ライフサイエンスコーパス: lipid peroxidation

1 methods (ORAC, TRAP, HORAC and inhibition of lipid peroxidation).

2 on mitochondrial potential and iron-induced lipid peroxidation.

3 eactive oxygen species levels and associated lipid peroxidation.

4 dopsis (Arabidopsis thaliana) leaves against lipid peroxidation.

5 ions between the chemistry and physiology of lipid peroxidation.

6 emary varieties correlated with tolerance to lipid peroxidation.

7 sly formed mutagenic DNA adduct derived from lipid peroxidation.

8 c nitro-radicals (ABTS, DPPH) and to inhibit lipid peroxidation.

9 aused by an increase in oxidative stress and lipid peroxidation.

10 g of TPB extract/kg of TTB) displayed higher lipid peroxidation.

11 fibrosis by removing fibrogenic products of lipid peroxidation.

12 ve stress and cell membrane damage caused by lipid peroxidation.

13 tion of nitric oxide, protein nitration, and lipid peroxidation.

14 totoxic aldehydes produced by metabolism and lipid peroxidation.

15 ancing vitamin E-mediated protection against lipid peroxidation.

16 70, presence of nitrotyrosine residues, and lipid peroxidation.

17 e iron could initiate carcinogenesis through lipid peroxidation.

18 icating a role for myeloperoxidase-dependent lipid peroxidation.

19 eduction observed in tissue inflammation and lipid peroxidation.

20 e via reactions that are well precedented in lipid peroxidation.

21 one, endogenous reactive oxygen species, and lipid peroxidation.

22 rolysate was the most effective inhibitor of lipid peroxidation.

23 elevates hydrogen peroxide accumulation and lipid peroxidation.

24 le significance of PUFA ratios in biological lipid peroxidation.

25 s were found in the extent of their membrane lipid peroxidation.

26 n peroxide and malondialdehyde, a product of lipid peroxidation.

27 ty acids that may be caused by activation of lipid peroxidation.

28 s of WM integrity and peripheral measures of lipid peroxidation.

29 eatic graft monitoring membrane fluidity and lipid peroxidation.

30 the steatosis score, oil red-O staining, and lipid peroxidation.

31 0% and 2.0%, possibly via modifying membrane lipid peroxidation.

32 rry extract was the most potent inhibitor of lipid peroxidation.

33 antioxidant activity by inhibiting membrane lipid peroxidation.

34 activity, decreased mitophagy, and increased lipid peroxidation.

35 was associated with suppression of (phospho)lipid peroxidation.

36 lar networks that induce and suppress lethal lipid peroxidation.

37 gulated cell death induced by iron-dependent lipid peroxidation.

38 d iron which results in oxidative stress and lipid peroxidation.

39 uced cell death, inhibiting NO formation and lipid peroxidation.

40 ed necrosis process driven by iron-dependent lipid peroxidation.

41 CM is generated during inflammation-mediated lipid peroxidation.

42 talloproteinases (MMP)-3 and -9 and enhanced lipid peroxidation.

43 HOCl signaling pathway that is finalized by lipid peroxidation.

44 by M1 Spastin is required to relieve LDs of lipid peroxidation.

45 thus abrogates the cell's protection towards lipid peroxidation.

46 ng DNA adduct formation, redox activity, and lipid peroxidation.

47 accumulation of reactive oxygen species and lipid peroxidation.

