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1 age was assessed by multipoint nonparametric linkage analyses.
2 e considered for genome-wide, non-parametric linkage analyses.
3 o either 17q23.1-23.2 or 17q11.1-12 based on linkage analyses.
4 metry techniques, along with composition and linkage analyses.
5 , and 3) how association analyses complement linkage analyses.
6 en developed explicitly for meta-analysis of linkage analyses.
7 le of men and women by using association and linkage analyses.
8 re characterized by glycosyl composition and linkage analyses.
9 us versions and validated our assemblies via linkage analyses.
10 ith various glycosidase digestions and GC-MS linkage analyses.
11 iabetes diagnosis, and BMI and nonparametric linkage analyses.
12 n the peak LOD score after adjustment in the linkage analyses.
13 se of these markers for whole-genome genetic linkage analyses.
14        By using parametric and nonparametric linkage analyses and allowing for genetic heterogeneity
15 ed heightened interest after a succession of linkage analyses and association studies identified mult
16 , genotype error detection and affected pair linkage analyses and can handle more markers than other
17 rome, and the collection of new families for linkage analyses and clinical assessments.
18                         Recent evidence from linkage analyses and follow-up candidate gene studies su
19                Candidate genes identified by linkage analyses and genome-wide association scans in ad
20 h a dense set of 31 markers using multipoint linkage analyses and monitoring for shared marker allele
21                                      Subsets linkage analyses and recurrence risk ratios in a combine
22  had previously been suggested based on gene linkage analyses and shared clinical features.
23         We concluded that the combination of linkage analyses and WGS to search for disease genes sti
24  was established by glycosyl composition and linkage analyses, and by one- and two-dimensional NMR sp
25 mericans, including studies of heritability, linkage analyses, and candidate genes.
26 ia, Europe, and South America, haplotype and linkage analyses, and case-control analyses, and determi
27                      Although animal models, linkage analyses, and genome-wide association studies ha
28                     Glycosyl composition and linkage analyses are important first steps toward unders
29                                              Linkage analyses are often limited by both sample size a
30 the available marker information, multilocus linkage analyses are performed.
31 e pursued both allele-sharing and parametric linkage analyses as implemented in Genehunter, version 2
32        We applied model-based and model-free linkage analyses, as well as the pedigree disequilibrium
33  performed Haseman-Elston and DeFries-Fulker linkage analyses, as well as transmission/disequilibrium
34                                Nonparametric linkage analyses at microsatellites surrounding D12S83 w
35 s and of genotype data can cause problems in linkage analyses based on genome-scan data.
36                                  Genome-wide linkage analyses, based on 1142 participants from the 31
37                            We have performed linkage analyses between the disorder and markers close
38 which involved non-parametric and parametric linkage analyses, both yielded significant linkage peaks
39                Stratification of genome-wide linkage analyses by disease associated variants is now p
40                                      Sibpair linkage analyses, by contrast, provided only marginal su
41                           QTL mapping and/or linkage analyses can establish a large genomic region (
42                     Phylodynamic and genetic linkage analyses can highlight populations and geographi
43                                Here, genetic linkage analyses combined with exome sequencing identifi
44                                              Linkage analyses conditioning on pedigrees in which one
45                                     Although linkage analyses confirmed the presence of a genetic sus
46                                   Multipoint linkage analyses confirmed this interval and generated a
47                                              Linkage analyses for COL2A1 locus markers were performed
48 hese findings support the utility of genetic linkage analyses for identification of novel risk factor
49                                   Multipoint linkage analyses for insulin sensitivity phenotypes were
50 e-components technique to conduct multipoint linkage analyses for localizing susceptibility genes tha
51 oint lod score and nonparametric (Zlr score) linkage analyses for social phobia were completed with A
52 e-components technique to conduct multipoint linkage analyses for two phenotypes: type 2 diabetes (a
53 bserved at marker D22S685, and nonparametric linkage analyses gave consistent results, with a P value
54  four major categories: association studies, linkage analyses, gene expression and literature search.
