ライフサイエンスコーパス: lipooligosaccharide

1 efficiently than those induced by wild-type lipooligosaccharide.

2 onserved component of lipopolysaccharide and lipooligosaccharide.

3 and synthesized a distinct, faster-migrating lipooligosaccharide.

4 ialic acid to a membrane acceptor resembling lipooligosaccharide.

5 iron-regulated proteins TbpB and CopB and to lipooligosaccharide.

6 serum samples contained new IgG directed at lipooligosaccharide.

7 was not reproduced by purified H. influenzae lipooligosaccharide.

8 reas the other aggregates contained only 14C-lipooligosaccharide.

9 ivation and attachment of sialic acid to the lipooligosaccharide.

10 defined but is not related to sialylation of lipooligosaccharide.

11 nfluenzae contributes to the toxicity of the lipooligosaccharide.

12 ntibody response directed against gonococcal lipooligosaccharide.

13 activation correlated with disaggregation of lipooligosaccharides.

14 , due to oligosaccharide moieties within the lipooligosaccharides.

15 Isolated N. gonorrhoeae lipooligosaccharide, a known virulence factor from this

16 The lipooligosaccharides, a major component of the outer mem

17 mnus, formalin-fixed H. somnus, nor purified lipooligosaccharide altered monolayer integrity within a

18 ATCC 43431 genes encode proteins involved in lipooligosaccharide and capsular biosynthesis, as expect

19 ho-N-acetylneuraminic acid, which sialylates lipooligosaccharide and converts to serum resistance gon

20 o major surface features, the outer membrane lipooligosaccharide and flagella, are highly variable an

21 rophoresis mobilities that resemble those of lipooligosaccharide and lipopolysaccharide.

22 lotting and mass spectrometry indicated that lipooligosaccharide and outer membrane proteins P2 and P

23 ng STAT to MSn data generated from bacterial lipooligosaccharides and an N-linked glycan.

24 sion by human epithelial cells than did NTHI lipooligosaccharides and envelope proteins.

25 C. jejuni growth by binding to GM1-mimicking lipooligosaccharides and increasing permeability of the

26 ipids change systematically from hydrophilic lipooligosaccharides and phenolic glycolipids to hydroph

27 ng the mutant in expression of Opa proteins, lipooligosaccharide, and pilin.

28 attach to peptides, proteins, carbohydrates, lipooligosaccharides, and DNA.

29 OMPs, fimbriae, pili, and lipooligosaccharide are several types of surface antigen

30 d (sialic acid) can be incorporated into the lipooligosaccharides as a terminal nonreducing sugar.

31 Thus, in addition to lipooligosaccharide assembly, Kdo is required for mening

32 An anti-lipooligosaccharide bactericidal monoclonal Ab (mAb) 2C7

33 bstitutions from the heptose II group of the lipooligosaccharide beta-chain did not impact levels of

34 sertional inactivation of the phase-variable lipooligosaccharide biosynthesis gene lgtC in R2866 augm

35 ied a PCR fragment with high homology to the lipooligosaccharide biosynthesis gene lic2B (originally

36 pervariable regions, such as the capsule and lipooligosaccharide biosynthesis regions.

37 unique genes in H. somnus 129Pt involved in lipooligosaccharide biosynthesis, carbohydrate uptake an

38 enes encode proteins involved in capsule and lipooligosaccharide biosynthesis, restriction and modifi

39 rain 2019 genes involved in carbohydrate and lipooligosaccharide biosynthesis.

40 phosphoglucomutase, an enzyme important for lipooligosaccharide biosynthesis.

41 coccal PorB.1B in the presence of sialylated lipooligosaccharide bound more fH, more effectively limi

42 Exogenous sialylation of gonococcal lipooligosaccharide causes resistance to serum bacterici

43 ids, reminiscent of the trehalose-containing lipooligosaccharide class of antigens but lacking the no

44 Lipooligosaccharide configurations were predicted in non

45 dium containing [(14)C]acetate yielded (14)C-lipooligosaccharides containing approximately 600 cpm/ng

46 iants within biofilms have on their surfaces lipooligosaccharides containing sialic acid (NeuAc) and

47 Gel sieving resolved 14C-lipooligosaccharide-containing aggregates with an estima

48 adds the terminal N-acetylglucosamine to the lipooligosaccharide core of Y. pestis.