48 l accumulation), oxidative/nitrative stress (lipid peroxidation, 3-nitrotyrosine formation, and expre

49 ificantly increased ROS content (31-46%) and lipid peroxidation (30-47%), concomitant with decreased

50 KC), neutrophil infiltration (MPO activity), lipid peroxidation (4-HNE), and nitric oxide (iNOS) - we

51 ), DNA oxidation (8-oxo-2'-desoxyguanosine), lipid peroxidation (4-hydroxy-2-nonenal, isoprostane), i

52 ctor alpha: 64% +/- 24% increase; P < 0.05), lipid peroxidation (4-hydroxynonenal, measured by ELISA:

53 roguanine (8-NO2Gua)) as well as products of lipid peroxidation (8-iso-prostaglandin F2alpha (8-isoPF

54 ion of nontransferrin bound iron, markers of lipid peroxidation-8alpha-isoprostanes, protein oxidatio

55 . 14% of trained mice) and oxidative stress (lipid peroxidation, 9.1 +/- 1.4 vs. 5.2 +/- 0.9 mumol mg

56 Recent advances in our understanding of lipid peroxidation, a degenerative process that is belie

57 o acids, whereas the Flt3 inhibitor prevents lipid peroxidation, a key mechanism of glutamate-mediate

58 Lipid peroxidation, a major consequence of oxidative str

59 The role of lipid peroxidation, a potent form of oxidative stress, i

60 scavenging ability, linoleic acid and plasma lipid peroxidation ability.

61 rated key hallmarks of ferroptosis including lipid peroxidation, abnormal iron metabolism, and hypers

62 r study identified the novel end-products of lipid peroxidation, accumulating in circulation in hyper

63 ung IR increased the tissue iron content and lipid peroxidation accumulation, along with key protein

64 depolarization, production of free radicals, lipid peroxidation, activation of phospholipase C, IP3 r

65 ibited a significant reducing power and anti-lipid peroxidation activities.

66 -l-cysteine, a potent antioxidant, abolished lipid peroxidation activity and ameliorated EAE in IFN-g

67 Importantly, "free" myelin debris and lipid peroxidation activity at CNS lesions was increased

68 and possess heightened levels of markers of lipid peroxidation after bacterial infection.

69 sed free iron, mitochondrial superoxide, and lipid peroxidation, all of which are important hallmarks

70 tures (~100-nm diameter) form rapidly due to lipid peroxidation, allowing calcium entry to initiate l

71 In general, AntiOxCINs derivatives prevented lipid peroxidation and acted as inhibitors of the mitoch

72 In addition, antioxidant, anti-lipid peroxidation and antibacterial activities were imp

73 of two tropical fruit juices (FA and FB) on lipid peroxidation and antioxidant enzymes in rats.

74 le to alkylation by the aldehyde products of lipid peroxidation and by the metabolic byproducts of vi

75 al, we found high consistency in measures of lipid peroxidation and circulating non-enzymatic antioxi

76 ge due to chronic exposure was found through lipid peroxidation and DNA damage assessments of liver,

77 FA-AKI in mice associates with lipid peroxidation and downregulation of glutathione met

78 ng capability and reduced levels of membrane lipid peroxidation and electrolyte leakage under drought

79 nfirmed by a marked reduction in VPA-induced lipid peroxidation and endoplasmic reticulum stress.

80 plants, as indicated by reduced ROS, lowered lipid peroxidation and enhanced photosynthesis.

81 , and as a consequence, promotes tumour cell lipid peroxidation and ferroptosis.

82 Less lipid peroxidation and higher alpha-amylase activity, hi

83 damage in IR-induced lung injury by reducing lipid peroxidation and increasing the glutathione and GP

84 reduced Gpx4 expression as well as increased lipid peroxidation and is likewise suppressed by Fer-1 t

85 Ferroptosis is triggered by lipid peroxidation and is tightly regulated by SLC7A11,

86 hat paralleled with increased levels of ROS, lipid peroxidation and lactate, depletion in glutathione

87 malondialdehyde (MDA) epitopes, products of lipid peroxidation and markers for enhanced oxidative st

88 Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the

89 Our observations suggest that lipid peroxidation and mitochondrial biogenesis are the

90 Also, lipid peroxidation and mitochondrial dysfunction appeare

91 Lipid peroxidation and mitochondrial function and struct

92 s were seen including elevated mitochondrial lipid peroxidation and mitochondrial membrane defects, a

93 MJ33 increased the levels of lipid peroxidation and mitochondrial O2(* horizontal lin

94 Levels of lipid peroxidation and of superoxide anion (O2(* horizon

95 sults identify 5-HT as a potent inhibitor of lipid peroxidation and offer a different perspective on

96 torage protein), that triggers iron-mediated lipid peroxidation and oligodendrocyte loss (via ferropt

97 of DNA and proteins, amino acid metabolism, lipid peroxidation and one carbon metabolism (1-C).

