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1 nerally consistent with the known profile of lisdexamfetamine.
4 inge eating and related psychopathology, and lisdexamfetamine and topiramate reduced weight in adults
5 amphetamine-based medication (dexamfetamine, lisdexamfetamine), and 24 controls (8 males, 16 females)
7 ntained when subgroups (ie, methylphenidate, lisdexamfetamine, and other amphetamines) were separatel
8 reen-detected populations; guided self-help, lisdexamfetamine, and topiramate were effective for redu
15 pen-label phase (dose optimization, 4 weeks [lisdexamfetamine dimesylate, 50 or 70 mg]; dose maintena
16 12) were randomized to placebo or continued lisdexamfetamine during a 26-week, double-blind, randomi
21 eness of cognitive-behavioral therapy (CBT), lisdexamfetamine (LDX), and combined CBT+LDX, for BED co
23 sdexamfetamine responders (placebo, n = 138; lisdexamfetamine, n = 137), the observed proportions of
24 nts and the central nervous system stimulant lisdexamfetamine reduce binge frequency in binge-eating
26 e, 50 or 70 mg]; dose maintenance, 8 weeks), lisdexamfetamine responders (</=1 binge eating day per w
29 months was lower in participants continuing lisdexamfetamine than in those randomized to placebo.
30 system arousal occurred more frequently with lisdexamfetamine than placebo (relative risk range, 1.63
32 [small effect size; 95% CI, -0.51 to -0.06]; lisdexamfetamine vs placebo, Hedges g = 0.57 [medium eff
33 ased on a Cox proportional hazards model for lisdexamfetamine vs placebo, was 0.09 (95% CI, 0.04-0.23