49 of intranasal immunization with a detoxified-lipooligosaccharide-cross-reactive mutant of diphtheria

50 An M. kansasii, DeltaMKAN27435 partially lipooligosaccharide-deficient mutant absorbed marginally

51 Detoxified-lipooligosaccharide (dLOS)-protein conjugates from nonty

52 oraxella catarrhalis strain 25238 detoxified lipooligosaccharide (dLOS)-protein conjugates induced a

53 e substitution from the lipid A component of lipooligosaccharide, due to insertional inactivation of

54 molecular determinants of host-meningococcal lipooligosaccharide (endotoxin) interactions at patho-ph

55 bacterial DNA, outer membrane proteins, and lipooligosaccharide (endotoxin).

56 e, but not TLR4 agonists including LPS or GC lipooligosaccharide enhanced HIV-1 infection of primary

57 lecules (PorB.1A or PorB.1B); sialylation of lipooligosaccharide enhances fH binding.

58 ed bactericidal Abs directed against the 2C7 lipooligosaccharide epitope as well as murine antigonoco

59 A gonococcal lipooligosaccharide epitope defined by the mAb 2C7 is be

60 l antibody (mAb) 2C7 recognizes a gonococcal lipooligosaccharide epitope that is expressed by >95% of

61 l antibody (MAb) 2C7 recognized a gonococcal lipooligosaccharide epitope, identified the epitope dire

62 not affect either the rate of growth or the lipooligosaccharide expression profile of this mutant.

63 The lipooligosaccharide from Neisseria gonorrhoeae (GC), con

64 unresponsive to protein-free preparations of lipooligosaccharide from Neisseria gonorrhoeae and LPS f

65 nsis LPS did not compete with a radiolabeled lipooligosaccharide from Neisseria meningitidis for bind

66 and composition analyses have shown that the lipooligosaccharides from three other H. ducreyi strains

67 The structures of the lipooligosaccharides fromBrucella melitensismutants affe

68 involve outer surface structures, including lipooligosaccharide glycans and outer surface proteins.

69 This strain produced the lipooligosaccharide glycoforms produced by the parental

70 We report the cloning of the gene (lgtG, lipooligosaccharide glycosyl transferase G) encoding the

71 resent study, the Neisseria gonorrhoeae lgtA lipooligosaccharide glycosyltransferase gene was used to

72 Nontypeable Haemophilus influenzae (NTHI) lipooligosaccharide htrB mutants exhibited greater than

73 sule expression decreased binding of an anti-lipooligosaccharide IgM mAb ( approximately 1.2- to 2-fo

74 were incubated with H. ducreyi or H. ducreyi lipooligosaccharide in vitro.

75 14C-Labeled capsule copurified with (14)C-lipooligosaccharides in peak B from NMBACE1, whereas the

76 Lipooligosaccharides in peaks A and B had the same fatty

77 emonstrate that the physical presentation of lipooligosaccharide, including possible interactions wit

78 acid and that the sialic acid content of the lipooligosaccharides increases concomitant with the tran

79 esults in the loss of phosphorylation of the lipooligosaccharide inner core and causes attenuation in

80 em results in loss of phosphorylation of the lipooligosaccharide inner core and causes attenuation in

81 derived sialic acid to Neisseria gonorrhoeae lipooligosaccharide is hypothesized to be an important m

82 suggesting that neither pili nor full-length lipooligosaccharide is required for H. ducreyi to bind t

83 hat, like the alpha chain, the beta chain of lipooligosaccharide is subject to antigenic variation.

84 he pathway of sialic acid incorporation into lipooligosaccharides is understood, the transporter resp

85 Structural studies demonstrated that the lipooligosaccharide isolated from the mutant strain lack

86 -time of flight mass spectometry analysis of lipooligosaccharides isolated from a biofilm, the plankt

87 onosaccharides (5-8), which are fragments of lipooligosaccharide IV (LOS-IV) from Mycobacterium marin

88 A mutant that produces truncated lipooligosaccharide (KDO(2)-lipid A) and a mutant defect

89 serves a dual role in modifying not only the lipooligosaccharide lipid anchor lipid A with pEtN, but

90 se membrane fibrils and blebs, which contain lipooligosaccharide (LOS) and immunoglobulin binding pro

91 . gonorrhoeae FA19 that produces a truncated lipooligosaccharide (LOS) and is non-transformable.

