ライフサイエンスコーパス: lipid metabolism

1 s one of the main enzymes in pro-ferroptotic lipid metabolism.

2 d in inflammation, extracellular matrix, and lipid metabolism.

3 and point to a coordinate mitotic control of lipid metabolism.

4 kinases to the control of cell survival and lipid metabolism.

5 ssential to a comprehensive understanding of lipid metabolism.

6 s in the metabolism of SCLC cells, including lipid metabolism.

7 es, such as the effects of the microbiome on lipid metabolism.

8 for inflammasomes in diseases with defective lipid metabolism.

9 is unknown whether p16 also controls hepatic lipid metabolism.

10 plication through direct effects on the host lipid metabolism.

11 ls and associated with perturbed glucose and lipid metabolism.

12 zing to the brain showed evidence of altered lipid metabolism.

13 ype dictates the response to drugs targeting lipid metabolism.

14 g PPARdelta and in turn affecting macrophage lipid metabolism.

15 idiabetic lipokines that connect glucose and lipid metabolism.

16 mediating insulin secretion, processing, and lipid metabolism.

17 as a substrate, otherwise unknown in animal lipid metabolism.

18 kines as endocrine regulators of glucose and lipid metabolism.

19 pathologies associated with impaired hepatic lipid metabolism.

20 nriched pathways related to inflammation and lipid metabolism.

21 sregulated inflammatory response and altered lipid metabolism.

22 erstand their effects in seeds especially on lipid metabolism.

23 ds, but not in the presence of regulators of lipid metabolism.

24 cy led to liver inflammation and deregulated lipid metabolism.

25 lls have alterations in glutamine, bile, and lipid metabolism.

26 or trait-associated DNA regions to regulate lipid metabolism.

27 sary to confirm their actual role in ruminal lipid metabolism.

28 ssociates with genes involved in glucose and lipid metabolism.

29 specifically targets APOB RNA in regulating lipid metabolism.

30 g an unrecognized role for PMP22 in cellular lipid metabolism.

31 insulin resistance and impaired glucose and lipid metabolism.

32 ctivity and S1P in AD to perturbed endosomal lipid metabolism.

33 gested that Mct6 plays a role in glucose and lipid metabolism.

34 nd rodent models through effects on cellular lipid metabolism.

35 oles in ER-associated protein biogenesis and lipid metabolism.

36 te phosphatase is a key regulatory enzyme in lipid metabolism.

37 onduit to integrate the circadian clock with lipid metabolism.

38 rapamycin cancelled the latter's effects on lipid metabolism.

39 oposed to be a dominant regulator of hepatic lipid metabolism.

40 genes involved in the regulation of hepatic lipid metabolism.

41 at M1, M3, and CB, signifying differences in lipid metabolism.

42 cell function can be modulated by changes in lipid metabolism.

43 e cross talk between glucose utilization and lipid metabolism.

44 ribosome biogenesis and function, as well as lipid metabolism.

45 7 metabolites) comprising diverse aspects of lipid metabolism.

46 by altering the BA pool, host signalling and lipid metabolism.

47 reprogramed to be hyperactive in glucose and lipid metabolism.

48 e that gut-derived bacterial SLs affect host lipid metabolism.

49 ocytosis, processing of neuronal debris, and lipid metabolism.

50 abilities in central carbon, amino acid, and lipid metabolism.

51 or-activated receptor) that regulate cardiac lipid metabolism.

52 mechanisms may involve impaired glucose and lipid metabolism.

53 Most tumours have an aberrantly activated lipid metabolism(1,2) that enables them to synthesize, e

54 ariants located in or near genes involved in lipid metabolism (ABCA1, CETP, APOE, and LIPC) with meta

55 ssion, but the commonality and difference of lipid metabolism among pan-cancer is not fully investiga

56 f the method and how current descriptions of lipid metabolism and ACP function are incomplete.

57 ceramide synthase 4 first disturbs epidermal lipid metabolism and adult epidermal barrier function, u

58 ggest that Mcp2 is involved in mitochondrial lipid metabolism and an increase of this involvement by

59 tients to ameliorate hyperglycaemia, adverse lipid metabolism and blood pressure.

