ライフサイエンスコーパス: lipemia

1 effects on postprandial vascular function or lipemia.

2 on of GLP-1 and GLP-2 decreased postprandial lipemia.

3 activity resulted in decreased postprandial lipemia.

4 ic acid (16:0) in the sn-2 position decrease lipemia.

5 of saturated fats may influence postprandial lipemia.

6 exposure, resulting in elevated postprandial lipemia.

7 s a primary role in determining postprandial lipemia.

8 en milk protein and milk fat on postprandial lipemia.

9 ent and an attenuated impact on postprandial lipemia.

10 ts with a focus on postprandial glycemia and lipemia.

11 ized SSOB resulted in 53% lower postprandial lipemia, 23% higher hepatic lipase activity, and a 25% l

12 pared with gluteofemoral fat on postprandial lipemia after a high-fat meal in individuals with obesit

13 usally associated with elevated postprandial lipemia after a high-fat meal, independent of fasting li

14 esterol concentrations and less postprandial lipemia after the low-fat than the high-fat diet.

15 relationship between increased postprandial lipemia and atherosclerosis.

16 differently, which could affect postprandial lipemia and contribute to the relation between abdominal

17 cturing dietary fat to modulate postprandial lipemia and lipid beta-oxidation in humans with differen

18 Through their effects on postprandial lipemia and oxidative stress, polyphenols may favorably

19 understanding the links between postprandial lipemia and the accumulation of lipid within vessels, a

20 t information on the control of postprandial lipemia and the biological effects of chylomicron remnan

21 ucoregulatory function, greater postprandial lipemia, and greater lipid oxidation rates than do their

22 ociations with glucoregulation, postprandial lipemia, and lipid oxidation rates.

23 ic stability on gastric emptying (GE), acute lipemia, and satiety.

24 ipoprotein particles; increased postprandial lipemia; and abnormal apolipoprotein A1 and B metabolism

25 dominal obesity and exaggerated postprandial lipemia are independent risk factors for cardiovascular

26 In children, postprandial lipemia, as indicated by the incremental area under the

27 meal macronutrients (3.6%) for postprandial lipemia, but not for postprandial glycemia (6.0% and 15.

28 d release (bioaccessibility) on postprandial lipemia by comparing lipid encapsulated by cell walls wi

29 erance (characterized by higher postprandial lipemia, cholesterol enrichment of triglyceride-rich lip

30 her the association of WHR with postprandial lipemia could be causal, we performed instrumental varia

31 ts on postprandial fibrinolytic activity and lipemia, factor VII coagulant (FVII:c) activity, and act

32 Despite the lower lipemia following the SFA-rich fats, increased proathero

33 se Western dietary habits cause postprandial lipemia for a major part of a day and, moreover, increas

34 Predictors of postprandial lipemia have not been explored in children.

35 ing herbs and spices attenuates postprandial lipemia, hyperglycemia, and oxidative stress, and improv

36 with an oral fat load enhances postprandial lipemia in animals and humans.

37 ates the impact of dairy fat on postprandial lipemia in healthy subjects.

38 d in the sn-2 position decrease postprandial lipemia in healthy subjects.

39 impact of the cheese matrix on postprandial lipemia in humans has not yet been evaluated.In healthy

40 sma lipids and lipoproteins and postprandial lipemia in premenopausal African American and white wome

41 In humans, postprandial lipemia increased plasma glucagon concentrations and led

42 We concluded that postprandial lipemia induces pancreatic alpha cell dysfunction charac

43 experimental data suggest that postprandial lipemia is an independent risk factor for atherosclerosi

44 High postprandial lipemia is associated with increased risk of cardiovascu

45 ffect of cell wall structure on postprandial lipemia is unknown.

46 d in glucomannan or glucomannan-spirulina on lipemia, liver glutathione status, antioxidant enzymes a

47 The greater HF morning lipemia occurred with elevated glucose, insulin, and non

48 n did not modify fat digestion, postprandial lipemia, or lipid metabolism measured by stable isotope

49 of TM6SF2 in the regulation of postprandial lipemia, potentially through a similar function for TM6S

50 Exaggerated postprandial lipemia (PPL) is a factor in atherogenesis, involving en

51 A compared with DHA only during postprandial lipemia relative to control high-oleic acid meals; the s

52 ir most recent fundus examination: untreated lipemia retinalis (abnormal fundus) and resolved lipemia

53 mia retinalis (abnormal fundus) and resolved lipemia retinalis (normal fundus).

54 The patients with history of lipemia retinalis (untreated and resolved) exhibited a h

55 comprised 10 eyes with documented history of lipemia retinalis and 10 participants as healthy control

56 Patients with untreated lipemia retinalis had a significantly higher retinal art

57 Moreover, patients with untreated lipemia retinalis had significantly smaller retinal arte

58 Patients with a confirmed history of lipemia retinalis were grouped into two cohorts based on

59 alteration in retinal oximetry, in untreated lipemia retinalis, and in retinal blood flow, in both th

60 acute pancreatitis, eruptive xanthomas, and lipemia retinalis.

61 ction despite the fundus changes observed in lipemia retinalis.

62 tion and retinal and choroidal blood flow in lipemia retinalis.

63 ower in patients with resolved and untreated lipemia retinalis.

64 tudy on 10 eyes (5 patients) with history of lipemia retinalis.

65 in may explain the reduction in postprandial lipemia seen in clinical trials of this agent and may pr

66 not associated with fasting or postprandial lipemia test triacylglycerol or free fatty acids (P > or

67 ubgroup (n = 36) also underwent postprandial lipemia tests with lipid oxidation rate measurements.

68 ke had more dramatic effects on postprandial lipemia than did MSF after fat intake, possibly because

69 agents also improve fasting and postprandial lipemia, the latter more significantly than the former.

70 depots show opposing effects on postprandial lipemia.This trial was registered at as ISRCTN25867281.

71 Vagal stimulation enhanced postprandial lipemia via effects on both chylomicron and VLDL metabol

72 oviral therapy predict enhanced postprandial lipemia, which is an emerging cardiovascular disease ris

73 , we tested the hypothesis that postprandial lipemia with its characteristic elevation of triglycerid