92 innate immune signaling pathways, including lipooligosaccharide (LOS) and other pathogen-derived mol

93 Outer membrane complex containing both lipooligosaccharide (LOS) and proteins and LOS from Neis

94 ilms differentially express both epitopes of lipooligosaccharide (LOS) and the outer membrane protein

95 y wherein the lacto-N-neotetraose termini of lipooligosaccharide (LOS) are "capped" by a surface LOS

96 Human IgGs that bind the neisserial L7 lipooligosaccharide (LOS) are bactericidal for L3,7 and

97 Outer membrane proteins (OMP) and lipooligosaccharide (LOS) are major surface antigens of

98 We identified Neisseria meningitidis lipooligosaccharide (LOS) as an acceptor for complement

99 is selection was observed in three different lipooligosaccharide (LOS) backgrounds, indicating that i

100 Lipooligosaccharide (LOS) based conjugate vaccines deriv

101 ylobacter CPS independent of the pathway for lipooligosaccharide (LOS) biosynthesis and identify a no

102 cant variation in the genetic content of the lipooligosaccharide (LOS) biosynthesis loci with concomi

103 The lipooligosaccharide (LOS) biosynthesis region is one of

104 d the role of cj1136 in C. jejuni virulence, lipooligosaccharide (LOS) biosynthesis, and host coloniz

105 Analysis of tetrameric repeat regions within lipooligosaccharide (LOS) biosynthetic genes in both str

106 terminal N-acetyllactosamine present on its lipooligosaccharide (LOS) by acquiring CMP-N-acetyl-5-ne

107 ransferase (Lst) that sialylates the surface lipooligosaccharide (LOS) by using exogenous (in all N.

108 nt study, we show that Neisseria gonorrhoeae lipooligosaccharide (LOS) can bind to the asialoglycopro

109 f Neisseria gonorrhoeae may express a single lipooligosaccharide (LOS) component on their cell surfac

110 uctural and antigenic phase variation in its lipooligosaccharide (LOS) components after in vivo and i

111 emophilus ducreyi strains examined contain a lipooligosaccharide (LOS) consisting of a single but var

112 lly transmitted disease chancroid produces a lipooligosaccharide (LOS) containing a terminal sialyl N

113 Purified lipooligosaccharide (LOS) containing lipid A devoid of t

114 The inner core of neisserial lipooligosaccharide (LOS) contains heptose residues that

115 Neisserial lipooligosaccharide (LOS) contains three oligosaccharide

116 of Campylobacter jejuni 81-176 lgtF and galT lipooligosaccharide (LOS) core mutants.

117 Three genes involved in biosynthesis of the lipooligosaccharide (LOS) core of Campylobacter jejuni M

118 a inhabiting mucosal surfaces, NTHi produces lipooligosaccharide (LOS) endotoxins that lack the O sid

119 NTHI produces lipooligosaccharide (LOS) endotoxins which lack polymeri

120 erparts in E. coli; (iii) sialylation of the lipooligosaccharide (LOS) epitope recognized by monoclon

121 A candidate vaccine that targets a lipooligosaccharide (LOS) epitope recognized mAb 2C7 att

122 asymmetry, with lipopolysaccharide (LPS) or lipooligosaccharide (LOS) exclusively in the outer leafl

123 ophilus ducreyi 35000 possessing a truncated lipooligosaccharide (LOS) failed to bind the LOS-specifi

124 lity, we found that the toxicity of purified lipooligosaccharide (LOS) from M986, a group B disease s

125 Lipooligosaccharide (LOS) from Moraxella catarrhalis has

126 We have previously shown that the lipooligosaccharide (LOS) from Neisseria meningitidis an

127 Lipooligosaccharide (LOS) from Neisseria meningitidis en

128 decyl sulfate-polyacrylamide gel analysis of lipooligosaccharide (LOS) from Neisseria meningitidis ha

129 The native lipooligosaccharide (LOS) from Neisseria meningitidis st

130 Lipooligosaccharide (LOS) glycoforms from Haemophilus in

131 Lipooligosaccharide (LOS) has been implicated in the adh

132 Lipooligosaccharide (LOS) has been shown to play a role

133 e alpha-oligosaccharide (alpha-OS) moiety of lipooligosaccharide (LOS) have been identified.