55                           Recent genome-wide linkage analyses have identified a locus encoding suscep
56                                        Human linkage analyses have implicated the MS4A2-containing ge
57  such as genome-wide association studies and linkage analyses, have been confounded by the genetic an
58                             Because previous linkage analyses identified chromosome 1q25-32, which ha
59                                              Linkage analyses identified QTLs associated with dietary
60                                   Genomewide linkage analyses identified the recessive IBM locus on c
61                           Quantitative trait linkage analyses in 1044 pairs of siblings, by using bot
62                                 We performed linkage analyses in 146 multiplex families ascertained t
63                           We carried out the linkage analyses in 22 Finnish multiplex MS families ori
64                                      Genetic linkage analyses in a family with autosomal dominant age
65   We undertook platelet function studies and linkage analyses in a pedigree of CSS-affected German sh
66 6 to maximize the informativeness of genetic linkage analyses in a region where they previously repor
67                       In addition, two-point linkage analyses in an enlarged family sample (n = 670)
68 omputationally practical option for accurate linkage analyses in genome scans with both large numbers
69 nic analysis and greater refinement of viral linkage analyses in HIV prevention studies.
70                           Here, we conducted linkage analyses in order to (1) create a platform for Q
71        This leads to specific strategies for linkage analyses in schizophrenia.
72                                        Prior linkage analyses in the Boston Early-Onset COPD Study ha
73  conducted age at diagnosis (AAD) stratified linkage analyses in the Diabetes UK Warren 2 sibpairs.
74                                              Linkage analyses in this subset of 65 pedigrees generate
75                            We have performed linkage analyses in two inter-related inbred kindreds, c
76        We performed nonparametric multipoint linkage analyses, in 152 families from the Autism Geneti
77 xamine this latter possibility, we performed linkage analyses, in 159 pedigrees affected by hereditar
78                        Cumulative multipoint linkage analyses indicate that an FHF gene is likely to
79                                              Linkage analyses indicate that FHL is genetically hetero
80                   Singlepoint and multipoint linkage analyses indicate that marker D10S1654 on chromo
81             However, following haplotype and linkage analyses indicate the presence of additional Pst
82                                     Glycosyl linkage analyses indicated that N-acetylglucosamine (Glc
83 identification of genes under selection, and linkage analyses involving association studies in natura
84              A common problem encountered in linkage analyses is that execution of the computer progr
85 apped to mouse chromosome 11 by conventional linkage analyses, its candidate region was broad and und
86                                              Linkage analyses mapped the defect to this type II kerat
87 as characterized by glycosyl composition and linkage analyses, mass spectrometry, and one- and two-di
88 S was determined by glycosyl composition and linkage analyses, matrix-assisted laser desorption-time
89                                     Previous linkage analyses of 19 cutaneous malignant melanoma/dysp
90    DESIGN, SETTING, AND PATIENTS: Model-free linkage analyses of 21 concordant-affected sibling pairs
91                     Association and sib-pair linkage analyses of a polymorphism in intron 7 of the TP
92  using segregation and joint segregation and linkage analyses of a quantitative trait.
93  statistic [Z] 2.98; P=.001), and subsequent linkage analyses of additional markers and association a
94 gram to provide parametric and nonparametric linkage analyses of affected individuals.
95                            After genome-wide linkage analyses of blood pressure levels, we resequence
96  sought to incorporate AAO as a covariate in linkage analyses of BP using two different methods, LODP
97                                        Valid linkage analyses of censored data require the developmen
98 pertension-related traits employs genomewide linkage analyses of families and association studies of
99 he findings from three successful genomewide linkage analyses of families segregating autosomal reces
100                                     Previous linkage analyses of families with multiple cases of schi
101 t chronic obstructive pulmonary disease, and linkage analyses of FEF(25-75%) and FEF(25-75%)/FVC were
102                                              Linkage analyses of genetic diseases and quantitative tr
103 add an important dimension of complexity for linkage analyses of human autoimmune disease.
104              In black individuals, bivariate linkage analyses of log serum creatinine and pulse press
105                                              Linkage analyses of melanoma pedigrees from many countri
106 een missed and deserves wider application in linkage analyses of quantitative traits.