98 -oxidant defence, subjugation of TBI-induced lipid peroxidation and phenotypic polarization of intest

99 Lipid peroxidation and polar compounds formation in sunf

100 Rice seedlings also exhibited severe lipid peroxidation and protein carbonylation, for oxidat

101 for cytoprotective activity on lymphocytes, lipid peroxidation and protein degradation.

102 ng sugars, sucrose, ethylene, ascorbic acid, lipid peroxidation and reactive oxygen species.

103 d that activation of SAT1 expression induces lipid peroxidation and sensitizes cells to undergo ferro

104 regulated cell death caused by unrestricted lipid peroxidation and subsequent membrane damage.

105 ls from mounting an adequate defense against lipid peroxidation and thereby promote ferroptosis.

106 GPX4 deficiency enhanced cellular lipid peroxidation and thus specifically inhibited the c

107 he membrane area increase corresponds to the lipids' peroxidation and is initiated by the delocalizat

108 Leaf proline content, lipid peroxidation, and activities of antioxidant enzyme

109 ative stress parameters such as glutathione, lipid peroxidation, and calcium levels along with the gl

110 scues respiratory deficiency, sensitivity to lipid peroxidation, and decreased Q(6) biosynthesis of t

111 elevations in pathogenic eicosanoid species, lipid peroxidation, and extracellular receptor kinase 1/

112 duction in senescence markers in older mice, lipid peroxidation, and fibrosis.

113 enhanced mitochondrial oxidative stress and lipid peroxidation, and induced ferroptosis.

114 eaf visible symptoms (i.e. cell death), less lipid peroxidation, and lower NADPH oxidase activity, in

115 tosynthetic pigment contents, plant biomass, lipid peroxidation, and membrane permeability) were not

116 ed, with altered antioxidant enzyme content, lipid peroxidation, and oxidative DNA adducts.

117 may indicate that lipoxygenase activity and lipid peroxidation are increased in those with colon pol

118 n sheaths, and that elevated serum levels of lipid peroxidation are reported in BD, these serum measu

119 induces cellular membrane damage, mainly via lipid peroxidation as a result of reactive oxygen specie

120 irect inhibition of TMEM16A or inhibition of lipid peroxidation as potentially powerful therapeutic a

121 storage roots showed delayed PPD and reduced lipid peroxidation as well as decreased H2O2 accumulatio

122 to disruption of the antioxidant system and lipid peroxidation, as well as alterations in lysosomal

123 re the most effective for protection against lipid peroxidation, as: P#3 (VHHA) > P#5 (LHALLL) > P#7

124 luding malondialdehyde (MDA) as a measure of lipid peroxidation, ascorbate, total phenolic concentrat

125 Endogenous lipid peroxidation (assay A) and induced lipid peroxidat

126 ous lipid peroxidation (assay A) and induced lipid peroxidation (assay B) were evaluated in liver hom

127 xygen radical absorbance capacity (ORAC) and lipid peroxidation assayed as thiobarbituric acid reacti

128 d proteomics data, enzymatic activities, and lipid peroxidation assays, we identified glutathione per

129 ivity of phenolic compounds in non-enzymatic lipid peroxidation assays.

130 scavenging, beta-carotene-linoleic acid and lipid peroxidation assays; the antibacterial activity wa

131 status (TUNEL and chromomycin A3 assay), and lipid peroxidation (Bodipy probe) in 18 infertile men wi

132 rably altered antioxidant enzyme content and lipid peroxidation, but better withstood insults.