134 Lipooligosaccharide (LOS) heptose (Hep) glycan substitut

135 The role of NTHi lipooligosaccharide (LOS) in adherence was examined usin

136 ere, we report on the role of the chlamydial lipooligosaccharide (LOS) in evading the immune response

137 Biosynthesis of the variable core domain of lipooligosaccharide (LOS) in Neisseria gonorrhoeae is me

138 The sialylation of lipooligosaccharide (LOS) in Neisseria meningitidis play

139 of nontypeable Haemophilus influenzae (NTHI) lipooligosaccharide (LOS) in the induction of proinflamm

140 ed nontypeable Haemophilus influenzae (NTHi) lipooligosaccharide (LOS) in the pathogenesis of otitis

141 Both live H. ducreyi and purified H. ducreyi lipooligosaccharide (LOS) induced significant interleuki

142 Lipooligosaccharide (LOS) is a component of the gonococc

143 Lipooligosaccharide (LOS) is a major surface antigen of

144 Lipooligosaccharide (LOS) is a major surface antigen of

145 Lipooligosaccharide (LOS) is a major surface antigen of

146 Lipooligosaccharide (LOS) is a major surface component o

147 C. jejuni lipooligosaccharide (LOS) is a potent activator of Toll-

148 The lipooligosaccharide (LOS) is an outer membrane component

149 Haemophilus ducreyi lipooligosaccharide (LOS) is capable of inducing an infl

150 Campylobacter jejuni 81-176 lipooligosaccharide (LOS) is composed of two covalently

151 The lipooligosaccharide (LOS) is considered to be a major vi

152 ganglioside mimicry in Campylobacter jejuni lipooligosaccharide (LOS) is considered to be involved i

153 To determine how lipooligosaccharide (LOS) modifications would affect the

154 To begin to understand the role of the lipooligosaccharide (LOS) molecule in chancroid infectio

155 s both express the lacto-N-neotetraose (LNT) lipooligosaccharide (LOS) molecule that can be sialylate

156 Moraxella catarrhalis isolates express lipooligosaccharide (LOS) molecules on their surface, wh

157 ct of a Haemophilus ducreyi pyocin-resistant lipooligosaccharide (LOS) mutant, HD35000R.

158 Neither lipooligosaccharide (LOS) nor lipid A could be isolated

159 Structural comparison of the lipooligosaccharide (LOS) of a lic2B mutant to that of t

160 The lipooligosaccharide (LOS) of H. influenzae has been impl

161 The lipooligosaccharide (LOS) of Haemophilus ducreyi contain

162 To define the role of the surface lipooligosaccharide (LOS) of Haemophilus ducreyi in the

163 The lipooligosaccharide (LOS) of Haemophilus ducreyi, the et

164 The lipooligosaccharide (LOS) of Haemophilus influenzae cont

165 The lipooligosaccharide (LOS) of Haemophilus somnus undergoe

166 In order to determine if the lipooligosaccharide (LOS) of M. catarrhalis has a role i

167 However, molecular mimicry of the lipooligosaccharide (LOS) of most C. jejuni strains with

168 Gal-Glc) moiety from heptose I (HepI) of the lipooligosaccharide (LOS) of Neisseria gonorrhoeae under

169 ransferases involved in the synthesis of the lipooligosaccharide (LOS) of Neisseria gonorrhoeae.

170 Lipooligosaccharide (LOS) of Neisseria meningitidis is t

171 required for inner-core biosynthesis of the lipooligosaccharide (LOS) of Neisseria meningitidis.

172 de gel electrophoresis demonstrated that the lipooligosaccharide (LOS) of the galU mutant migrated fa

173 ined the effect of sialylation of gonococcal lipooligosaccharide (LOS) on MBL function.

