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2 ynthesis of linoleate [18:2(9,12)] and gamma-linolenate [18:3(6,9,12)] on this phospholipid is simila
3 s, which has reduced levels of linoleate and linolenate and elevated oleate in cytosolic phospholipid
4 nergy as linoleate, 0.08% of energy as alpha-linolenate, and 0.06% of energy as arachidonic acid) to
5 nergy as linoleate, 1.06% of energy as alpha-linolenate, and 0.08% of energy as arachidonic acid).
11 periments on thermal degradation of d5-ethyl linolenate indicate that off-aroma volatiles originate t
12 he essential fatty acids linoleate and alpha-linolenate into long-chain polyunsaturated fatty acids.
14 tivity in the GLU (7-hydroxycoumarinyl-gamma-linolenate) micelle assay, which contains lipid and dete
15 ), docosahexaenoate (P-interaction = 0.001), linolenate (n-3 or n-6, P-interaction = 0.005) and docos
16 e other hand, the Omega-3 polyunsaturated FA linolenate neither induced insulin resistance nor promot
17 ters (FAME: stearate, oleate, linoletate and linolenate) on a mixture of three plant sterols (PS: cam
18 6; erucate, p < 0.001; nervonate, p < 0.001; linolenate, p < 0.001), and plant sterols (campesterol,
19 vitamins C and E and in which linoleate and linolenate represented 1.4% and 0.08% of the total calor
20 eate, stearate, oleate, linoleate, and alpha-linolenate, representing >80% of the FFA fraction in the
21 ed to their double bonds; with linoleate and linolenate substrates, abstraction of the bis-allylic H
23 s reveals marked deficiency in linoleate and linolenate when compared with wild type (WT) or overexpr