophilic neurons in discrete regions of the limbic system; within 24 hr of seizures, a significant p
254 reduced the accumulation of alpha-syn in the limbic system without any significant deleterious effect
255 of the amygdala (CeA), a major output of the limbic system; yet, the neuronal signaling pathways unde

 
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