174 The lipooligosaccharide (LOS) on the cell surface of NTHi di

175 The lipooligosaccharide (LOS) outer core represents a protot

176 of lacto-N-neotetraose-expressing gonococcal lipooligosaccharide (LOS) play an important role in inva

177 hosphoethanolamine on the lipid A portion of lipooligosaccharide (LOS) plays an important role in Tol

178 The lipooligosaccharide (LOS) present in the outer membrane

179 ningitidis utilizes capsular polysaccharide, lipooligosaccharide (LOS) sialic acid, factor H binding

180 ), factor H (fH) binding protein (fHbp), and lipooligosaccharide (LOS) sialylation.

181 H binding to N. gonorrhoeae independently of lipooligosaccharide (LOS) sialylation.

182 most serotypes, which was homologous to the lipooligosaccharide (LOS) sialyltransferase gene, lst, o

183 nd a range of isogenic mutants revealed that lipooligosaccharide (LOS) structure and capsulation infl

184 corrected] Galbeta1 --> 4Glcbeta1 --> 4HepI) lipooligosaccharide (LOS) structure exclusively; the oth

185 N. meningitidis resulted in expression of a lipooligosaccharide (LOS) structure that contained only

186 beta-1,4 N-acetylglucosamine (Gal-GlcNAc)-R lipooligosaccharide (LOS) structure.

187 lic-3 and lgtC, that generate phase-variable lipooligosaccharide (LOS) structures.

188 Strain 35000HP produces a lipooligosaccharide (LOS) that contains D-glycero-D-mann

189 ic meningococcal case strains were of the L8 lipooligosaccharide (LOS) type; four of these were both

190 Neisseria meningitidis lipooligosaccharide (LOS) was a potent direct inducer of

191 ulin G3), specific for Moraxella catarrhalis lipooligosaccharide (LOS) was evaluated for its function

192 s fH than those of their parent strains when lipooligosaccharide (LOS) was sialylated, whereas PorB m

193 the lacto-N-neotetraose (LNnT) structure on lipooligosaccharide (LOS) were phagocytosed by neutrophi

194 the causative agent of chancroid, produces a lipooligosaccharide (LOS) which terminates in N-acetylla

195 immune evasion strategy involves capping of lipooligosaccharide (LOS) with sialic acid by gonococcal

196 oethanolaminylation of lipid A on neisserial lipooligosaccharide (LOS), a major cell-surface antigen,

197 Lipooligosaccharide (LOS), a major outer membrane compon

198 Lipooligosaccharide (LOS), a major surface antigen of th

199 Lipooligosaccharide (LOS), a predominant surface-exposed

200 to an alternative outer membrane structure, lipooligosaccharide (LOS), because disseminated disease

201 eisseria meningitidis and sialylates surface lipooligosaccharide (LOS), facilitating resistance to co

202 aluate the impact on inflammation induced by lipooligosaccharide (LOS), Haemophilus influenzae type b

203 A major surface-exposed component of NTHi, lipooligosaccharide (LOS), is a virulence factor as well

204 size that multiple NTHi molecules, including lipooligosaccharide (LOS), mediate cellular interactions

205 Lipopolysaccharide, lipooligosaccharide (LOS), or endotoxin is important in

206 nic bacteria, truncation of surface glycans, lipooligosaccharide (LOS), or lipopolysaccharide (LPS) h

207 Various membrane markers including lipooligosaccharide (LOS), the 16-kDa outer membrane lip

208 predominant NTHI outer membrane protein, and lipooligosaccharide (LOS), the specific endotoxin of NTH

209 n C. jejuni suggest that all strains produce lipooligosaccharide (LOS), with about one-third of strai

210 netically and structurally defined capsule-, lipooligosaccharide (LOS)-, and sialylation-altered muta

211 adhesins, including structures found in the lipooligosaccharide (LOS).

212 type, including two that contained truncated lipooligosaccharide (LOS).

213 igenic and structural phase variation in its lipooligosaccharide (LOS).

214 cterium lost the ability to bind to purified lipooligosaccharide (LOS).

215 A major virulence factor is cell wall lipooligosaccharide (LOS).

216 lar rod protein, FlgG; the lipid A domain of lipooligosaccharide (LOS); and several N-linked glycans.