60 d changes in expression of genes involved in lipid metabolism and calcium handling in cells of the pe

61 -dependent mechanism.IMPORTANCE MDV disrupts lipid metabolism and causes atherosclerosis in MDV-infec

62 organic compounds (VOCs) that were linked to lipid metabolism and cholesterol synthesis were elevated

63 We investigated links between lipid metabolism and cognitive function in mice and huma

64 e show here that PGAM5 inhibition attenuates lipid metabolism and colorectal tumorigenesis in mice.

65 bolic adaptations, through the regulation of lipid metabolism and contributes to fasting-induced free

66 rplay between hepatic glucagon signaling and lipid metabolism and describe a new posttranscriptional

67 abolic consequences such as impaired hepatic lipid metabolism and development of nonalcoholic fatty l

68 affects expression of genes associated with lipid metabolism and diapause preparation in C. finmarch

69 rmation processing, carbohydrate metabolism, lipid metabolism and digestive and endocrine system path

70 abolic regulator, is sufficient to reprogram lipid metabolism and drive progression of RAS mutant can

71 vo lipid synthesis, providing a link between lipid metabolism and generating and maintaining NE ident

72 IR in humans to examine pathways controlling lipid metabolism and gluconeogenesis.METHODSCross-sectio

73 lisin/kexin type 9 is a central regulator of lipid metabolism and has been implicated in regulating t

74 Furthermore, HFD altered lipid metabolism and HDL oxidation and also induced live

75 These effects manifest in altered lipid metabolism and IBA responses leading to disrupted

76 Genes related to lipid metabolism and immune response that are associated

77 , indicating the potential crosstalk between lipid metabolism and immune response.

78 as key regulators of hepatic bile acid (BA)/lipid metabolism and inflammation.

79 odimer partner (Shp) plays a complex role in lipid metabolism and inflammation.

80 ncrease our understanding of endogenous leaf lipid metabolism and its ability to adapt to metabolic e

81 lipidome could provide better insights into lipid metabolism and its link to cardiovascular diseases

82 ne, a regulator of metformin effects on host lipid metabolism and lifespan.

83 Both lipid metabolism and lipid synthesis are activated in DE

84 The relationship between lipid metabolism and longevity remains unclear.

85 lar compartments, including reprogramming of lipid metabolism and mitochondria bioenergetics.

86 nts is associated with beneficial effects on lipid metabolism and other markers of cardiovascular dis

87 ovel mechanism that is crucial for normal ER lipid metabolism and protects the ER from dysfunction.

88 ciprocal kinase-substrate regulation whereby lipid metabolism and proteomic stability intertwine.

89 ves as a rich resource for probing mammalian lipid metabolism and provides opportunities for the disc

90 In vivo, DECR1 deletion impairs lipid metabolism and reduces CRPC tumour growth, emphasi

91 To better understand lipid metabolism and seed oil synthesis in canola (Brass

92 The lipid metabolism and specifically the sub pathways invol

93 dentification of candidate genes involved in lipid metabolism and the assembly of a proposed pathway

94 loss and blocked hepatocyte proliferation on lipid metabolism and the consequent impact on whole-body

95 substantially reduced or no side effects on lipid metabolism and the immune system.

96 haracteristics are likely to influence human lipid metabolism and the subsequent effect on health fol

97 for genome replication by manipulating host lipid metabolism and trafficking, but in most cases, we

98 XRbeta), nuclear receptors known to regulate lipid metabolism and tumor-immune interaction, are highl

99 changes, by longitudinally interrogating the lipid metabolism and white blood cell transcriptomic mar

100 onset disease is encoded by loci involved in lipid metabolism and/or encoded by microglia.

101 nal studies implicate both Abeta and altered lipid metabolism and/or signaling as key pathogenic driv

102 ays (regulating mitochondrial biogenesis and lipid metabolism) and indicators of bone metabolism.

103 ed in hepatocytes as a critical regulator of lipid metabolism, and clinical trials targeting PCSK9 re

104 function through effects on vasoreactivity, lipid metabolism, and inflammation.

105 bited decreased energy expenditure, impaired lipid metabolism, and insulin resistance.

106 sitizing mechanisms, including adipogenesis, lipid metabolism, and insulin signaling.

107 ion of antioxidants, inflammatory cytokines, lipid metabolism, and organ growth of largemouth bass (L

108 aired insulin sensitivity, glycemic control, lipid metabolism, and sympathetic output in females but

109 ic processes and provides a link between CR, lipid metabolism, and the circadian clock.