107     Nonparametric, sibling-pair, genome-wide linkage analyses of refraction (spherical equivalent adj
108                                  Genome-wide linkage analyses of schizophrenia have identified severa
109    We performed parametric and nonparametric linkage analyses of seven genetic markers on chromosome
110                                              Linkage analyses of the 12 endophenotypes collectively i
111                                              Linkage analyses of the 6,055 single-nucleotide polymorp
112  sample of 126 ASPs to 270 ASPs and provides linkage analyses of the entire sample, using polymorphic
113                          However, additional linkage analyses of these crosses showed that loci contr
114                      We conducted genomewide linkage analyses on 1,152 individuals from 250 families
115            We therefore performed multipoint linkage analyses on both GFR measures using models that
116  To overcome these issues, we have performed linkage analyses on members of 301 HPC families genotype
117 basis for this strain-dependent sensitivity, linkage analyses on the progeny of a B6CBAF1 intercross
118  We conducted multipoint variance components linkage analyses on these factors with the program SOLAR
119 ined geographical regions could be useful in linkage analyses or quantitative trait locus studies.
120 vidence for linkage was observed by sib-pair linkage analyses (P values ranged from .24 to .81).
121 ozygosity rates in individuals, and powerful linkage analyses, particularly in coding regions.
122                                  Genome-wide linkage analyses performed in a Finnish study sample hav
123 overing the entire X chromosome were used in linkage analyses performed on 42 genomic DNA samples (13
124  6 novel baboon microsatellites were used in linkage analyses performed with the MultiMap expert syst
125  opportunity to identify the disease gene by linkage analyses, positional cloning, and analysis of ca
126     Both pedigree-based and population-based linkage analyses rely on estimating recent IBD, and evid
127                                   Stratified linkage analyses resulted in a marked increase in eviden
128                                              Linkage analyses revealed 8 QTLs linked to number of DNs
129               Multipoint variance components linkage analyses revealed suggestive linkage on chromoso
130                                              Linkage analyses revealed that genes influencing risk fo
131                                 Pedigree and linkage analyses revealed that the Class III phenotype (
132                                      Genetic linkage analyses show a TBC1D1 R125W missense variant co
133                                    Two-point linkage analyses showed linkage to markers on 16q22 with
134                                              Linkage analyses showed suggestive linkage to the COL2A1
135                       Model-free, multipoint linkage analyses (SIBPAL2, SAGE version 4.0) and exclusi
136                                    Bivariate linkage analyses strongly rejected both the null hypothe
137                                        These linkage analyses suggest that a genetic element on chrom
138                                   Parametric linkage analyses suggested a low-penetrance, dominant mo
139                               In genome-wide linkage analyses, suggestive linkage (logarithm of odds
140                                     Applying linkage analyses that assume linkage equilibrium to dens
141 e now present the results of our genome-wide linkage analyses that provide evidence that regions on c
142                                   By genetic linkage analyses, the gene responsible for this disease,
143                       After over a decade of linkage analyses, the identification of non-major histoc
144                                         From linkage analyses, the location of elg on chromosome 4, b
145                                    In SIBPAL linkage analyses, the maximum number of sibpairs availab
146 the application of traditional nonparametric linkage analyses to complex human traits and diseases.
147 nomic regions shared by cluster members, and linkage analyses to establish their co-inheritance patte
148 nn, we carried out variance components-based linkage analyses to evaluate the contribution of variati
149 mosome 6q, we conducted bivariate multipoint linkage analyses to map the correlated IRS phenotypes.
150 linked by numerous genome-screen studies and linkage analyses to markers on human chromosome 5q31-q34
151 ci, we performed genomewide parent-of-origin linkage analyses under an allele-sharing model for discr
152 the data, using parametric and nonparametric linkage analyses under both a narrow phenotype model (DS
153                  Ophthalmic examinations and linkage analyses using a variety of polymorphisms were p
154                                Nonparametric linkage analyses using GENEHUNTER and ASPEX were perform
155                               The multipoint linkage analyses using Genehunter and SIMWALK 2.40 provi
156                     Two-point and multipoint linkage analyses using informative markers excluded most
157 ilies (599 sibpairs) identified through tree-linkage analyses using interacting covariates of age, se
158  merging difficulties by simply carrying out linkage analyses using laboratory-specific allele labels
159 rmed oligogenic simultaneous segregation and linkage analyses using Markov Chain Monte Carlo methods
160 r three diagnostic models, we have conducted linkage analyses using parametric (heterogeneity LOD [HL
161                                 We performed linkage analyses using parametric and allele-sharing mod
162                                              Linkage analyses using scores from the Social Responsive
163                                              Linkage analyses using the resultant component scores id
164                             After performing linkage analyses using various methods implemented in th
165                                   Model-free linkage analyses, using a dichotomous definition for dia
166                                        Using linkage analyses, we discovered a quantitative trait loc
167                                  In previous linkage analyses, we identified a major locus on chromos
168 rming standard parametric and non-parametric linkage analyses, we observed a 'highly significant' lin
169      Using both nonparametric and parametric linkage analyses, we obtained evidence for a small numbe
170 cluding 1,249 sib pairs) that are useful for linkage analyses, we performed a whole-genome linkage sc
171 ere genotyped, and multipoint allele-sharing linkage analyses were carried out.