217 s with pathogenic Neisseria or with purified lipooligosaccharides (LOS) after previous exposure to LO

218 uter leaflet of lipopolysaccharides (LPS) or lipooligosaccharides (LOS) and an inner leaflet of glyce

219 irected at a number of surface proteins, and lipooligosaccharides (LOS) and detoxified LOS may be an

220 Lipopolysaccharides (LPS) and lipooligosaccharides (LOS) are the main lipid components

221 Lipooligosaccharides (LOS) from the bacterium Rhizobium

222 nd contain outer membrane proteins (OMP) and lipooligosaccharides (LOS) in their natural conformation

223 . ducreyi culture supernatant and H. ducreyi lipooligosaccharides (LOS) induced IDO expression, which

224 the carbohydrate sialic acid into C. jejuni lipooligosaccharides (LOS) is associated with increased

225 Experiments utilizing endotoxin aggregates, lipooligosaccharides (LOS) isolated from metabolically l

226 Cell surface lipooligosaccharides (LOS) of H. ducreyi are thought to

227 The lipooligosaccharides (LOS) of Haemophilus ducreyi are hi

228 The outer cores of the lipooligosaccharides (LOS) of many strains of Campylobac

229 Neu5Gc can be incorporated into cell surface lipooligosaccharides (LOS) of nontypeable Haemophilus in

230 ccurring during NTHi disease is initiated by lipooligosaccharides (LOS) on the bacterial surface.

231 Modification of structures of lipooligosaccharides (LOS) represents one prevalent mech

232 the etiologic agent of chancroid, expresses lipooligosaccharides (LOS) that are highly sialylated.

233 , NTHi has on its surface a diverse array of lipooligosaccharides (LOS) that influence host-bacterial

234 Neisseria gonorrhoeae make relatively large lipooligosaccharides (LOS) that structurally resemble hu

235 atty acid acylation, we compared endotoxins (lipooligosaccharides (LOS)) from hexa-acylated wild-type

236 Lipooligosaccharides (LOS), composed of hydrophilic olig

237 of nontypeable H. influenzae is dominated by lipooligosaccharides (LOS), many of which incorporate si

238 2 strain which produces a complex mixture of lipooligosaccharides (LOS), the mutant strains 281.25 an

239 on in the oligosaccharide component of their lipooligosaccharides (LOS).

240 with different unusual free lipids, such as lipooligosaccharides (LOS).

241 performance of two EIAs (adsorption EIA and lipooligosaccharide [LOS] EIA) that detect antibodies to

242 sts (adsorption enzyme immunoassay [EIA] and lipooligosaccharide [LOS] EIA) to detect current Haemoph

243 Neisserial lipooligosaccharides (LOSs) are a family of complex cell

244 Lipooligosaccharides (LOSs) are cell surface-exposed gly

245 Neisseria gonorrhoeae lipooligosaccharides (LOSs) induce immunoglobulin G that

246 Unique glycolipids called lipopolysaccharide/lipooligosaccharide (LPS/LOS) are enriched in the cell-s

247 m-negative bacterial lipopolysaccharides and lipooligosaccharides (LPSs or LOSs, endotoxin) within in

248 nzae cannot incorporate sialic acid into its lipooligosaccharides, making the organism unable to surv

249 nally, these data suggest that the 1291 msbB lipooligosaccharide may suppress cytokine induction.

250 t are found in Campylobacter jejuni includes lipooligosaccharides mimicking human glycolipids, capsul

251 Analysis of lipooligosaccharide moieties using an enzyme-linked immu

252 matory responses elicited by the lptA mutant lipooligosaccharide more efficiently than those induced

253 wed no changes in sensitivity as a result of lipooligosaccharide mutations in oligosaccharide and lip

254 Neither purified lipooligosaccharide nor PorB from N. lactamica is likely

255 Purified lipooligosaccharide of C. jejuni appears to be the major

256 d with purified outer membranes and purified lipooligosaccharide of homologous infecting strains and

257 The lipooligosaccharide of N. gonorrhoeae has a hexa-acylate

258 s studies have shown that the outer membrane lipooligosaccharides of H. ducreyi contain terminal sial