133 VL), and kidney dysfunction; Fer-1 inhibited lipid peroxidation, but not mitochondrial reactive oxyge

134 that 5-HT contributes to the termination of lipid peroxidation by direct interaction with active gro

135 These lipids are the substrates for lipid peroxidation by lipoxygenase enzymes.

136 ted lipids, the rate-limiting substrates for lipid peroxidation, by activating the expression of hypo

137 correlated significantly with (1) increased lipid peroxidation byproducts and endoplasmic reticulum

138 XBP1 activation, fueled by lipid peroxidation byproducts, induced a triglyceride bi

139 cological targeting of increased endothelial lipid peroxidation can attenuate diabetes-induced comorb

140 solute scale of inhibition properties of the lipid peroxidation can be devised.

141 We found that ferroptotic cell death and lipid peroxidation can be inhibited by treatments that i

142 Increases in lipid peroxidation can cause ferroptosis, a form of cell

143 ith the control, and decreased the degree of lipid peroxidation compared with the HF diet.

144 The pex11a line showed higher levels of lipid peroxidation content and lower expression of genes

145 t involve ROS generation, carnosol inhibited lipid peroxidation, contrary to carnosic acid.

146 Lipid peroxidation correlated negatively with ascorbate,

147 Lipid peroxidation correlated with increased cyst volume

148 Strategies to decrease lipid peroxidation could improve mitochondrial energy ge

149 4) mitochondrial function by measuring H2O2, lipid peroxidation, cytochrome c oxidase activity and mi

150 of AhR that protects RPE cells in vitro from lipid peroxidation cytotoxicity mediated by 4-hydroxynon

151 eactive aldehydes, like those generated from lipid peroxidation damage, the contributions of these en

152 ssociated with a decrease in age-exacerbated lipid peroxidation, demyelination and axon loss.

153 easing levels of reactive oxygen species and lipid peroxidation, depleting and oxidizing glutathione

154 sis model of assessment (HOMA), and systemic lipid peroxidation determined by plasma F2-isoprostane l

155 dings indicate that activation of TMEM16A by lipid peroxidation drives growth of renal cysts.

156 rsus UW: 8.5 +/- 4.4 days, P=0.1357), nor in lipid peroxidation during 16-hr cold ischemia (P=0.672),

157 We report here the mechanism of lipid peroxidation during ferroptosis, which involves ph

158 isolevuglandins (isoLGs), are generated from lipid peroxidation during the inflammatory response and

159 mistry underpinning the cascade reactions of lipid peroxidation (enzymatic or free radical), the reac

160 ve summary of fundamental concepts regarding lipid peroxidation, experimental tools for the study of

161 Our data suggest that lipid peroxidation functions as a spatial redox relay th

162 f the olefinic band elicited seizure-induced lipid peroxidation further confirmed by the thiobarbitur

163 Lipid peroxidation generates reactive dicarbonyls includ

164 eam (bran) and have the potential to inhibit lipid peroxidation given their phenolic structure.

165 hosphate pathway (G6PD), and defense against lipid peroxidation (GPX4) scored high as synthetic sick/

166 donic acid converged on half-millimetre-long lipid peroxidation gradients that promoted leukocyte att

167 en by cellular metabolism and iron-dependent lipid peroxidation, has been implicated in diseases such

168 otic cell death that is induced by excessive lipid peroxidation, has been recently identified as a ne

169 t surprisingly, the root and consequences of lipid peroxidation have garnered increasing attention fr

170 Lipid peroxidation have not changed significantly howeve

171 onstrated increased reactive oxygen species, lipid peroxidation, histological evidence of balloon deg

172 IC50=56.51 +/- 3.6 mug/mL) and inhibition of lipid peroxidation (IC50=12.34 +/- 2.3 mug/mL) as compar

173 The ability of these compounds to inhibit lipid peroxidation in a liposome membrane system was exa

174 Additionally, NCX inhibited lipid peroxidation in an emulsified system of Sacha inch

175 Our understanding of lipid peroxidation in biology and medicine is rapidly ev

176 advances promise to help clarify the role of lipid peroxidation in cell death and human disease.