110 f K337 strongly influences NADPH generation, lipid metabolism, and the susceptibility to colorectal t

111 set of plant genes, including carbohydrate, lipid-metabolism, and defence-related genes, differentia

112 a large number of molecules associated with lipid metabolism are known to be implicated in MNDs, the

113 We have also shown that alterations in lipid metabolism are mediated by adrenomedullin (ADM) in

114 ing systemic infection, data on the need for lipid metabolism are mixed.

115 he same nanoparticle, synergistic effects on lipid metabolism are observed in vitro and vivo.

116 genome sequences reveals photosynthesis and lipid metabolism are preferentially retained after a rec

117 s response remain unclear and the effects on lipid metabolism are relatively unexplored.

118 in resistance, oxidative stress, and altered lipid metabolism are some of the mechanisms by which NAF

119 Changes to lipid metabolism are tightly associated with the onset a

120 ropose a tenet that mitochondrial fusion and lipid metabolism are tightly linked to regulate AEC2 cel

121 Cell cycle progression and lipid metabolism are well-coordinated processes required

122 associated with MacTel and implicate altered lipid metabolism as a contributor to this retinal neurod

123 lthough recent studies have depicted altered lipid metabolism as an underlying feature, the detailed

124 dentify G0S2 as a dual-function regulator of lipid metabolism as well as a novel mechanism whereby he

125 ying pivotal roles in regulating glucose and lipid metabolism as well as inflammation.

126 as a result of the dysregulated glucose and lipid metabolism associated with diabetes mellitus, whic

127 associated with type 2 diabetes, glucose and lipid metabolism, associated with fasting glucose level

128 n proteins and genes involved in glucose and lipid metabolism-associated pathways.

129 growth, regeneration, as well as glucose and lipid metabolism at resting state, even when fed a high-

130 These data identify a new MEK5-ERK5-lipid metabolism axis that promotes the growth of SCLC.

131 otherwise nutrient-poor TME, access to using lipid metabolism becomes particularly valuable for susta

132 erentiation, down-regulate genes involved in lipid metabolism, block cofactor recruitment to PPARgamm

133 of these nutritional factors on glucose and lipid metabolism, body-fat distribution, and liver fat c

134 nuclear envelope is a site of regulation of lipid metabolism but there is limited appreciation of th

135 ne transcripts, not directly associated with lipid metabolism, but with immune function, signalling a

136 ilisin/kexin 9 (PCSK9) is a key regulator of lipid metabolism by degrading liver LDL receptors.

137 sterol metabolism, fatty acid biosynthesis, lipid metabolism, carotenoid metabolism, protein glycosy

138 nalysis indicates that genes associated with lipid metabolism, cholesterol biosynthesis, and circadia

139 human brain development suggest that altered lipid metabolism contributes to intellectual disability.

140 of adipose tissue expansion and glucose and lipid metabolism control, which should be taken into acc

141 t RabGAP-mediated control of skeletal muscle lipid metabolism converges with glucose metabolism at th

142 , was performed to understand the balance of lipid metabolism, conversion, and catabolism in this stu

143 e endoplasmic reticulum is a cellular hub of lipid metabolism, coordinating lipid synthesis with cont

144 We questioned how modifying intracellular lipid metabolism could be a possible means for alleviati

145 metabolism, such as Carbohydrate metabolism, Lipid metabolism, Drug metabolism, Nucleotide metabolism

146 We assessed how lipid metabolism during development from the C4 stage to

147 previously demonstrated to be important for lipid metabolism during HCMV infection: fatty acid elong

148 actor kappaB (NF-kappaB)/Relish in governing lipid metabolism during metabolic adaptation to fasting.

149 o functions are crucial in the regulation of lipid metabolism during metabolic adaptation, which may

150 s revealed altered amino acid metabolism and lipid metabolism, especially for the tryptophan-kynureni

151 fer protein (MTP) plays an essential role in lipid metabolism, especially in the biogenesis of very l

152 mmatory cytokines (TNF-alpha and TGF-beta1), lipid metabolism (FASN and CYP7A1), and organ growth (IG

153 ffected in both strains related to increased lipid metabolism, fatty acid metabolism, lipid and fatty

154 Furthermore, we observed profound changes in lipid metabolism following VLCD but not in response to R

155 ese data shed light on essential and complex lipid metabolism for soybean nodulation and nodule devel