259 few high-carbon sugars are also found in the lipooligosaccharides of the outer cell walls of Gram-neg

260 We previously identified lipooligosaccharide on Neisseria meningitidis as an acce

261 tures by means of specific subpopulations of lipooligosaccharides on the bacterial surface that are d

262 eltaNspA Deltalst mutant unable to sialylate lipooligosaccharide or bind human fH via the known fH li

263 f sialic acid as a component of cell surface lipooligosaccharides or capsular polysaccharides has bee

264 The lipooligosaccharide, outer membrane protein patterns, an

265 pe- and species-specific glycopeptidolipids, lipooligosaccharides, phenolic glycolipids, and the genu

266 spectroscopy, the molecular elasticities of lipooligosaccharides present on NTHI cell surfaces were

267 ylglucosamine and distal sugars found in the lipooligosaccharide produced by the parental strain.

268 had similar outer membrane protein (OMP) and lipooligosaccharide profiles and growth rates except for

269 ad similar growth rates in broth and similar lipooligosaccharide profiles.

270 diator modulates the composition of the NTHI lipooligosaccharides, resulting in effects on biofilm ma

271 e degree of sialylation or expression of the lipooligosaccharide sialic acid acceptor, lacto-N-neotet

272 mbrane-bound factor H (fH; AP inhibitor) and lipooligosaccharide sialic acid, the meningococcal fH li

273 The aim of this study was to determine how lipooligosaccharide sialylation affects the serum sensit

274 Both endogenous and exogenous lipooligosaccharide sialylation are associated with incr

275 een polysialic acid capsule biosynthesis and lipooligosaccharide sialylation pathways in group B Neis

276 d in the loss of encapsulation and intrinsic lipooligosaccharide sialylation that may promote adheren

277 essential for survivability of NTHI in vivo, lipooligosaccharide sialylation was indispensable.

278 meningococcal fH ligands (fHbp and NspA) and lipooligosaccharide sialylation were deleted in all stra

279 isolates are rendered resistant in vitro by lipooligosaccharide sialylation.

280 and NspA (DeltafHbp DeltaNspA mutant) or the lipooligosaccharide sialyltransferase (Deltalst mutant)

281 main determinant of serum resistance in F62, lipooligosaccharide sialyltransferase (Lst).

282 The receptor kinase LIPOOLIGOSACCHARIDE-SPECIFIC REDUCED ELICITATION (LORE)

283 is not fully understood, but alterations in lipooligosaccharide structure can affect such resistance

284 a C. jejuni isolate capable of altering its lipooligosaccharide structure selects for variants lacki

285 The hldA mutant has a truncated lipooligosaccharide structure, contains lipid A in its o

286 of selected surface-associated proteins and lipooligosaccharide structure, known to contribute to vi

287 ption of PhoPQ-mediated modifications to the lipooligosaccharide structure.

288 19 and two FA 19 derivatives with truncated lipooligosaccharide structures were more susceptible to

289 h plasmids containing Haemophilus influenzae lipooligosaccharide synthesis genes (lsg).

290 om m-Tn3(Cm) insertions into the 7.4-kb lsg (lipooligosaccharide synthesis genes) region of Hib DNA,

291 translation, and the inhibition of bacterial lipooligosaccharide synthesis.

292 d cell wall hydrolase lead to alterations in lipooligosaccharide synthesis.

293 protein and soluble CD14-induced delivery of lipooligosaccharides to endothelial cells and cell activ

294 structure of a recently identified bacterial lipooligosaccharide, to appear foreign to the immune sys

295 ins, polysialic acid capsule or a particular lipooligosaccharide variant.

296 2, HMW1/2, and Hap or expressing a truncated lipooligosaccharide was compared to that of parental str

297 forms were two- to fourfold greater when the lipooligosaccharide was derived from planktonic or biofi

298 cance of phosphoethanolamine extensions from lipooligosaccharide, we found that phosphoethanolamine s

299 n, FlgG, and the lipid A domain of C. jejuni lipooligosaccharide with a pEtN residue.

300 zae (NTHi) is dependent on the decoration of lipooligosaccharide with sialic acid.