177 lar glutathione concentrations and decreased lipid peroxidation in cultured hepatocytes.

178 moglobin / iron overload, ROS production and lipid peroxidation in ectopic lesions.

179 n of mitochondrial biogenesis, and increased lipid peroxidation in female mouse offspring exposed to

180 concentration was negatively correlated with lipid peroxidation in foliar tissue under ozone stress a

181 on and prevents anemia, ROS accumulation and lipid peroxidation in Gpx4-deficient cells remain high.

182 conventional red grape juice consumption on lipid peroxidation in healthy individuals.

183 at extracts of P. trunciflora fruits prevent lipid peroxidation in HepG2 cells with higher efficacy t

184 related with significantly increased cardiac lipid peroxidation in HFD-fed WT mice relative to GCN5L1

185 (SA), and correlated with the inhibition of lipid peroxidation in human erythrocytes (LP) and total

186 These flavonoids were also found to prevent lipid peroxidation in L6 myoblast.

187 egarding the mechanisms of inhibition of the lipid peroxidation in micelles, in view of bibliographic

188 ioxidant properties, preventing iron-induced lipid peroxidation in mitochondria.

189 eration, mitochondrial hyperpolarization and lipid peroxidation in neuronal cells, but they do so by

190 beta3pE-42 has an enhanced capacity to cause lipid peroxidation in primary cortical mouse neurons com

191 We used it to probe the importance of lipid peroxidation in progression of NASH beyond simple

192 MERTK expression and lipid peroxidation in synaptoneurosomes also increased t

193 -2 fad8 quadruple mutants prevents increased lipid peroxidation in the vte2 background and restores p

194 roscopy, to co-localize amyloid deposits and lipid peroxidation in tissue slides from patients affect

195 NE) is a main endogenous product of cellular lipid peroxidation in tissues and is reported to play pa

196 CD8(+) T cells enhance ferroptosis-specific lipid peroxidation in tumour cells, and that increased f

197 nd 4-hydroxy-2,6-alkadienals), biomarkers of lipid peroxidation, in exhaled breath condensate of thre

198 acks while, reducing power and inhibition of lipid peroxidation increased.

199 tathione-to-glutathione disulfide ratio, and lipid peroxidation indicated that HFD-induced oxidative

200 en species production and hence the level of lipid peroxidation, indicating a role of TAG in protecti

201 ties of glutathione reductase, catalase, and lipid peroxidation, indicating increased antioxidant def

202 We found no significant differences in lipid peroxidation, indicating that oxidative stress may

203 tested fractions of sea buckthorn inhibited lipid peroxidation induced by H2O2, however, the non-pol

204 iggers formation of ethylene as a product of lipid peroxidation induced by the respiratory burst.

205 a consequence of sequence-specific repair of lipid peroxidation-induced DNA adduct, 1, N(6)-ethenoade

206 pe juices produced in Southern Brazil showed lipid peroxidation inhibition abilities in healthy subje

207 arkable antiradical activity and significant lipid peroxidation inhibition activities, with their IC5

208 TCC), free radical scavenging activities and lipid peroxidation inhibition activities.

209 Superoxide radical scavenging, lipid peroxidation inhibition and cupric ion reducing ac

210 polar dicaffeoylquinic acid; whereas higher lipid peroxidation inhibition was attributed to the pres

211 ity (scavenging activity, reducing power and lipid peroxidation inhibition) and individual phenolic p

212 xidant properties (mainly reducing power and lipid peroxidation inhibition), antibacterial activity a

213 Lipid peroxidation is a major consequence of oxidative s

214 Lipid peroxidation is a salient feature of ferroptosis,

215 Lipid peroxidation is connected to increases in mitochon

216 How BD affects lipid peroxidation is not known.

217 The rate-determining step in free radical lipid peroxidation is the propagation of the peroxyl rad

218 light and is photoprotective, as it reduces lipid peroxidation levels.