156 We identified enrichment in hepatic and lipid metabolism gene pathways and enriched expression o

157 1) but showed characteristic alterations in lipid metabolism genes (ELOVL fatty acid elongase 3 and

158 ed that PPARgamma binding to several crucial lipid metabolism genes (fatty acid synthase, stearoyl-co

159 This is mediated by misregulation of lipid metabolism genes and is rescued by modulating CE s

160 that play a central role in regulating these lipid metabolism genes during NAFLD, including peroxisom

161 tumor microenvironment and the expression of lipid metabolism genes revealed immune-related different

162 Parameters of lipid metabolism, glucose homeostasis, and mitochondrial

163 reduced Aqp7 revealed significantly altered lipid metabolism, glutathione metabolism, and urea/argin

164 Tumour lipid metabolism has gained increasing attention over th

165 The overriding theme is that lipid metabolism has numerous but complex functions in i

166 s of subcellular disruption to autophagy and lipid metabolism have been limited to in vitro investiga

167 RP) supplementation on tissue lipid profile, lipid metabolism, health indices, oxidative stability, a

168 own adipocytes, mTORC2 regulates glucose and lipid metabolism, however the mechanism has been unclear

169 investigating the emerging role of RNF213 in lipid metabolism, hypoxia, and angiogenesis.

170 e expression that reprogram carbohydrate and lipid metabolism, impair muscle mitochondrial respirator

171 nofibrate, on liver and kidney autophagy and lipid metabolism in 5-day-old G6pc -/- mice serving as a

172 criptomic analyses to interrogate changes in lipid metabolism in a transgenic zebrafish model of onco

173 tissue, ultimately lead to abnormalities in lipid metabolism in Adipo-Drp1(flx/flx) mice.

174 first in vivo evidence that CTRP2 regulates lipid metabolism in adipose tissue and liver.

175 Thus, upon HFD feeding, Utx regulates lipid metabolism in adipose tissues by influencing the l

176 hway induces expression of genes involved in lipid metabolism in adipose tissues, and both peripheral

177 emerging mechanisms involving adipocytes and lipid metabolism in altering the response to cancer trea

178 taining ER-PM junctions in regulating PtdIns lipid metabolism in brain neurons.

179 ith minimum interruption and to characterize lipid metabolism in breast cancer cells via quantitative

180 tant (ClkDelta19/Delta19) protein on hepatic lipid metabolism in C57BL/6 Clkwt/wt and apolipoprotein

181 d n-3 PUFA on expression of genes related to lipid metabolism in dairy cows.

182 FN response genes, steroid biosynthesis, and lipid metabolism in dendritic cells and iron homeostasis

183 Recent studies suggest that POPs affect the lipid metabolism in female polar bears; however, the mec

184 Given the importance of lipid metabolism in ferroptosis and the key role of lipi

185 study strengthens the hypothesis of altered lipid metabolism in HH and susceptibility to nonalcoholi

186 nhibitors resulted in different responses on lipid metabolism in human myotubes, indicating that the

187 ctive inhibitors of DGAT1 and DGAT2 affected lipid metabolism in human primary skeletal muscle cells.

188 ations and repeated markers of adiposity and lipid metabolism in infancy.

189 dly lymphoma and induces an imbalance of the lipid metabolism in infected chickens.

190 ight into the plasticity and control of acyl lipid metabolism in leaves.

191 luable resource for further investigation of lipid metabolism in liver of chicken.

192 targeting Treg cells or their modulation of lipid metabolism in M2-like TAMs could improve cancer im

193 macrophages, 2) GW9662 significantly affects lipid metabolism in macrophages, 3) careful validation o

194 rtant than disturbed haemodynamic factors or lipid metabolism in MCI pathogenesis.

195 intestinal homeostasis, gut defence and host lipid metabolism in mice.

196 Here, we describe a role for peroxisomal lipid metabolism in mitochondrial dynamics in brown and

197 ilable that addressed the topic of targeting lipid metabolism in PCa.

198 ver, the exact mechanisms of how cholesterol/lipid metabolism in peripheral nervous system (PNS) cont

199 tle is known about the role for autophagy in lipid metabolism in plants.

200 Lipid metabolism in pregnancy delivers PUFAs from matern

201 and efferocytosis as well as deregulation of lipid metabolism in residual tumor cells.

202 evealed higher oxidative phosphorylation and lipid metabolism in responders than in non-responders in

203 tion rates and elucidated effects of altered lipid metabolism in T47D breast cancer cells.