172                    Multipoint, nonparametric linkage analyses were conducted in affected relative pai
173                                   Genomewide linkage analyses were conducted of serum creatinine, est
174                                              Linkage analyses were conducted to quantify the evidence
175                         Two- and three-point linkage analyses were conducted using a previously repor
176                                              Linkage analyses were conducted using data from a genome
177                                              Linkage analyses were conducted using model-free methods
178                     Homozygosity mapping and linkage analyses were conducted using two pedigrees deri
179     Parametric and nonparametric genome-wide linkage analyses were conducted with Morgan and Merlin i
180                                              Linkage analyses were conducted with parametric and nonp
181                     Nonparametric multipoint linkage analyses were conducted, and the strongest evide
182 ped for 335 markers, and multipoint sib pair linkage analyses were conducted.
183                                   Genomewide linkage analyses were first performed separately in each
184 , and a recessive model with 85% penetrance, linkage analyses were performed assuming a narrow (BP an
185                              Segregation and linkage analyses were performed for adult height in a po
186                     Two-point and multipoint linkage analyses were performed for all 218 families and
187                                              Linkage analyses were performed on 1201 samples from 10
188                            Affected sib-pair linkage analyses were performed on 98 diabetic sibling p
189           Genomewide, model-free, multipoint linkage analyses were performed separately for each popu
190 to localize susceptibility genes, stratified linkage analyses were performed using clinical criteria
191                                   Multipoint linkage analyses were performed using data from a 10-cM-
192                                     Standard linkage analyses were performed using GENEHUNTER and Has
193 c and nonparametric two-point and multipoint linkage analyses were performed using the FASTLINK, HOMO
194                                              Linkage analyses were performed using three methods (sin
195                                              Linkage analyses were performed with 11 candidate genes,
196                 Parametric and nonparametric linkage analyses were performed with a denser map of mar
197                                              Linkage analyses were performed with an "affecteds-only"
198                     Multipoint nonparametric linkage analyses were performed with diabetes, and diabe
199                     Genome-wide sibling-pair linkage analyses were performed with the phenotypes como
200                                      Genetic linkage analyses were performed with the use of microsat
201       Two-point and multipoint nonparametric linkage analyses were performed, and 1-parameter allele-
202                Both two-point and multipoint linkage analyses were performed, by use of affected/unaf
203                    Multipoint non-parametric linkage analyses were performed, with further stratifica
204       Nonparametric multipoint and two-point linkage analyses were performed.
205                     Two-point and multipoint linkage analyses were performed.
206 ellite markers in 12 families, and two-point linkage analyses were performed.
207                     Nonparametric multipoint linkage analyses were the primary approach, although par
208                                   Multipoint linkage analyses were undertaken using both non-parametr
209                                   Multipoint linkage analyses were undertaken using both nonparametri
210 ed from 46 U.S. families in which multipoint linkage analyses were undertaken using nonparametric (mo
211 g followed by polygenic risk score (PRS) and linkage analyses were used to identify the genetic etiol
212 ith bipolar disorder and (2) association and linkage analyses with a common silent exonic polymorphis
213              The authors conducted lod score linkage analyses with both phenotypes using both a domin
214                    Clinical examinations and linkage analyses with polymerase chain reaction (PCR) po
215                                              Linkage analyses yielded a total of 95 linkage groups, s

 
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