219 A2-VIA is strongly associated with increased lipid peroxidation levels.

220 ted foliar reactive oxygen species (ROS) and lipid peroxidation levels.

221 idemia by sensing a wide range of endogenous lipid peroxidation ligands and activating innate immune

222 A (and/or RD) in BD, and also examined serum lipid peroxidation (lipid hydroperoxides, LPH and 4-hydr

223 athione peroxidase 4 (GPX4) protects against lipid peroxidation (LPO) and cell death termed ferroptos

224 tes leads to production of ROS, resulting in lipid peroxidation (LPO) and steatosis in the absence of

225 owever, PUFAs are susceptible to the noxious lipid peroxidation (LPO) chain reaction, which is a comm

226 ons produced endogenously via reactions with lipid peroxidation (LPO) products.

227 yde (MDA), which is a significant product of lipid peroxidation (LPO), total oxidant status (TOS), to

228 he left maxilla was used for the analysis of lipid peroxidation (malondialdehyde [MDA]) and antioxida

229 Resistance to lipid peroxidation maps genetically to transmembrane and

230 and 450 +/- 360%, respectively, and urinary lipid peroxidation marker malondialdehyde was decreased

231 serum levels of malondialdehyde (MDA), as a lipid peroxidation marker, and 8-hydroxydeoxyguanosine (

232 ctions of several SPs, we found increases in lipid peroxidation markers in Trsp-deficient epithelial

233 Brain death (BD)-related lipid peroxidation, measured as serum malondialdehyde (M

234 The mechanism of the effect of lipid peroxidation, mediated by 4-hydroxynonenal ([4HNE]

235 therapeutic uses for ferrostatins, and that lipid peroxidation mediates diverse disease phenotypes.

236 ve oxygen species (ROS) generation, membrane lipid peroxidation, membrane fluidity, intracellular cal

237 Increased lipid peroxidation, mitochondrial calcium overload, and

238 loss of normal PLA2G6 gene activity leads to lipid peroxidation, mitochondrial dysfunction and subseq

239 ore polyphenols and were more active against lipid peroxidation, NO production, and tumour cells grow

240 oated-NPs exposed snails did not undergo any lipid peroxidation nor change in the antioxidant content

241 Lower lipid peroxidation occurred in the meat of animals that

242 d by 4-hydroxynonenal ([4HNE] a byproduct of lipid peroxidation) on mitochondrial function and struct

243 locking ferroptosis, either by inhibition of lipid peroxidation or by limiting iron retention, mitiga

244 s free radicals by mitochondria thus causing lipid peroxidation, oxidative and acidic stress, which c

245 < 0.0001), blood pressure (P < 0.0001), and lipid peroxidation (P = 0.001) were also observed for th

246 We here identified that targeting the lipid peroxidation product 12(S)-hydroxyeicosatetraenoic

247 CaARP-expressing plants showed lower lipid peroxidation product content in presence or absenc

248 ng is activated by 4-hydroxynonenal (HNE), a lipid peroxidation product generated naturally during ox

249 Lipid peroxidation product-mediated crosslinking of prot

250 of genotoxic N-nitroso compounds (NOCs) and lipid peroxidation products (LPOs) in the gut.

251 These data suggest that lipid peroxidation products play a role in progression o

252 the diet and traditional medicines and from lipid peroxidation products, in human prostate and renal

253 formed from 2,3-epoxyaldehydes of endogenous lipid peroxidation products, were present in all subject

254 e available for the generation of neurotoxic lipid peroxidation products.

255 ating antibodies against protein adducted by lipid peroxidation products.

256 -type HCV replicase is uniquely regulated by lipid peroxidation, providing a mechanism for attenuatin

257 lucose (P < 0.01), and reduced intracellular lipid peroxidation, reactive oxygen species (ROS), and c

258 Lipid peroxidation, regulated in part through sphingosin

259 displayed an increased membrane leakage and lipid peroxidation relative to Cu-GGH and OV-3 alone.