204 In summary, we highlight the importance of lipid metabolism in the differentiation and function of

205 This study highlights the role of lipid metabolism in the differentiation and function of

206 is in different organs, its specific role in lipid metabolism in the liver and its contribution to th

207 estigate the role of fructose in glucose and lipid metabolism in the liver, heart, skeletal muscle, a

208 ated genes were reported with involvement in lipid metabolism in the liver, implying the direct effec

209 transcriptional corepressor of bile acid and lipid metabolism in the liver; however, its function in

210 criptomic signatures in posterior thigh, and lipid metabolism in the upper back.

211 Perturbation of lipid metabolism in these cells curbed brain metastasis

212 ues for biomarkers or therapeutic targets of lipid metabolism in tumors.

213 l types, they exhibit different functions on lipid metabolism in various tissues.

214 The aim of this study was to investigate lipid metabolism including lipoproteins (HDL, LDL), neut

215 onal reprogramming associated with increased lipid metabolism, including elevated expression of the t

216 Peroxisomes perform essential functions in lipid metabolism, including fatty acid oxidation and pla

217 ithin the intestinal lumen that can modulate lipid metabolism, insulin secretion, and energy expendit

218 nctions of Gpr27 by illuminating its role in lipid metabolism, insulin signaling, and glucose homeost

219 mor subset characterized by reprogramming of lipid metabolism, invasive and metastatic progression, a

220 Strong evidence suggests that dysregulated lipid metabolism involving dysfunction of the retinal pi

221 genotype 1-, 2-, or 3-specific regulation of lipid metabolism, involving transforming growth factor b

222 , WNT signaling, TGF-beta and BMP signaling, lipid metabolism, iron homeostasis, and membrane transpo

223 This study demonstrates that lipid metabolism is a sensitive endpoint for the interac

224 Lipid metabolism is abnormal in Alzheimer's disease (AD)

225 However, recent work reveals that lipid metabolism is also controlled by the tumor microen

226 lthough the biochemical and genetic basis of lipid metabolism is clear in Arabidopsis, there is limit

227 Lipid metabolism is critical for adaptation to external

228 ese observations conclude that LAL-regulated lipid metabolism is essential to maintain antitumor immu

229 Lipid metabolism is exceedingly important in maintaining

230 The importance of lipin 1 to lipid metabolism is exemplified by cellular defects and

231 The impact of iron on lipid metabolism is so far not fully understood.

232 nterventions for diseases where dysregulated lipid metabolism is the underlying cause.

233 lin phagocytosis, but its role in microglial lipid metabolism is unknown.

234 in particular, in the context of an altered lipid metabolism, is only poorly understood.

235 C18, mtpn), gonad development (nell2, tcp4), lipid metabolism (ldah, at11b, nltp), reproduction (cyb5

236 s have the potential to rebalance disordered lipid metabolism, leading to improvements in nonalcoholi

237 In addition to their roles in lipid metabolism, LXRs participate in the transcriptiona

238 ATP supply, with the consequence of favoring lipid metabolism, maintaining muscle mass, and reducing

239 effect on CAD through immune, glycemic, and lipid metabolism, making it a candidate of interest for

240 Altering lipid metabolism may be a new therapeutic avenue in ccRC

241 etary intake of foods that can alter hepatic lipid metabolism may influence circulating ceramide conc

242 ar processes, including calcium homeostasis, lipid metabolism, membrane biogenesis and organelle remo

243 signalling and lipid signalling), autophagy, lipid metabolism, membrane dynamics, cellular stress res

244 ferential activation of pathways involved in lipid metabolism, morphology of oligodendrocytes, inflam

245 3 decreased proteins were mainly involved in lipid metabolism (n = 13, 59.10%) and included butyrophi

246 plets (LDs) carry information to measure the lipid metabolism of live cells.

247 MDD was associated with disorders of lipid metabolism (OR 1.22, 95% CI 1.12-1.34) and ischaem

248 hisms previously assigned to the complement, lipid metabolism, or extracellular matrix (ECM) pathways

249 associated with immune system functions and lipid metabolism (parental-like genes).