260 arbituric acid (TBA) number, an indicator of lipid peroxidation responsible for off-flavour generatio

261 ane integrity by inducing ROS generation and lipid peroxidation, resulting in decreased membrane flui

262 t counteracts the respiratory deficiency and lipid peroxidation sensitivity phenotypes of the coq10De

263 dicate that photoexcited SWCNTs can catalyze lipid peroxidation similarly to lipoxygenases.

264 Thus, cellular stress-induced lipid peroxidation specifically attenuates the STING DNA

265 ts are respiratory-incompetent, sensitive to lipid peroxidation stress, and unable to synthesize Q(6)

266 Reactive oxygen species and lipid peroxidation strongly activated TMEM16A, leading t

267 0) were predominantly associated with higher lipid peroxidation (TBARS) [exp(beta) = 1.09-1.78, p < 0

268 ntioxidant activity (FRAP, ABTS), as well as lipid peroxidation (TBARS) were determined at the end of

269 ids (PUFA-ePLs), which act as substrates for lipid peroxidation that, in turn, results in the inducti

270 metabolite, and crotonaldehyde, a product of lipid peroxidation, these findings further implicate inf

271 PSEE exhibited a protection of lipid peroxidation threefold higher than a positive cont

272 titudes, which paralleled with reductions in lipid peroxidation, thus suggesting plants from the high

273 be used in TTB as a protective agent against lipid peroxidation to extend its shelf-life up to two mo

274 Lipid peroxidation took place after 3 weeks of storage i

275 optosis, iron-mediated free radicals trigger lipid peroxidation under conditions of glutathione insuf

276 dehyde levels in both endogenous and induced lipid peroxidation up to 35% and 70%, respectively.

277 Increased lipid peroxidation via oxidative stress was also detecte

278 Reducing LD accumulation in glia and lipid peroxidation via targeted lipase overexpression an

279 Hepatic lipid peroxidation was also elevated in the nose-only gr

280 tioxidant status (SOD, CAT, GPX and GSH) and lipid peroxidation was also studied.

281 mages were captured by a digital camera, and lipid peroxidation was monitored using colorimetric dete

282 stress as measured by glutathione levels and lipid peroxidation was significantly reduced in rapamyci

283 rmance, egg quality, blood chemistry and egg lipid peroxidation was studied.

284 nce on the antioxidant role of Vitamin E, as lipid peroxidation was suppressed in HeLa cells both und

285 Muscle lipid peroxidation was unaffected by the dietary treatme

286 Urinary malondialdehyde (MDA), a marker of lipid peroxidation, was measured in 24 hour urine collec

287 To determine possible underlying causes of lipid peroxidation, we investigated the renal redox bala

288 ipid accumulation, apoptosis, and changes in lipid peroxidation were attenuated.

289 to the non-treated samples, and H(2)O(2) and lipid peroxidation were concomitantly increased.

290 DNA fragmentation, protamine deficiency, and lipid peroxidation were significantly higher in infertil

291 onylation, amount of glutathione stores, and lipid peroxidation were similar irrespective of the insu

292 d polyunsaturated fatty acids, which inhibit lipid peroxidation, were able to partially rescue the lo

293 ow-density lipoprotein, serum amyloid A, and lipid peroxidation, were significantly altered by polyba

294 l, traps lipid peroxyl radicals that mediate lipid peroxidation, whereas FSP1 catalyses the regenerat

295 ate, ascorbic acid degradation, and membrane lipid peroxidation, which enhanced total phenolics conte

296 y, GPX4 inactivation increased production of lipid peroxidation, which led to STING carbonylation at

297 tor that inhibits cellular NO production and lipid peroxidation, which set the stage for further expl

298 ntensifies Nrf2-driven transcription to fuel lipid peroxidation while inactivating Nrf2-mediated anti

299 s an early and integral component of in vivo lipid peroxidation with important clinical implications

300 also respiratory-deficient and sensitive to lipid peroxidation, yet it continues to produce Q(6) at