250 rtant signaling pathways for cancer, such as lipid metabolism pathway, PI3K/AKT and MAPK signaling pa

251 Mycobacterium tuberculosis lipid metabolism pathways facilitate access to carbon an

252 TCGA, we found that the most-widely altered lipid metabolism pathways in pan-cancer are fatty acid m

253 owed that loss of MEK5/ERK5 perturbs several lipid metabolism pathways, including the mevalonate path

254 the up-stream transcription factors for the lipid metabolism pathways.

255 ant associations with host genes involved in lipid metabolism pathways.

256 s host metabolism by targeting two prominent lipid metabolism pathways: glycerophospholipid metabolis

257 Independent of its role in lipid metabolism, PCSK9 levels associate with endothelia

258 With a primary role in lipid metabolism, peroxisomes share a niche with lipid d

259 evious genetic studies of MetS, finding that lipid metabolism plays a key role in pathology of the sy

260 liver diseases that arise from dysregulated lipid metabolism, proliferation is diminished.

261 ation of various molecular processes such as lipid metabolism, proliferation, and cell survival.

262 nt for proper myelination through control of lipid metabolism, protein glycosylation, and organizatio

263 lipidome with corresponding upregulation of lipid metabolism proteins.

264 irtuin-1 signaling important for glucose and lipid metabolism regulation.

265 type; (2) Aberrant activation of the glucose/lipid metabolism regulator peroxisome proliferator-activ

266 tworks using six seed oil-related traits, 52 lipid metabolism-related metabolites and 54 294 SNPs in

267 sizing that loss-of-function mutation in the lipid-metabolism-related genes pcsk9, apoc3, and angptl3

268 ysis; however, its mechanism on the cellular lipid metabolism remains largely unclear.

269 Lipid metabolism reprogramming is a hallmark for tumor w

270 The lipid metabolism response to predation risk differed dep

271 meostasis and metabolic activities involving lipid metabolism, specifically the synthesis of pigments

272 s implicated in muscle wasting and perturbed lipid metabolism, speculating that both can be prevented

273 e levels, and expression of genes related to lipid metabolism, stress, and immunity.

274 ucleoside, amino acid, neurotransmitter, and lipid metabolism, suggesting that modular glucosides, li

275 Here we demonstrate that the Arf1-mediated lipid metabolism sustains cells enriched with CSCs and i

276 7BL/6J mice expressed more genes involved in lipid metabolism than BALB/cJ mice.

277 s compromised, we identified pathways beyond lipid metabolism that are necessary to maintain ER integ

278 ings establish ILRUN as a novel regulator of lipid metabolism that promotes hepatic lipoprotein produ

279 tes, proteins, and lipids), feeding affected lipid metabolism the most.

280 at FATP2 provides an important role in liver lipid metabolism through its transport or activation act

281 How adult neurons coordinate lipid metabolism to regenerate axons remains elusive.

282 Oncogenic transformation alters lipid metabolism to sustain tumor growth.

283 which Cav-1 enables rewiring of cancer cell lipid metabolism towards a program of 1) exogenous sphin

284 highlight previously undescribed factors in lipid metabolism, transport and signaling that coordinat

285 ific mRNAs are associated with neurogenesis, lipid metabolism, ubiquitination, chromatin regulation a

286 This signaling pathway regulates lipid metabolism, unfolded protein responses, secretion

287 ix, G protein-coupled receptor signaling and lipid metabolism using both SKAT-O and RQTest.

288 state cancer progression is dysregulation of lipid metabolism via overexpression of fatty acid syntha

289 Lipid metabolism was affected, causing an increase in pr

290 The interplay between fluid flow and lipid metabolism was confirmed in vivo using a unilatera

291 Functional analyses also indicated that GMB lipid metabolism was enriched in HIV-infected women.

292 Functional analyses also indicated that GMB lipid metabolism was enriched in women with HIV.

293 hat the expression levels of genes linked to lipid metabolism were altered between sensitive and resi

294 uced beta-cell function and abnormal hepatic lipid metabolism were associated with GDM; these cluster

295 xpenditure was not altered but adiposity and lipid metabolism were both increased, even under caloric

296 The group reports major defects in hepatic lipid metabolism when the torsin system is compromised i

297 e development, growth, energy metabolism and lipid metabolism, which may be associated with body weig

298 ene set signature to drive tissue repair and lipid metabolism, which was distinct from other CD8(+) T

299 Overall, our data indicate that increased lipid metabolism, while impairing their function, is a f

300 tional reprogramming of pathways involved in lipid metabolism, with the subsequent reduction of fatty